On the public citation graph
Cardiomyocyte-specific deficiency of HSPB1 worsens cardiac dysfunction by activating NFκB-mediated leucocyte recruitment after myocardial infarction
0 signed · 107 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/s00395-004-0483-6openalex-snapshot
Heat shock protein 27 protects the heart against myocardial infarction
inferredcites →https://doi.org/10.1016/j.ejheart.2007.03.007openalex-snapshot
Over-Expression of Heat Shock Protein 27 Attenuates Doxorubicin-Induced Cardiac Dysfunction in Mice
inferredcites →https://doi.org/10.1016/j.pharmthera.2011.12.010openalex-snapshot
Post-infarct cardiac rupture: Recent insights on pathogenesis and therapeutic interventions
inferredcites →https://doi.org/10.1016/j.tcm.2012.02.002openalex-snapshot
Role of the Inflammasome in Myocardial Infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2007.06.011openalex-snapshot
Differences in inflammation, MMP activation and collagen damage account for gender difference in murine cardiac rupture following myocardial infarction
inferredcites →https://doi.org/10.1016/j.yjmcc.2015.02.018openalex-snapshot
High-mobility group box 1-mediated heat shock protein beta 1 expression attenuates mitochondrial dysfunction and apoptosis
inferredcites →https://doi.org/10.1038/nm.2354openalex-snapshot
GDF-15 is an inhibitor of leukocyte integrin activation required for survival after myocardial infarction in mice
inferredcites →https://doi.org/10.1038/nrcardio.2014.28openalex-snapshot
The inflammatory response in myocardial injury, repair, and remodelling
inferredcites →https://doi.org/10.1038/nrd2070openalex-snapshot
Targeting signal transduction as a strategy to treat inflammatory diseases
inferredcites →https://doi.org/10.1038/srep33636openalex-snapshot
HSPA12B Attenuated Acute Myocardial Ischemia/reperfusion Injury via Maintaining Endothelial Integrity in a PI3K/Akt/mTOR-dependent Mechanism
inferredcites →https://doi.org/10.1073/pnas.1108586108openalex-snapshot
The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse
inferredcites →https://doi.org/10.1074/jbc.m112.358929openalex-snapshot
Deficiency of Capn4 Gene Inhibits Nuclear Factor-κB (NF-κB) Protein Signaling/Inflammation and Reduces Remodeling after Myocardial Infarction
inferredcites →https://doi.org/10.1093/cvr/cvq274openalex-snapshot
Cardiomyocyte NF-κB p65 promotes adverse remodelling, apoptosis, and endoplasmic reticulum stress in heart failure
inferredcites →https://doi.org/10.1093/cvr/cvt139openalex-snapshot
HSPA12B attenuates cardiac dysfunction and remodelling after myocardial infarction through an eNOS-dependent mechanism
inferredcites →https://doi.org/10.1093/eurjhf/hfn043openalex-snapshot
Inflammation as a Therapeutic Target in Heart Failure? A Scientific Statement from the Translational Research Committee of the Heart Failure Association of the European Society of Cardiology
inferredcites →https://doi.org/10.1097/crd.0b013e3181bf4ab4openalex-snapshot
Cardiac Rupture After Myocardial Infarction
inferredcites →https://doi.org/10.1097/shk.0b013e318199165dopenalex-snapshot
CARDIAC-SPECIFIC EXPRESSION OF HEAT SHOCK PROTEIN 27 ATTENUATED ENDOTOXIN-INDUCED CARDIAC DYSFUNCTION AND MORTALITY IN MICE THROUGH A PI3K/AKT-DEPENDENT MECHANISM
inferredcites →https://doi.org/10.1097/tp.0000000000000170openalex-snapshot
Heat Shock Protein-27 Delays Acute Rejection After Cardiac Transplantation
inferredcites →https://doi.org/10.1111/micc.12305openalex-snapshot
Immune cells in repair of the infarcted myocardium
inferredcites →https://doi.org/10.1152/ajpheart.01175.2005openalex-snapshot
Blockade of NF-κB improves cardiac function and survival after myocardial infarction
inferredcites →https://doi.org/10.1155/2016/2586706openalex-snapshot
HSP27 Alleviates Cardiac Aging in Mice via a Mechanism Involving Antioxidation and Mitophagy Activation
inferredcites →https://doi.org/10.1161/01.cir.0000148825.99184.50openalex-snapshot
Overexpression of Wild-Type Heat Shock Protein 27 and a Nonphosphorylatable Heat Shock Protein 27 Mutant Protects Against Ischemia/Reperfusion Injury in a Transgenic Mouse Model
inferredcites →https://doi.org/10.1161/01.cir.60.7.1532openalex-snapshot
Expansion of transmural myocardial infarction: a pathophysiologic factor in cardiac rupture.
