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Upregulating CD4+CD25+FOXP3+ Regulatory T Cells in Pancreatic Lymph Nodes in Diabetic NOD Mice by Adjuvant Immunotherapy

https://doi.org/10.1097/tp.0b013e3181933261

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Naturally Arising Foxp3-Expressing CD25+CD4+ Regulatory T Cells in Self-Tolerance and Autoimmune Disease

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Adult pancreatic β-cells are formed by self-duplication rather than stem-cell differentiation

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Visualizing regulatory T cell control of autoimmune responses in nonobese diabetic mice

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TGF-β-dependent mechanisms mediate restoration of self-tolerance induced by antibodies to CD3 in overt autoimmune diabetes

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TGF-β regulates in vivo expansion of Foxp3-expressing CD4 + CD25 + regulatory T cells responsible for protection against diabetes

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CD4 + CD25 + T regulatory cells control anti-islet CD8 + T cells through TGF-β–TGF-β receptor interactions in type 1 diabetes

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Regulatory T Cells in Immunologic Self-Tolerance and Autoimmune Disease

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In Vitro–expanded Antigen-specific Regulatory T Cells Suppress Autoimmune Diabetes

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Where CD4 + CD25 + T reg cells impinge on autoimmune diabetes

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In vivo apoptosis of diabetogenic T cells in NOD mice by IFN- /TNF-

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Systemic Transforming Growth Factor-??1 Gene Therapy Induces Foxp3+ Regulatory Cells, Restores Self-Tolerance, and Facilitates Regeneration Of Beta Cell Function in Overtly Diabetic Nonobese Diabetic Mice

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Immunological Reversal of Autoimmune Diabetes Without Hematopoietic Replacement of ß Cells

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Reversal of Diabetes in Non-Obese Diabetic Mice Without Spleen Cell-Derived ß Cell Regeneration

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Islet Recovery and Reversal of Murine Type 1 Diabetes in the Absence of Any Infused Spleen Cell Contribution

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Regulatory T Cells in Autoimmmunity

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TRANSFORMING GROWTH FACTOR-β REGULATION OF IMMUNE RESPONSES

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TGF- signaling is required for the function of insulin-reactive T regulatory cells

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Glucagon-Like Peptide-1 Induces Cell Proliferation and Pancreatic-Duodenum Homeobox-1 Expression and Increases Endocrine Cell Mass in the Pancreas of Old, Glucose-Intolerant Rats

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Minireview: Glucagon-Like Peptides Regulate Cell Proliferation and Apoptosis in the Pancreas, Gut, and Central Nervous System

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Exendin-4 Modulates Diabetes Onset in Nonobese Diabetic Mice

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Islet growth and development in the adult

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Harnessing the Therapeutic Potential of Glucagon-Like Peptide-1

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Effects of Autoimmunity and Immune Therapy on β-Cell Turnover in Type 1 Diabetes

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Patients With Chronic Pancreatitis Have Islet Progenitor Cells in Their Ducts, but Reversal of Overt Diabetes in NOD Mice by Anti-CD3 Shows No Evidence for Islet Regeneration

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No Alterations in the Frequency of FOXP3+ Regulatory T-Cells in Type 1 Diabetes

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Prevention of Type I Diabetes in NOD Mice by Adjuvant Immunotherapy

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Prevention of Insulitis and Diabetes Onset by Treatment With Complete Freund's Adjuvant in NOD Mice

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Prevention of Recurrence of IDDM in Islet-Transplanted Diabetic NOD Mice by Adjuvant Immunotherapy

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Exendin-4 stimulates both beta-cell replication and neogenesis, resulting in increased beta-cell mass and improved glucose tolerance in diabetic rats.

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beta-cell turnover: its assessment and implications.

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Glucagon-Like Peptide 1 Induces Differentiation of Islet Duodenal Homeobox-1–Positive Pancreatic Ductal Cells Into Insulin-Secreting Cells

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Glucagon-Like Peptide-1 and Exendin-4 Stimulate β-Cell Neogenesis in Streptozotocin-Treated Newborn Rats Resulting in Persistently Improved Glucose Homeostasis at Adult Age

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Role of Endogenous Glucagon-Like Peptide-1 in Islet Regeneration After Partial Pancreatectomy

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Cure of Overt Diabetes in NOD Mice by Transient Treatment With Anti-Lymphocyte Serum and Exendin-4

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Regulation of Diabetes Development by Regulatory T Cells in Pancreatic Islet Antigen-Specific TCR Transgenic Nonobese Diabetic Mice

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Regulation of Autoimmune Diabetes by Complete Freund’s Adjuvant Is Mediated by NK Cells

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Cyclophosphamide-Induced Type-1 Diabetes in the NOD Mouse Is Associated with a Reduction of CD4+CD25+Foxp3+ Regulatory T Cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Glucagon Like Peptide-1: More than Glucose Control and Weight Reduction

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Protective effects of carbonyl iron against multiple low‐dose streptozotocin‐induced diabetes in rodents

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The Antidiabetic Effect of Mesenchymal Stem Cells Is Unrelated to Their Transdifferentiation Potential But to Their Capability to Restore Th1/Th2 Balance and to Modify the Pancreatic Microenvironment

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Targeting of IL-2 receptor with a caspase fusion protein disrupts autoimmunity in prediabetic and diabetic NOD mice

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Glucagon-like peptide-1 receptor signalling selectively regulates murine lymphocyte proliferation and maintenance of peripheral regulatory T cells

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Use of Glucagon-Like Peptide-1 Agonists to Improve Islet Graft Performance

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Prevention of Type 1 Diabetes and Its Recurrence by Immunotherapy with Mycobacterial Adjuvants

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Reg2 treatment is protective but the induced Reg2 autoantibody is destructive to the islets in NOD mice

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Endogenous Expansion of Regulatory T Cells Leads to Long-Term Islet Graft Survival in Diabetic NOD Mice

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