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Glucocorticoids employ the monomeric glucocorticoid receptor to potentiate vitamin D 3 and parathyroid hormone‐induced osteoclastogenesis

https://doi.org/10.1096/fj.201802729rrr
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/1529-0131(199911)42:11<2309::aid-anr8>3.0.co;2-kopenalex-snapshot
Risedronate therapy prevents corticosteroid-induced bone loss : A twelve-month, multicenter, randomized, double-blind, placebo-controlled, parallel-group study
inferredcites →https://doi.org/10.1002/art.11283openalex-snapshot
Bone density threshold and other predictors of vertebral fracture in patients receiving oral glucocorticoid therapy
inferredcites →https://doi.org/10.1002/art.21896openalex-snapshot
The ratio of circulating osteoprotegerin to RANKL in early rheumatoid arthritis predicts later joint destruction
inferredcites →https://doi.org/10.1002/art.23454openalex-snapshot
Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis: A longitudinal study of gene expression in bone tissue from glucocorticoid‐treated mice
inferredcites →https://doi.org/10.1002/art.24445openalex-snapshot
Prevention of glucocorticoid‐induced bone loss in mice by inhibition of RANKL
inferredcites →https://doi.org/10.1002/jbmr.113openalex-snapshot
Glucocorticoids maintain human osteoclasts in the active mode of their resorption cycle
inferredcites →https://doi.org/10.1006/bbrc.2000.4223openalex-snapshot
Regulation of Osteoprotegerin Secretion from Primary Cultures of Human Bone Marrow Stromal Cells
inferredcites →https://doi.org/10.1006/bbrc.2001.6251openalex-snapshot
Vitamin D3 Augments Osteoclastogenesis via Vitamin D-Responsive Element of Mouse RANKL Gene Promoter
inferredcites →https://doi.org/10.1007/978-1-4939-2895-8_8openalex-snapshot
Glucocorticoid-Induced Osteoporosis
inferredcites →https://doi.org/10.1007/bf00675619openalex-snapshot
Differential effects of glucocorticoids on cortical appendicular and cortical vertebral bone mineral content
inferredcites →https://doi.org/10.1007/s00198-007-0394-0openalex-snapshot
Glucocorticoid-induced osteoporosis: pathophysiology and therapy
inferredcites →https://doi.org/10.1007/s001980200108openalex-snapshot
The Epidemiology of Corticosteroid-Induced Osteoporosis: a Meta-analysis
inferredcites →https://doi.org/10.1007/s00223-012-9684-4openalex-snapshot
Acute Effects of Glucocorticoids on Serum Markers of Osteoclasts, Osteoblasts, and Osteocytes
inferredcites →https://doi.org/10.1007/s002230001146openalex-snapshot
Effects of Risedronate Treatment on Bone Density and Vertebral Fracture in Patients on Corticosteroid Therapy
inferredcites →https://doi.org/10.1007/s12018-018-9242-3openalex-snapshot
Glucocorticoid Excess in Bone and Muscle
inferredcites →https://doi.org/10.1016/0014-5793(91)81129-vopenalex-snapshot
Antihormones in health and disease
inferredcites →https://doi.org/10.1016/j.bone.2005.10.004openalex-snapshot
Effects of dissociated glucocorticoids on OPG and RANKL in osteoblastic cells
inferredcites →https://doi.org/10.1016/j.bone.2006.02.005openalex-snapshot
High prevalence of asymptomatic vertebral fractures in post-menopausal women receiving chronic glucocorticoid therapy: A cross-sectional outpatient study
inferredcites →https://doi.org/10.1016/j.bone.2010.08.011openalex-snapshot
A bisphosphonate that does not affect osteoclasts prevents osteoblast and osteocyte apoptosis and the loss of bone strength induced by glucocorticoids in mice
inferredcites →https://doi.org/10.1016/j.bone.2014.12.004openalex-snapshot
A joined role of canopy and reversal cells in bone remodeling — Lessons from glucocorticoid-induced osteoporosis
inferredcites →https://doi.org/10.1016/j.bone.2016.08.024openalex-snapshot
Activation of dimeric glucocorticoid receptors in osteoclast progenitors potentiates RANKL induced mature osteoclast bone resorbing activity
inferredcites →https://doi.org/10.1016/j.cmet.2010.05.005openalex-snapshot
