On the public citation graph

Suppression of MDA5-mediated antiviral immune responses by NSP8 of SARS-CoV-2

https://doi.org/10.1101/2020.08.12.247767

0 signed · 53 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/jmv.25681openalex-snapshot

Emerging coronaviruses: Genome structure, replication, and pathogenesis

inferredcites →https://doi.org/10.1016/j.celrep.2017.10.020openalex-snapshot

The E3 Ubiquitin Ligase TRIM40 Attenuates Antiviral Immune Responses by Targeting MDA5 and RIG-I

inferredcites →https://doi.org/10.1016/j.chom.2016.01.007openalex-snapshot

Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice

inferredcites →https://doi.org/10.1016/j.chom.2019.09.012openalex-snapshot

Zika Virus NS3 Mimics a Cellular 14-3-3-Binding Motif to Antagonize RIG-I- and MDA5-Mediated Innate Immunity

inferredcites →https://doi.org/10.1016/j.ebiom.2016.06.015openalex-snapshot

Regulation of Retinoic Acid Inducible Gene-I (RIG-I) Activation by the Histone Deacetylase 6

inferredcites →https://doi.org/10.1016/j.immuni.2012.03.022openalex-snapshot

Ubiquitin-Induced Oligomerization of the RNA Sensors RIG-I and MDA5 Activates Antiviral Innate Immune Response

inferredcites →https://doi.org/10.1016/j.jmii.2015.07.002openalex-snapshot

Phage display technique identifies the interaction of severe acute respiratory syndrome coronavirus open reading frame 6 protein with nuclear pore complex interacting protein NPIPB3 in modulating Type I interferon antagonism

inferredcites →https://doi.org/10.1016/s0140-6736(20)30183-5openalex-snapshot

Clinical features of patients infected with 2019 novel coronavirus in Wuhan, China

inferredcites →https://doi.org/10.1016/s0140-6736(20)30211-7openalex-snapshot

Epidemiological and clinical characteristics of 99 cases of 2019 novel coronavirus pneumonia in Wuhan, China: a descriptive study

inferredcites →https://doi.org/10.1016/s0165-2478(02)00228-6openalex-snapshot

Recognition of pathogen-associated molecular patterns by TLR family

inferredcites →https://doi.org/10.1038/ni.1979openalex-snapshot

Ribose 2′-O-methylation provides a molecular signature for the distinction of self and non-self mRNA dependent on the RNA sensor Mda5

inferredcites →https://doi.org/10.1038/ni.2239openalex-snapshot

NLRP4 negatively regulates type I interferon signaling by targeting the kinase TBK1 for degradation via the ubiquitin ligase DTX4

inferredcites →https://doi.org/10.1080/22221751.2020.1747363openalex-snapshot

Transcriptomic characteristics of bronchoalveolar lavage fluid and peripheral blood mononuclear cells in COVID-19 patients

inferredcites →https://doi.org/10.1093/bioinformatics/btu097openalex-snapshot

ZDOCK server: interactive docking prediction of protein–protein complexes and symmetric multimers

inferredcites →https://doi.org/10.1093/nar/gku340openalex-snapshot

SWISS-MODEL: modelling protein tertiary and quaternary structure using evolutionary information

inferredcites →https://doi.org/10.1126/science.abc8665openalex-snapshot

Structural basis for translational shutdown and immune evasion by the Nsp1 protein of SARS-CoV-2

inferredcites →https://doi.org/10.1128/jvi.01700-12openalex-snapshot

Severe Acute Respiratory Syndrome Coronavirus nsp1 Facilitates Efficient Propagation in Cells through a Specific Translational Shutoff of Host mRNA

inferredcites →https://doi.org/10.1128/jvi.02220-08openalex-snapshot

Severe Acute Respiratory Syndrome Coronavirus Papain-Like Protease Ubiquitin-Like Domain and Catalytic Domain Regulate Antagonism of IRF3 and NF-κB Signaling

inferredcites →https://doi.org/10.1146/annurev-med-051215-031152openalex-snapshot

Middle East Respiratory Syndrome: Emergence of a Pathogenic Human Coronavirus

inferredcites →https://doi.org/10.1146/annurev.immunol.23.021704.115843openalex-snapshot

TYPE I INTERFERONS (α/β) IN IMMUNITY AND AUTOIMMUNITY

inferredcites →https://doi.org/10.1371/journal.pone.0005466openalex-snapshot

MAVS-Mediated Apoptosis and Its Inhibition by Viral Proteins

inferredcites →https://doi.org/10.1371/journal.ppat.1002059openalex-snapshot

Crystal Structure and Functional Analysis of the SARS-Coronavirus RNA Cap 2′-O-Methyltransferase nsp10/nsp16 Complex

inferredcites →https://doi.org/10.1371/journal.ppat.1002294openalex-snapshot

Biochemical and Structural Insights into the Mechanisms of SARS Coronavirus RNA Ribose 2′-O-Methylation by nsp16/nsp10 Protein Complex

inferredcites →https://doi.org/10.1371/journal.ppat.1002433openalex-snapshot

SARS Coronavirus nsp1 Protein Induces Template-Dependent Endonucleolytic Cleavage of mRNAs: Viral mRNAs Are Resistant to nsp1-Induced RNA Cleavage

inferredcites →https://doi.org/10.1371/journal.ppat.1006838openalex-snapshot

MERS-CoV 4b protein interferes with the NF-κB-dependent innate immune response during infection

inferredcites →https://doi.org/10.1371/journal.ppat.1006852openalex-snapshot

Herpesvirus deconjugases inhibit the IFN response by promoting TRIM25 autoubiquitination and functional inactivation of the RIG-I signalosome

inferredcites →https://doi.org/10.3389/fimmu.2020.00939openalex-snapshot

Pangolins Lack IFIH1/MDA5, a Cytoplasmic RNA Sensor That Initiates Innate Immune Defense Upon Coronavirus Infection

inferredcites →https://doi.org/10.4049/jimmunol.1303196openalex-snapshot

SARS-Coronavirus Open Reading Frame-9b Suppresses Innate Immunity by Targeting Mitochondria and the MAVS/TRAF3/TRAF6 Signalosome

