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Synthetic lethality targeting LKB1 mutant and EGFR wild type human non-small cell lung cancer cells by glucose starvation and SGLT2 inhibition
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/ijc.31091openalex-snapshot
Value of a molecular screening program to support clinical trial enrollment in Asian cancer patients: The Integrated Molecular Analysis of Cancer (IMAC) Study
inferredcites →https://doi.org/10.1002/ijc.31193openalex-snapshot
Sodium glucose cotransporter 2 inhibitor canagliflozin attenuates liver cancer cell growth and angiogenic activity by inhibiting glucose uptake
inferredcites →https://doi.org/10.1006/bbrc.2001.5544openalex-snapshot
The Regulation of AMP-Activated Protein Kinase by H2O2
inferredcites →https://doi.org/10.1007/s00280-014-2610-xopenalex-snapshot
Addition of bevacizumab enhances antitumor activity of erlotinib against non-small cell lung cancer xenografts depending on VEGF expression
inferredcites →https://doi.org/10.1007/s00424-014-1619-7openalex-snapshot
Localizations of Na+-d-glucose cotransporters SGLT1 and SGLT2 in human kidney and of SGLT1 in human small intestine, liver, lung, and heart
inferredcites →https://doi.org/10.1007/s00428-014-1566-xopenalex-snapshot
Mutation status of somatic EGFR and KRAS genes in Chinese patients with prostate cancer (PCa)
inferredcites →https://doi.org/10.1007/s12551-015-0186-2openalex-snapshot
Glucose transporters: physiological and pathological roles
inferredcites →https://doi.org/10.1007/s12551-016-0237-3openalex-snapshot
The regulatory role of DNA supercoiling in nucleoprotein complex assembly and genetic activity
inferredcites →https://doi.org/10.1016/j.bbcan.2012.12.004openalex-snapshot
Expression of glucose transporters in cancers
inferredcites →https://doi.org/10.1016/j.canlet.2014.01.018openalex-snapshot
The AMP-activated protein kinase (AMPK) and cancer: Many faces of a metabolic regulator
inferredcites →https://doi.org/10.1016/j.ccell.2015.04.001openalex-snapshot
LKB1 Inactivation Elicits a Redox Imbalance to Modulate Non-small Cell Lung Cancer Plasticity and Therapeutic Response
inferredcites →https://doi.org/10.1016/j.ccell.2017.01.003openalex-snapshot
A UBE2O-AMPKα2 Axis that Promotes Tumor Initiation and Progression Offers Opportunities for Therapy
inferredcites →https://doi.org/10.1016/j.ccr.2012.12.008openalex-snapshot
LKB1 Inactivation Dictates Therapeutic Response of Non-Small Cell Lung Cancer to the Metabolism Drug Phenformin
inferredcites →https://doi.org/10.1016/j.cell.2012.08.029openalex-snapshot
Mapping the Hallmarks of Lung Adenocarcinoma with Massively Parallel Sequencing
inferredcites →https://doi.org/10.1016/j.cell.2012.12.016openalex-snapshot
Differential Regulation of Distinct Vps34 Complexes by AMPK in Nutrient Stress and Autophagy
inferredcites →https://doi.org/10.1016/j.cell.2015.01.034openalex-snapshot
Degradation of AMPK by a Cancer-Specific Ubiquitin Ligase
inferredcites →https://doi.org/10.1016/j.celrep.2017.09.026openalex-snapshot
AMPK Maintains Cellular Metabolic Homeostasis through Regulation of Mitochondrial Reactive Oxygen Species
inferredcites →https://doi.org/10.1016/j.cmet.2013.12.013openalex-snapshot
A Redox-Dependent Mechanism for Regulation of AMPK Activation by Thioredoxin1 during Energy Starvation
inferredcites →https://doi.org/10.1016/j.cmet.2014.06.014openalex-snapshot
The Lysosomal v-ATPase-Ragulator Complex Is a Common Activator for AMPK and mTORC1, Acting as a Switch between Catabolism and Anabolism
