On the public citation graph

Cardiac nuclear high mobility group box 1 prevents the development of cardiac hypertrophy and heart failure

0 signed · 115 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/jama.292.3.344openalex-snapshot

Trends in Heart Failure Incidence and Survival in a Community-Based Population

inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb00387.xopenalex-snapshot

Evidence for a shared structural role for HMG1 and linker histones B4 and H1 in organizing chromatin.

inferredcites →https://doi.org/10.1007/s00011-006-6112-0openalex-snapshot

HMGB1 modulates inflammatory responses in LPS-activated macrophages

inferredcites →https://doi.org/10.1016/j.amjcard.2003.09.050openalex-snapshot

Development of a depressed left ventricular ejection fraction in patients with left ventricular hypertrophy and a normal ejection fraction

inferredcites →https://doi.org/10.1016/j.bbrc.2008.08.108openalex-snapshot

Age-dependent change of HMGB1 and DNA double-strand break accumulation in mouse brain

inferredcites →https://doi.org/10.1016/j.cmet.2011.04.008openalex-snapshot

High-Mobility Group Box 1 Is Essential for Mitochondrial Quality Control

inferredcites →https://doi.org/10.1016/j.molcel.2007.06.029openalex-snapshot

HMGB1 Is a Cofactor in Mammalian Base Excision Repair

inferredcites →https://doi.org/10.1016/s0140-6736(05)66621-4openalex-snapshot

Heart failure

inferredcites →https://doi.org/10.1021/bi035615yopenalex-snapshot

In Vitro Acetylation of HMGB-1 and -2 Proteins by CBP: the Role of the Acidic Tail

inferredcites →https://doi.org/10.1021/bi9004304openalex-snapshot

Nucleosome Binding Properties and Co-Remodeling Activities of Native and in Vivo Acetylated HMGB-1 and HMGB-2 Proteins

inferredcites →https://doi.org/10.1038/10338openalex-snapshot

The lack of chromosomal protein Hmg1 does not disrupt cell growth but causes lethal hypoglycaemia in newborn mice

inferredcites →https://doi.org/10.1038/nature00858openalex-snapshot

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

inferredcites →https://doi.org/10.1038/ncb1553openalex-snapshot

Proteome analysis of soluble nuclear proteins reveals that HMGB1/2 suppress genotoxic stress in polyglutamine diseases

inferredcites →https://doi.org/10.1038/nri1594openalex-snapshot

High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

inferredcites →https://doi.org/10.1042/bj20071613openalex-snapshot

A critical role in structure-specific DNA binding for the acetylatable lysine residues in HMGB1

inferredcites →https://doi.org/10.1046/j.1365-2796.2003.01204.xopenalex-snapshot

High mobility group 1 B‐box mediates activation of human endothelium

inferredcites →https://doi.org/10.1056/nejmra021498openalex-snapshot

Heart Failure

inferredcites →https://doi.org/10.1073/pnas.0803181105openalex-snapshot

High mobility group protein B1 enhances DNA repair and chromatin modification after DNA damage

inferredcites →https://doi.org/10.1073/pnas.1003893107openalex-snapshot

A critical cysteine is required for HMGB1 binding to Toll-like receptor 4 and activation of macrophage cytokine release

inferredcites →https://doi.org/10.1074/jbc.m110.128348openalex-snapshot

High Mobility Group Box 1 Release from Hepatocytes during Ischemia and Reperfusion Injury Is Mediated by Decreased Histone Deacetylase Activity

inferredcites →https://doi.org/10.1074/jbc.m602369200openalex-snapshot

ERK5 Activation Inhibits Inflammatory Responses via Peroxisome Proliferator-activated Receptor δ (PPARδ) Stimulation

inferredcites →https://doi.org/10.1091/mbc.e02-09-0581openalex-snapshot

Association of Chromatin Proteins High Mobility Group Box (HMGB) 1 and HMGB2 with Mitotic Chromosomes

