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Bradykinin and high glucose promote renal tubular inflammation
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/jcla.2057openalex-snapshot
Urinary levels of monocyte chemoattractant protein‐1 (MCP‐1) and interleukin‐8 (IL‐8), and renal injuries in patients with type 2 diabetic nephropathy
inferredcites →https://doi.org/10.1007/bf00211772openalex-snapshot
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Impact of the population at risk of diabetes on projections of diabetes burden in the United States: an epidemic on the way
inferredcites →https://doi.org/10.1016/j.bbrc.2006.04.127openalex-snapshot
High glucose increases B1-kinin receptor expression and signaling in endothelial cells
inferredcites →https://doi.org/10.1016/s0162-3109(97)00028-3openalex-snapshot
Localisation of tissue kallikrein in the kidney of black African women with early onset pre-eclampsia: a pilot study
inferredcites →https://doi.org/10.1016/s1567-5769(02)00188-1openalex-snapshot
Diabetes modulates the expression of glomerular kinin receptors
inferredcites →https://doi.org/10.1016/s1567-5769(02)00273-4openalex-snapshot
Kinins are involved in the antiproteinuric effect of angiotensin-converting enzyme inhibition in experimental diabetic nephropathy
inferredcites →https://doi.org/10.1038/414782aopenalex-snapshot
Global and societal implications of the diabetes epidemic
inferredcites →https://doi.org/10.1038/414813aopenalex-snapshot
Biochemistry and molecular cell biology of diabetic complications
inferredcites →https://doi.org/10.1038/86490openalex-snapshot
Vascular endothelial growth factor enhances atherosclerotic plaque progression
inferredcites →https://doi.org/10.1038/ki.1995.339openalex-snapshot
Cellular localization of tissue kallikrein and kallistatin mRNAs in human kidney
inferredcites →https://doi.org/10.1038/labinvest.2008.60openalex-snapshot
The pivotal role of VEGF on glomerular macrophage infiltration in advanced diabetic nephropathy
inferredcites →https://doi.org/10.1038/ncpneph0757openalex-snapshot
Mechanisms of Disease: the hypoxic tubular hypothesis of diabetic nephropathy
inferredcites →https://doi.org/10.1038/nrd1522openalex-snapshot
Bradykinin receptor ligands: therapeutic perspectives
inferredcites →https://doi.org/10.1038/sj.ki.5000014openalex-snapshot
Monocyte chemoattractant protein-1 promotes the development of diabetic renal injury in streptozotocin-treated mice
inferredcites →https://doi.org/10.1042/cs20070462openalex-snapshot
Role of PPARγ in renoprotection in Type 2 diabetes: molecular mechanisms and therapeutic potential
inferredcites →https://doi.org/10.1046/j.1523-1755.2002.00475.xopenalex-snapshot
Inhibition of IGF-I–induced Erk 1 and 2 activation and mitogenesis in mesangial cells by bradykinin
inferredcites →https://doi.org/10.1073/pnas.0405449101openalex-snapshot
Diabetic nephropathy is markedly enhanced in mice lacking the bradykinin B2 receptor
inferredcites →https://doi.org/10.1093/rheumatology/kep016openalex-snapshot
Kallikreins, kininogens and kinin receptors on circulating and synovial fluid neutrophils: role in kinin generation in rheumatoid arthritis
inferredcites →https://doi.org/10.1111/j.1523-1755.2004.00494.xopenalex-snapshot
The role of bradykinin in the antifibrotic actions of perindoprilat on human mesangial cells
inferredcites →https://doi.org/10.1111/j.1523-1755.2004.00653.xopenalex-snapshot
Proteinuria, a target for renoprotection in patients with type 2 diabetic nephropathy: Lessons from RENAAL
inferredcites →https://doi.org/10.1111/j.1523-1755.2005.00200.xopenalex-snapshot
IL-10, IL-6, and TNF-α: Central factors in the altered cytokine network of uremia—The good, the bad, and the ugly
inferredcites →https://doi.org/10.1126/science.3283939openalex-snapshot
The Steroid and Thyroid Hormone Receptor Superfamily
inferredcites →https://doi.org/10.1152/ajpheart.2001.280.4.h1537openalex-snapshot
Regulation of B2-kinin receptors by glucose in vascular smooth muscle cells
inferredcites →https://doi.org/10.1152/ajprenal.00165.2004openalex-snapshot
Mechanisms through which bradykinin promotes glomerular injury in diabetes
inferredcites →https://doi.org/10.1152/ajprenal.00203.2007openalex-snapshot
Targeted deletion of B2-kinin receptors protects against the development of diabetic nephropathy
inferredcites →https://doi.org/10.1152/ajprenal.00495.2006openalex-snapshot
Uncoupling of the VEGF-endothelial nitric oxide axis in diabetic nephropathy: an explanation for the paradoxical effects of VEGF in renal disease
