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Mammalian Base Excision Repair: the Forgotten Archangel

https://doi.org/10.1093/nar/gkt076
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/bies.1063openalex-snapshot
Mammalian DNA single‐strand break repair: an X‐ra(y)ted affair
inferredcites →https://doi.org/10.1002/em.20312openalex-snapshot
XRCC1 down‐regulation in human cells leads to DNA‐damaging agent hypersensitivity, elevated sister chromatid exchange, and reduced survival of BRCA2 mutant cells
inferredcites →https://doi.org/10.1002/j.1460-2075.1996.tb01056.xopenalex-snapshot
Reconstitution of DNA base excision‐repair with purified human proteins: interaction between DNA polymerase beta and the XRCC1 protein.
inferredcites →https://doi.org/10.1006/abbi.2000.1773openalex-snapshot
The Human OGG1 Gene: Structure, Functions, and Its Implication in the Process of Carcinogenesis
inferredcites →https://doi.org/10.1007/978-1-4419-8740-2_1openalex-snapshot
Poly ADP-ribosylation: A DNA break signal mechanism
inferredcites →https://doi.org/10.1007/s00018-011-0839-7openalex-snapshot
Haploinsufficiency in mouse models of DNA repair deficiency: modifiers of penetrance
inferredcites →https://doi.org/10.1007/s00412-011-0347-4openalex-snapshot
DNA glycosylases: in DNA repair and beyond
inferredcites →https://doi.org/10.1016/0165-1110(90)90022-4openalex-snapshot
Repair of intrinsic DNA lesions
inferredcites →https://doi.org/10.1016/0921-8777(94)00035-5openalex-snapshot
Overexpression of N-methylpurine-DNA glycosylase in Chinese hamster ovary cells renders them more sensitive to the production of chromosomal aberrations by methylating agents — a case of imbalanced DNA repair
inferredcites →https://doi.org/10.1016/j.cbpa.2011.08.009openalex-snapshot
DNA replication and repair bypass machines
inferredcites →https://doi.org/10.1016/j.cell.2005.01.028openalex-snapshot
DNA Repair, Genome Stability, and Aging
inferredcites →https://doi.org/10.1016/j.cell.2005.03.037openalex-snapshot
ARF-BP1/Mule Is a Critical Mediator of the ARF Tumor Suppressor
inferredcites →https://doi.org/10.1016/j.dnarep.2003.08.007openalex-snapshot
Rescue of Xrcc1 knockout mouse embryo lethality by transgene-complementation
inferredcites →https://doi.org/10.1016/j.dnarep.2008.03.009openalex-snapshot
Poly ADP-ribose polymerase-1: An international molecule of mystery
inferredcites →https://doi.org/10.1016/j.dnarep.2008.03.017openalex-snapshot
Poly(ADP-ribose) polymerase-1 modulates DNA repair capacity and prevents formation of DNA double strand breaks
inferredcites →https://doi.org/10.1016/j.febslet.2011.03.069openalex-snapshot
Ubiquitylation of DNA polymerase λ
inferredcites →https://doi.org/10.1016/j.molcel.2004.06.003openalex-snapshot
AP Endonuclease-Independent DNA Base Excision Repair in Human Cells
inferredcites →https://doi.org/10.1016/j.molcel.2007.12.027openalex-snapshot
CHIP-Mediated Degradation and DNA Damage-Dependent Stabilization Regulate Base Excision Repair Proteins
inferredcites →https://doi.org/10.1016/j.molcel.2010.01.038openalex-snapshot
Frameshift Mutagenesis and Microsatellite Instability Induced by Human Alkyladenine DNA Glycosylase
inferredcites →https://doi.org/10.1016/j.mrfmmm.2003.05.001openalex-snapshot
DNA N-glycosylase deficient mice: a tale of redundancy
inferredcites →https://doi.org/10.1016/j.mrfmmm.2009.10.017openalex-snapshot
Uracil-DNA glycosylase: Structural, thermodynamic and kinetic aspects of lesion search and recognition
inferredcites →https://doi.org/10.1016/j.tibs.2011.01.006openalex-snapshot
Tidying up loose ends: the role of polynucleotide kinase/phosphatase in DNA strand break repair
inferredcites →https://doi.org/10.1016/s0079-6603(08)60800-4openalex-snapshot
DNA Glycosylases, Endonucleases for Apurinic/Apyrimidinic Sites, and Base Excision-Repair
inferredcites →https://doi.org/10.1016/s0960-9822(00)00245-1openalex-snapshot
Reconstitution of the DNA base excision—repair pathway
inferredcites →https://doi.org/10.1021/bi050085mopenalex-snapshot
DNA Polymerase β Promotes Recruitment of DNA Ligase IIIα−XRCC1 to Sites of Base Excision Repair
inferredcites →https://doi.org/10.1021/bi962281mopenalex-snapshot
XRCC1 Protein Interacts with One of Two Distinct Forms of DNA Ligase III
inferredcites →https://doi.org/10.1023/a:1006975002262openalex-snapshot
Poly ADP-ribosylation: A DNA break signal mechanism
inferredcites →https://doi.org/10.1038/35000249openalex-snapshot
DNA-bound structures and mutants reveal abasic DNA binding by APE1 DNA repair and coordination
inferredcites →https://doi.org/10.1038/35006693openalex-snapshot
Erratum: DNA-bound structures and mutants reveal abasic DNA binding by APE1 and DNA repair coordination
inferredcites →https://doi.org/10.1038/356356a0openalex-snapshot
Role of poly(ADP-ribose) formation in DNA repair
inferredcites →https://doi.org/10.1038/362709a0openalex-snapshot
Instability and decay of the primary structure of DNA
inferredcites →https://doi.org/10.1038/379183a0openalex-snapshot
Requirement of mammalian DNA polymerase-β in base-excision repair
inferredcites →https://doi.org/10.1038/73260openalex-snapshot
Passing the baton in base excision repair.
