On the public citation graph

Impairment of mitophagy and autophagy accompanies calcific aortic valve stenosis favouring cell death and the severity of disease

0 signed · 79 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

1 of the 29 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-21): 10.1074/jbc.m800102200 (notice 2023-08-07).

Dataset join, best-effort; this page does not check the publisher record. Retraction Watch data via Crossref Labs, used with credit; OpenCitations Index (CC0) and Meta (CC BY 4.0); dataset doi:10.7910/DVN/WT8VOA (CC BY 4.0).

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/ana.24410openalex-snapshot

Syndromic parkinsonism and dementia associated with OPA 1 missense mutations

inferredcites →https://doi.org/10.1002/cphy.c170005openalex-snapshot

Mitophagy as a Protective Mechanism against Myocardial Stress

inferredcites →https://doi.org/10.1016/j.atherosclerosis.2018.08.029openalex-snapshot

Inflammatory and metabolic mechanisms underlying the calcific aortic valve disease

inferredcites →https://doi.org/10.1016/j.jamcollsurg.2012.12.034openalex-snapshot

Activation of the Homeostatic Intracellular Repair Response During Cardiac Surgery

inferredcites →https://doi.org/10.1016/j.jss.2017.05.088openalex-snapshot

Autophagy negatively regulates pro-osteogenic activity in human aortic valve interstitial cells

inferredcites →https://doi.org/10.1016/s0140-6736(06)69208-8openalex-snapshot

Burden of valvular heart diseases: a population-based study

inferredcites →https://doi.org/10.1038/s41418-017-0012-4openalex-snapshot

Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018

inferredcites →https://doi.org/10.1038/s41569-018-0074-0openalex-snapshot

Targeting mitochondria for cardiovascular disorders: therapeutic potential and obstacles

inferredcites →https://doi.org/10.1038/s41580-018-0052-8openalex-snapshot

The machineries, regulation and cellular functions of mitochondrial calcium

inferredcites →https://doi.org/10.1038/s41598-019-56614-5openalex-snapshot

Autophagy and mitophagy biomarkers are reduced in sera of patients with Alzheimer’s disease and mild cognitive impairment

inferredcites →https://doi.org/10.1073/pnas.0911187107openalex-snapshot

PINK1-dependent recruitment of Parkin to mitochondria in mitophagy

inferredcites →https://doi.org/10.1074/jbc.273.7.3963openalex-snapshot

Tor, a Phosphatidylinositol Kinase Homologue, Controls Autophagy in Yeast

inferredcites →https://doi.org/10.1074/jbc.m800102200openalex-snapshot

Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to Hypoxia

inferredcites →https://doi.org/10.1093/cvr/cvz233openalex-snapshot

Comprehensive autophagy evaluation in cardiac disease models

inferredcites →https://doi.org/10.1136/jnnp-2017-316234openalex-snapshot

Autophagy and mitophagy elements are increased in body fluids of multiple sclerosis-affected individuals

inferredcites →https://doi.org/10.1152/ajpcell.00137.2007openalex-snapshot

Expression of functional Toll-like receptors 2 and 4 in human aortic valve interstitial cells: potential roles in aortic valve inflammation and stenosis

inferredcites →https://doi.org/10.1161/01.cir.0000070591.21548.69openalex-snapshot

Human Aortic Valve Calcification Is Associated With an Osteoblast Phenotype

inferredcites →https://doi.org/10.1161/circulationaha.115.020502openalex-snapshot