inferredcites →https://doi.org/10.1159/000104089openalex-snapshot
Renoprotection Provided by Losartan in Combination with Pioglitazone Is Superior to Renoprotection Provided by Losartan Alone in Patients with Type 2 Diabetic Nephropathy
inferredcites →https://doi.org/10.1172/jci11951openalex-snapshot
Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE)
inferredcites →https://doi.org/10.1172/jci16079openalex-snapshot
Albumin stimulates interleukin-8 expression in proximal tubular epithelial cells in vitro and in vivo
inferredcites →https://doi.org/10.1681/asn.2005080859openalex-snapshot
Leukocyte Recruitment and Vascular Injury in Diabetic Nephropathy
inferredcites →https://doi.org/10.1681/asn.2005101113openalex-snapshot
Activation of Tubular Epithelial Cells in Diabetic Nephropathy and the Role of the Peroxisome Proliferator–Activated Receptor-γ Agonist
inferredcites →https://doi.org/10.1681/asn.2006050525openalex-snapshot
Connective Tissue Growth Factor Plays an Important Role in Advanced Glycation End Product–Induced Tubular Epithelial-to-Mesenchymal Transition
inferredcites →https://doi.org/10.1681/asn.2007050581openalex-snapshot
The Death Ligand TRAIL in Diabetic Nephropathy
inferredcites →https://doi.org/10.1681/asn.v1061204openalex-snapshot
Induction of Monocyte Chemoattractant Protein-1 by Albumin Is Mediated by Nuclear Factor κB in Proximal Tubule Cells
inferredcites →https://doi.org/10.1681/asn.v125993openalex-snapshot
Antibodies against Vascular Endothelial Growth Factor Improve Early Renal Dysfunction in Experimental Diabetes
inferredcites →https://doi.org/10.2337/diab.43.6.836openalex-snapshot
Glycation and Diabetic Complications
inferredcites →https://doi.org/10.2337/diab.43.8.1046openalex-snapshot
Clinical and Histological Correlations of Decline in Renal Function in Diabetic Patients With Proteinuria
inferredcites →https://doi.org/10.2337/diabetes.51.12.3532openalex-snapshot
Activation of Tubular Epithelial Cells in Diabetic Nephropathy
inferredcites →https://doi.org/10.2337/diacare.26.10.2903openalex-snapshot
Signs of Nephropathy May Occur Early in Young Adults With Diabetes Despite Modern Diabetes Management
inferredcites →https://doi.org/10.2337/diacare.27.1.256openalex-snapshot
Thiazolidinedione Use, Fluid Retention, and Congestive Heart Failure
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3965383openalex-extracted
Mannose Binding Lectin Activates the Nuclear Factor-κB and Renal Inflammation in the Progression of Diabetic Nephropathy
inferred← citeshttps://doi.org/10.1002/mco2.70506openalex-snapshot
Tissue Kallikrein as a Mediator in Stroke Outcomes among Patients With Metabolic Syndrome: A Multicenter Study Integrating Big Data and Biomarkers
inferred← citeshttps://doi.org/10.1007/978-3-319-06683-7_3openalex-snapshot
Tissue Kallikrein-Kinin Therapy in Hypertension and Organ Damage
inferred← citeshttps://doi.org/10.1007/s00018-025-05988-zopenalex-snapshot
Inhibition of FXIIa attenuates kidney fibrosis in mice with unilateral ureteral obstruction
inferred← citeshttps://doi.org/10.1007/s10157-010-0296-xopenalex-snapshot
Overexpression of acetyl-coenzyme A carboxylase beta increases proinflammatory cytokines in cultured human renal proximal tubular epithelial cells
inferred← citeshttps://doi.org/10.1016/b978-0-443-28907-1.00015-3openalex-snapshot
Bradykinin in diabetes and obesity
inferred← citeshttps://doi.org/10.1016/j.amjcard.2021.06.014openalex-snapshot
Usefulness of Adding Pre-procedural Glycemia to the Mehran Score to Enhance Its Ability to Predict Contrast-induced Kidney Injury in Patients Undergoing Percutaneous Coronary Intervention Development and Validation of a Predictive Model
inferred← citeshttps://doi.org/10.1016/j.biopha.2023.115660openalex-snapshot
Inhibition of MD2 by natural product-drived JM-9 attenuates renal inflammation and diabetic nephropathy in mice
inferred← citeshttps://doi.org/10.1016/j.carpath.2014.08.006openalex-snapshot
Angiotensin converting enzyme is involved in the cardiac hypertrophy induced by sinoaortic denervation in rats
inferred← citeshttps://doi.org/10.1016/j.ejphar.2020.173503openalex-snapshot
Targeting NLRP3 inflammasome as a promising approach for treatment of diabetic nephropathy: Preclinical evidences with therapeutic approaches
inferred← citeshttps://doi.org/10.1016/j.ejphar.2021.174378openalex-snapshot
Diabetic proximal tubulopathy: Can we mimic the disease for in vitro screening of SGLT inhibitors?
