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Cell-nonautonomously tunable actomyosin flows orient distinct cell division axes
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Outgoing claims: the papers this work cites, builds on, or argues with.
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Selective inhibition of catalytic activity of smooth muscle myosin light chain kinase.
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Chapter 13 Blastomere Culture and Analysis
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Signaling Networks that Regulate Cell Migration
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A complex of LIN-5 and GPR proteins regulates G protein signaling and spindle function in C. elegans
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Wnt pathway components orient a mitotic spindle in the early Caenorhabditis elegans embryo without requiring gene transcription in the responding cell
inferredcites →https://doi.org/10.1111/j.1600-0854.2008.00837.xopenalex-snapshot
Multiple Mechanisms for Accumulation of Myosin II Filaments at the Equator During Cytokinesis
inferredcites →https://doi.org/10.1126/science.1067765openalex-snapshot
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Triggering a Cell Shape Change by Exploiting Preexisting Actomyosin Contractions
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Cortical Flow in Animal Cells
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Regulation of spindle orientation and neural stem cell fate in the Drosophila optic lobe
inferredcites →https://doi.org/10.1242/dev.00563openalex-snapshot
Establishment of POP-1 asymmetry in earlyC. elegansembryos
inferredcites →https://doi.org/10.1242/dev.045823openalex-snapshot
Symmetry breaking and polarization of the C. elegans zygote by the polarity protein PAR-2
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Time-dependent responses to glp-1-mediated inductions in early C. elegans embryos
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An asymmetric attraction model for the diversity and robustness of cell arrangement in nematodes
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Features of cell lineage in preimplantation mouse development
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Polarity establishment, asymmetric division and segregation of fate determinants in early C. elegans embryos
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Notch signaling in the C. elegans embryo
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Active torque generation by the actomyosin cell cortex drives left–right symmetry breaking
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Cortical flow aligns actin filaments to form a furrow
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