Drp1-Dependent Mitochondrial Autophagy Plays a Protective Role Against Pressure Overload–Induced Mitochondrial Dysfunction and Heart Failure

inferredcites →https://doi.org/10.1172/jci122035openalex-snapshot

An alternative mitophagy pathway mediated by Rab9 protects the heart against ischemia

inferredcites →https://doi.org/10.1186/s12974-019-1526-0openalex-snapshot

Correlation between auto/mitophagic processes and magnetic resonance imaging activity in multiple sclerosis patients

inferredcites →https://doi.org/10.1242/jcs.142026openalex-snapshot

Rapamycin increases mitochondrial efficiency by mtDNA-dependent reprogramming of mitochondrial metabolism in Drosophila

inferredcites →https://doi.org/10.1371/journal.pone.0123257openalex-snapshot

Endothelial-Derived Oxidative Stress Drives Myofibroblastic Activation and Calcification of the Aortic Valve

inferredcites →https://doi.org/10.15252/emmm.201708799openalex-snapshot

Rapamycin rescues mitochondrial myopathy via coordinated activation of autophagy and lysosomal biogenesis

inferredcites →https://doi.org/10.18632/oncotarget.4370openalex-snapshot

The endoplasmic reticulum mitochondrial calcium cross talk is downregulated in malignant pleural mesothelioma cells and plays a critical role in apoptosis inhibition

inferredcites →https://doi.org/10.3389/fphys.2018.00201openalex-snapshot

Toll-Like Receptors, Inflammation, and Calcific Aortic Valve Disease

inferredcites →https://doi.org/10.3390/ijms20061486openalex-snapshot

Upregulated Autophagy in Calcific Aortic Valve Stenosis Confers Protection of Valvular Interstitial Cells

inferredcites →https://doi.org/10.3390/ijms21144899openalex-snapshot

Aortic Valve Stenosis and Mitochondrial Dysfunctions: Clinical and Molecular Perspectives

inferredcites →https://doi.org/10.3390/jcm9030892openalex-snapshot

Mitophagy in Cardiovascular Diseases

inferredcites →https://doi.org/10.4161/auto.26501openalex-snapshot

MitoTimer

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.5101318openalex-extracted

Palmatine Retards Calcific Aortic Valve Disease Progression Targeting Gapdh K263 Lactylation Interfering with Glycolytic Metabolism

inferred← citeshttps://doi.org/10.1002/advs.77368openalex-snapshot

KLF5 Downregulation Links Impaired BNIP3‐Mediated Mitophagy to Inflammatory Valve Remodeling in Calcific Aortic Valve Disease

inferred← citeshttps://doi.org/10.1002/biof.2091openalex-snapshot

ErbB2 ‐ NOTCH1 axis controls autophagy in cardiac cells

inferred← citeshttps://doi.org/10.1002/jcp.31264openalex-snapshot

Empagliflozin, a sodium‐glucose cotransporter inhibitor enhancing mitochondrial action and cardioprotection in metabolic syndrome

inferred← citeshttps://doi.org/10.1007/s11033-026-12294-9openalex-snapshot

Inflammatory and hormonal crosstalk linking rheumatic fever to chronic valvular heart disease

inferred← citeshttps://doi.org/10.1007/s13105-026-01176-0openalex-snapshot

Identification of mitochondria-related genes in calcific aortic valve disease by integrated analysis of single-cell and bulk transcriptomic atlases

inferred← citeshttps://doi.org/10.1016/j.bbamcr.2025.119911openalex-snapshot

MARCH5 ameliorates aortic valve calcification via RACGAP1-DRP1 associated mitochondrial quality control

inferred← citeshttps://doi.org/10.1016/j.celrep.2022.111124openalex-snapshot

Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy

inferred← citeshttps://doi.org/10.1016/j.ejphar.2025.178355openalex-snapshot

NF-κB/TOM6/PINK1-mediated mitophagy attenuates vascular calcification: Luteolin as a therapeutic modulator

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2026.09.013openalex-snapshot

