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Claudin-4, a core component of the tight-junctional complex along the collecting system, is induced in nephrotic syndrome

https://doi.org/10.1101/2022.06.01.494142

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.kint.2018.05.001openalex-snapshot

A mouse model of pseudohypoaldosteronism type II reveals a novel mechanism of renal tubular acidosis

inferredcites →https://doi.org/10.1038/ki.2012.180openalex-snapshot

The pathophysiology of edema formation in the nephrotic syndrome

inferredcites →https://doi.org/10.1038/ki.2013.14openalex-snapshot

Rapid dephosphorylation of the renal sodium chloride cotransporter in response to oral potassium intake in mice

inferredcites →https://doi.org/10.1038/s41598-017-15108-yopenalex-snapshot

Identification of claudin-4 binder that attenuates tight junction barrier function by TR-FRET-based screening assay

inferredcites →https://doi.org/10.1073/pnas.071516798openalex-snapshot

Pendrin, encoded by the Pendred syndrome gene, resides in the apical region of renal intercalated cells and mediates bicarbonate secretion

inferredcites →https://doi.org/10.1073/pnas.1009399107openalex-snapshot

Claudin-4 forms paracellular chloride channel in the kidney and requires claudin-8 for tight junction localization

inferredcites →https://doi.org/10.1073/pnas.1406741111openalex-snapshot

The Cap1–claudin-4 regulatory pathway is important for renal chloride reabsorption and blood pressure regulation

inferredcites →https://doi.org/10.1073/pnas.1421441112openalex-snapshot

KLHL3 regulates paracellular chloride transport in the kidney by ubiquitination of claudin-8

inferredcites →https://doi.org/10.1074/jbc.m116.724690openalex-snapshot

Oxidative Stress and Nuclear Factor κB (NF-κB) Increase Peritoneal Filtration and Contribute to Ascites Formation in Nephrotic Syndrome

inferredcites →https://doi.org/10.1074/jbc.m212388200openalex-snapshot

Dual Influence of Aldosterone on AQP2 Expression in Cultured Renal Collecting Duct Principal Cells

inferredcites →https://doi.org/10.1096/fj.201901345ropenalex-snapshot

Time‐course of sodium transport along the nephron in nephrotic syndrome: The role of potassium

inferredcites →https://doi.org/10.1097/mnh.0b013e328332bda4openalex-snapshot

Regulation of sodium transport by ENaC in the kidney

inferredcites →https://doi.org/10.1113/jphysiol.2011.209692openalex-snapshot

Inhibition of K + secretion in the distal nephron in nephrotic syndrome: possible role of albuminuria

inferredcites →https://doi.org/10.1152/ajprenal.00155.2007openalex-snapshot

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null mice

inferredcites →https://doi.org/10.1152/ajprenal.00207.2021openalex-snapshot

Expression of claudin-8 is induced by aldosterone in renal collecting duct principal cells

inferredcites →https://doi.org/10.1152/ajprenal.00277.2003openalex-snapshot

Increased expression and apical targeting of renal ENaC subunits in puromycin aminonucleoside-induced nephrotic syndrome in rats

inferredcites →https://doi.org/10.1152/ajprenal.00450.2009openalex-snapshot

Renal salt wasting and chronic dehydration in claudin-7-deficient mice

inferredcites →https://doi.org/10.1152/ajprenal.00613.2007openalex-snapshot

Mouse model of type II Bartter's syndrome. II. Altered expression of renal sodium- and water-transporting proteins

inferredcites →https://doi.org/10.1152/ajprenal.1984.247.2.f291openalex-snapshot

Microelectrode assessment of chloride-conductive properties of cortical collecting duct

inferredcites →https://doi.org/10.1161/01.hyp.0000088321.67254.b7openalex-snapshot

Deoxycorticosterone Upregulates PDS ( Slc26a4 ) in Mouse Kidney

inferredcites →https://doi.org/10.1161/01.hyp.0000145863.96091.89openalex-snapshot

NaCl Restriction Upregulates Renal Slc26a4 Through Subcellular Redistribution

inferredcites →https://doi.org/10.1172/jci110756openalex-snapshot

Role for intrarenal mechanisms in the impaired salt excretion of experimental nephrotic syndrome.

inferredcites →https://doi.org/10.1172/jci40145openalex-snapshot

The Na+-dependent chloride-bicarbonate exchanger SLC4A8 mediates an electroneutral Na+ reabsorption process in the renal cortical collecting ducts of mice

inferredcites →https://doi.org/10.1172/jci83942openalex-snapshot

Paracellular epithelial sodium transport maximizes energy efficiency in the kidney

inferredcites →https://doi.org/10.1242/jcs.040113openalex-snapshot

Claudin-10 exists in six alternatively spliced isoforms that exhibit distinct localization and function

inferredcites →https://doi.org/10.14814/phy2.12169openalex-snapshot

Epithelial sodium channel abundance is decreased by an unfolded protein response induced by hyperosmolality

inferredcites →https://doi.org/10.1681/asn.2005040363openalex-snapshot

Hyperaldosteronemia and Activation of the Epithelial Sodium Channel Are Not Required for Sodium Retention in Puromycin-Induced Nephrosis

inferredcites →https://doi.org/10.1681/asn.2008040364openalex-snapshot

Plasmin in Nephrotic Urine Activates the Epithelial Sodium Channel

inferredcites →https://doi.org/10.1681/asn.2012040373openalex-snapshot

NADPH-Oxidase 4 Protects against Kidney Fibrosis during Chronic Renal Injury

inferredcites →https://doi.org/10.1681/asn.2012040414openalex-snapshot

Adiponectin Promotes Functional Recovery after Podocyte Ablation

inferredcites →https://doi.org/10.1681/asn.2013040429openalex-snapshot

Sodium Transport Is Modulated by p38 Kinase–Dependent Cross-Talk between ENaC and Na,K-ATPase in Collecting Duct Principal Cells

inferredcites →https://doi.org/10.1681/asn.2014030284openalex-snapshot

Claudins and the Kidney

inferredcites →https://doi.org/10.1681/asn.2019080790openalex-snapshot

Interaction between Epithelial Sodium Channel γ-Subunit and Claudin-8 Modulates Paracellular Sodium Permeability in Renal Collecting Duct

inferredcites →https://doi.org/10.1681/asn.v114604openalex-snapshot

Collecting Duct Na+/K+-ATPase Activity Is Correlated with Urinary Sodium Excretion in Rat Nephrotic Syndromes

inferredcites →https://doi.org/10.1681/asn.v123598openalex-snapshot

Collecting Duct Is a Site of Sodium Retention in PAN Nephrosis

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1097/mnh.0000000000001003openalex-snapshot

The role of claudins in renal transepithelial transport and kidney disease

inferred← citeshttps://doi.org/10.1111/apha.70216openalex-snapshot

Low‐Salt Diet Induces Claudin‐3 Expression and Drives Adaptive Changes in Collecting Duct of Claudin‐3‐Deficient Mice

inferred← citeshttps://doi.org/10.3389/fphys.2026.1869073openalex-snapshot

Claudin-3 is expressed at the macula densa and is associated with altered glomerular filtration in claudin-3 knockout mice

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