inferredcites →https://doi.org/10.1038/82818openalex-snapshot
Functional sites of interaction between release factor RF1 and the ribosome.
inferredcites →https://doi.org/10.1038/cr.2008.7openalex-snapshot
XRCC1 and DNA polymerase β in cellular protection against cytotoxic DNA single-strand breaks
inferredcites →https://doi.org/10.1038/emboj.2009.243openalex-snapshot
Ubiquitin ligase ARF‐BP1/Mule modulates base excision repair
inferredcites →https://doi.org/10.1038/nature01408openalex-snapshot
DNA damage and repair
inferredcites →https://doi.org/10.1038/nature03314openalex-snapshot
Defective DNA single-strand break repair in spinocerebellar ataxia with axonal neuropathy-1
inferredcites →https://doi.org/10.1038/nature03482openalex-snapshot
DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis
inferredcites →https://doi.org/10.1038/nature05164openalex-snapshot
The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates
inferredcites →https://doi.org/10.1038/nature05843openalex-snapshot
8-oxo-guanine bypass by human DNA polymerases in the presence of auxiliary proteins
inferredcites →https://doi.org/10.1038/nature06420openalex-snapshot
Poly(ADP-ribose)-binding zinc finger motifs in DNA repair/checkpoint proteins
inferredcites →https://doi.org/10.1038/nature08444openalex-snapshot
A human 5′-tyrosyl DNA phosphodiesterase that repairs topoisomerase-mediated DNA damage
inferredcites →https://doi.org/10.1038/nature09773openalex-snapshot
DNA ligase III is critical for mtDNA integrity but not Xrcc1-mediated nuclear DNA repair
inferredcites →https://doi.org/10.1038/nrg2380openalex-snapshot
Single-strand break repair and genetic disease
inferredcites →https://doi.org/10.1038/sj.onc.1207824openalex-snapshot
p14ARF is a component of the p53 response following ionizing irradiation of normal human fibroblasts
inferredcites →https://doi.org/10.1042/bst0310247openalex-snapshot
Protein–protein interactions during mammalian DNA single-strand break repair
inferredcites →https://doi.org/10.1046/j.1432-1327.2000.01266.xopenalex-snapshot
Overexpression of enzymes that repair endogenous damage to DNA
inferredcites →https://doi.org/10.1073/pnas.0406048101openalex-snapshot
In situ analysis of repair processes for oxidative DNA damage in mammalian cells
inferredcites →https://doi.org/10.1073/pnas.1110449109openalex-snapshot
Regulation of oxidative DNA damage repair by DNA polymerase λ and MutYH by cross-talk of phosphorylation and ubiquitination
inferredcites →https://doi.org/10.1073/pnas.1111237108openalex-snapshot
Strandwise translocation of a DNA glycosylase on undamaged DNA
inferredcites →https://doi.org/10.1073/pnas.131009198openalex-snapshot
Single-strand interruptions in replicating chromosomes cause double-strand breaks
inferredcites →https://doi.org/10.1073/pnas.94.1.109openalex-snapshot
Cloning and characterization of a functional human homolog of Escherichia coli endonuclease III
inferredcites →https://doi.org/10.1073/pnas.94.14.7303openalex-snapshot
Requirement of poly(ADP-ribose) polymerase in recovery from DNA damage in mice and in cells
inferredcites →https://doi.org/10.1073/pnas.97.7.3266openalex-snapshot
Differential requirement for p19ARF in the p53-dependent arrest induced by DNA damage, microtubule disruption, and ribonucleotide depletion
inferredcites →https://doi.org/10.1074/jbc.271.16.9573openalex-snapshot
Two Pathways for Base Excision Repair in Mammalian Cells
inferredcites →https://doi.org/10.1074/jbc.271.27.16000openalex-snapshot
Specific Interaction of DNA Polymerase β and DNA Ligase I in a Multiprotein Base Excision Repair Complex from Bovine Testis
inferredcites →https://doi.org/10.1074/jbc.272.38.23970openalex-snapshot
Involvement of XRCC1 and DNA Ligase III Gene Products in DNA Base Excision Repair
inferredcites →https://doi.org/10.1074/jbc.273.50.33811openalex-snapshot
Repair Pathways for Processing of 8-Oxoguanine in DNA by Mammalian Cell Extracts
inferredcites →https://doi.org/10.1074/jbc.274.20.13741openalex-snapshot
Role of DNA Polymerase β in the Excision Step of Long Patch Mammalian Base Excision Repair
inferredcites →https://doi.org/10.1074/jbc.275.16.11809openalex-snapshot
Single Nucleotide Patch Base Excision Repair Is the Major Pathway for Removal of Thymine Glycol from DNA in Human Cell Extracts
