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Cholesterol accumulation in macrophages drives NETosis in atherosclerotic plaques via IL-1β secretion

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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TET2 -Driven Clonal Hematopoiesis and Response to Canakinumab

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Modulation of Myelopoiesis Progenitors Is an Integral Component of Trained Immunity

inferredcites →https://doi.org/10.1016/j.cmet.2005.01.002openalex-snapshot

ABCG1 has a critical role in mediating cholesterol efflux to HDL and preventing cellular lipid accumulation

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Comparative analysis of the efficiency and specificity of myeloid-Cre deleting strains using ROSA-EYFP reporter mice

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Regulation of Hematopoietic Stem and Progenitor Cell Mobilization by Cholesterol Efflux Pathways

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Plasma metabolite profiles, cellular cholesterol efflux, and non-traditional cardiovascular risk in patients with CKD

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NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

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Reciprocal coupling of coagulation and innate immunity via neutrophil serine proteases

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Externalized histone H4 orchestrates chronic inflammation by inducing lytic cell death

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Structure of the NLRP3 decamer bound to the cytokine release inhibitor CRID3

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Therapeutic Targeting of Neutrophil Extracellular Traps in Atherogenic Inflammation

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Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease

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ATP-binding cassette transporters G1 and G4 mediate cellular cholesterol efflux to high-density lipoproteins

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Cancers predispose neutrophils to release extracellular DNA traps that contribute to cancer-associated thrombosis

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Specific Binding of ApoA-I, Enhanced Cholesterol Efflux, and Altered Plasma Membrane Morphology in Cells Expressing ABC1

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Stage-dependent differential effects of interleukin-1 isoforms on experimental atherosclerosis

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Clonal hematopoiesis associated with TET2 deficiency accelerates atherosclerosis development in mice

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Defective cholesterol clearance limits remyelination in the aged central nervous system

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Noncanonical inflammasome signaling elicits gasdermin D–dependent neutrophil extracellular traps

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Elevated Levels of Circulating DNA and Chromatin Are Independently Associated With Severe Coronary Atherosclerosis and a Prothrombotic State

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Deficiency of ATP-Binding Cassette Transporters A1 and G1 in Macrophages Increases Inflammation and Accelerates Atherosclerosis in Mice

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Roles of PAD4 and NETosis in Experimental Atherosclerosis and Arterial Injury

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Neutrophil Extracellular Traps Participate in Cardiovascular Diseases

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Increased Inflammatory Gene Expression in ABC Transporter–Deficient Macrophages

inferredcites →https://doi.org/10.1161/circulationaha.117.032636openalex-snapshot

Cholesterol Efflux Pathways Suppress Inflammasome Activation, NETosis, and Atherogenesis

inferredcites →https://doi.org/10.1161/circulationaha.117.033098openalex-snapshot

Double-Strand DNA Sensing Aim2 Inflammasome Regulates Atherosclerotic Plaque Vulnerability

inferredcites →https://doi.org/10.1161/circulationaha.121.056414openalex-snapshot

Oxidized Phospholipids Promote NETosis and Arterial Thrombosis in LNK(SH2B3) Deficiency

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Addressing dyslipidemic risk beyond LDL-cholesterol

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Targeted inactivation of hepatic Abca1 causes profound hypoalphalipoproteinemia and kidney hypercatabolism of apoA-I

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Targeted inactivation of hepatic Abca1 causes profound hypoalphalipoproteinemia and kidney hypercatabolism of apoA-I

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Endothelial cell activation leads to neutrophil transmigration as supported by the sequential roles of ICAM-2, JAM-A, and PECAM-1

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Type 2 Diabetes Is Associated with Reduced ATP-Binding Cassette Transporter A1 Gene Expression, Protein and Function

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Neutrophil Extracellular Trap Formation Is Associated with IL-1β and Autophagy-Related Signaling in Gout

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inferredcites →https://doi.org/10.3389/fimmu.2018.01680openalex-snapshot

Myeloid-Specific Deletion of Peptidylarginine Deiminase 4 Mitigates Atherosclerosis

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NLRP3 Inflammasome Assembly in Neutrophils Is Supported by PAD4 and Promotes NETosis Under Sterile Conditions

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Interleukin-1β Induces Tissue Factor Expression in A549 Cells via EGFR-Dependent and -Independent Mechanisms

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Cholesterol trafficking to the ER leads to the activation of CaMKII/JNK/NLRP3 and promotes atherosclerosis

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Additional file 1 of SYK promotes the formation of neutrophil extracellular traps by inducing PKM2 nuclear translocation and promoting STAT3 phosphorylation to exacerbate hepatic ischemia-reperfusion injury and tumor recurrence

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inferred← citeshttps://doi.org/10.7717/peerj.21485openalex-snapshot

Machine learning and single-cell RNA sequencing identify shared diagnostic genes and mechanistic links between antiphospholipid syndrome and carotid atherosclerosis

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