Dysfunctional mitophagy in Alzheimer's disease: evidence from peripheral optineurin

inferred← citeshttps://doi.org/10.1016/j.jacbts.2023.01.009openalex-snapshot

Focusing on the Native Matrix Proteins in Calcific Aortic Valve Stenosis

inferred← citeshttps://doi.org/10.1016/j.jot.2025.08.002openalex-snapshot

Targeting ANT1 to regulate PINK1/Parkin-mediated mitophagy is an effective treatment of trauma-induced tendon heterotopic ossification

inferred← citeshttps://doi.org/10.1016/j.jri.2022.103781openalex-snapshot

Recent insight into autophagy and immunity at the maternal-fetal interface

inferred← citeshttps://doi.org/10.1016/j.mito.2023.07.003openalex-snapshot

Mitochondrial dysfunction and skeletal muscle atrophy: Causes, mechanisms, and treatment strategies

inferred← citeshttps://doi.org/10.1016/j.redox.2026.104097openalex-snapshot

Mitochondrial permeability transition pore desensitization by a novel dispiranic derivative prevents cardiac reperfusion injury

inferred← citeshttps://doi.org/10.1038/s41392-026-02624-5openalex-snapshot

Cyclic guanosine monophosphate-protein kinase G signaling attenuates aortic valve calcification through ULK1-mediated autophagy

inferred← citeshttps://doi.org/10.1038/s41419-022-04906-6openalex-snapshot

Mitochondrial autophagy: molecular mechanisms and implications for cardiovascular disease

inferred← citeshttps://doi.org/10.1038/s41598-022-11870-wopenalex-snapshot

Increase of Parkin and ATG5 plasmatic levels following perinatal hypoxic-ischemic encephalopathy

inferred← citeshttps://doi.org/10.1038/s41598-023-47774-6openalex-snapshot

Metformin ameliorates valve interstitial cell calcification by promoting autophagic flux

inferred← citeshttps://doi.org/10.1038/s41598-024-75115-8openalex-snapshot

Multiomics identification of ALDH9A1 as a crucial immunoregulatory molecule involved in calcific aortic valve disease

inferred← citeshttps://doi.org/10.1073/pnas.2504409122openalex-snapshot

A fixed mutation in the respiratory complex I impairs mitochondrial bioenergetics in the endangered Apennine brown bear

inferred← citeshttps://doi.org/10.1080/15548627.2024.2341588openalex-snapshot

The Wolfram-like variant WFS1 E864K destabilizes MAM and compromises autophagy and mitophagy in human and mice

inferred← citeshttps://doi.org/10.1080/15548627.2025.2469315openalex-snapshot

Autophagy regulates cellular senescence by mediating the degradation of CDKN1A/p21 and CDKN2A/p16 through SQSTM1/p62-mediated selective autophagy in myxomatous mitral valve degeneration

inferred← citeshttps://doi.org/10.1089/ars.2022.0122openalex-snapshot

Calcium Homeostasis in the Control of Mitophagy

inferred← citeshttps://doi.org/10.1093/ehjvshd/xwag018openalex-snapshot

Molecular mechanisms of aortic stenosis: deciphering the role of cellular metabolism in the pursuit of novel therapeutic targets

inferred← citeshttps://doi.org/10.1096/fj.202402831ropenalex-snapshot

Uric acid promotes aortic valve calcification via mediating valve interstitial cell osteogenic differentiation and endothelial dysfunction

inferred← citeshttps://doi.org/10.1111/apm.70014openalex-snapshot

Faecalibacterium prausnitzii Suppresses Mitophagy to Alleviate Muscle Atrophy in Chronic Renal Failure With Protein‐Energy Wasting

inferred← citeshttps://doi.org/10.1111/eci.14331openalex-snapshot

Modulation of mitochondrial permeability transition pores in reperfusion injury: Mechanisms and therapeutic approaches

inferred← citeshttps://doi.org/10.1155/2022/3858871openalex-snapshot

[Retracted] Identification of MMP9 as a Novel Biomarker to Mitochondrial Metabolism Disorder and Oxidative Stress in Calcific Aortic Valve Stenosis

inferred← citeshttps://doi.org/10.1155/cdr/2277191openalex-snapshot

Inhibition of lnc‐COL6A1‐6‐Alleviated Osteogenic Differentiation of Valvular Interstitial Cells During Aortic Valve Calcification

inferred← citeshttps://doi.org/10.1159/000537816openalex-snapshot

Association of Circulating Autophagy Proteins ATG5 and Beclin 1 with Acute Myocardial Infarction in a Case-Control Study

