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Phosphatidylinositol 5 phosphate 4-kinase regulates plasma-membrane PIP 3 turnover and insulin sensitivity in Drosophila

https://doi.org/10.1101/333153
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1 of the 47 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1042/bj20030577 (notice 2025-04-01).

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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00018-015-2063-3openalex-snapshot
Insulin/IGF signaling in Drosophila and other insects: factors that regulate production, release and post-release action of the insulin-like peptides
inferredcites →https://doi.org/10.1007/s00424-011-1008-4openalex-snapshot
Involvement of phosphatidylinositol 5-phosphate in insulin-stimulated glucose uptake in the L6 myotube model of skeletal muscle
inferredcites →https://doi.org/10.1016/j.advenzreg.2009.10.006openalex-snapshot
Localization, regulation and function of Type II phosphatidylinositol 5-phosphate 4-kinases
inferredcites →https://doi.org/10.1016/j.bbalip.2014.10.010openalex-snapshot
Phosphoinositide signalling in Drosophila
inferredcites →https://doi.org/10.1016/j.bbalip.2014.12.006openalex-snapshot
PI3K signalling in inflammation
inferredcites →https://doi.org/10.1016/j.biochi.2004.10.019openalex-snapshot
Positive and negative regulation of insulin signaling through IRS-1 phosphorylation
inferredcites →https://doi.org/10.1016/j.cell.2006.01.016openalex-snapshot
TOR Signaling in Growth and Metabolism
inferredcites →https://doi.org/10.1016/j.cell.2013.09.057openalex-snapshot
Depletion of a Putatively Druggable Class of Phosphatidylinositol Kinases Inhibits Growth of p53-Null Tumors
inferredcites →https://doi.org/10.1016/j.cmet.2010.08.012openalex-snapshot
TRPM Channels Mediate Zinc Homeostasis and Cellular Growth during Drosophila Larval Development
inferredcites →https://doi.org/10.1016/j.cub.2004.08.026openalex-snapshot
Inappropriate Activation of the TSC/Rheb/mTOR/S6K Cassette Induces IRS1/2 Depletion, Insulin Resistance, and Cell Survival Deficiencies
inferredcites →https://doi.org/10.1016/j.molcel.2017.09.024openalex-snapshot
PTEN Regulates PI(3,4)P2 Signaling Downstream of Class I PI3K
inferredcites →https://doi.org/10.1016/s0014-5793(98)01255-1openalex-snapshot
The SH2 domain containing inositol 5‐phosphatase SHIP2 displays phosphatidylinositol 3,4,5‐trisphosphate and inositol 1,3,4,5‐tetrakisphosphate 5‐phosphatase activity
inferredcites →https://doi.org/10.1016/s0960-9822(01)00068-9openalex-snapshot
An evolutionarily conserved function of the Drosophila insulin receptor and insulin-like peptides in growth control
inferredcites →https://doi.org/10.1016/s0960-9822(03)00506-2openalex-snapshot
Tuberous Sclerosis Complex Gene Products, Tuberin and Hamartin, Control mTOR Signaling by Acting as a GTPase-Activating Protein Complex toward Rheb
inferredcites →https://doi.org/10.1016/s0962-8924(01)02129-8openalex-snapshot
Insulin resistance: a phosphorylation-based uncoupling of insulin signaling
inferredcites →https://doi.org/10.1016/s1535-6108(03)00248-4openalex-snapshot
Targeting the PI3K-Akt pathway in human cancer
inferredcites →https://doi.org/10.1038/35051094openalex-snapshot
The lipid phosphatase SHIP2 controls insulin sensitivity
inferredcites →https://doi.org/10.1038/351033a0openalex-snapshot
Pathway of phosphatidylinositol(3,4,5)-trisphosphate synthesis in activated neutrophils
inferredcites →https://doi.org/10.1038/36621openalex-snapshot
A new pathway for synthesis of phosphatidylinositol-4,5-bisphosphate
inferredcites →https://doi.org/10.1038/embor.2011.257openalex-snapshot
Regulation of TOR by small GTPases
inferredcites →https://doi.org/10.1038/nature02866openalex-snapshot
Absence of S6K1 protects against age- and diet-induced obesity while enhancing insulin sensitivity
inferredcites →https://doi.org/10.1038/ncb999openalex-snapshot
Rheb is a direct target of the tuberous sclerosis tumour suppressor proteins
inferredcites →https://doi.org/10.1038/nmeth.1564openalex-snapshot
Quantification of PtdInsP3 molecular species in cells and tissues by mass spectrometry
inferredcites →https://doi.org/10.1042/bcj20160069openalex-snapshot