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2009.02.019openalex-snapshot
Hypoxic activation of AMPK is dependent on mitochondrial ROS but independent of an increase in AMP/ATP ratio
inferredcites →https://doi.org/10.1016/j.jep.2016.11.004openalex-snapshot
Bufalin inhibits gefitinib resistant NCI-H460 human lung cancer cell migration and invasion in vitro
inferredcites →https://doi.org/10.1016/j.jtcvs.2008.11.029openalex-snapshot
Expression of LKB1 tumor suppressor in non–small cell lung cancer determines sensitivity to 2-deoxyglucose
inferredcites →https://doi.org/10.1016/j.lungcan.2009.11.020openalex-snapshot
KRAS mutations and resistance to EGFR-TKIs treatment in patients with non-small cell lung cancer: A meta-analysis of 22 studies
inferredcites →https://doi.org/10.1016/j.lungcan.2016.05.021openalex-snapshot
Precision medicine in age-specific non-small-cell-lung-cancer patients: Integrating biomolecular results into clinical practice—A new approach to improve personalized translational research
inferredcites →https://doi.org/10.1016/j.molcel.2008.03.003openalex-snapshot
AMPK Phosphorylation of Raptor Mediates a Metabolic Checkpoint
inferredcites →https://doi.org/10.1016/j.molcel.2018.01.001openalex-snapshot
Nuclear Receptor Nur77 Facilitates Melanoma Cell Survival under Metabolic Stress by Protecting Fatty Acid Oxidation
inferredcites →https://doi.org/10.1016/j.molmet.2016.08.014openalex-snapshot
The diabetes medication Canagliflozin reduces cancer cell proliferation by inhibiting mitochondrial complex-I supported respiration
inferredcites →https://doi.org/10.1016/j.redox.2019.101154openalex-snapshot
Critical role of AMPK in redox regulation under glucose starvation
inferredcites →https://doi.org/10.1016/s0092-8674(03)00929-2openalex-snapshot
TSC2 Mediates Cellular Energy Response to Control Cell Growth and Survival
inferredcites →https://doi.org/10.1038/34432openalex-snapshot
A serine/threonine kinase gene defective in Peutz–Jeghers syndrome
inferredcites →https://doi.org/10.1038/bjc.2014.210openalex-snapshot
Coexistence of EGFR with KRAS, or BRAF, or PIK3CA somatic mutations in lung cancer: a comprehensive mutation profiling from 5125 Chinese cohorts
inferredcites →https://doi.org/10.1038/bjc.2014.401openalex-snapshot
Coexistence of KRAS mutation with mutant but not wild-type EGFR predicts response to tyrosine-kinase inhibitors in human lung cancer
inferredcites →https://doi.org/10.1038/bjc.2015.261openalex-snapshot
Targeting LKB1 in cancer – exposing and exploiting vulnerabilities
inferredcites →https://doi.org/10.1038/cdd.2009.49openalex-snapshot
Superoxide is the major reactive oxygen species regulating autophagy
inferredcites →https://doi.org/10.1038/cddis.2013.312openalex-snapshot
Atorvastatin overcomes gefitinib resistance in KRAS mutant human non-small cell lung carcinoma cells
inferredcites →https://doi.org/10.1038/emboj.2008.92openalex-snapshot
Downregulation of AMP‐activated protein kinase by Cidea‐mediated ubiquitination and degradation in brown adipose tissue
inferredcites →https://doi.org/10.1038/msb.2012.20openalex-snapshot
Glucose deprivation activates a metabolic and signaling amplification loop leading to cell death
inferredcites →https://doi.org/10.1038/nature01045openalex-snapshot
Loss of the Lkb1 tumour suppressor provokes intestinal polyposis but resistance to transformation
inferredcites →https://doi.org/10.1038/nature06030openalex-snapshot
LKB1 modulates lung cancer differentiation and metastasis
inferredcites →https://doi.org/10.1038/nature07423openalex-snapshot
Somatic mutations affect key pathways in lung adenocarcinoma