inferredcites →https://doi.org/10.1074/jbc.m006520200openalex-snapshot
Poly(ADP-ribose) Binds to Specific Domains in DNA Damage Checkpoint Proteins
inferredcites →https://doi.org/10.1093/carcin/20.6.1049openalex-snapshot
DNA polymerase expression differences in selected human tumors and cell lines
inferredcites →https://doi.org/10.1093/emboj/16.11.3341openalex-snapshot
Second pathway for completion of human DNA base excision‐repair: reconstitution with purified proteins and requirement for DNase IV (FEN1)
inferredcites →https://doi.org/10.1093/emboj/20.6.1477openalex-snapshot
Human DNA polymerase β initiates DNA synthesis during long‐patch repair of reduced AP sites in DNA
inferredcites →https://doi.org/10.1093/mutage/gel020openalex-snapshot
Base excision repair fidelity in normal and cancer cells
inferredcites →https://doi.org/10.1093/mutage/gel024openalex-snapshot
TDP1-dependent DNA single-strand break repair and neurodegeneration
inferredcites →https://doi.org/10.1093/mutage/gel070openalex-snapshot
Overexpression of DNA polymerase results in an increased rate of frameshift mutations during base excision repair
inferredcites →https://doi.org/10.1093/nar/23.23.4836openalex-snapshot
Characterization of the XRCC1-DNA ligase III complex in vitro and its absence from mutant hamster cells
inferredcites →https://doi.org/10.1093/nar/gkh872openalex-snapshot
Repair of U/G and U/A in DNA by UNG2-associated repair complexes takes place predominantly by short-patch repair both in proliferating and growth-arrested cells
inferredcites →https://doi.org/10.1093/nar/gki845openalex-snapshot
Phosphorylation of human DNA polymerase by the cyclin-dependent kinase Cdk2/cyclin A complex is modulated by its association with proliferating cell nuclear antigen
inferredcites →https://doi.org/10.1098/rstb.1995.0009openalex-snapshot
Enzymes acting at strand interruptions in DNA
inferredcites →https://doi.org/10.1101/gad.9.5.509openalex-snapshot
Mice lacking ADPRT and poly(ADP-ribosyl)ation develop normally but are susceptible to skin disease.
inferredcites →https://doi.org/10.1111/j.1742-4658.2005.04628.xopenalex-snapshot
Poly(ADP‐ribose) polymerase‐1 protects excessive DNA strand breaks from deterioration during repair in human cell extracts
inferredcites →https://doi.org/10.1111/j.1742-4658.2005.04962.xopenalex-snapshot
End‐damage‐specific proteins facilitate recruitment or stability of X‐ray cross‐complementing protein 1 at the sites of DNA single‐strand break repair
inferredcites →https://doi.org/10.1126/science.7624801openalex-snapshot
Excision of Deoxyribose Phosphate Residues by DNA Polymerase β During DNA Repair
inferredcites →https://doi.org/10.1128/mcb.00457-08openalex-snapshot
Removal of Oxidative DNA Damage via FEN1-Dependent Long-Patch Base Excision Repair in Human Cell Mitochondria
inferredcites →https://doi.org/10.1128/mcb.12.4.1605openalex-snapshot
Generation of Single-Nucleotide Repair Patches Following Excision of Uracil Residues from DNA
inferredcites →https://doi.org/10.1128/mcb.14.9.6187openalex-snapshot
Proliferating cell nuclear antigen-dependent abasic site repair in Xenopus laevis oocytes: an alternative pathway of base excision DNA repair.
inferredcites →https://doi.org/10.1128/mcb.14.9.6187-6197.1994openalex-snapshot
Proliferating Cell Nuclear Antigen-Dependent Abasic Site Repair in Xenopus laevis Oocytes: an Alternative Pathway of Base Excision DNA Repair
inferredcites →https://doi.org/10.1128/mcb.23.11.3974-3981.2003openalex-snapshot
Spatial and Temporal Cellular Responses to Single-Strand Breaks in Human Cells
inferredcites →https://doi.org/10.1128/mcb.23.15.5472.2003openalex-snapshot
Spatial and Temporal Cellular Responses to Single-Strand Breaks in Human Cells
inferredcites →https://doi.org/10.1142/7667openalex-snapshot
DNA Polymerases
inferredcites →https://doi.org/10.1142/7667#t=tocopenalex-snapshot
DNA Polymerases: Discovery, Characterization and Functions in Cellular DNA Transactions
inferredcites →https://doi.org/10.1146/annurev.bi.63.070194.004411openalex-snapshot
REPAIR OF OXIDATIVE DAMAGE TO DNA: Enzymology and Biology
inferredcites →https://doi.org/10.18388/abp.2003_3724openalex-snapshot
Poly(ADP-ribose) polymerase in base excision repair: always engaged, but not essential for DNA damage processing.