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1002/jmv.28680openalex-snapshot

SARS‐CoV‐2 NSP8 suppresses type I and III IFN responses by modulating the RIG‐I/MDA5, TRIF, and STING signaling pathways

inferred← citeshttps://doi.org/10.1007/s00705-022-05365-2openalex-snapshot

The ins and outs of SARS-CoV-2 variants of concern (VOCs)

inferred← citeshttps://doi.org/10.1007/s10753-021-01519-7openalex-snapshot

Severe Acute Respiratory Syndrome Coronavirus 2: The Role of the Main Components of the Innate Immune System

inferred← citeshttps://doi.org/10.1007/s12275-022-1525-1openalex-snapshot

SARS-CoV-2-mediated evasion strategies for antiviral interferon pathways

inferred← citeshttps://doi.org/10.1016/j.crviro.2021.100013openalex-snapshot

Devil's tools: SARS-CoV-2 antagonists against innate immunity

inferred← citeshttps://doi.org/10.1016/j.intimp.2022.108531openalex-snapshot

Immunoediting in SARS-CoV-2: Mutual relationship between the virus and the host

inferred← citeshttps://doi.org/10.1016/j.it.2021.12.005openalex-snapshot

K63 ubiquitination in immune signaling

inferred← citeshttps://doi.org/10.1016/j.jmb.2023.168008openalex-snapshot

Selenoprotein S Interacts with the Replication and Transcription Complex of SARS-CoV-2 by Binding Nsp7

inferred← citeshttps://doi.org/10.1038/s12276-021-00602-1openalex-snapshot

Innate immune sensing of coronavirus and viral evasion strategies

inferred← citeshttps://doi.org/10.1038/s41423-020-00602-7openalex-snapshot

Regulation of RIG-I-like receptor-mediated signaling: interaction between host and viral factors

inferred← citeshttps://doi.org/10.1038/s41423-020-00619-yopenalex-snapshot

SARS-CoV-2 nsp12 attenuates type I interferon production by inhibiting IRF3 nuclear translocation

inferred← citeshttps://doi.org/10.1038/s41423-023-01104-yopenalex-snapshot

SARS-CoV-2 and innate immunity: the good, the bad, and the “goldilocks”

inferred← citeshttps://doi.org/10.1038/s41589-022-01128-xopenalex-snapshot

A C-terminal glutamine recognition mechanism revealed by E3 ligase TRIM7 structures

inferred← citeshttps://doi.org/10.1089/jir.2021.0060openalex-snapshot

Control of Innate Immune Activation by Severe Acute Respiratory Syndrome Coronavirus 2 and Other Coronaviruses

inferred← citeshttps://doi.org/10.1101/2022.03.24.485560openalex-snapshot

Structural insights into the viral proteins binding by TRIM7 reveal a general C-terminal glutamine recognition mechanism

inferred← citeshttps://doi.org/10.14348/molcells.2021.0026openalex-snapshot

Molecular Perspectives of SARS-CoV-2: Pathology, Immune Evasion, and Therapeutic Interventions

inferred← citeshttps://doi.org/10.3389/fimmu.2022.940756openalex-snapshot

Roles and functions of SARS-CoV-2 proteins in host immune evasion

inferred← citeshttps://doi.org/10.3389/fimmu.2024.1363572openalex-snapshot

New insights into the pathogenesis of SARS-CoV-2 during and after the COVID-19 pandemic

inferred← citeshttps://doi.org/10.3389/fmicb.2021.770656openalex-snapshot

Innate Immunity Evasion Strategies of Highly Pathogenic Coronaviruses: SARS-CoV, MERS-CoV, and SARS-CoV-2

inferred← citeshttps://doi.org/10.3390/ijms23115966openalex-snapshot

Could a Lower Toll-like Receptor (TLR) and NF-κB Activation Due to a Changed Charge Distribution in the Spike Protein Be the Reason for the Lower Pathogenicity of Omicron?

inferred← citeshttps://doi.org/10.3390/v13071373openalex-snapshot

Immunity and Viral Infections: Modulating Antiviral Response via CRISPR–Cas Systems

inferred← citeshttps://doi.org/10.3390/v14020189openalex-snapshot

Induction of Innate Immune Response by TLR3 Agonist Protects Mice against SARS-CoV-2 Infection

inferred← citeshttps://doi.org/10.3390/v14030530openalex-snapshot

Escape and Over-Activation of Innate Immune Responses by SARS-CoV-2: Two Faces of a Coin

inferred← citeshttps://doi.org/10.3390/v14091991openalex-snapshot

SARS-CoV-2 Non-Structural Proteins and Their Roles in Host Immune Evasion

inferred← citeshttps://doi.org/10.3390/v17040569openalex-snapshot

Impact of SARS-CoV-2 Wuhan and Omicron Variant Proteins on Type I Interferon Response

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.