inferredcites →https://doi.org/10.1161/01.res.0000209968.66606.10openalex-snapshot
IP-10 Blocks Vascular Endothelial Growth Factor-Induced Endothelial Cell Motility and Tube Formation via Inhibition of Calpain
inferredcites →https://doi.org/10.1161/cir.0000000000000485openalex-snapshot
Heart Disease and Stroke Statistics—2017 Update: A Report From the American Heart Association
inferredcites →https://doi.org/10.1161/circheartfailure.113.000604openalex-snapshot
Interleukin-23 Deficiency Leads to Impaired Wound Healing and Adverse Prognosis After Myocardial Infarction
inferredcites →https://doi.org/10.1161/circheartfailure.114.002029openalex-snapshot
Cardiomyocyte A Disintegrin And Metalloproteinase 17 (ADAM17) Is Essential in Post–Myocardial Infarction Repair by Regulating Angiogenesis
inferredcites →https://doi.org/10.1161/circresaha.109.199471openalex-snapshot
Induction of the CXC Chemokine Interferon-γ-Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction
inferredcites →https://doi.org/10.1161/circresaha.111.300502openalex-snapshot
Matrix Metalloproteinase-28 Deletion Exacerbates Cardiac Dysfunction and Rupture After Myocardial Infarction in Mice by Inhibiting M2 Macrophage Activation
inferredcites →https://doi.org/10.1161/circresaha.113.302593openalex-snapshot
Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation
inferredcites →https://doi.org/10.1161/circresaha.116.303577openalex-snapshot
The Biological Basis for Cardiac Repair After Myocardial Infarction
inferredcites →https://doi.org/10.1161/circulationaha.105.602094openalex-snapshot
Factor XIII Deficiency Causes Cardiac Rupture, Impairs Wound Healing, and Aggravates Cardiac Remodeling in Mice With Myocardial Infarction
inferredcites →https://doi.org/10.1161/circulationaha.111.083675openalex-snapshot
Cross-Talk Between the Toll-Like Receptor 4 and Notch1 Pathways Augments the Inflammatory Response in the Interstitial Cells of Stenotic Human Aortic Valves
inferredcites →https://doi.org/10.1172/jci117505openalex-snapshot
Activation and attenuation of transcription factor NF-kB in mouse glomerular mesangial cells in response to tumor necrosis factor-alpha, immunoglobulin G, and adenosine 3':5'-cyclic monophosphate. Evidence for involvement of reactive oxygen species.
