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Genetic risk for autism spectrum disorders and neuropsychiatric variation in the general population
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archgenpsychiatry.2011.119openalex-snapshot
Evidence That Autistic Traits Show the Same Etiology in the General Population and at the Quantitative Extremes (5%, 2.5%, and 1%)
inferredcites →https://doi.org/10.1001/archgenpsychiatry.2011.144openalex-snapshot
Autism Spectrum Disorders and Autisticlike Traits
inferredcites →https://doi.org/10.1001/jama.2015.4845openalex-snapshot
Copy Number Variations and Cognitive Phenotypes in Unselected Populations
inferredcites →https://doi.org/10.1001/jamapsychiatry.2014.2147openalex-snapshot
The Role of Parental Cognitive, Behavioral, and Motor Profiles in Clinical Variability in Individuals With Chromosome 16p11.2 Deletions
inferredcites →https://doi.org/10.1016/j.ajhg.2010.11.011openalex-snapshot
GCTA: A Tool for Genome-wide Complex Trait Analysis
inferredcites →https://doi.org/10.1016/j.biopsych.2014.04.021openalex-snapshot
The Cognitive and Behavioral Phenotype of the 16p11.2 Deletion in a Clinically Ascertained Population
inferredcites →https://doi.org/10.1016/j.jaac.2011.01.005openalex-snapshot
Stability of Autistic Traits in the General Population: Further Evidence for a Continuum of Impairment
inferredcites →https://doi.org/10.1016/j.neuron.2010.10.006openalex-snapshot
The Simons Simplex Collection: A Resource for Identification of Autism Genetic Risk Factors
inferredcites →https://doi.org/10.1037/t15164-000openalex-snapshot
Vineland Adaptive Behavior Scales, Second Edition
inferredcites →https://doi.org/10.1038/mp.2013.2openalex-snapshot
Genome-wide study of association and interaction with maternal cytomegalovirus infection suggests new schizophrenia loci
inferredcites →https://doi.org/10.1038/mp.2015.12openalex-snapshot
Common polygenic risk for autism spectrum disorder (ASD) is associated with cognitive ability in the general population
inferredcites →https://doi.org/10.1038/mp.2015.126openalex-snapshot
Phenome-wide analysis of genome-wide polygenic scores
inferredcites →https://doi.org/10.1038/nature12818openalex-snapshot
CNVs conferring risk of autism or schizophrenia affect cognition in controls
inferredcites →https://doi.org/10.1038/nature13595openalex-snapshot
Biological insights from 108 schizophrenia-associated genetic loci
inferredcites →https://doi.org/10.1038/nature13772openalex-snapshot
Synaptic, transcriptional and chromatin genes disrupted in autism
inferredcites →https://doi.org/10.1038/nature13908openalex-snapshot
The contribution of de novo coding mutations to autism spectrum disorder
inferredcites →https://doi.org/10.1038/ng.2711openalex-snapshot
Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs
inferredcites →https://doi.org/10.1038/ng.3039openalex-snapshot
Most genetic risk for autism resides with common variation
inferredcites →https://doi.org/10.1038/ng.3050openalex-snapshot
A framework for the interpretation of de novo mutation in human disease
inferredcites →https://doi.org/10.1038/ng.3211openalex-snapshot
LD Score regression distinguishes confounding from polygenicity in genome-wide association studies
inferredcites →https://doi.org/10.1038/ng.3303openalex-snapshot
Excess of rare, inherited truncating mutations in autism
inferredcites →https://doi.org/10.1038/ng.3406openalex-snapshot
An atlas of genetic correlations across human diseases and traits
inferredcites →https://doi.org/10.1038/ni.1747openalex-snapshot
Mina, an Il4 repressor, controls T helper type 2 bias
inferredcites →https://doi.org/10.1038/nmeth0410-248openalex-snapshot
A method and server for predicting damaging missense mutations
inferredcites →https://doi.org/10.1038/nrg3585openalex-snapshot
The role of de novo mutations in the genetics of autism spectrum disorders