inferredcites →https://doi.org/10.1093/cvr/cvn163openalex-snapshot

High-mobility group box 1 restores cardiac function after myocardial infarction in transgenic mice

inferredcites →https://doi.org/10.1093/emboj/cdg516openalex-snapshot

Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion

inferredcites →https://doi.org/10.1093/emboj/cdg601openalex-snapshot

NRSF regulates the fetal cardiac gene program and maintains normal cardiac structure and function

inferredcites →https://doi.org/10.1093/nar/gkh550openalex-snapshot

Direct interaction of NRSF with TBP: chromatin reorganization and core promoter repression for neuron-specific gene transcription

inferredcites →https://doi.org/10.1096/fj.10-162636openalex-snapshot

Novel role of C terminus of Hsc70-interacting protein (CHIP) ubiquitin ligase on inhibiting cardiac apoptosis and dysfunction via regulating ERK5-mediated degradation of inducible cAMP early repressor

inferredcites →https://doi.org/10.1096/fj.11-197525openalex-snapshot

Nutrient deprivation regulates DNA damage repair in cardiomyocytes via loss of the base‐excision repair enzyme OGG1

inferredcites →https://doi.org/10.1111/j.1365-2362.2010.02465.xopenalex-snapshot

8-Hydroxy-2′-deoxyguanosine is a prognostic mediator for cardiac event

inferredcites →https://doi.org/10.1111/j.1365-2796.2011.02369.xopenalex-snapshot

HMGB1 as a predictor of infarct transmurality and functional recovery in patients with myocardial infarction

inferredcites →https://doi.org/10.1128/mcb.01862-08openalex-snapshot

Chromatin-Specific Remodeling by HMGB1 and Linker Histone H1 Silences Proinflammatory Genes during Endotoxin Tolerance

inferredcites →https://doi.org/10.1128/mcb.21.6.2085-2097.2001openalex-snapshot

The Neuron-Restrictive Silencer Element–Neuron-Restrictive Silencer Factor System Regulates Basal and Endothelin 1-Inducible Atrial Natriuretic Peptide Gene Expression in Ventricular Myocytes

inferredcites →https://doi.org/10.1161/01.res.0000186276.06104.04openalex-snapshot

Exogenous High-Mobility Group Box 1 Protein Induces Myocardial Regeneration After Infarction via Enhanced Cardiac C-Kit + Cell Proliferation and Differentiation

inferredcites →https://doi.org/10.1161/circresaha.107.168138openalex-snapshot

Effects of MEK5/ERK5 Association on Small Ubiquitin-Related Modification of ERK5: Implications for Diabetic Ventricular Dysfunction After Myocardial Infarction

inferredcites →https://doi.org/10.1161/circresaha.111.254730openalex-snapshot

p90RSK Targets the ERK5-CHIP Ubiquitin E3 Ligase Activity in Diabetic Hearts and Promotes Cardiac Apoptosis and Dysfunction

inferredcites →https://doi.org/10.1161/circulationaha.108.769331openalex-snapshot

High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart

inferredcites →https://doi.org/10.1189/jlb.0306165openalex-snapshot

Pivotal Advances: High-mobility group box 1 protein—a cytokine with a role in cardiac repair

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.3753798openalex-extracted

inferred← citeshttps://doi.org/10.1002/alz.13490openalex-snapshot

Senescence, brain inflammation, and oligomeric tau drive cognitive decline in Alzheimer's disease: Evidence from clinical and preclinical studies

inferred← citeshttps://doi.org/10.1002/cbin.10984openalex-snapshot

Angiotensin II enhances the acetylation and release of HMGB1 in RAW264.7 macrophage

inferred← citeshttps://doi.org/10.1007/s00011-018-1160-9openalex-snapshot

sRAGE attenuates angiotensin II-induced cardiomyocyte hypertrophy by inhibiting RAGE-NFκB-NLRP3 activation

