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Amyloid-driven disruption of default mode network connectivity in cognitively healthy individuals
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Outgoing claims: the papers this work cites, builds on, or argues with.
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inferredcites →https://doi.org/10.1523/jneurosci.3067-17.2018openalex-snapshot
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inferredcites →https://doi.org/10.1523/jneurosci.3189-09.2009openalex-snapshot
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Clinical Dementia Rating (CDR)
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inferredcites →https://doi.org/10.3233/jad-2010-101020openalex-snapshot
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What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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The Intrinsic Connectivity between the Default Mode and Dorsal Attention Networks Is a Marker of Alzheimer's Pathology Burden Independent of Cognitive Reserve
inferred← citeshttps://doi.org/10.1002/alz.13102openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/alz.14051openalex-snapshot
Neuroimaging correlates of Alzheimer's disease biomarker concentrations in a racially diverse high‐risk cohort of middle‐aged adults
inferred← citeshttps://doi.org/10.1002/alz.71025openalex-snapshot
Amyloid‐related default mode network hyperconnectivity and longitudinal decline in network distinctiveness in preclinical Alzheimer's disease
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The intrinsic connectivity between the default mode and dorsal attention networks is an independent fMRI biomarker of Alzheimer's disease pathology burden
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Past and future episodic detail retrieval is reduced among clinically normal older adults at higher genetic risk for late-onset Alzheimer’s disease.
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Auditory gamma-band entrainment enhances default mode network connectivity in dementia patients
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Individualized Functional Deviation Mapping: Linking Heterogeneous Structural Atrophy to Convergent Network Disruption in Preclinical Alzheimer’s Disease
inferred← citeshttps://doi.org/10.64898/2026.09.28.754989openalex-snapshot
Normative Modeling of Molecular-Enriched Functional Connectivity for Detecting Deviations from Healthy Brain Aging
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