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Alkylating‐Agent Cytotoxicity Associated with O 6‐Methylguanine
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The Principles and Drivers of Cancer
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Specificity of CRISPR‐Cas9 Gene Editing
inferred← citeshttps://doi.org/10.1002/bies.70092openalex-snapshot
The Double Face of Base Excision Repair: Preventing and Triggering Double‐Strand Breaks
inferred← citeshttps://doi.org/10.1002/biot.70303openalex-snapshot
Nucleases and Their Inhibitors: Exploring Biological Roles, Industrial Applications, and Challenges in Heterologous Expression
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Development of the adverse outcome pathway “alkylation of DNA in male premeiotic germ cells leading to heritable mutations” using the OECD 's users' handbook supplement
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Mechanisms of DNA damage, repair, and mutagenesis
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Tandem Affinity Purification and Mass-Spectrometric Analysis of FACT and Associated Proteins
inferred← citeshttps://doi.org/10.1007/978-1-4939-9434-2_11openalex-snapshot
Biochemical and Cellular Assays to Assess the Effects of Acetylation on Base Excision Repair Enzymes
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Magnetic Nanoparticle Hyperthermia
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Inhibition of DNA Repair System Activity
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Clinical Relevance of BRCA1/2 Pathogenic Variants and Impaired DNA Repair Pathways in Ovarian Carcinomas
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The Role of H. pylori in Gastric Carcinogenesis
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Genetic Engineering: A Powerful Tool for Crop Improvement
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Imaging the DNA damage response with PET and SPECT
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Transcriptional alteration of DNA repair genes in Philadelphia chromosome negative myeloproliferative neoplasms
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A glance at genome editing with CRISPR–Cas9 technology
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CRISPR-Cas orthologues and variants: optimizing the repertoire, specificity and delivery of genome engineering tools
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Current aspects of DNA damage and repair in ecotoxicology: a mini-review
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Harnessing the Potential of Human Pluripotent Stem Cells and Gene Editing for the Treatment of Retinal Degeneration
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Helicobacter pylori infection induced genome instability and gastric cancer
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Tagging Endogenous Loci for Live-Cell Fluorescence Imaging and Molecule Counting Using ZFNs, TALENs, and Cas9
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Genome Editing Using Cas9 Nickases
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DNA Repair and Its Influence on Points of Departure for Alkylating Agent Genotoxicity
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Genome Stability in Caenorhabditis elegans
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Understanding the basics for translating the base excision repair pathway from benchtop to bedside in cancer treatment
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Forward and Reverse Genetics to Model Human Diseases in the Mouse
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Neurodegenerative Diseases and the Aging Brain
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CRISPR-Cas orthologs and variants
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Gene editing and central nervous system regeneration
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Advances and application of CRISPR-Cas systems
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Error-Prone DNA Mismatch and Base Excision Repair in the Somatic Hypermutation of Antibody Variable Regions
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Genome stability in Caenorhabditis elegans
inferred← citeshttps://doi.org/10.1016/b978-0-443-27416-9.00004-6openalex-snapshot
CRISPR/Cas9 gene editing for treating gene and protein disorders
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Enzymology of mitochondrial DNA repair
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Endonucleases: new tools to edit the mouse genome
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Crystal structure of the apurinic/apyrimidinic endonuclease XthA (HP1526 protein) from Helicobacter pylori
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Helicity of the bridge helix of Cas12a regulates on-target DNA cleavage efficiency and off-target cleavage propensity
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Potential effects of metformin in DNA BER system based on oxidative status in type 2 diabetes
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Coordination between human DNA polymerase β and apurinic/apyrimidinic endonuclease 1 in the course of DNA repair
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DNA repair and genomic stability in lungs affected by acute injury
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Altération de la réparation de l’ADN et cancer
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Insights into mechanism of DNA damage and repair-apoptosis in digestive gland of female scallop Chlamys farreri under benzo[a]pyrene exposure during reproductive stage
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Double Nicking by RNA-Guided CRISPR Cas9 for Enhanced Genome Editing Specificity
inferred← citeshttps://doi.org/10.1016/j.cell.2014.05.010openalex-snapshot
Development and Applications of CRISPR-Cas9 for Genome Engineering
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.113654openalex-snapshot
NEIL1 drives the initiation of colorectal cancer through transcriptional regulation of COL17A1
inferred← citeshttps://doi.org/10.1016/j.celrep.2025.115718openalex-snapshot
Cas12h is a crRNA-guided DNA nickase that can be utilized for precise gene editing
inferred← citeshttps://doi.org/10.1016/j.celrep.2026.118109openalex-snapshot
Oxidative DNA damage drives centromere fragility and chromosome instability
inferred← citeshttps://doi.org/10.1016/j.chemphys.2022.111456openalex-snapshot
A novel ligand swing-mediated active site coordination change of human apurinic/apyrimidinic endonuclease 1: A potential cytotoxic mechanism of nickel ion in the base excision repair
inferred← citeshttps://doi.org/10.1016/j.copbio.2017.06.012openalex-snapshot
Genetic engineering as a tool for the generation of mouse models to understand disease phenotypes and gene function
inferred← citeshttps://doi.org/10.1016/j.dcmed.2024.09.007openalex-snapshot
Integrative analysis of bone-formation associated genes and immune cell infiltration in osteoporosis, and the prediction of active ingredients in targeted traditional Chinese medicine
inferred← citeshttps://doi.org/10.1016/j.dnarep.2014.03.030openalex-snapshot
Base excision repair: A critical player in many games
inferred← citeshttps://doi.org/10.1016/j.dnarep.2015.03.007openalex-snapshot
Cell cycle regulation of human DNA repair and chromatin remodeling genes
inferred← citeshttps://doi.org/10.1016/j.dnarep.2015.09.004openalex-snapshot
XPC: Going where no DNA damage sensor has gone before
inferred← citeshttps://doi.org/10.1016/j.dnarep.2016.12.007openalex-snapshot
Processing of the abasic sites clustered with the benzo[a]pyrene adducts by the base excision repair enzymes
inferred← citeshttps://doi.org/10.1016/j.dnarep.2017.02.011openalex-snapshot
A role for the base excision repair enzyme NEIL3 in replication-dependent repair of interstrand DNA cross-links derived from psoralen and abasic sites
inferred← citeshttps://doi.org/10.1016/j.dnarep.2017.06.008openalex-snapshot
Unveiling the non-repair face of the Base Excision Repair pathway in RNA processing: A missing link between DNA repair and gene expression?