inferred← citeshttps://doi.org/10.1016/j.intimp.2014.06.025openalex-snapshot
Kinin B2 receptor deletion and blockage ameliorates cisplatin-induced acute renal injury
inferred← citeshttps://doi.org/10.1016/j.intimp.2025.114562openalex-snapshot
Targeting inflammation and necroptosis in diabetic kidney disease: A novel approach via PPARα modulation
inferred← citeshttps://doi.org/10.1016/j.isci.2024.110075openalex-snapshot
Bradykinin induces acute kidney injury after hypothermic circulatory arrest through the repression of the Nrf2-xCT pathway
inferred← citeshttps://doi.org/10.1016/j.jdiacomp.2015.04.014openalex-snapshot
Renal tubular damage may contribute more to acute hyperglycemia induced kidney injury in non-diabetic conscious rats
inferred← citeshttps://doi.org/10.1016/j.jpha.2023.09.003openalex-snapshot
The role of innate immunity in diabetic nephropathy and their therapeutic consequences
inferred← citeshttps://doi.org/10.1016/j.lfs.2012.07.020openalex-snapshot
The high glucose-induced stimulation of B1R and B2R expression via CB1R activation is involved in rat podocyte apoptosis
inferred← citeshttps://doi.org/10.1016/j.lfs.2019.116957openalex-snapshot
BMP-7 inhibits renal fibrosis in diabetic nephropathy via miR-21 downregulation
inferred← citeshttps://doi.org/10.1016/j.ymthe.2021.03.012openalex-snapshot
MicroRNA-10 negatively regulates inflammation in diabetic kidney via targeting activation of the NLRP3 inflammasome
inferred← citeshttps://doi.org/10.1021/acs.jproteome.4c00267openalex-snapshot
Application of Proteomics and Machine Learning Methods to Study the Pathogenesis of Diabetic Nephropathy and Screen Urinary Biomarkers
inferred← citeshttps://doi.org/10.1038/ki.2013.11openalex-snapshot
The TLR4 antagonist CRX-526 protects against advanced diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/ki.2013.8openalex-snapshot
Plasma kininogen and kininogen fragments are biomarkers of progressive renal decline in type 1 diabetes
inferred← citeshttps://doi.org/10.1038/ki.2015.331openalex-snapshot
Kallistatin protects against diabetic nephropathy in db/db mice by suppressing AGE-RAGE-induced oxidative stress
inferred← citeshttps://doi.org/10.1038/labinvest.2011.81openalex-snapshot
Additive renoprotective effects of B2-kinin receptor blocker and PPAR-γ agonist in uninephrectomized db/db mice
inferred← citeshttps://doi.org/10.1038/s41467-024-52214-8openalex-snapshot
Factor XII signaling via uPAR-integrin β1 axis promotes tubular senescence in diabetic kidney disease
inferred← citeshttps://doi.org/10.1038/s41581-019-0234-4openalex-snapshot
Innate immunity in diabetic kidney disease
inferred← citeshttps://doi.org/10.1038/s41598-019-56263-8openalex-snapshot
Associations of ACE I/D polymorphism with the levels of ACE, kallikrein, angiotensin II and interleukin-6 in STEMI patients
inferred← citeshttps://doi.org/10.1038/s41598-026-45999-9openalex-snapshot
Non-linear associations between red blood cell distribution width/serum albumin ratio with diabetic kidney disease: results from two nationwide studies in the United States and China
inferred← citeshttps://doi.org/10.1039/c4fo00379aopenalex-snapshot
Soy β-conglycinin retards progression of diabetic nephropathy via modulating the insulin sensitivity and angiotensin-converting enzyme activity in rats fed with high salt diet
inferred← citeshttps://doi.org/10.1042/cs20140401openalex-snapshot
BMP7 reduces inflammation and oxidative stress in diabetic tubulopathy
inferred← citeshttps://doi.org/10.1053/j.jrn.2014.10.020openalex-snapshot
Diabetic Nephropathy and Proximal Tubular Damage
inferred← citeshttps://doi.org/10.1055/a-0630-0173openalex-snapshot
MicroRNA-204-3p Attenuates High Glucose-Induced MPC5 Podocytes Apoptosis by Targeting Braykinin B2 Receptor