inferredcites →https://doi.org/10.1038/nature11066openalex-snapshot
AMPK regulates NADPH homeostasis to promote tumour cell survival during energy stress
inferredcites →https://doi.org/10.1038/nature11404openalex-snapshot
Comprehensive genomic characterization of squamous cell lung cancers
inferredcites →https://doi.org/10.1038/nature13385openalex-snapshot
Comprehensive molecular profiling of lung adenocarcinoma
inferredcites →https://doi.org/10.1038/nature23275openalex-snapshot
Fructose-1,6-bisphosphate and aldolase mediate glucose sensing by AMPK
inferredcites →https://doi.org/10.1038/nature25183openalex-snapshot
The biology and management of non-small cell lung cancer
inferredcites →https://doi.org/10.1038/ncb2152openalex-snapshot
AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1
inferredcites →https://doi.org/10.1038/nchembio.1069openalex-snapshot
The orphan nuclear receptor Nur77 regulates LKB1 localization and activates AMPK
inferredcites →https://doi.org/10.1038/ng.2330openalex-snapshot
Activation of the AXL kinase causes resistance to EGFR-targeted therapy in lung cancer
inferredcites →https://doi.org/10.1038/ng.3564openalex-snapshot
Distinct patterns of somatic genome alterations in lung adenocarcinomas and squamous cell carcinomas
inferredcites →https://doi.org/10.1038/nprot.2013.143openalex-snapshot
Genome engineering using the CRISPR-Cas9 system
inferredcites →https://doi.org/10.1038/nrc2088openalex-snapshot
Epidermal growth factor receptor mutations in lung cancer
inferredcites →https://doi.org/10.1038/nutd.2014.40openalex-snapshot
Efficacy, safety and regulatory status of SGLT2 inhibitors: focus on canagliflozin
inferredcites →https://doi.org/10.1038/onc.2011.98openalex-snapshot
Frequent homozygous deletion of the LKB1/STK11 gene in non-small cell lung cancer
inferredcites →https://doi.org/10.1038/onc.2015.140openalex-snapshot
LKB1 deficiency enhances sensitivity to energetic stress induced by erlotinib treatment in non-small-cell lung cancer (NSCLC) cells
inferredcites →https://doi.org/10.1038/onc.2015.375openalex-snapshot
Differential association of STK11 and TP53 with KRAS mutation-associated gene expression, proliferation and immune surveillance in lung adenocarcinoma
inferredcites →https://doi.org/10.1038/s41388-018-0145-zopenalex-snapshot
Controlling the master—upstream regulation of the tumor suppressor LKB1
inferredcites →https://doi.org/10.1038/s41419-018-0273-yopenalex-snapshot
Canagliflozin mediated dual inhibition of mitochondrial glutamate dehydrogenase and complex I: an off-target adverse effect
inferredcites →https://doi.org/10.1038/sj.bjc.6600547openalex-snapshot
Evaluation of 2-deoxy-D-glucose as a chemotherapeutic agent: mechanism of cell death
inferredcites →https://doi.org/10.1038/sj.bjc.6604515openalex-snapshot
Role of LKB1 in lung cancer development
inferredcites →https://doi.org/10.1038/sj.emboj.7600667openalex-snapshot
Deficiency of LKB1 in skeletal muscle prevents AMPK activation and glucose uptake during contraction
inferredcites →https://doi.org/10.1038/sj.onc.1210418openalex-snapshot
Prevalence and specificity of LKB1 genetic alterations in lung cancers
inferredcites →https://doi.org/10.1042/bj20081386openalex-snapshot
How mitochondria produce reactive oxygen species
inferredcites →https://doi.org/10.1042/cs20120239openalex-snapshot
Mitochondrial reactive oxygen species enhance AMP-activated protein kinase activation in the endothelium of patients with coronary artery disease and diabetes
inferredcites →https://doi.org/10.1053/j.gastro.2010.04.055openalex-snapshot