inferred← citeshttps://doi.org/10.1016/j.dnarep.2017.09.007openalex-snapshot
Not breathing is not an option: How to deal with oxidative DNA damage
inferred← citeshttps://doi.org/10.1016/j.dnarep.2019.02.001openalex-snapshot
Deployment of DNA polymerases beta and lambda in single-nucleotide and multinucleotide pathways of mammalian base excision DNA repair
inferred← citeshttps://doi.org/10.1016/j.dnarep.2019.102665openalex-snapshot
Diverse functions of DNA glycosylases processing oxidative base lesions in brain
inferred← citeshttps://doi.org/10.1016/j.dnarep.2019.102679openalex-snapshot
Polyphenols: Major regulators of key components of DNA damage response in cancer
inferred← citeshttps://doi.org/10.1016/j.dnarep.2019.102740openalex-snapshot
Sp1-independent downregulation of NHEJ in response to BER deficiency
inferred← citeshttps://doi.org/10.1016/j.dnarep.2020.102951openalex-snapshot
DNA repair pathways as guardians of the genome: Therapeutic potential and possible prognostic role in hematologic neoplasms
inferred← citeshttps://doi.org/10.1016/j.dnarep.2021.103133openalex-snapshot
Coping with RNA damage with a focus on APE1, a BER enzyme at the crossroad between DNA damage repair and RNA processing/decay
inferred← citeshttps://doi.org/10.1016/j.dnarep.2023.103450openalex-snapshot
Fluorescently labeled human apurinic/apyrimidinic endonuclease APE1 reveals effects of DNA polymerase β on the APE1–DNA interaction
inferred← citeshttps://doi.org/10.1016/j.dnarep.2024.103756openalex-snapshot
C-terminal residues of DNA polymerase β and E3 ligase required for ubiquitin-linked proteolysis of oxidative DNA-protein crosslinks
inferred← citeshttps://doi.org/10.1016/j.dnarep.2025.103827openalex-snapshot
Modulation of OGG1 enzymatic activities by small molecules, promising tools and current challenges
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2016.11.025openalex-snapshot
Risky repair: DNA-protein crosslinks formed by mitochondrial base excision DNA repair enzymes acting on free radical lesions
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2016.11.042openalex-snapshot
Repair of 8-oxo-7,8-dihydroguanine in prokaryotic and eukaryotic cells: Properties and biological roles of the Fpg and OGG1 DNA N-glycosylases
inferred← citeshttps://doi.org/10.1016/j.gene.2019.04.072openalex-snapshot
LIG3 gene polymorphisms and risk of gastric cancer in a Southern Chinese population
inferred← citeshttps://doi.org/10.1016/j.gene.2020.145225openalex-snapshot
An overview of currently available molecular Cas-tools for precise genome modification
inferred← citeshttps://doi.org/10.1016/j.genrep.2020.100832openalex-snapshot
A decade in unravelling the etiology of gastric carcinogenesis in Kashmir, India – A high risk region
inferred← citeshttps://doi.org/10.1016/j.ica.2016.01.021openalex-snapshot
Targeting DNA mismatches with rhodium metalloinsertors
inferred← citeshttps://doi.org/10.1016/j.ijpharm.2021.120446openalex-snapshot
The APEX1/miRNA-27a-5p axis plays key roles in progression, metastasis and targeted chemotherapy of gastric cancer
inferred← citeshttps://doi.org/10.1016/j.jbc.2023.104636openalex-snapshot
Visualizing the coordination of apurinic/apyrimidinic endonuclease (APE1) and DNA polymerase β during base excision repair
inferred← citeshttps://doi.org/10.1016/j.jgg.2015.06.005openalex-snapshot
The Application of CRISPR-Cas9 Genome Editing in Caenorhabditis elegans
inferred← citeshttps://doi.org/10.1016/j.jmb.2014.03.014openalex-snapshot
APE1 Incision Activity at Abasic Sites in Tandem Repeat Sequences
inferred← citeshttps://doi.org/10.1016/j.jmb.2019.05.028openalex-snapshot
A Single-Molecule Atomic Force Microscopy Study of PARP1 and PARP2 Recognition of Base Excision Repair DNA Intermediates
inferred← citeshttps://doi.org/10.1016/j.jmb.2020.03.020openalex-snapshot
RanBP2-Mediated SUMOylation Promotes Human DNA Polymerase Lambda Nuclear Localization and DNA Repair
inferred← citeshttps://doi.org/10.1016/j.jmb.2020.166806openalex-snapshot
CUT Domains Stimulate Pol β Enzymatic Activities to Accelerate Completion of Base Excision Repair
inferred← citeshttps://doi.org/10.1016/j.lfs.2015.08.019openalex-snapshot
Molecular mechanism of adenomatous polyposis coli-induced blockade of base excision repair pathway in colorectal carcinogenesis
inferred← citeshttps://doi.org/10.1016/j.lfs.2022.120852openalex-snapshot
Molecular chaperones in DNA repair mechanisms: Role in genomic instability and proteostasis in cancer
inferred← citeshttps://doi.org/10.1016/j.mce.2017.06.004openalex-snapshot
NR1D1 enhances oxidative DNA damage by inhibiting PARP1 activity
inferred← citeshttps://doi.org/10.1016/j.molonc.2014.01.001openalex-snapshot
DNA polymerase β deficiency is linked to aggressive breast cancer: A comprehensive analysis of gene copy number, mRNA and protein expression in multiple cohorts
inferred← citeshttps://doi.org/10.1016/j.molonc.2014.08.001openalex-snapshot
Targeting BRCA1‐BER deficient breast cancer by ATM or DNA‐PKcs blockade either alone or in combination with cisplatin for personalized therapy
inferred← citeshttps://doi.org/10.1016/j.mrfmmm.2015.01.003openalex-snapshot