inferredcites →https://doi.org/10.1073/pnas.1409204111openalex-snapshot
Autism spectrum disorder severity reflects the average contribution of de novo and familial influences
inferredcites →https://doi.org/10.1093/ije/dys064openalex-snapshot
Cohort Profile: The ‘Children of the 90s’—the index offspring of the Avon Longitudinal Study of Parents and Children
inferredcites →https://doi.org/10.1093/ije/dys066openalex-snapshot
Cohort Profile: The Avon Longitudinal Study of Parents and Children: ALSPAC mothers cohort
inferredcites →https://doi.org/10.1097/01.chi.0000215325.13058.9dopenalex-snapshot
Genetic Heterogeneity Between the Three Components of the Autism Spectrum: A Twin Study
inferredcites →https://doi.org/10.1101/016527openalex-snapshot
Contrasting regional architectures of schizophrenia and other complex diseases using fast variance components analysis
inferredcites →https://doi.org/10.1176/appi.ajp.2010.09101470openalex-snapshot
Sibling Recurrence and the Genetic Epidemiology of Autism
inferredcites →https://doi.org/10.1186/1471-2156-12-58openalex-snapshot
Robustness of genome-wide scanning using archived dried blood spot samples as a DNA source
inferredcites →https://doi.org/10.1186/2040-2392-5-18openalex-snapshot
Variability in the common genetic architecture of social-communication spectrum phenotypes during childhood and adolescence
inferredcites →https://doi.org/10.1192/bjp.187.6.568openalex-snapshot
Measuring autistic traits: heritability, reliability and validity of the Social and Communication Disorders Checklist
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1002/aur.2393openalex-snapshot
Reduced Language Lateralization in Autism and the Broader Autism Phenotype as Assessed with Robust Individual‐Subjects Analyses
inferred← citeshttps://doi.org/10.1007/s10803-019-04001-4openalex-snapshot
Social Cognition in Autism and Other Neurodevelopmental Disorders: A Co-twin Control Study
inferred← citeshttps://doi.org/10.1007/s10803-020-04667-1openalex-snapshot
Distributional Properties and Criterion Validity of a Shortened Version of the Social Responsiveness Scale: Results from the ECHO Program and Implications for Social Communication Research
inferred← citeshttps://doi.org/10.1007/s10803-020-04710-1openalex-snapshot
Peripheral Blood S100B Levels in Autism Spectrum Disorder: A Systematic Review and Meta-Analysis
inferred← citeshttps://doi.org/10.1016/j.biopsych.2016.08.035openalex-snapshot
The Pleiotropic MET Receptor Network: Circuit Development and the Neural-Medical Interface of Autism
inferred← citeshttps://doi.org/10.1016/j.biopsych.2023.11.017openalex-snapshot
Phenotypic and Genetic Associations Between Preschool Fine Motor Skills and Later Neurodevelopment, Psychopathology, and Educational Achievement
inferred← citeshttps://doi.org/10.1038/s41380-020-00876-3openalex-snapshot
Exons as units of phenotypic impact for truncating mutations in autism
inferred← citeshttps://doi.org/10.1038/s41380-021-01026-zopenalex-snapshot
Polygenic burden has broader impact on health, cognition, and socioeconomic outcomes than most rare and high-risk copy number variants
inferred← citeshttps://doi.org/10.1038/s41380-021-01204-zopenalex-snapshot
Patterns of brain asymmetry associated with polygenic risks for autism and schizophrenia implicate language and executive functions but not brain masculinization
inferred← citeshttps://doi.org/10.1038/s41386-020-0768-yopenalex-snapshot
Leveraging large genomic datasets to illuminate the pathobiology of autism spectrum disorders
inferred← citeshttps://doi.org/10.1038/s41398-020-00906-2openalex-snapshot
Quantitative genome-wide association study of six phenotypic subdomains identifies novel genome-wide significant variants in autism spectrum disorder
inferred← citeshttps://doi.org/10.1038/s41398-022-01833-0openalex-snapshot