inferred← citeshttps://doi.org/10.1007/s00018-018-2930-9openalex-snapshot

The Janus face of HMGB1 in heart disease: a necessary update

inferred← citeshttps://doi.org/10.1007/s00018-024-05464-0openalex-snapshot

Long noncoding RNA AK144717 exacerbates pathological cardiac hypertrophy through modulating the cellular distribution of HMGB1 and subsequent DNA damage response

inferred← citeshttps://doi.org/10.1007/s00210-018-1462-zopenalex-snapshot

Endothelin-1-induced hypertrophic alterations and heme oxygenase-1 expression in cardiomyoblasts are counteracted by beta estradiol: in vitro and in vivo studies

inferred← citeshttps://doi.org/10.1007/s00604-017-2295-zopenalex-snapshot

A voltammetric study on the interaction between isoproterenol and cardiomyocyte DNA by using a glassy carbon electrode modified with carbon nanotubes, polyaniline and gold nanoparticles

inferred← citeshttps://doi.org/10.1007/s10753-022-01668-3openalex-snapshot

Role of High Mobility Group Box 1 in Cardiovascular Diseases

inferred← citeshttps://doi.org/10.1007/s12012-024-09919-5openalex-snapshot

High Mobility Group Box 1 and Cardiovascular Diseases: Study of Act and Connect

inferred← citeshttps://doi.org/10.1007/s12272-020-01290-7openalex-snapshot

Protective effect of lncRNA CRNDE on myocardial cell apoptosis in heart failure by regulating HMGB1 cytoplasm translocation through PARP-1

inferred← citeshttps://doi.org/10.1016/b978-0-12-803111-7.00003-8openalex-snapshot

Endothelin-1 as a Cardiac-Derived Autocrine, Paracrine and Intracrine Factor in Heart Health and Disease

inferred← citeshttps://doi.org/10.1016/j.bbrc.2013.11.083openalex-snapshot

Midkine exacerbates pressure overload-induced cardiac remodeling

inferred← citeshttps://doi.org/10.1016/j.bbrc.2022.04.053openalex-snapshot

HMGB1 in macrophage nucleus protects against pressure overload induced cardiac remodeling via regulation of macrophage differentiation and inflammatory response

inferred← citeshttps://doi.org/10.1016/j.bcp.2025.117527openalex-snapshot

PRC1 mediates downstream PI3K-AKT activation through regulating PTEN ubiquitination degradation in TAC-induced cardiac remodeling

inferred← citeshttps://doi.org/10.1016/j.biopha.2018.10.035openalex-snapshot

Nrf2 activation and down-regulation of HMGB1 and MyD88 expression by amnion membrane extracts in response to the hypoxia-induced injury in cardiac H9c2 cells

inferred← citeshttps://doi.org/10.1016/j.biopha.2021.111555openalex-snapshot

High-mobility group box 1 serves as an inflammation driver of cardiovascular disease

inferred← citeshttps://doi.org/10.1016/j.cellsig.2014.04.020openalex-snapshot

P21-activated kinase in inflammatory and cardiovascular disease

inferred← citeshttps://doi.org/10.1016/j.celrep.2021.109419openalex-snapshot

Tau oligomer induced HMGB1 release contributes to cellular senescence and neuropathology linked to Alzheimer’s disease and frontotemporal dementia

inferred← citeshttps://doi.org/10.1016/j.clinbiochem.2016.11.005openalex-snapshot

Relevance of pre-analytical blood management on the emerging cardiovascular protein biomarkers TWEAK and HMGB1 and on miRNA serum and plasma profiling

inferred← citeshttps://doi.org/10.1016/j.intimp.2023.109757openalex-snapshot

Cytoplasmic HMGB1 induces renal tubular ferroptosis after ischemia/reperfusion

inferred← citeshttps://doi.org/10.1016/j.isci.2022.103973openalex-snapshot

Paracrine signal emanating from stressed cardiomyocytes aggravates inflammatory microenvironment in diabetic cardiomyopathy

