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Modeling epistasis in mice and yeast using the proportion of two or more distinct genetic backgrounds: evidence for “polygenic epistasis”

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1 of the 47 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1038/nature13005 (notice 2021-08-11).

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Using Heterogeneous Stocks for Fine-Mapping Genetically Complex Traits

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Hybrid mouse diversity panel: a panel of inbred mouse strains suitable for analysis of complex genetic traits

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Resistance to Noise-Induced Hearing Loss in 129S6 and MOLF Mice: Identification of Independent, Overlapping, and Interacting Chromosomal Regions

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The Alternative Heart: Impact of Alternative Splicing in Heart Disease

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Key molecules in lymphatic development, function, and identification

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Are Interactions between cis-Regulatory Variants Evidence for Biological Epistasis or Statistical Artifacts?

inferredcites →https://doi.org/10.1016/j.cels.2016.10.016openalex-snapshot

Systems Genetics Approach Identifies Gene Pathways and Adamts2 as Drivers of Isoproterenol-Induced Cardiac Hypertrophy and Cardiomyopathy in Mice

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Genetic Architecture of Insulin Resistance in the Mouse

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Emerging insights into the role of matrix metalloproteases as therapeutic targets in fibrosis

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Epistasis in sporadic Alzheimer's disease

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Genetic Background Limits Generalizability of Genotype-Phenotype Relationships

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Decreased RARβ expression induces abundant inflammation and cervical precancerous lesions

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Genomic structure and expression profile of LPHH1, a 7TM gene variably expressed in breast cancer cell lines

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Dissection of epistasis in oligogenic Bardet–Biedl syndrome

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Genetic incompatibilities are widespread within species

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RETRACTED ARTICLE: Detection and replication of epistasis influencing transcription in humans

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Meta-analysis of the heritability of human traits based on fifty years of twin studies

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The Collaborative Cross, a community resource for the genetic analysis of complex traits

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Identification of the molecular basis of doxorubicin-induced cardiotoxicity

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Epistasis dominates the genetic architecture of Drosophila quantitative traits

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The Cysteine-rich Secretory Protein Domain of Tpx-1 Is Related to Ion Channel Toxins and Regulates Ryanodine Receptor Ca2+ Signaling

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Enhancer of Polycomb1, a Novel Homeodomain Only Protein-binding Partner, Induces Skeletal Muscle Differentiation

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GeneAnalytics: An Integrative Gene Set Analysis Tool for Next Generation Sequencing, RNAseq and Microarray Data

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Epigenetic factor EPC1 is a master regulator of DNA damage response by interacting with E2F1 to silence death and activate metastasis-related gene signatures

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The expanded BXD family of mice: A cohort for experimental systems genetics and precision medicine

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A high-resolution association mapping panel for the dissection of complex traits in mice

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Genetic analysis in the Collaborative Cross breeding population

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Genetic dissection of a model complex trait using the Drosophila Synthetic Population Resource

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Contrasting genetic architectures in different mouse reference populations used for studying complex traits

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Determining epistatic selection in admixed populations

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A global genetic interaction network maps a wiring diagram of cellular function

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Interacting genetic loci cause airway hyperresponsiveness

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Fibrosin, a Novel Fibrogenic Protein: Discovery, Cloning and Implications for Fibrotic Disorders

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Mapping Genetic Contributions to Cardiac Pathology Induced by Beta-Adrenergic Stimulation in Mice

inferredcites →https://doi.org/10.1186/gm561openalex-snapshot

Why epistasis is important for tackling complex human disease genetics

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The Hybrid Mouse Diversity Panel: a resource for systems genetics analyses of metabolic and cardiovascular traits

inferredcites →https://doi.org/10.1371/journal.pgen.1000008openalex-snapshot

Data and Theory Point to Mainly Additive Genetic Variance for Complex Traits

inferredcites →https://doi.org/10.1371/journal.pgen.1005805openalex-snapshot

Weak Epistasis Generally Stabilizes Phenotypes in a Mouse Intercross

inferredcites →https://doi.org/10.1371/journal.pgen.1005849openalex-snapshot

Accounting for Population Structure in Gene-by-Environment Interactions in Genome-Wide Association Studies Using Mixed Models

inferredcites →https://doi.org/10.1371/journal.pgen.1006038openalex-snapshot

Genetic Dissection of Cardiac Remodeling in an Isoproterenol-Induced Heart Failure Mouse Model

inferredcites →https://doi.org/10.1534/genetics.107.080101openalex-snapshot

Efficient Control of Population Structure in Model Organism Association Mapping

inferredcites →https://doi.org/10.1534/genetics.114.165282openalex-snapshot

Influence of Gene Interaction on Complex Trait Variation with Multilocus Models

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Genomes of the Mouse Collaborative Cross

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Epistasis: Searching for Interacting Genetic Variants Using Crosses

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Rare variants contribute disproportionately to quantitative trait variation in yeast

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1007/978-1-0716-0947-7_5openalex-snapshot

The Combined Analysis of Pleiotropy and Epistasis (CAPE)

inferred← citeshttps://doi.org/10.1073/pnas.1922305118openalex-snapshot

A model and test for coordinated polygenic epistasis in complex traits

inferred← citeshttps://doi.org/10.1101/2020.02.14.949883openalex-snapshot

Coordinated Interaction: A model and test for globally signed epistasis in complex traits

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