inferred← citeshttps://doi.org/10.1080/1547691x.2018.1532974openalex-snapshot
Bradykinin receptor in immune-mediated renal tubular injury in trichloroethylene-sensitized mice: Impact on NF-κB signaling pathway
inferred← citeshttps://doi.org/10.1093/ndt/gfs260openalex-snapshot
The pathogenic role of the renal proximal tubular cell in diabetic nephropathy
inferred← citeshttps://doi.org/10.1096/fj.202100913rrropenalex-snapshot
Nicotinamide N‐methyltransferase ameliorates renal fibrosis by its metabolite 1‐methylnicotinamide inhibiting the TGF‐β1/Smad3 pathway
inferred← citeshttps://doi.org/10.1096/fj.202101852ropenalex-snapshot
Mannose‐binding lectin activates the nuclear factor‐κB and renal inflammation in the progression of diabetic nephropathy
inferred← citeshttps://doi.org/10.1097/fjc.0000000000001329openalex-snapshot
Contrast-induced Acute Kidney Injury in Diabetic Patients and SGLT-2 Inhibitors: A Preventive Opportunity or Promoting Element?
inferred← citeshttps://doi.org/10.1097/md.0000000000038417openalex-snapshot
Efficacy and safety of Huangkui capsule for diabetic nephropathy: A systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1111/j.1440-1797.2010.01437.xopenalex-snapshot
Differential effects of advanced glycation end‐products on renal tubular cell inflammation
inferred← citeshttps://doi.org/10.1111/jcmm.13314openalex-snapshot
The classic signalling and trans‐signalling of interleukin‐6 are both injurious in podocyte under high glucose exposure
inferred← citeshttps://doi.org/10.1152/ajprenal.00437.2011openalex-snapshot
Bradykinin inhibits high glucose- and growth factor-induced collagen synthesis in mesangial cells through the B2-kinin receptor
inferred← citeshttps://doi.org/10.1155/2017/6483572openalex-snapshot
PGC1αActivators Mitigate Diabetic Tubulopathy by Improving Mitochondrial Dynamics and Quality Control
inferred← citeshttps://doi.org/10.1159/000316056openalex-snapshot
Renoprotection by Rosiglitazone in Accelerated Type 2 Diabetic Nephropathy: Role of STAT1 Inhibition and Nephrin Restoration
inferred← citeshttps://doi.org/10.1159/000324513openalex-snapshot
Enalaprilat-Mediated Activation of Kinin B1 Receptors and Vasodilation in the Rat Isolated Perfused Kidney
inferred← citeshttps://doi.org/10.1159/000325647openalex-snapshot
Diabetic Tubulopathy: An Emerging Entity
inferred← citeshttps://doi.org/10.1159/000354473openalex-snapshot
Bone Marrow-Derived Mononuclear Cells Promote Improvement in Glomerular Function in Rats with Early Diabetic Nephropathy
inferred← citeshttps://doi.org/10.11606/t.5.2016.tde-11042016-115124openalex-snapshot
A expressão dos genes codificantes da proteína de interação com tiorredoxina, da beta 2 microglobulina e do transportador de tiamina 1, correlaciona-se com marcadores clínicos da doença renal em pacientes com diabetes tipo 1
inferred← citeshttps://doi.org/10.1186/s12882-017-0620-8openalex-snapshot
Performance of urinary neutrophil gelatinase-associated lipocalin, clusterin, and cystatin C in predicting diabetic kidney disease and diabetic microalbuminuria: a consecutive cohort study
inferred← citeshttps://doi.org/10.1186/s13287-019-1424-5openalex-snapshot
Human mesenchymal stromal cells broadly modulate high glucose-induced inflammatory responses of renal proximal tubular cell monolayers
inferred← citeshttps://doi.org/10.1371/journal.pone.0088894openalex-snapshot
Tissue Kallikrein Mediates Pro-Inflammatory Pathways and Activation of Protease-Activated Receptor-4 in Proximal Tubular Epithelial Cells
inferred← citeshttps://doi.org/10.1371/journal.pone.0090883openalex-snapshot
Mesenchymal Stem Cells Modulate Albumin-Induced Renal Tubular Inflammation and Fibrosis
