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Targeting Epsins to Inhibit FGF Signaling while Potentiating TGF-β Signaling Constrains Endothelial-to-Mesenchymal-Transition in Atherosclerosis

https://doi.org/10.1101/2022.08.09.503324
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1 of the 47 references listed on this page is in the Retraction Watch database as retracted (export 2026-10-21): 10.2337/db09-1637 (notice 2026-07-20).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s10456-018-9639-0openalex-snapshot
The therapeutic potential of targeting the endothelial-to-mesenchymal transition
inferredcites →https://doi.org/10.1016/j.ijmm.2017.03.001openalex-snapshot
Chlamydia pneumoniae infection promotes vascular endothelial cell angiogenesis through an IQGAP1-related signaling pathway
inferredcites →https://doi.org/10.1016/j.jacc.2015.10.092openalex-snapshot
Myocardial Infarction Alters Adaptation of the Tethered Mitral Valve
inferredcites →https://doi.org/10.1016/j.jacc.2015.11.036openalex-snapshot
Mitral Valve Adaptation in Ischemic Heart Disease∗
inferredcites →https://doi.org/10.1016/j.jacc.2017.07.734openalex-snapshot
Effect of Losartan on Mitral Valve Changes After Myocardial Infarction
inferredcites →https://doi.org/10.1016/j.jacc.2017.07.751openalex-snapshot
New Pharmacological Target to Treat Ischemic Mitral Regurgitation
inferredcites →https://doi.org/10.1016/j.jacc.2018.09.089openalex-snapshot
Endothelial to Mesenchymal Transition in Cardiovascular Disease
inferredcites →https://doi.org/10.1038/29555openalex-snapshot
Epsin is an EH-domain-binding protein implicated in clathrin-mediated endocytosis
inferredcites →https://doi.org/10.1038/35025203openalex-snapshot
Atherosclerosis
inferredcites →https://doi.org/10.1038/nature01020openalex-snapshot
Curvature of clathrin-coated pits driven by epsin
inferredcites →https://doi.org/10.1038/nbt.4096openalex-snapshot
Integrating single-cell transcriptomic data across different conditions, technologies, and species
inferredcites →https://doi.org/10.1038/ncomms11853openalex-snapshot
Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability
inferredcites →https://doi.org/10.1038/nm720openalex-snapshot
A tumor-homing peptide with a targeting specificity related to lymphatic vessels
inferredcites →https://doi.org/10.1038/s41467-020-17848-4openalex-snapshot
Epsin-mediated degradation of IP3R1 fuels atherosclerosis
inferredcites →https://doi.org/10.1038/s41569-018-0023-yopenalex-snapshot
Reassessing endothelial-to-mesenchymal transition in cardiovascular diseases
inferredcites →https://doi.org/10.1038/s42255-019-0100-5openalex-snapshot
Transforming endothelial cells in atherosclerosis
inferredcites →https://doi.org/10.1038/s42255-019-0102-3openalex-snapshot
Endothelial TGF-β signalling drives vascular inflammation and atherosclerosis
inferredcites →https://doi.org/10.1038/s42255-020-00338-8openalex-snapshot
Multiple cell types contribute to the atherosclerotic lesion fibrous cap by PDGFRβ and bioenergetic mechanisms
inferredcites →https://doi.org/10.1073/pnas.0907008106openalex-snapshot
Embryonic arrest at midgestation and disruption of Notch signaling produced by the absence of both epsin 1 and epsin 2 in mice
inferredcites →https://doi.org/10.1073/pnas.1104540108openalex-snapshot
Specific penetration and accumulation of a homing peptide within atherosclerotic plaques of apolipoprotein E-deficient mice
inferredcites →https://doi.org/10.1093/cvr/cvx253openalex-snapshot
Endothelial–mesenchymal transition in atherosclerosis
inferredcites →https://doi.org/10.1093/cvr/cvz211openalex-snapshot
Non-coding RNA in endothelial-to-mesenchymal transition
inferredcites →https://doi.org/10.1126/science.111.2877.166openalex-snapshot
The Role of Lipids and Lipoproteins in Atherosclerosis
inferredcites →https://doi.org/10.1126/scisignal.2005413openalex-snapshot
