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Interleukin-17A Secreted from the Lung–infiltrating T Helper 17 Cells Renders Protective Immunity to Pulmonary Cryptococcus neoformans Infection

https://doi.org/10.1101/328005
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s11046-005-3091-6openalex-snapshot
The isolation, characterization and antifungal susceptibilities of Cryptococcus neoformans from bird excreta in Klang Valley, Malaysia
inferredcites →https://doi.org/10.1016/j.ajpath.2011.04.025openalex-snapshot
Eosinophils Contribute to IL-4 Production and Shape the T-Helper Cytokine Profile and Inflammatory Response in Pulmonary Cryptococcosis
inferredcites →https://doi.org/10.1016/j.ajpath.2011.12.038openalex-snapshot
IL-23 Dampens the Allergic Response to Cryptococcus neoformans through IL-17–Independent and –Dependent Mechanisms
inferredcites →https://doi.org/10.1016/j.cub.2006.09.032openalex-snapshot
Expulsion of Live Pathogenic Yeast by Macrophages
inferredcites →https://doi.org/10.1016/j.cub.2006.09.061openalex-snapshot
Phagosome Extrusion and Host-Cell Survival after Cryptococcus neoformans Phagocytosis by Macrophages
inferredcites →https://doi.org/10.1016/j.cytogfr.2010.10.001openalex-snapshot
C-type lectins, fungi and Th17 responses
inferredcites →https://doi.org/10.1016/s0378-1097(00)00128-2openalex-snapshot
Reduced host resistance and Th1 response to Cryptococcus neoformans in interleukin-18 deficient mice
inferredcites →https://doi.org/10.1038/nrmicro.2015.6openalex-snapshot
Cryptococcus: from environmental saprophyte to global pathogen
inferredcites →https://doi.org/10.1038/srep11046openalex-snapshot
Suppression of cell division-associated genes by Helicobacter pylori attenuates proliferation of RAW264.7 monocytic macrophage cells
inferredcites →https://doi.org/10.1038/srep21489openalex-snapshot
Cryptococcus neoformans Intracellular Proliferation and Capsule Size Determines Early Macrophage Control of Infection
inferredcites →https://doi.org/10.1074/jbc.m116.738187openalex-snapshot
Modulation of Macrophage Inflammatory Nuclear Factor κB (NF-κB) Signaling by Intracellular Cryptococcus neoformans
inferredcites →https://doi.org/10.1080/13693780802140766openalex-snapshot
Th17 cells in the setting ofAspergillusinfection and pathology
inferredcites →https://doi.org/10.1084/jem.173.3.755openalex-snapshot
Intrapulmonary growth and dissemination of an avirulent strain of Cryptococcus neoformans in mice depleted of CD4+ or CD8+ T cells.
inferredcites →https://doi.org/10.1084/jem.173.4.793openalex-snapshot
Immunity to a pulmonary Cryptococcus neoformans infection requires both CD4+ and CD8+ T cells.
inferredcites →https://doi.org/10.1084/jem.20081463openalex-snapshot
Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis
inferredcites →https://doi.org/10.1086/422329openalex-snapshot
Requirement of Interleukin‐17A for Systemic Anti– Candida albicans Host Defense in Mice
inferredcites →https://doi.org/10.1086/592690openalex-snapshot
Immunocompromised Hosts: Cryptococcosis in Solid Organ Transplant Recipients: Current State of the Science
inferredcites →https://doi.org/10.1093/infdis/jit099openalex-snapshot
The Phenotype of the Cryptococcus-Specific CD4+ Memory T-Cell Response Is Associated With Disease Severity and Outcome in HIV-Associated Cryptococcal Meningitis
inferredcites →https://doi.org/10.1097/qad.0b013e3282a4a64dopenalex-snapshot
HIV-associated cryptococcal meningitis
inferredcites →https://doi.org/10.1111/j.1348-0421.2000.tb02573.xopenalex-snapshot
Th1‐Th2 Cytokine Kinetics in the Bronchoalveolar Lavage Fluid of Mice Infected with Cryptococcus neoformans of Different Virulences
inferredcites →https://doi.org/10.1111/j.1365-2141.2005.05397.xopenalex-snapshot
The immune response to fungal infections
inferredcites →https://doi.org/10.1111/j.1365-2567.2009.03211.xopenalex-snapshot
Anti‐Aspergillus human host defence relies on type 1 T helper (Th1), rather than type 17 T helper (Th17), cellular immunity
inferredcites →https://doi.org/10.1128/cmr.8.4.515openalex-snapshot
Cryptococcosis in the era of AIDS--100 years after the discovery of Cryptococcus neoformans
inferredcites →https://doi.org/10.1128/ec.00039-10openalex-snapshot
Cryptococcal Interactions with the Host Immune System
inferredcites →https://doi.org/10.1128/iai.00809-09openalex-snapshot