inferred← citeshttps://doi.org/10.1507/endocrj.ej20-0783openalex-snapshot
Rosiglitazone attenuates high glucose-induced proliferation, inflammation, oxidative stress and extracellular matrix accumulation in mouse mesangial cells through the Gm26917/miR-185-5p pathway
inferred← citeshttps://doi.org/10.1590/2175-8239-jbn-2023-0187enopenalex-snapshot
SGLT2 inhibitors and NLRP3 inflammasome: potential target in diabetic kidney disease
inferred← citeshttps://doi.org/10.1590/2175-8239-jbn-2023-0187ptopenalex-snapshot
Inibidores de SGLT2 e inflamassoma NLRP3: alvo potencial na doença renal do diabetes
inferred← citeshttps://doi.org/10.1681/asn.2010111210openalex-snapshot
Toll-Like Receptor 4 Promotes Tubular Inflammation in Diabetic Nephropathy
inferred← citeshttps://doi.org/10.1681/asn.2013070811openalex-snapshot
Tribbles Homolog 3 Attenuates Mammalian Target of Rapamycin Complex-2 Signaling and Inflammation in the Diabetic Kidney
inferred← citeshttps://doi.org/10.1681/asn.2021101286openalex-snapshot
Activation of Stimulator of IFN Genes (STING) Causes Proteinuria and Contributes to Glomerular Diseases
inferred← citeshttps://doi.org/10.20517/2574-1209.2024.21openalex-snapshot
Role of inflammation in the progression of diabetic kidney disease
inferred← citeshttps://doi.org/10.2147/dmso.s353717openalex-snapshot
Glucagon-Like Peptide 1 Receptor Agonist Improves Renal Tubular Damage in Mice with Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.2147/ijnrd.s436791openalex-snapshot
The Molecular Mechanism of Renal Tubulointerstitial Inflammation Promoting Diabetic Nephropathy
inferred← citeshttps://doi.org/10.2174/0929867329666220922104654openalex-snapshot
The Selective NLRP3-Inflammasome Inhibitor CY-09 Ameliorates Kidney Injury in Diabetic Nephropathy by Inhibiting NLRP3- inflammasome Activation
inferred← citeshttps://doi.org/10.2478/dine-2021-0004openalex-snapshot
Pathogenesis of diabetic kidney disease
inferred← citeshttps://doi.org/10.3389/fendo.2023.1232790openalex-snapshot
Advances in understanding and treating diabetic kidney disease: focus on tubulointerstitial inflammation mechanisms
inferred← citeshttps://doi.org/10.3389/fendo.2025.1564170openalex-snapshot
Diagnostic accuracy of neutrophil-to-lymphocyte ratio in type 2 diabetic nephropathy: a meta-analysis
inferred← citeshttps://doi.org/10.3389/fimmu.2020.578952openalex-snapshot
The Emerging Role of Renal Tubular Epithelial Cells in the Immunological Pathophysiology of Lupus Nephritis
inferred← citeshttps://doi.org/10.3389/fphar.2021.662706openalex-snapshot
Inhibition of Renal Tubular Epithelial Mesenchymal Transition and Endoplasmic Reticulum Stress-Induced Apoptosis with Shenkang Injection Attenuates Diabetic Tubulopathy
inferred← citeshttps://doi.org/10.3390/ijms24010570openalex-snapshot
What’s New in the Molecular Mechanisms of Diabetic Kidney Disease: Recent Advances
inferred← citeshttps://doi.org/10.3390/ijms25063086openalex-snapshot
Recent Advances in the Management of Diabetic Kidney Disease: Slowing Progression
inferred← citeshttps://doi.org/10.3892/mmr.2015.4145openalex-snapshot
Peroxisome proliferator-activated receptor γ prevents the production of NOD-like receptor family, pyrin domain containing 3 inflammasome and interleukin 1β in HK-2 renal tubular epithelial cells stimulated by monosodium urate crystals
inferred← citeshttps://doi.org/10.4239/wjd.v14.i10.1450openalex-snapshot
Multifaceted relationship between diabetes and kidney diseases: Beyond diabetes
inferred← citeshttps://doi.org/10.5772/34541openalex-snapshot
Immunoinflammation in Diabetic Nephropathy: Molecular Mechanisms and Therapeutic Options
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