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p53 Controls Murine Gammaherpesvirus Latency and Prevents Infection-Associated IgH/c-Myc Translocations

https://doi.org/10.1101/2020.08.02.233148

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Outgoing claims: the papers this work cites, builds on, or argues with.

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Deregulation of c-Myc in primary effusion lymphoma by Kaposi's sarcoma herpesvirus latency-associated nuclear antigen

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Infectious Mononucleosis

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Epstein–Barr virus‐mediated B‐cell proliferation is dependent upon latent membrane protein 1, which simulates an activated CD40 receptor

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Vaccine potential of a murine gammaherpesvirus-68 mutant deficient for ORF73

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E1A signaling to p53 involves the p19ARFtumor suppressor

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Stabilization of the p53 tumor suppressor is induced by adenovirus 5 E1A and accompanies apoptosis.

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Isolation of High-Molecular-Weight DNA from Mammalian Blood Using Proteinase K and Phenol

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Isolation of High-molecular-weight DNA from Mammalian Cells Using Proteinase K and Phenol

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Hematoxylin and Eosin Staining of Tissue and Cell Sections

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Mediation of c-Myc-Induced Apoptosis by p53

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ORF73-Null Murine Gammaherpesvirus 68 Reveals Roles for mLANA and p53 in Virus Replication

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Myc Is Required for the Maintenance of Kaposi's Sarcoma-Associated Herpesvirus Latency

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Endosomal Toll-Like Receptors 7 and 9 Cooperate in Detection of Murine Gammaherpesvirus 68 Infection

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Functional p53 Signaling in Kaposi's Sarcoma-Associated Herpesvirus Lymphomas: Implications for Therapy

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Epstein-Barr Virus Nuclear Antigen 3C Augments Mdm2-Mediated p53 Ubiquitination and Degradation by Deubiquitinating Mdm2

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The Intersection of Epstein-Barr Virus with the Germinal Center

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Latent Gammaherpesvirus 68 Infection Induces Distinct Transcriptional Changes in Different Organs

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Murine Gammaherpesvirus 68 LANA and SOX Homologs Counteract ATM-Driven p53 Activity during Lytic Viral Replication

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Mature B cells are required for acute splenic infection, but not for establishment of latency, by murine gammaherpesvirus 68

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B Cells Regulate Murine Gammaherpesvirus 68 Latency

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The Murine Gammaherpesvirus 68 v-Cyclin Gene Is an Oncogene That Promotes Cell Cycle Progression in Primary Lymphocytes

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Cloning and Mutagenesis of the Murine Gammaherpesvirus 68 Genome as an Infectious Bacterial Artificial Chromosome

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Establishment and Maintenance of Gammaherpesvirus Latency Are Independent of Infective Dose and Route of Infection

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NF-κB Inhibits Gammaherpesvirus Lytic Replication

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The Gammaherpesvirus 68 Latency-Associated Nuclear Antigen Homolog Is Critical for the Establishment of Splenic Latency

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Terminal Differentiation into Plasma Cells Initiates the Replicative Cycle of Epstein-Barr Virus In Vivo

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KSHV infects a subset of human tonsillar B cells, driving proliferation and plasmablast differentiation

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Gammaherpesvirus-Driven Plasma Cell Differentiation Regulates Virus Reactivation from Latently Infected B Lymphocytes

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Stabilization of Myc through Heterotypic Poly-Ubiquitination by mLANA Is Critical for γ-Herpesvirus Lymphoproliferation

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Murine Gammaherpesvirus M2 Protein Induction of IRF4 via the NFAT Pathway Leads to IL-10 Expression in B Cells

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