inferred← citeshttps://doi.org/10.1016/j.jacbts.2018.11.011openalex-snapshot

Cardiac Nuclear High-Mobility Group Box 1 Ameliorates Pathological Cardiac Hypertrophy by Inhibiting DNA Damage Response

inferred← citeshttps://doi.org/10.1016/j.jacbts.2019.03.008openalex-snapshot

Nuclear Hmgb1

inferred← citeshttps://doi.org/10.1016/j.jacbts.2026.101623openalex-snapshot

Macrophage-Specific SPP1 Contributes to Pressure Overload–Induced Cardiac Dysfunction and Maladaptive Remodeling

inferred← citeshttps://doi.org/10.1016/j.lfs.2014.01.062openalex-snapshot

Innate immunity and cardiomyocytes in ischemic heart disease

inferred← citeshttps://doi.org/10.1016/j.lfs.2020.118203openalex-snapshot

HMGB1 in kidney diseases

inferred← citeshttps://doi.org/10.1016/j.mam.2014.05.001openalex-snapshot

HMGB1 in health and disease

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2018.12.005openalex-snapshot

HMGB1 and repair: focus on the heart

inferred← citeshttps://doi.org/10.1016/j.phrs.2019.03.025openalex-snapshot

Molecular targets of fenofibrate in the cardiovascular-renal axis: A unifying perspective of its pleiotropic benefits

inferred← citeshttps://doi.org/10.1016/j.prp.2023.154889openalex-snapshot

Apoptosis and long non-coding RNAs: Focus on their roles in Heart diseases

inferred← citeshttps://doi.org/10.1016/j.redox.2018.07.008openalex-snapshot

Over-expression of a cardiac-specific human dopamine D5 receptor mutation in mice causes a dilated cardiomyopathy through ROS over-generation by NADPH oxidase activation and Nrf2 degradation

inferred← citeshttps://doi.org/10.1016/j.yjmcc.2015.02.018openalex-snapshot

High-mobility group box 1-mediated heat shock protein beta 1 expression attenuates mitochondrial dysfunction and apoptosis

inferred← citeshttps://doi.org/10.1021/acs.jmedchem.4c01912openalex-snapshot

Targeting HMGB1 and Its Interaction with Receptors: Challenges and Future Directions

inferred← citeshttps://doi.org/10.1038/s41401-018-0044-4openalex-snapshot

PARP1 interacts with HMGB1 and promotes its nuclear export in pathological myocardial hypertrophy

inferred← citeshttps://doi.org/10.1038/s41401-024-01436-zopenalex-snapshot

SNX3 mediates heart failure by interacting with HMGB1 and subsequently facilitating its nuclear-cytoplasmic translocation

inferred← citeshttps://doi.org/10.1038/s41420-020-00340-9openalex-snapshot

Cardiomyocyte-restricted high-mobility group box 1 (HMGB1) deletion leads to small heart and glycolipid metabolic disorder through GR/PGC-1α signalling

inferred← citeshttps://doi.org/10.1038/s44325-026-00119-4openalex-snapshot

Scoping review of preclinical and clinical studies on the role of HMGB1 in heart disease

inferred← citeshttps://doi.org/10.1080/10641963.2021.1996588openalex-snapshot

AT1 receptor blocker inhibits HMGB1 expression in pressure overload-induced acute cardiac dysfunction by suppressing the MAPK/NF-κB signaling pathway

inferred← citeshttps://doi.org/10.1089/dna.2018.4592openalex-snapshot

Regulation of Acetylation in High Mobility Group Protein B1 Cytosol Translocation

inferred← citeshttps://doi.org/10.1093/cvr/cvac080openalex-snapshot

Targeting DNA damage response in cardiovascular diseases: from pathophysiology to therapeutic implications

inferred← citeshttps://doi.org/10.1111/ajt.13560openalex-snapshot

The Effect of High-Mobility Group Box 1 in Rat Steatotic and Nonsteatotic Liver Transplantation From Donors After Brain Death