Effects of post mortem interval and gender in DNA base excision repair activities in rat brains
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2022.503447openalex-snapshot
A pooled analysis of molecular epidemiological studies on modulation of DNA repair by host factors
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2022.503462openalex-snapshot
Dose response to methylating agents in the γH2AX, SCE and colony formation assays: Effect of MGMT and MPG overexpression
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2023.503685openalex-snapshot
Comparison of the transgenic rodent mutation assay, error corrected next generation duplex sequencing, and the alkaline comet assay to detect dose-related mutations following exposure to N-nitrosodiethylamine
inferred← citeshttps://doi.org/10.1016/j.mrrev.2015.05.001openalex-snapshot
Theoretical considerations for thresholds in chemical carcinogenesis
inferred← citeshttps://doi.org/10.1016/j.mrrev.2021.108376openalex-snapshot
Mitotic syndicates Aurora Kinase B (AURKB) and mitotic arrest deficient 2 like 2 (MAD2L2) in cohorts of DNA damage response (DDR) and tumorigenesis
inferred← citeshttps://doi.org/10.1016/j.ncrna.2024.03.010openalex-snapshot
Long non-coding RNA LIP interacts with PARP-1 influencing the efficiency of base excision repair
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2014.10.002openalex-snapshot
Patient-specific induced pluripotent stem cells (iPSCs) for the study and treatment of retinal degenerative diseases
inferred← citeshttps://doi.org/10.1016/j.redox.2025.103770openalex-snapshot
Sirt2 ablation exacerbates Sod1 knockout-induced progeroid phenotype in mice
inferred← citeshttps://doi.org/10.1016/j.str.2024.02.014openalex-snapshot
Structural insights into the catalytic mechanism of the AP endonuclease AtARP
inferred← citeshttps://doi.org/10.1016/j.tranon.2024.101977openalex-snapshot
HMGB2 is a biomarker associated with poor prognosis promoting radioresistance in glioma by targeting base excision repair pathway
inferred← citeshttps://doi.org/10.1016/j.virusres.2013.10.017openalex-snapshot
Repair of base damage and genome maintenance in the nucleo-cytoplasmic large DNA viruses
inferred← citeshttps://doi.org/10.1016/j.ygeno.2025.111091openalex-snapshot
High-resolution genome-wide profiling reveals intricate relationship between DNA damage and sperm physiology
inferred← citeshttps://doi.org/10.1016/j.ymgme.2021.08.002openalex-snapshot
Current advances in overcoming obstacles of CRISPR/Cas9 off-target genome editing
inferred← citeshttps://doi.org/10.1016/j.ymthe.2017.07.005openalex-snapshot
COL7A1 Editing via CRISPR/Cas9 in Recessive Dystrophic Epidermolysis Bullosa
inferred← citeshttps://doi.org/10.1016/j.ymthe.2017.08.015openalex-snapshot
Cut and Paste: Efficient Homology-Directed Repair of a Dominant Negative KRT14 Mutation via CRISPR/Cas9 Nickases
inferred← citeshttps://doi.org/10.1016/j.ymthe.2024.03.006openalex-snapshot
On- and off-target effects of paired CRISPR-Cas nickase in primary human cells
inferred← citeshttps://doi.org/10.1021/acs.analchem.5c06141openalex-snapshot
Single Molecular Anchor-Assisted Nanopore Profiling of DNA Abasic Site Reactivity Dynamics
inferred← citeshttps://doi.org/10.1021/acs.chemrestox.6b00128openalex-snapshot
Oxidative DNA Damage and Repair in the Radioresistant Archaeon Thermococcus gammatolerans
inferred← citeshttps://doi.org/10.1021/acs.chemrev.6b00191openalex-snapshot
Role of Base Excision “Repair” Enzymes in Erasing Epigenetic Marks from DNA
inferred← citeshttps://doi.org/10.1021/acs.est.5c06946openalex-snapshot
Mechanistic Insights into Adenovirus Resistance to UV: Dose-Dependent Repair and Delayed Suppression of Host DNA Damage Response
inferred← citeshttps://doi.org/10.1021/acs.jctc.8b00645openalex-snapshot
Vertical Ionization Energies and Electron Affinities of Native and Damaged DNA Bases, Nucleotides, and Pairs from Density Functional Theory Calculations: Model Assessment and Implications for DNA Damage Recognition and Repair
inferred← citeshttps://doi.org/10.1021/acs.jpclett.4c00161openalex-snapshot
Enhanced Sampling for Conformational Changes and Molecular Mechanisms of Human NTHL1
inferred← citeshttps://doi.org/10.1021/acschembio.7b00259openalex-snapshot
Synergistic Effects of an Irreversible DNA Polymerase Inhibitor and DNA Damaging Agents on HeLa Cells
inferred← citeshttps://doi.org/10.1021/ar400275aopenalex-snapshot
A Chemical and Kinetic Perspective on Base Excision Repair of DNA
inferred← citeshttps://doi.org/10.1021/jacs.7b05048openalex-snapshot
Time-Dependent Extension from an 8-Oxoguanine Lesion by Human DNA Polymerase Beta
inferred← citeshttps://doi.org/10.1038/mtna.2016.95openalex-snapshot
CRISPR-Cas9 System as a Versatile Tool for Genome Engineering in Human Cells
inferred← citeshttps://doi.org/10.1038/ncomms10806openalex-snapshot
Biochemical reconstitution of TET1–TDG–BER-dependent active DNA demethylation reveals a highly coordinated mechanism
inferred← citeshttps://doi.org/10.1038/nrc3805openalex-snapshot