inferredcites →https://doi.org/10.1172/jci6709openalex-snapshot
Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium
inferredcites →https://doi.org/10.1536/ihj.13-388openalex-snapshot
NLRP3 Inflammasome as a Novel Player in Myocardial Infarction
inferredcites →https://doi.org/10.1536/ihj.14-140openalex-snapshot
Letter by Mezzaroma, et al Regarding Article, “NLRP3 Inflammasome as a Therapeutic Target in Myocardial Infarction”
inferredcites →https://doi.org/10.2174/13816128113199990449openalex-snapshot
Targeting the Chemokines in Cardiac Repair
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/mco2.503openalex-snapshot
Reticulon 3 deficiency ameliorates post‐myocardial infarction heart failure by alleviating mitochondrial dysfunction and inflammation
inferred← citeshttps://doi.org/10.1007/s12012-025-09982-6openalex-snapshot
Downregulation of HSPB1 and MGST1 Promotes Ferroptosis and Impacts Immune Infiltration in Diabetic Cardiomyopathy
inferred← citeshttps://doi.org/10.1007/s12035-026-05754-7openalex-snapshot
Pharmacological Inhibition of the HSPB1-NF-κB Axis Alleviates Brain Injury in a Rat Veno-Venous Extracorporeal Membrane Oxygenation Model
inferred← citeshttps://doi.org/10.1016/j.abb.2022.109415openalex-snapshot
Sacubitril/valsartan attenuates myocardial ischemia/reperfusion injury via inhibition of the GSK3β/NF-κB pathway in cardiomyocytes
inferred← citeshttps://doi.org/10.1016/j.abb.2025.110400openalex-snapshot
HSPB1 suppresses oxLDL-induced vascular smooth muscle cell ferroptosis by inhibiting DPP4
inferred← citeshttps://doi.org/10.1016/j.apsb.2023.02.005openalex-snapshot
The traditional Chinese medicines treat chronic heart failure and their main bioactive constituents and mechanisms
inferred← citeshttps://doi.org/10.1016/j.arr.2025.102757openalex-snapshot
Intercellular communication after myocardial infarction: Macrophage as the centerpiece
inferred← citeshttps://doi.org/10.1016/j.bioadv.2025.214614openalex-snapshot
Cell mechanical microenvironment modulates the secretion phenotype of mesenchymal stem cell-derived exosomes for diabetic wound therapy
inferred← citeshttps://doi.org/10.1016/j.biopha.2021.112225openalex-snapshot
HSPB1 inhibitor J2 attenuates lung inflammation through direct modulation of Ym1 production and paracrine signaling
inferred← citeshttps://doi.org/10.1016/j.carbpol.2021.118996openalex-snapshot
Chondroitin sulfate zinc with antibacterial properties and anti-inflammatory effects for skin wound healing
inferred← citeshttps://doi.org/10.1016/j.cstres.2024.03.004openalex-snapshot
Roles of heat shock protein A12A in the development of diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.heliyon.2023.e18324openalex-snapshot
Insights gained from single-cell RNA analysis of murine endothelial cells in aging hearts
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2025.140793openalex-snapshot
Integration of single-cell and bulk RNA sequencing data using machine learning identifies oxidative stress-related genes LUM and PCOLCE2 as potential biomarkers for heart failure
inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2025.143099openalex-snapshot
Advanced nanofibers integrating vitamin D3 and cerium oxide nanoparticles for enhanced diabetic wound healing: Co-electrospun silk fibroin-collagen and chitosan-PVA systems
inferred← citeshttps://doi.org/10.1016/j.ijpharm.2023.123389openalex-snapshot
Effective myocardial infarction treatment by targeted accumulation of Sulforaphane using porous magnetic silica nanoparticles
inferred← citeshttps://doi.org/10.1016/j.isci.2023.107759openalex-snapshot
Mapping the cellular and molecular landscape of cardiac non-myocytes in murine diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.1016/j.isci.2026.115028openalex-snapshot
Cardiomyocyte-derived HSPB1 regulates TGF-β1 maturation and inhibits endothelial-to-mesenchymal transition in myocardial fibrosis
inferred← citeshttps://doi.org/10.1016/j.jare.2024.01.012openalex-snapshot
HSPA12A acts as a scaffolding protein to inhibit cardiac fibroblast activation and cardiac fibrosis
inferred← citeshttps://doi.org/10.1016/j.jot.2024.12.002openalex-snapshot