Association of cumulative early medical factors with autism and autistic symptoms in a population-based twin sample
inferred← citeshttps://doi.org/10.1038/s41398-023-02407-4openalex-snapshot
Candidate biomarkers from the integration of methylation and gene expression in discordant autistic sibling pairs
inferred← citeshttps://doi.org/10.1038/s41467-020-20443-2openalex-snapshot
Risk variants and polygenic architecture of disruptive behavior disorders in the context of attention-deficit/hyperactivity disorder
inferred← citeshttps://doi.org/10.1038/s41588-022-01072-5openalex-snapshot
Genetic correlates of phenotypic heterogeneity in autism
inferred← citeshttps://doi.org/10.1038/s41593-019-0489-xopenalex-snapshot
A perturbed gene network containing PI3K–AKT, RAS–ERK and WNT–β-catenin pathways in leukocytes is linked to ASD genetics and symptom severity
inferred← citeshttps://doi.org/10.1038/s41598-021-95447-zopenalex-snapshot
Understanding the impact of SNPs associated with autism spectrum disorder on biological pathways in the human fetal and adult cortex
inferred← citeshttps://doi.org/10.1093/med/9780197640654.003.0060openalex-snapshot
Diagnosis and Epidemiology of Pediatric Psychiatric Disorders
inferred← citeshttps://doi.org/10.1093/med/9780197640654.003.0069openalex-snapshot
The Present and Future of Psychiatric Diagnosis
inferred← citeshttps://doi.org/10.1093/oso/9780190645472.003.0014openalex-snapshot
The Spectrum of Twice Exceptional and Autistic Learners and Suggestions for Their Learning Styles
inferred← citeshttps://doi.org/10.1101/2020.06.01.127688openalex-snapshot
Reproducible functional connectivity endophenotype confers high risk of ASD diagnosis in a subset of individuals
inferred← citeshttps://doi.org/10.1111/jcpp.13473openalex-snapshot
Etiological links between autism and difficulties in initiating and maintaining sleep: a familial co‐aggregation and twin study
inferred← citeshttps://doi.org/10.1111/jcpp.13724openalex-snapshot
Preferential looking to eyes versus mouth in early infancy: heritability and link to concurrent and later development
inferred← citeshttps://doi.org/10.1111/nyas.14880openalex-snapshot
Autistic traits heighten sensitivity to rejection‐induced social pain
inferred← citeshttps://doi.org/10.20944/preprints201805.0359.v1openalex-snapshot
Pathogenicity of Endocrine Dysregulation in Autism
inferred← citeshttps://doi.org/10.21203/rs.3.rs-325654/v1openalex-snapshot
The Contribution of Autism-Related Common Variants to the Familial Aggregation of Quantitative Autistic Traits
inferred← citeshttps://doi.org/10.21203/rs.3.rs-3346245/v1openalex-snapshot
SOX7: Novel Autistic Gene Identified by Analysis of Multi-Omics Data
inferred← citeshttps://doi.org/10.2139/ssrn.4028542openalex-snapshot
Neuropsychiatric Risk in Children With Intellectual Disability of Genetic Origin: IMAGINE - The UK National Cohort Study
inferred← citeshttps://doi.org/10.3410/f.732397732.793566104openalex-snapshot
Faculty Opinions recommendation of The Dynamic Landscape of Open Chromatin during Human Cortical Neurogenesis.
inferred← citeshttps://doi.org/10.3410/f.733081240.793545433openalex-snapshot
Faculty Opinions recommendation of Paternally inherited cis-regulatory structural variants are associated with autism.
inferred← citeshttps://doi.org/10.3410/f.733320749.793549010openalex-snapshot
Faculty Opinions recommendation of Quantifying the Impact of Rare and Ultra-rare Coding Variation across the Phenotypic Spectrum.
inferred← citeshttps://doi.org/10.52852/tcncyh.v163i2.1372openalex-snapshot
Tỷ lệ rối loạn phổ tự kỷ ở trẻ 24 - 72 tháng tuổi bằng tiêu chuẩn DSMM-5
inferred← citeshttps://doi.org/10.58837/chula.the.2020.407openalex-snapshot
Association between pm2.5 concentrations and hospitalization cases for schizophrenia and mood disorders in Ho Chi Minh city, Vietnam from 2019 to 2020
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