CUX1, a haploinsufficient tumour suppressor gene overexpressed in advanced cancers
inferred← citeshttps://doi.org/10.1038/nsmb.3105openalex-snapshot
Capturing snapshots of APE1 processing DNA damage
inferred← citeshttps://doi.org/10.1038/s12276-022-00822-zopenalex-snapshot
8-Oxoguanine: from oxidative damage to epigenetic and epitranscriptional modification
inferred← citeshttps://doi.org/10.1038/s41388-021-01710-yopenalex-snapshot
Molecular disruption of DNA polymerase β for platinum sensitisation and synthetic lethality in epithelial ovarian cancers
inferred← citeshttps://doi.org/10.1038/s41392-021-00648-7openalex-snapshot
DNA damage repair: historical perspectives, mechanistic pathways and clinical translation for targeted cancer therapy
inferred← citeshttps://doi.org/10.1038/s41392-023-01309-7openalex-snapshot
CRISPR/Cas9 therapeutics: progress and prospects
inferred← citeshttps://doi.org/10.1038/s41392-023-01548-8openalex-snapshot
A new wave of innovations within the DNA damage response
inferred← citeshttps://doi.org/10.1038/s41392-026-03032-5openalex-snapshot
RNA therapeutics: current status and future directions
inferred← citeshttps://doi.org/10.1038/s41467-018-03159-2openalex-snapshot
PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation
inferred← citeshttps://doi.org/10.1038/s41467-019-09006-2openalex-snapshot
CRISPR-Cas9 genome editing induces megabase-scale chromosomal truncations
inferred← citeshttps://doi.org/10.1038/s41467-022-33594-1openalex-snapshot
Complex genomic patterns of abasic sites in mammalian DNA revealed by a high-resolution SSiNGLe-AP method
inferred← citeshttps://doi.org/10.1038/s41467-024-54141-0openalex-snapshot
Loss of cytoplasmic actin filaments raises nuclear actin levels to drive INO80C-dependent chromosome fragmentation
inferred← citeshttps://doi.org/10.1038/s41573-023-00775-6openalex-snapshot
Therapeutic approaches for Duchenne muscular dystrophy
inferred← citeshttps://doi.org/10.1038/s41598-020-59072-6openalex-snapshot
Alkyladenine DNA glycosylase deficiency uncouples alkylation-induced strand break generation from PARP-1 activation and glycolysis inhibition
inferred← citeshttps://doi.org/10.1038/s41598-020-62040-9openalex-snapshot
Cervical carcinoma risk associate with genetic polymorphisms of NEIL2 gene in Chinese population and its significance as predictive biomarker
inferred← citeshttps://doi.org/10.1038/s41598-021-97523-wopenalex-snapshot
Versatile cell-based assay for measuring DNA alkylation damage and its repair
inferred← citeshttps://doi.org/10.1038/s41598-026-46639-yopenalex-snapshot
Increased vulnerability of colorectal cancer models with high alkylation damage to NTHL1 inactivation
inferred← citeshttps://doi.org/10.1038/s44321-023-00008-8openalex-snapshot
Efficient and safe therapeutic use of paired Cas9-nickases for primary hyperoxaluria type 1
inferred← citeshttps://doi.org/10.1038/srep10710openalex-snapshot
CRISPR/Cas9-mediated reporter knock-in in mouse haploid embryonic stem cells
inferred← citeshttps://doi.org/10.1038/srep17066openalex-snapshot
Tumor-selective use of DNA base excision repair inhibition in pancreatic cancer using the NQO1 bioactivatable drug, β-lapachone
inferred← citeshttps://doi.org/10.1038/srep25516openalex-snapshot
CT-Finder: A Web Service for CRISPR Optimal Target Prediction and Visualization
inferred← citeshttps://doi.org/10.1038/srep41783openalex-snapshot
Polymorphism of apyrimidinic DNA structures in the nucleosome
inferred← citeshttps://doi.org/10.1039/c3sc51228bopenalex-snapshot
Luminescent oligonucleotide-based detection of enzymes involved with DNA repair
inferred← citeshttps://doi.org/10.1039/c5ay01776aopenalex-snapshot
A dual enzymatic amplified strategy for the detection of endonuclease V activity
inferred← citeshttps://doi.org/10.1073/pnas.1402391111openalex-snapshot
Differential regulation of S-region hypermutation and class-switch recombination by noncanonical functions of uracil DNA glycosylase
inferred← citeshttps://doi.org/10.1073/pnas.1416031112openalex-snapshot
ATM prevents DSB formation by coordinating SSB repair and cell cycle progression
inferred← citeshttps://doi.org/10.1073/pnas.2111404119openalex-snapshot
A DNA repair-independent role for alkyladenine DNA glycosylase in alkylation-induced unfolded protein response
inferred← citeshttps://doi.org/10.1073/pnas.2118940119openalex-snapshot
Interlocking activities of DNA polymerase β in the base excision repair pathway
inferred← citeshttps://doi.org/10.1074/jbc.m114.629907openalex-snapshot
Influence of Oxidized Purine Processing on Strand Directionality of Mismatch Repair
inferred← citeshttps://doi.org/10.1074/jbc.m116.735696openalex-snapshot
Special AT-rich Sequence-binding Protein 1 (SATB1) Functions as an Accessory Factor in Base Excision Repair
inferred← citeshttps://doi.org/10.1074/jbc.ra118.004548openalex-snapshot
Poly(ADP-ribose) polymerase 1 (PARP1) promotes oxidative stress–induced association of Cockayne syndrome group B protein with chromatin
inferred← citeshttps://doi.org/10.1080/09670262.2017.1358827openalex-snapshot
Random mutagenesis and precise gene editing technologies: applications in algal crop improvement and functional genomics