Glucocorticoids Suppress Bone Formation by Attenuating Osteoblast Differentiation via the Monomeric Glucocorticoid Receptor
inferredcites →https://doi.org/10.1016/j.jaci.2013.09.007openalex-snapshot
The biology of the glucocorticoid receptor: New signaling mechanisms in health and disease
inferredcites →https://doi.org/10.1016/j.mce.2007.07.003openalex-snapshot
Crosstalk between the glucocorticoid receptor and other transcription factors: Molecular aspects
inferredcites →https://doi.org/10.1016/j.molmed.2012.04.005openalex-snapshot
Glucocorticoid receptor signaling in bone cells
inferredcites →https://doi.org/10.1016/j.tem.2006.03.009openalex-snapshot
Glucocorticoid-induced osteoporosis: an update
inferredcites →https://doi.org/10.1016/j.tem.2012.05.005openalex-snapshot
New insights into osteoclastogenic signaling mechanisms
inferredcites →https://doi.org/10.1016/s0092-8674(00)81183-6openalex-snapshot
DNA Binding of the Glucocorticoid Receptor Is Not Essential for Survival
inferredcites →https://doi.org/10.1016/s0092-8674(00)81569-xopenalex-snapshot
Osteoprotegerin Ligand Is a Cytokine that Regulates Osteoclast Differentiation and Activation
inferredcites →https://doi.org/10.1016/s0167-4781(99)00032-9openalex-snapshot
Promoter structure of mouse RANKL/TRANCE/OPGL/ODF gene
inferredcites →https://doi.org/10.1038/s41467-018-05244-yopenalex-snapshot
Soluble RANKL contributes to osteoclast formation in adult mice but not ovariectomy-induced bone loss
inferredcites →https://doi.org/10.1073/pnas.95.7.3597openalex-snapshot
Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL
inferredcites →https://doi.org/10.1074/jbc.274.19.13613openalex-snapshot
Evidence for a Role of a Tumor Necrosis Factor-α (TNF-α)-converting Enzyme-like Protease in Shedding of TRANCE, a TNF Family Member Involved in Osteoclastogenesis and Dendritic Cell Survival
inferredcites →https://doi.org/10.1074/jbc.m111.247734openalex-snapshot
Retinoids Stimulate Periosteal Bone Resorption by Enhancing the Protein RANKL, a Response Inhibited by Monomeric Glucocorticoid Receptor
inferredcites →https://doi.org/10.1074/jbc.m510160200openalex-snapshot
Inhibition of Hormone and Cytokine-stimulated Osteoclastogenesis and Bone Resorption by Interleukin-4 and Interleukin-13 Is Associated with Increased Osteoprotegerin and Decreased RANKL and RANK in a STAT6-dependent Pathway
inferredcites →https://doi.org/10.1074/jbc.m606656200openalex-snapshot
Negative Regulation of Osteoclastogenesis by Ectodomain Shedding of Receptor Activator of NF-κB Ligand
inferredcites →https://doi.org/10.1084/jem.20150088openalex-snapshot
Oxysterols and EBI2 promote osteoclast precursor migration to bone surfaces and regulate bone mass homeostasis
inferredcites →https://doi.org/10.1101/gr.188581.114openalex-snapshot
Genomic redistribution of GR monomers and dimers mediates transcriptional response to exogenous glucocorticoid in vivo
inferredcites →https://doi.org/10.1111/jcpe.12134openalex-snapshot
Comparison of circulating tumour necrosis factor superfamily cytokines in periodontitis patients undergoing supportive therapy: a case‐controlled cross‐sectional study comparing smokers and non‐smokers in health and disease
inferredcites →https://doi.org/10.1152/physrev.00036.2016openalex-snapshot
Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal Systems
inferredcites →https://doi.org/10.1172/jci0214538openalex-snapshot
Promotion of osteoclast survival and antagonism of bisphosphonate-induced osteoclast apoptosis by glucocorticoids
inferredcites →https://doi.org/10.1172/jci14538openalex-snapshot
Promotion of osteoclast survival and antagonism of bisphosphonate-induced osteoclast apoptosis by glucocorticoids
inferredcites →https://doi.org/10.1172/jci2799openalex-snapshot
Inhibition of osteoblastogenesis and promotion of apoptosis of osteoblasts and osteocytes by glucocorticoids. Potential mechanisms of their deleterious effects on bone.
