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Dark selection for JAK/STAT-inhibitor resistance in chronic myelomonocytic leukemia

https://doi.org/10.1101/211151
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Outgoing claims: the papers this work cites, builds on, or argues with.

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Non-cell-autonomous driving of tumour growth supports sub-clonal heterogeneity
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Mutation allele burden remains unchanged in chronic myelomonocytic leukaemia responding to hypomethylating agents
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Non-genetic heterogeneity — a mutation-independent driving force for the somatic evolution of tumours
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Heterogeneity for IGF-II production maintained by public goods dynamics in neuroendocrine pancreatic cancer
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Tumor Hypoxia: Definitions and Current Clinical, Biologic, and Molecular Aspects
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Edge effects in game-theoretic dynamics of spatially structured tumours
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Fibroblasts and Alectinib switch the evolutionary games played by non-small cell lung cancer
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A Multi-Institution Phase I Trial of Ruxolitinib in Patients with Chronic Myelomonocytic Leukemia (CMML)
inferredcites →https://doi.org/10.1182/blood-2009-03-209262openalex-snapshot
The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes
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The Concentration of Oxygen Dissolved in Tissues at the Time of Irradiation as a Factor in Radiotherapy
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Oxygen diffusion and the distribution of cellular radiosensitivity in tumours
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StochPy: A Comprehensive, User-Friendly Tool for Simulating Stochastic Biological Processes
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The Role of Oxygen in Avascular Tumor Growth
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Hypoxia Signaling and the Metastatic Phenotype
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Analysis of the Effects of Oxygen Supply and Demand on Hypoxic Fraction in Tumors
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