inferred← citeshttps://doi.org/10.1080/10408444.2017.1423462openalex-snapshot
Development of an adverse outcome pathway for chemically induced hepatocellular carcinoma: case study of AFB1, a human carcinogen with a mutagenic mode of action
inferred← citeshttps://doi.org/10.1080/10409238.2020.1869173openalex-snapshot
The complexity and regulation of repair of alkylation damage to nucleic acids
inferred← citeshttps://doi.org/10.1080/1354750x.2016.1204006openalex-snapshot
Base excision repair pathway:PARP1genotypes as modulators of therapy response in cervical cancer patients
inferred← citeshttps://doi.org/10.1080/14656566.2021.1952983openalex-snapshot
Overview of Olaparib as a treatment option for metastatic castration-resistant prostate cancer
inferred← citeshttps://doi.org/10.1080/15476286.2021.1874161openalex-snapshot
Molecular correction of Duchenne muscular dystrophy by splice modulation and gene editing
inferred← citeshttps://doi.org/10.1080/17460794.2025.2465178openalex-snapshot
The relationship between KSHV and the cellular DNA damage response
inferred← citeshttps://doi.org/10.1083/jcb.201312078openalex-snapshot
DNA breaks and chromosomal aberrations arise when replication meets base excision repair
inferred← citeshttps://doi.org/10.1089/ars.2013.5492openalex-snapshot
Human Apurinic/Apyrimidinic Endonuclease 1
inferred← citeshttps://doi.org/10.1089/ars.2014.6077openalex-snapshot
Is There a Role for Base Excision Repair in Estrogen/Estrogen Receptor-Driven Breast Cancers?
inferred← citeshttps://doi.org/10.1089/omi.2015.0023openalex-snapshot
CRISPR-Cas9 Based Genome Engineering: Opportunities in Agri-Food-Nutrition and Healthcare
inferred← citeshttps://doi.org/10.1091/mbc.e20-10-0680openalex-snapshot
Cytoskeleton integrity influences XRCC1 and PCNA dynamics at DNA damage
inferred← citeshttps://doi.org/10.1093/bfgp/elw038openalex-snapshot
Molecular basis, applications and challenges of CRISPR/Cas9: a continuously evolving tool for genome editing
inferred← citeshttps://doi.org/10.1093/carcin/bgu225openalex-snapshot
Base excision repair capacity in informing healthspan
inferred← citeshttps://doi.org/10.1093/mutage/geab011openalex-snapshot
NEK1 deficiency affects mitochondrial functions and the transcriptome of key DNA repair pathways
inferred← citeshttps://doi.org/10.1093/mutage/gez051openalex-snapshot
New perspectives in cancer biology from a study of canonical and non-canonical functions of base excision repair proteins with a focus on early steps
inferred← citeshttps://doi.org/10.1093/nar/gkaa1002openalex-snapshot
A unified model for the G1/S cell cycle transition
inferred← citeshttps://doi.org/10.1093/nar/gkac695openalex-snapshot
Processing oxidatively damaged bases at DNA strand breaks by APE1
inferred← citeshttps://doi.org/10.1093/nar/gkad1037openalex-snapshot
The function of BCL11B in base excision repair contributes to its dual role as an oncogene and a haplo-insufficient tumor suppressor gene
inferred← citeshttps://doi.org/10.1093/nar/gkae190openalex-snapshot
SIRT2 promotes base excision repair by transcriptionally activating OGG1 in an ATM/ATR-dependent manner
inferred← citeshttps://doi.org/10.1093/nar/gkaf706openalex-snapshot
The function of MYC in base excision repair protects against RAS-induced senescence
inferred← citeshttps://doi.org/10.1093/nar/gkag921openalex-snapshot
Abasic site repair in APE1-deficient mouse embryonic stem cells: a matter for the MRN complex and ATM signaling
inferred← citeshttps://doi.org/10.1093/nar/gkag939openalex-snapshot
Structural basis of PARP2 recognition of nucleosomal DNA breaks and inhibitor trapping
inferred← citeshttps://doi.org/10.1093/nar/gkt1185openalex-snapshot
ARF induction in response to DNA strand breaks is regulated by PARP1
inferred← citeshttps://doi.org/10.1093/nar/gkt635openalex-snapshot
Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptation
inferred← citeshttps://doi.org/10.1093/nar/gku131openalex-snapshot
ARTD2 activity is stimulated by RNA
inferred← citeshttps://doi.org/10.1093/nar/gku251openalex-snapshot
High-efficiency genome editing via 2A-coupled co-expression of fluorescent proteins and zinc finger nucleases or CRISPR/Cas9 nickase pairs
inferred← citeshttps://doi.org/10.1093/nar/gkv121openalex-snapshot
A novel endonuclease that may be responsible for damaged DNA base repair in Pyrococcus furiosus
inferred← citeshttps://doi.org/10.1093/nar/gkv1520openalex-snapshot
3CAPS – a structural AP–site analogue as a tool to investigate DNA base excision repair
inferred← citeshttps://doi.org/10.1093/nar/gkv222openalex-snapshot
Cells deficient in base-excision repair reveal cancer hallmarks originating from adjustments to genetic instability
inferred← citeshttps://doi.org/10.1093/nar/gkw015openalex-snapshot
p53 coordinates base excision repair to prevent genomic instability
inferred← citeshttps://doi.org/10.1093/nar/gkw1300openalex-snapshot
Proximity to AGCT sequences dictates MMR-independent versus MMR-dependent mechanisms for AID-induced mutationviaUNG2
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