Temporal and spatial regulation of epsin abundance and VEGFR3 signaling are required for lymphatic valve formation and function
inferredcites →https://doi.org/10.1126/scisignal.2005504openalex-snapshot
Fibroblast growth factor receptor 1 is a key inhibitor of TGFβ signaling in the endothelium
inferredcites →https://doi.org/10.1126/scitranslmed.3006927openalex-snapshot
TGF-β Signaling Mediates Endothelial-to-Mesenchymal Transition (EndMT) During Vein Graft Remodeling
inferredcites →https://doi.org/10.1146/annurev.pathol.1.110304.100100openalex-snapshot
INFLAMMATION AND ATHEROSCLEROSIS
inferredcites →https://doi.org/10.1158/0008-5472.can-07-6752openalex-snapshot
Mitochondrial/Cell-Surface Protein p32/gC1qR as a Molecular Target in Tumor Cells and Tumor Stroma
inferredcites →https://doi.org/10.1161/01.res.0000245188.41002.2copenalex-snapshot
Human Pulmonary Valve Progenitor Cells Exhibit Endothelial/Mesenchymal Plasticity in Response to Vascular Endothelial Growth Factor-A and Transforming Growth Factor-β 2
inferredcites →https://doi.org/10.1161/atvbaha.113.302586openalex-snapshot
Genetic Reduction of Vascular Endothelial Growth Factor Receptor 2 Rescues Aberrant Angiogenesis Caused by Epsin Deficiency
inferredcites →https://doi.org/10.1161/circresaha.115.306301openalex-snapshot
Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis
inferredcites →https://doi.org/10.1161/circresaha.115.307679openalex-snapshot
Selective Targeting of a Novel Epsin–VEGFR2 Interaction Promotes VEGF-Mediated Angiogenesis
inferredcites →https://doi.org/10.1161/circresaha.118.313028openalex-snapshot
Myeloid-Specific Deletion of Epsins 1 and 2 Reduces Atherosclerosis by Preventing LRP-1 Downregulation
inferredcites →https://doi.org/10.1161/circresaha.119.314813openalex-snapshot
Endothelial-to-Mesenchymal Transition
inferredcites →https://doi.org/10.1161/circulationaha.108.846782openalex-snapshot
Active Adaptation of the Tethered Mitral Valve
inferredcites →https://doi.org/10.1161/circulationaha.109.900928openalex-snapshot
Reduction of AMP-Activated Protein Kinase α2 Increases Endoplasmic Reticulum Stress and Atherosclerosis In Vivo
inferredcites →https://doi.org/10.1172/jci129374openalex-snapshot
Epsins 1 and 2 promote NEMO linear ubiquitination via LUBAC to drive breast cancer development
inferredcites →https://doi.org/10.1172/jci64537openalex-snapshot
Endothelial epsin deficiency decreases tumor growth by enhancing VEGF signaling
inferredcites →https://doi.org/10.1172/jci66171openalex-snapshot
Are epsins a therapeutic target for tumor angiogenesis?
inferredcites →https://doi.org/10.1172/jci80349openalex-snapshot
Motif mimetic of epsin perturbs tumor growth and metastasis
inferredcites →https://doi.org/10.1172/jci82719openalex-snapshot
Endothelial-to-mesenchymal transition drives atherosclerosis progression
inferredcites →https://doi.org/10.1172/jci98215openalex-snapshot
Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heart
inferredcites →https://doi.org/10.1177/1479164116666762openalex-snapshot
Role of endoplasmic reticulum stress signalling in diabetic endothelial dysfunction and atherosclerosis
inferredcites →https://doi.org/10.1186/s12864-018-4772-0openalex-snapshot
Slingshot: cell lineage and pseudotime inference for single-cell transcriptomics
inferredcites →https://doi.org/10.2337/db09-1637openalex-snapshot
Activation of AMP-Activated Protein Kinase Inhibits Oxidized LDL-Triggered Endoplasmic Reticulum Stress In Vivo
inferredcites →https://doi.org/10.3389/fcvm.2020.00053openalex-snapshot
Endothelial-to-Mesenchymal Transition, Vascular Inflammation, and Atherosclerosis
inferredcites →https://doi.org/10.3390/cells10081918openalex-snapshot
Epsins Negatively Regulate Aortic Endothelial Cell Function by Augmenting Inflammatory Signaling
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