Control of diverse subcellular processes by a single multi-functional lipid phosphatidylinositol 4,5-bisphosphate [PI(4,5)P2]
inferredcites →https://doi.org/10.1042/bj20021848openalex-snapshot
Interfacial kinetic analysis of the tumour suppressor phosphatase, PTEN: evidence for activation by anionic phospholipids
inferredcites →https://doi.org/10.1042/bj20030577openalex-snapshot
Insulin-induced Drosophila S6 kinase activation requires phosphoinositide 3-kinase and protein kinase B
inferredcites →https://doi.org/10.1042/bj20100129openalex-snapshot
A novel HPLC-based approach makes possible the spatial characterization of cellular PtdIns5 P and other phosphoinositides
inferredcites →https://doi.org/10.1042/bst0340647openalex-snapshot
Signalling through Class I PI3Ks in mammalian cells
inferredcites →https://doi.org/10.1073/pnas.011318098openalex-snapshot
PDK1 regulates growth through Akt and S6K in Drosophila
inferredcites →https://doi.org/10.1073/pnas.0706517104openalex-snapshot
Identification of IRS-1 Ser-1101 as a target of S6K1 in nutrient- and obesity-induced insulin resistance
inferredcites →https://doi.org/10.1073/pnas.1219333110openalex-snapshot
Phosphatidylinositol 5-phosphate 4-kinase (PIP4K) regulates TOR signaling and cell growth during Drosophila development
inferredcites →https://doi.org/10.1073/pnas.1522478113openalex-snapshot
In B cells, phosphatidylinositol 5-phosphate 4-kinase–α synthesizes PI(4,5)P 2 to impact mTORC2 and Akt signaling
inferredcites →https://doi.org/10.1073/pnas.1600934113openalex-snapshot
Deletion of the gene Pip4k2c , a novel phosphatidylinositol kinase, results in hyperactivation of the immune system
inferredcites →https://doi.org/10.1073/pnas.1734038100openalex-snapshot
The phosphatidylinositol (PI)-5-phosphate 4-kinase type II enzyme controls insulin signaling by regulating PI-3,4,5-trisphosphate degradation
inferredcites →https://doi.org/10.1083/jcb.200403069openalex-snapshot
The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins
inferredcites →https://doi.org/10.1096/fj.12-218842openalex-snapshot
PtdIns5 P is an oxidative stress‐induced second messenger that regulates PKB activation
inferredcites →https://doi.org/10.1101/gad.13.11.1438openalex-snapshot
A PDK1 homolog is necessary and sufficient to transduce AGE-1 PI3 kinase signals that regulate diapause in Caenorhabditis elegans
inferredcites →https://doi.org/10.1126/scisignal.2005191openalex-snapshot
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
inferredcites →https://doi.org/10.1128/mcb.24.11.5080-5087.2004openalex-snapshot
Increased Insulin Sensitivity and Reduced Adiposity in Phosphatidylinositol 5-Phosphate 4-Kinase β−/− Mice
inferredcites →https://doi.org/10.1128/mcb.26.1.63-76.2006openalex-snapshot
Nutrients Suppress Phosphatidylinositol 3-Kinase/Akt Signaling via Raptor-Dependent mTOR-Mediated Insulin Receptor Substrate 1 Phosphorylation
inferredcites →https://doi.org/10.1152/ajpendo.00296.2003openalex-snapshot
Insulin/IGF-I-signaling pathway: an evolutionarily conserved mechanism of longevity from yeast to humans
inferredcites →https://doi.org/10.1210/me.14.6.783openalex-snapshot
A Rapamycin-Sensitive Pathway Down-Regulates Insulin Signaling via Phosphorylation and Proteasomal Degradation of Insulin Receptor Substrate-1
inferredcites →https://doi.org/10.1242/dmm.007948openalex-snapshot
A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila
inferredcites →https://doi.org/10.1371/journal.pbio.0030289openalex-snapshot
The Temporal Requirements for Insulin Signaling During Development in Drosophila
inferredcites →https://doi.org/10.1371/journal.pgen.1000901openalex-snapshot
Distinct Roles of Hand2 in Initiating Polarity and Posterior Shh Expression during the Onset of Mouse Limb Bud Development
inferredcites →https://doi.org/10.1371/journal.pone.0035495openalex-snapshot
Perceptions and Experiences of Research Participants on Gender-Based Violence Community Based Survey: Implications for Ethical Guidelines
inferredcites →https://doi.org/10.2337/diabetes.53.7.1900openalex-snapshot
Polymorphisms in Type II SH2 Domain–Containing Inositol 5-Phosphatase (INPPL1, SHIP2) Are Associated With Physiological Abnormalities of the Metabolic Syndrome
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