Humanin reduces nucleus pulposus cells ferroptosis to alleviate intervertebral disc degeneration: An in vitro and in vivo study
inferred← citeshttps://doi.org/10.1016/j.phrs.2020.105162openalex-snapshot
Therapeutic perspectives of heat shock proteins and their protein-protein interactions in myocardial infarction
inferred← citeshttps://doi.org/10.1016/j.yjmcc.2022.09.004openalex-snapshot
Peli1 Contributes to Myocardial Ischemia/Reperfusion Injury by Impairing Autophagy Flux Via its E3 Ligase Mediated Ubiquitination of P62
inferred← citeshttps://doi.org/10.1021/acsptsci.4c00272openalex-snapshot
Mechanisms and Therapeutic Strategies for Myocardial Ischemia-Reperfusion Injury in Diabetic States
inferred← citeshttps://doi.org/10.1038/s41401-023-01170-yopenalex-snapshot
Cardiac-specific deletion of BRG1 ameliorates ventricular arrhythmia in mice with myocardial infarction
inferred← citeshttps://doi.org/10.1080/13880209.2020.1840597openalex-snapshot
Latifolin protects against myocardial infarction by alleviating myocardial inflammatory via the HIF-1α/NF-κB/IL-6 pathway
inferred← citeshttps://doi.org/10.1080/21691401.2019.1669617openalex-snapshot
Macrophage-derived exosomes accelerate wound healing through their anti-inflammation effects in a diabetic rat model
inferred← citeshttps://doi.org/10.1089/scd.2018.0242openalex-snapshot
Exosomes Derived from Human Umbilical Cord Mesenchymal Stem Cells Promote Fibroblast-to-Myofibroblast Differentiation in Inflammatory Environments and Benefit Cardioprotective Effects
inferred← citeshttps://doi.org/10.1093/cvr/cvaa067openalex-snapshot
Sectm1a deficiency aggravates inflammation-triggered cardiac dysfunction through disruption of LXRα signalling in macrophages
inferred← citeshttps://doi.org/10.1101/2023.02.22.529500openalex-snapshot
Identification of novel prognostic targets in coronary artery disease and related complications using bioinformatics and next generation sequencing data analysis
inferred← citeshttps://doi.org/10.1101/2024.08.15.608116openalex-snapshot
The Identification of Key Genes and Biological Pathways in Cardiac Arrest by Integrated Bioinformatics and Next Generation Sequencing Data Analysis
inferred← citeshttps://doi.org/10.1101/2025.08.14.670204openalex-snapshot
Rapid CE–MS with Real-Time Eco–AI Resolves Proteomic Heterogeneity Among Single Human Neutrophils
inferred← citeshttps://doi.org/10.1111/acer.14471openalex-snapshot
Proteomic Profiling Reveals Roles of Stress Response, Ca2+ Transient Dysregulation, and Novel Signaling Pathways in Alcohol‐Induced Cardiotoxicity
inferred← citeshttps://doi.org/10.1111/jcmm.16876openalex-snapshot
MiR‐363‐5p modulates regulatory T cells through STAT4‐HSPB1‐Notch1 axis and is associated with the immunological abnormality in Graves' disease
inferred← citeshttps://doi.org/10.1152/ajpheart.00318.2020openalex-snapshot
Validation of diagnostic criteria and histopathological characterization of cardiac rupture in the mouse model of nonreperfused myocardial infarction
inferred← citeshttps://doi.org/10.1155/2021/5542815openalex-snapshot
Bmal1 Regulates the Redox Rhythm of HSPB1, and Homooxidized HSPB1 Attenuates the Oxidative Stress Injury of Cardiomyocytes
inferred← citeshttps://doi.org/10.1155/2022/2333848openalex-snapshot
HSPA12A Stimulates p38/ERK‐AP‐1 Signaling to Promote Angiogenesis and Is Required for Functional Recovery Postmyocardial Infarction
inferred← citeshttps://doi.org/10.1172/jci.insight.169125openalex-snapshot
HSPA12A maintains aerobic glycolytic homeostasis and Histone3 lactylation in cardiomyocytes to attenuate myocardial ischemia/reperfusion injury
inferred← citeshttps://doi.org/10.1186/s12014-024-09474-9openalex-snapshot
Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.12307/2021.307openalex-snapshot
Hypoxia-inducible factor and coronary heart disease: antagonism and protection
inferred← citeshttps://doi.org/10.17513/ejnh.34258openalex-snapshot
SEARCH FOR PREDICTORS OF POST-INFARCTION MYOCARDIAL RUPTURE IN CLINICAL PRACTICE
inferred← citeshttps://doi.org/10.21203/rs.3.rs-2364455/v1openalex-snapshot
Porous magnetic Silica nanoparticles loaded with sulforaphane mitigate myocardial infarction injury through upregulating HSP70
