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Role of somatic HCN in epileptiform activity in subicular neurons

https://doi.org/10.1101/2024.07.26.604395

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Outgoing claims: the papers this work cites, builds on, or argues with.

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H-Current

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HCN hyperpolarization-activated cation channels inhibit EPSPs by interactions with M-type K+ channels

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Trafficking and Surface Expression of Hyperpolarization-activated Cyclic Nucleotide-gated Channels in Hippocampal Neurons

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Hyperpolarization-Activated Cyclic Nucleotide-Gated (HCN) Channels in Epilepsy

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Progression of Brain Damage after Status Epilepticus and Its Association with Epileptogenesis: A Quantitative MRI Study in a Rat Model of Temporal Lobe Epilepsy

inferredcites →https://doi.org/10.1111/j.1460-9568.2008.06519.xopenalex-snapshot

Epileptiform activity induces distance‐dependent alterations of the Ca2+ extrusion mechanism in the apical dendrites of subicular pyramidal neurons

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Mode of action of bradycardic agent, S 16257, on ionic currents of rabbit sinoatrial node cells

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Hyperpolarization‐activated cation currents in human epileptogenic neocortex

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The hyperpolarization‐activated current (Ih) and its contribution to pacemaker activity in rat CA1 hippocampal stratum oriens‐alveus interneurones.

inferredcites →https://doi.org/10.1113/jphysiol.2006.113944openalex-snapshot

Different levels of Ih determine distinct temporal integration in bursting and regular‐spiking neurons in rat subiculum

inferredcites →https://doi.org/10.1113/jphysiol.2006.122028openalex-snapshot

Inherited cortical HCN1 channel loss amplifies dendritic calcium electrogenesis and burst firing in a rat absence epilepsy model

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Light-Induced Structural and Functional Plasticity in Drosophila Larval Visual System

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Decreased Hippocampal Inhibition and a Selective Loss of Interneurons in Experimental Epilepsy

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Persistent Sodium Current in Mammalian Central Neurons

inferredcites →https://doi.org/10.1152/jn.00850.2002openalex-snapshot

Development of Inward Rectification and Control of Membrane Excitability in Mesencephalic V Neurons

inferredcites →https://doi.org/10.1152/jn.01333.2005openalex-snapshot

Axo-Somatic and Apical Dendritic Kv7/M Channels Differentially Regulate the Intrinsic Excitability of Adult Rat CA1 Pyramidal Cells

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Quantification of membrane properties of trigeminal root ganglion neurons in guinea pigs

inferredcites →https://doi.org/10.1152/jn.2001.86.1.261openalex-snapshot

Contribution of the Hyperpolarization-Activated Current (I h) to Membrane Potential and GABA Release in Hippocampal Interneurons

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Novel HCN2 Mutation Contributes to Febrile Seizures by Shifting the Channel's Kinetics in a Temperature-Dependent Manner

inferredcites →https://doi.org/10.1523/jneurosci.0309-04.2004openalex-snapshot

Synaptic Contributions to Focal and Widespread Spatiotemporal Dynamics in the Isolated Rat SubiculumIn Vitro

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Suppression of Hyperpolarization-Activated Cyclic Nucleotide-Gated Channel Function in Thalamocortical Neurons Prevents Genetically Determined and Pharmacologically Induced Absence Seizures

inferredcites →https://doi.org/10.1523/jneurosci.1148-11.2011openalex-snapshot

Rapid Loss of Dendritic HCN Channel Expression in Hippocampal Pyramidal Neurons following Status Epilepticus

inferredcites →https://doi.org/10.1523/jneurosci.1531-09.2009openalex-snapshot

Loss of Dendritic HCN1 Subunits Enhances Cortical Excitability and Epileptogenesis

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Developmental Febrile Seizures Modulate Hippocampal Gene Expression of Hyperpolarization-Activated Channels in an Isoform- and Cell-Specific Manner

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Upregulation of the Hyperpolarization-Activated Cation Current after Chronic Compression of the Dorsal Root Ganglion

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inferredcites →https://doi.org/10.1523/jneurosci.3605-07.2007openalex-snapshot

Progressive Dendritic HCN Channelopathy during Epileptogenesis in the Rat Pilocarpine Model of Epilepsy

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Recessive Loss-of-Function Mutation in the Pacemaker HCN2 Channel Causing Increased Neuronal Excitability in a Patient with Idiopathic Generalized Epilepsy

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The Contribution of HCN Channelopathies in Different Epileptic Syndromes, Mechanisms, Modulators, and Potential Treatment Targets: A Systematic Review

inferredcites →https://doi.org/10.3389/neuro.03.002.2008openalex-snapshot

Upregulated H-Current in hyperexcitable CA1 dendrites after febrile seizures

inferredcites →https://doi.org/10.4161/chan.22209openalex-snapshot

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Contributions of T-type calcium channel isoforms to neuronal firing

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