inferred← citeshttps://doi.org/10.1186/s12893-025-03383-wopenalex-snapshot

The correlation between the systemic immune-inflammatory index and the risk of major adverse cardiovascular events after transcatheter aortic valve replacement: a retrospective cohort study

inferred← citeshttps://doi.org/10.1186/s12967-022-03502-8openalex-snapshot

Extracellular vesicle miR-32 derived from macrophage promotes arterial calcification in mice with type 2 diabetes via inhibiting VSMC autophagy

inferred← citeshttps://doi.org/10.1186/s12967-025-07377-3openalex-snapshot

Radiation meets inflammation: NLRP3 inflammasome at the core of radiation-induced cardiac injury

inferred← citeshttps://doi.org/10.2147/dmso.s336882openalex-snapshot

Mitophagy Disequilibrium, a Prominent Pathological Mechanism in Metabolic Heart Diseases

inferred← citeshttps://doi.org/10.3389/fcell.2022.1082095openalex-snapshot

Perspectives on mitochondrial relevance in cardiac ischemia/reperfusion injury

inferred← citeshttps://doi.org/10.3389/fcell.2023.1240207openalex-snapshot

Editorial: Mitochondrial remodeling and dynamic inter-organellar contacts in cardiovascular physiopathology-Volume II

inferred← citeshttps://doi.org/10.3389/fcvm.2022.918056openalex-snapshot

Endoplasmic Reticulum Stress and Pathogenesis of Vascular Calcification

inferred← citeshttps://doi.org/10.3389/fcvm.2023.1135938openalex-snapshot

Different heart failure phenotypes of valvular heart disease: the role of mitochondrial dysfunction

inferred← citeshttps://doi.org/10.3389/fcvm.2025.1664067openalex-snapshot

Macrophages as key modulators of calcific aortic valve disease

inferred← citeshttps://doi.org/10.3389/fphar.2025.1431311openalex-snapshot

Bibliometric analysis of treatment modalities in calcific aortic valve stenosis

inferred← citeshttps://doi.org/10.3389/fphys.2022.1008517openalex-snapshot

The molecular mechanisms and intervention strategies of mitophagy in cardiorenal syndrome

inferred← citeshttps://doi.org/10.3390/biomedicines10030726openalex-snapshot

Some Insights into the Regulation of Cardiac Physiology and Pathology by the Hippo Pathway

inferred← citeshttps://doi.org/10.3390/cancers13225622openalex-snapshot

Understanding the Role of Autophagy in Cancer Formation and Progression Is a Real Opportunity to Treat and Cure Human Cancers

inferred← citeshttps://doi.org/10.3390/ijms24043414openalex-snapshot

Comprehensive Analysis of Mitochondrial Dynamics Alterations in Heart Diseases

inferred← citeshttps://doi.org/10.3390/ijms24098181openalex-snapshot

The Tricky Connection between Extracellular Vesicles and Mitochondria in Inflammatory-Related Diseases

inferred← citeshttps://doi.org/10.3390/ijms241311105openalex-snapshot

The Complex Relationship between Hypoxia Signaling, Mitochondrial Dysfunction and Inflammation in Calcific Aortic Valve Disease: Insights from the Molecular Mechanisms to Therapeutic Approaches

inferred← citeshttps://doi.org/10.6084/m9.figshare.20250444openalex-snapshot

Additional file 1 of Extracellular vesicle miR-32 derived from macrophage promotes arterial calcification in mice with type 2 diabetes via inhibiting VSMC autophagy

inferred← citeshttps://doi.org/10.6084/m9.figshare.20250447openalex-snapshot

Additional file 2 of Extracellular vesicle miR-32 derived from macrophage promotes arterial calcification in mice with type 2 diabetes via inhibiting VSMC autophagy

inferred← citeshttps://doi.org/10.7717/peerj.14307openalex-snapshot

M1 macrophage-derived extracellular vesicle containing tsRNA-5006c promotes osteogenic differentiation of aortic valve interstitial cells through regulating mitophagy

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.