inferredcites →https://doi.org/10.1186/1471-2164-11-359openalex-snapshot
Prednisolone-induced differential gene expression in mouse liver carrying wild type or a dimerization-defective glucocorticoid receptor
inferredcites →https://doi.org/10.1210/en.2003-0990openalex-snapshot
Glucocorticoids Act Directly on Osteoblasts and Osteocytes to Induce Their Apoptosis and Reduce Bone Formation and Strength
inferredcites →https://doi.org/10.1210/en.2005-0717openalex-snapshot
Glucocorticoid Regulation of Osteoclast Differentiation and Expression of Receptor Activator of Nuclear Factor-κB (NF-κB) Ligand, Osteoprotegerin, and Receptor Activator of NF-κB in Mouse Calvarial Bones
inferredcites →https://doi.org/10.1210/en.2006-0459openalex-snapshot
Glucocorticoids Act Directly on Osteoclasts to Increase Their Life Span and Reduce Bone Density
inferredcites →https://doi.org/10.1210/endo-123-5-2600openalex-snapshot
OSTEOBLASTIC CELLS ARE INVOLVED IN OSTEOCLAST FORMATION
inferredcites →https://doi.org/10.1210/endo.140.10.7034openalex-snapshot
Stimulation of Osteoprotegerin Ligand and Inhibition of Osteoprotegerin Production by Glucocorticoids in Human Osteoblastic Lineage Cells: Potential Paracrine Mechanisms of Glucocorticoid-Induced Osteoporosis1
inferredcites →https://doi.org/10.1359/jbmr.041233openalex-snapshot
Influence of Glucocorticoids on Human Osteoclast Generation and Activity
inferredcites →https://doi.org/10.1359/jbmr.081244openalex-snapshot
A Simple Risk Score for the Assessment of Absolute Fracture Risk in General Practice Based on Two Longitudinal Studies
inferredcites →https://doi.org/10.1359/jbmr.2001.16.1.97openalex-snapshot
Comparison of Trabecular Bone Microarchitecture and Remodeling in Glucocorticoid-Induced and Postmenopausal Osteoporosis
inferredcites →https://doi.org/10.1677/joe.0.1550513openalex-snapshot
Differential effects of glucocorticoids on bone resorption in neonatal mouse calvariae stimulated by peptide and steroid-like hormones
inferredcites →https://doi.org/10.1677/joe.0.1590191openalex-snapshot
Osteoprotegerin mRNA is expressed in primary human osteoblast-like cells: down-regulation by glucocorticoids
inferredcites →https://doi.org/10.3109/08820139.2013.822766openalex-snapshot
The role of cytokines in inflammatory bone loss
inferredcites →https://doi.org/10.3171/jns.1964.21.4.0318openalex-snapshot
The Basophil Adenomas of the Pituitary Body and Their Clinical Manifestations (Pituitary Basophilism)

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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inferred← citeshttps://doi.org/10.12659/msm.925474openalex-snapshot
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inferred← citeshttps://doi.org/10.21037/atm-20-7607openalex-snapshot
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inferred← citeshttps://doi.org/10.3390/ijms22031054openalex-snapshot
Glucocorticoid Receptor Regulates TNFSF11 Transcription by Binding to Glucocorticoid Responsive Element in TNFSF11 Proximal Promoter Region
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