inferred← citeshttps://doi.org/10.1111/imcb.12660openalex-snapshot

Cardiomyocyte‐derived HMGB1 takes a protective role in CVB3‐induced viral myocarditis via inhibiting cardiac apoptosis

inferred← citeshttps://doi.org/10.1111/jcmm.12743openalex-snapshot

Extracellular high‐mobility group box 1 mediates pressure overload‐induced cardiac hypertrophy and heart failure

inferred← citeshttps://doi.org/10.1155/2014/541394openalex-snapshot

HMGB1 Is Involved in the Protective Effect of the PPARαAgonist Fenofibrate against Cardiac Hypertrophy

inferred← citeshttps://doi.org/10.1155/2015/430185openalex-snapshot

Correlation between Serum Levels of High Mobility Group Box-1 Protein and Pancreatitis: A Meta-Analysis

inferred← citeshttps://doi.org/10.1155/2016/2612743openalex-snapshot

PPAR Ligands Function as Suppressors That Target Biological Actions of HMGB1

inferred← citeshttps://doi.org/10.1155/2020/7182428openalex-snapshot

Peptidomics Analysis Reveals Peptide PDCryab1 Inhibits Doxorubicin-Induced Cardiotoxicity

inferred← citeshttps://doi.org/10.1155/2021/6687096openalex-snapshot

NETosis as a Pathogenic Factor for Heart Failure

inferred← citeshttps://doi.org/10.1159/000362954openalex-snapshot

FIP200 is Involved in Murine Pseudomonas Infection by Regulating HMGB1 Intracellular Translocation

inferred← citeshttps://doi.org/10.1159/000373970openalex-snapshot

MicroRNA-223 Displays a Protective Role Against Cardiomyocyte Hypertrophy by Targeting Cardiac Troponin I-Interacting Kinase

inferred← citeshttps://doi.org/10.11606/t.42.2014.tde-19112014-133201openalex-snapshot

Treinamento aeróbio x disfunção autonômica na hipertensão espontânea: uma abordagem molecular em núcleos centrais de regulação.

inferred← citeshttps://doi.org/10.1161/circresaha.123.323030openalex-snapshot

T-Cell MyD88 Is a Novel Regulator of Cardiac Fibrosis Through Modulation of T-Cell Activation

inferred← citeshttps://doi.org/10.1161/hypertensionaha.115.06922openalex-snapshot

Midkine Deteriorates Cardiac Remodeling via Epidermal Growth Factor Receptor Signaling in Chronic Kidney Disease

inferred← citeshttps://doi.org/10.1161/hypertensionaha.115.07089openalex-snapshot

SmgGDS as a Crucial Mediator of the Inhibitory Effects of Statins on Cardiac Hypertrophy and Fibrosis

inferred← citeshttps://doi.org/10.1161/hypertensionaha.120.15487openalex-snapshot

HECT (Homologous to the E6-AP Carboxyl Terminus)-Type Ubiquitin E3 Ligase ITCH Attenuates Cardiac Hypertrophy by Suppressing the Wnt/β-Catenin Signaling Pathway

inferred← citeshttps://doi.org/10.1161/jaha.115.002485openalex-snapshot

HECT‐Type Ubiquitin E3 Ligase ITCH Interacts With Thioredoxin‐Interacting Protein and Ameliorates Reactive Oxygen Species–Induced Cardiotoxicity

inferred← citeshttps://doi.org/10.1161/jaha.124.035608openalex-snapshot

Diacylglycerol Kinase ζ Attenuates Doxorubicin‐Induced Cardiotoxicity Through p53 Degradation

inferred← citeshttps://doi.org/10.1186/s13287-026-04970-1openalex-snapshot

HMGB1-mediated enhancement of glycolysis activates hepatic stellate cells by inhibiting ferroptosis in alcoholic hepatic fibrosis