Th2 but Not Th1 Immune Bias Results in Altered Lung Functions in a Murine Model of PulmonaryCryptococcus neoformansInfection
inferredcites →https://doi.org/10.1128/iai.00845-10openalex-snapshot
Interleukin-17 Is Not Required for Classical Macrophage Activation in a Pulmonary Mouse Model ofCryptococcus neoformansInfection
inferredcites →https://doi.org/10.1128/iai.00929-09openalex-snapshot
Precarious Balance: Th17 Cells in Host Defense
inferredcites →https://doi.org/10.1128/iai.01171-09openalex-snapshot
Extracellular Vesicles from Cryptococcus neoformans Modulate Macrophage Functions
inferredcites →https://doi.org/10.1128/iai.01270-10openalex-snapshot
Effect of Cytokine Interplay on Macrophage Polarization during Chronic Pulmonary Infection with Cryptococcus neoformans
inferredcites →https://doi.org/10.1128/iai.01773-14openalex-snapshot
Cryptococcus gattii Infection Dampens Th1 and Th17 Responses by Attenuating Dendritic Cell Function and Pulmonary Chemokine Expression in the Immunocompetent Hosts
inferredcites →https://doi.org/10.1128/iai.66.10.4994-5000.1998openalex-snapshot
Interleukin-12 Is Essential for a Protective Th1 Response in Mice Infected withCryptococcus neoformans
inferredcites →https://doi.org/10.1128/mbio.00167-11openalex-snapshot
Nonlytic Exocytosis of Cryptococcus neoformans from Macrophages Occurs In Vivo and Is Influenced by Phagosomal pH
inferredcites →https://doi.org/10.1128/mbio.00264-13openalex-snapshot
Macrophage M1/M2 Polarization Dynamically Adapts to Changes in Cytokine Microenvironments in Cryptococcus neoformans Infection
inferredcites →https://doi.org/10.1159/000430375openalex-snapshot
Cryptococcus Neoformans Infection and Immune Cell Regulation in Human Monocytes
inferredcites →https://doi.org/10.1371/journal.pone.0006854openalex-snapshot
Insights into the Mechanisms of Protective Immunity against Cryptococcus neoformans Infection Using a Mouse Model of Pulmonary Cryptococcosis
inferredcites →https://doi.org/10.1371/journal.pone.0017204openalex-snapshot
Role of IL-17A on Resolution of Pulmonary C. neoformans Infection
inferredcites →https://doi.org/10.1371/journal.pone.0137457openalex-snapshot
Genome-Wide Transcription Study of Cryptococcus neoformans H99 Clinical Strain versus Environmental Strains
inferredcites →https://doi.org/10.1371/journal.ppat.1001041openalex-snapshot
The Human Fungal Pathogen Cryptococcus neoformans Escapes Macrophages by a Phagosome Emptying Mechanism That Is Inhibited by Arp2/3 Complex-Mediated Actin Polymerisation
inferredcites →https://doi.org/10.3389/fimmu.2017.01987openalex-snapshot
Contribution of IL-1RI Signaling to Protection against Cryptococcus neoformans 52D in a Mouse Model of Infection
inferredcites →https://doi.org/10.3389/fmicb.2016.00360openalex-snapshot
Triclosan Demonstrates Synergic Effect with Amphotericin B and Fluconazole and Induces Apoptosis-Like Cell Death in Cryptococcus neoformans
inferredcites →https://doi.org/10.4049/jimmunol.0804250openalex-snapshot
Requisite Role for the Dectin-1 β-Glucan Receptor in Pulmonary Defense against Aspergillus fumigatus
inferredcites →https://doi.org/10.4049/jimmunol.1402719openalex-snapshot
Cryptococcal Heat Shock Protein 70 Homolog Ssa1 Contributes to Pulmonary Expansion of Cryptococcus neoformans during the Afferent Phase of the Immune Response by Promoting Macrophage M2 Polarization
inferredcites →https://doi.org/10.4049/jimmunol.144.4.1472openalex-snapshot
Depletion of CD4+ (L3T4+) lymphocytes in vivo impairs murine host defense to Cryptococcus neoformans.
inferredcites →https://doi.org/10.4049/jimmunol.176.2.1098openalex-snapshot
IL-23 Enhances the Inflammatory Cell Response in Cryptococcus neoformans Infection and Induces a Cytokine Pattern Distinct from IL-12
inferredcites →https://doi.org/10.4049/jimmunol.177.8.5509openalex-snapshot
In the Absence of Reactive Oxygen Species, T Cells Default to a Th1 Phenotype and Mediate Protection against Pulmonary Cryptococcus neoformans Infection
inferredcites →https://doi.org/10.4049/jimmunol.179.8.5367openalex-snapshot
IL-13 Induces Disease-Promoting Type 2 Cytokines, Alternatively Activated Macrophages and Allergic Inflammation during Pulmonary Infection of Mice with Cryptococcus neoformans
inferredcites →https://doi.org/10.4049/jimmunol.180.6.4022openalex-snapshot
Lack of Toll IL-1R8 Exacerbates Th17 Cell Responses in Fungal Infection
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