inferred← citeshttps://doi.org/10.2147/jir.s492465openalex-snapshot
PLL-g-HPA Hydrogel Loaded Human Umbilical Cord Mesenchymal Stem Cells Promote Burn Wound Healing in Rat Model by Regulating Inflammation Response
inferred← citeshttps://doi.org/10.26481/dis.20221122tnopenalex-snapshot
The way forward in multi-omics data analyses
inferred← citeshttps://doi.org/10.3389/fcell.2022.817819openalex-snapshot
Ferroptosis Related Genes in Ischemic and Idiopathic Cardiomyopathy: Screening for Potential Pharmacological Targets
inferred← citeshttps://doi.org/10.3389/fcell.2022.950927openalex-snapshot
Exercise-induced signaling pathways to counteracting cardiac apoptotic processes
inferred← citeshttps://doi.org/10.3389/fcvm.2021.633013openalex-snapshot
Heat Shock Proteins: Potential Modulators and Candidate Biomarkers of Peripartum Cardiomyopathy
inferred← citeshttps://doi.org/10.3389/fcvm.2021.647785openalex-snapshot
Post-ischemic Myocardial Inflammatory Response: A Complex and Dynamic Process Susceptible to Immunomodulatory Therapies
inferred← citeshttps://doi.org/10.3389/fcvm.2023.1195464openalex-snapshot
Mechanistic insights into heat shock protein 27, a potential therapeutic target for cardiovascular diseases
inferred← citeshttps://doi.org/10.3389/fgene.2021.695625openalex-snapshot
Translational Proteomics Analysis of Anthracycline-Induced Cardiotoxicity From Cardiac Microtissues to Human Heart Biopsies
inferred← citeshttps://doi.org/10.3389/fgene.2022.870222openalex-snapshot
Identification of molecular subtypes of coronary artery disease based on ferroptosis- and necroptosis-related genes
inferred← citeshttps://doi.org/10.3389/fimmu.2023.1163350openalex-snapshot
Risk prediction model construction for post myocardial infarction heart failure by blood immune B cells
inferred← citeshttps://doi.org/10.3389/fphar.2021.725186openalex-snapshot
Dangshen Erling Decoction Ameliorates Myocardial Hypertrophy via Inhibiting Myocardial Inflammation
inferred← citeshttps://doi.org/10.3389/fphar.2024.1494205openalex-snapshot
Brg1 and RUNX1 synergy in regulating TRPM4 channel in mouse cardiomyocytes
inferred← citeshttps://doi.org/10.3389/fphys.2020.00033openalex-snapshot
Dexmedetomidine Exerted Anti-arrhythmic Effects in Rat With Ischemic Cardiomyopathy via Upregulation of Connexin 43 and Reduction of Fibrosis and Inflammation
inferred← citeshttps://doi.org/10.3390/biomedicines9010052openalex-snapshot
SIRT1 Activation Attenuates the Cardiac Dysfunction Induced by Endothelial Cell-Specific Deletion of CRIF1
inferred← citeshttps://doi.org/10.6084/m9.figshare.25329754openalex-snapshot
Additional file 1 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.25329835openalex-snapshot
Additional file 2 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687699openalex-snapshot
Additional file 3 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687702openalex-snapshot
Additional file 4 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687705openalex-snapshot
Additional file 5 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687708openalex-snapshot
Additional file 6 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687711openalex-snapshot
Additional file 7 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687714openalex-snapshot
Additional file 8 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687717openalex-snapshot
Additional file 9 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.6084/m9.figshare.26687720openalex-snapshot
Additional file 10 of Proteomic analysis of plasma proteins from patients with cardiac rupture after acute myocardial infarction using TMT-based quantitative proteomics approach
inferred← citeshttps://doi.org/10.7150/thno.77694openalex-snapshot
Targeting WWP1 ameliorates cardiac ischemic injury by suppressing KLF15-ubiquitination mediated myocardial inflammation
inferred← citeshttps://doi.org/10.7150/thno.82607openalex-snapshot
Hepatocyte HSPA12A inhibits macrophage chemotaxis and activation to attenuate liver ischemia/reperfusion injury via suppressing glycolysis-mediated HMGB1 lactylation and secretion of hepatocytes
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.