inferred← citeshttps://doi.org/10.1186/s43088-024-00515-8openalex-snapshot

Association between circulatory microRNA-218 expression, serum PCSK9 levels, inflammatory markers, and monocyte subsets in coronary artery disease patients: impact of statin therapy

inferred← citeshttps://doi.org/10.1253/circj.cj-18-0925openalex-snapshot

Loss of Endogenous HMGB2 Promotes Cardiac Dysfunction and Pressure Overload-Induced Heart Failure in Mice

inferred← citeshttps://doi.org/10.1371/journal.pone.0158514openalex-snapshot

HMGB1-RAGE Axis Makes No Contribution to Cardiac Remodeling Induced by Pressure-Overload

inferred← citeshttps://doi.org/10.1536/ihj.15-188openalex-snapshot

Plasma Levels of Receptor for Advanced Glycation End-Products and High-Mobility Group Box 1 in Patients With Pulmonary Hypertension

inferred← citeshttps://doi.org/10.1536/ihj.20-384openalex-snapshot

Angiotensin II Increases HMGB1 Expression in the Myocardium Through AT1 and AT2 Receptors When Under Pressure Overload

inferred← citeshttps://doi.org/10.18103/mra.v11i12.4952openalex-snapshot

Genome wide hypomethylation and youth-associated DNA gap reduction promoting DNA damage and senescence-associated pathogenesis

inferred← citeshttps://doi.org/10.3389/fcvm.2020.579036openalex-snapshot

Key Player in Cardiac Hypertrophy, Emphasizing the Role of Toll-Like Receptor 4

inferred← citeshttps://doi.org/10.3389/fncel.2019.00319openalex-snapshot

Amelioration of Mouse Retinal Degeneration After Blue LED Exposure by Glycyrrhizic Acid-Mediated Inhibition of Inflammation

inferred← citeshttps://doi.org/10.3390/cells13231946openalex-snapshot

High Mobility Group Box 1 (HMGB1): Molecular Signaling and Potential Therapeutic Strategies

inferred← citeshttps://doi.org/10.3390/ijms17040542openalex-snapshot

Deficiency of Senescence Marker Protein 30 Exacerbates Cardiac Injury after Ischemia/Reperfusion

inferred← citeshttps://doi.org/10.3390/jpm12061007openalex-snapshot

Regulation of Key Immune-Related Genes in the Heart Following Burn Injury

inferred← citeshttps://doi.org/10.3892/etm.2015.2373openalex-snapshot

Protective effect of olmesartan against cardiac ischemia/reperfusion injury in spontaneously hypertensive rats

inferred← citeshttps://doi.org/10.3892/ijmm.2019.4276openalex-snapshot

HMGB1 enhances mechanical stress-induced cardiomyocyte hypertrophy in�vitro via the RAGE/ERK1/2 signaling pathway

inferred← citeshttps://doi.org/10.3892/mmr.2015.4308openalex-snapshot

Expression and purification of functional HMGB1 A box by fusion with SUMO

inferred← citeshttps://doi.org/10.3892/mmr.2016.4755openalex-snapshot

Netrin-1 prevents the development of cardiac hypertrophy and heart failure

inferred← citeshttps://doi.org/10.3892/mmr.2021.11853openalex-snapshot

Recombinant high‑mobility group box 1 induces cardiomyocyte hypertrophy by regulating the 14‑3‑3η, PI3K and nuclear factor of activated T cells signaling pathways

inferred← citeshttps://doi.org/10.4081/ejh.2022.3426openalex-snapshot

MALAT1 regulates hypertrophy of cardiomyocytes by modulating the miR-181a/HMGB2 pathway

inferred← citeshttps://doi.org/10.4172/2157-7412.1000178openalex-snapshot

Hemagglutinating Virus of Japan Envelope Vectors as High-Performance Vehicles for Delivery of Small RNAs

inferred← citeshttps://doi.org/10.5551/jat.26740openalex-snapshot

Deficiency of Long Pentraxin PTX3 Promoted Neointimal Hyperplasia after Vascular Injury

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.