LKB1 Haploinsufficiency Cooperates With Kras to Promote Pancreatic Cancer Through Suppression of p21-Dependent Growth Arrest
inferredcites →https://doi.org/10.1056/nejmoa044238openalex-snapshot
EGFR Mutation and Resistance of Non–Small-Cell Lung Cancer to Gefitinib
inferredcites →https://doi.org/10.1056/nejmoa050736openalex-snapshot
Erlotinib in Lung Cancer — Molecular and Clinical Predictors of Outcome
inferredcites →https://doi.org/10.1073/pnas.0308061100openalex-snapshot
The tumor suppressor LKB1 kinase directly activates AMP-activated kinase and regulates apoptosis in response to energy stress
inferredcites →https://doi.org/10.1073/pnas.1312570111openalex-snapshot
Loss of the tumor suppressor LKB1 promotes metabolic reprogramming of cancer cells via HIF-1α
inferredcites →https://doi.org/10.1073/pnas.1511698112openalex-snapshot
Functional expression of sodium-glucose transporters in cancer
inferredcites →https://doi.org/10.1074/jbc.m110.143685openalex-snapshot
Exposure to Hydrogen Peroxide Induces Oxidation and Activation of AMP-activated Protein Kinase*
inferredcites →https://doi.org/10.1074/jbc.m509723200openalex-snapshot
Reactive Lipid Species from Cyclooxygenase-2 Inactivate Tumor Suppressor LKB1/STK11
inferredcites →https://doi.org/10.1074/jbc.ra118.002579openalex-snapshot
Mitochondria-derived ROS activate AMP-activated protein kinase (AMPK) indirectly
inferredcites →https://doi.org/10.1089/ars.2010.3284openalex-snapshot
High-Glucose Stimulation Increases Reactive Oxygen Species Production Through the Calcium and Mitogen-Activated Protein Kinase-Mediated Activation of Mitochondrial Fission
inferredcites →https://doi.org/10.1093/jnci/dji055openalex-snapshot
Clinical and Biological Features Associated With Epidermal Growth Factor Receptor Gene Mutations in Lung Cancers
inferredcites →https://doi.org/10.1093/nar/18.12.3587openalex-snapshot
Advanced mammalian gene transfer: high titre retroviral vectors with multiple drug selection markers and a complementary helper-free packaging cell line
inferredcites →https://doi.org/10.1097/jto.0b013e318262de4aopenalex-snapshot
A Phase I Study of Erlotinib and Hydroxychloroquine in Advanced Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1111/j.1349-7006.2001.tb01175.xopenalex-snapshot
SGLT Gene Expression in Primary Lung Cancers and Their Metastatic Lesions
inferredcites →https://doi.org/10.1111/j.1432-1033.1988.tb14201.xopenalex-snapshot
Identification by amino acid sequencing of three major regulatory phosphorylation sites on rat acetyl‐CoA carboxylase
inferredcites →https://doi.org/10.1111/j.1582-4934.2003.tb00202.xopenalex-snapshot
Glucose deprivation induces mitochondrial dysfunction and oxidative stress in PC12 cell line
inferredcites →https://doi.org/10.1126/science.1120781openalex-snapshot
The Kinase LKB1 Mediates Glucose Homeostasis in Liver and Therapeutic Effects of Metformin
inferredcites →https://doi.org/10.1126/science.1174229openalex-snapshot
Glucose Deprivation Contributes to the Development of KRAS Pathway Mutations in Tumor Cells
inferredcites →https://doi.org/10.1126/science.1196371openalex-snapshot
Phosphorylation of ULK1 (hATG1) by AMP-Activated Protein Kinase Connects Energy Sensing to Mitophagy
inferredcites →https://doi.org/10.1126/science.1211485openalex-snapshot
Inhibition of Pyruvate Kinase M2 by Reactive Oxygen Species Contributes to Cellular Antioxidant Responses
inferredcites →https://doi.org/10.1126/science.aaa1348openalex-snapshot
Mutational landscape determines sensitivity to PD-1 blockade in non–small cell lung cancer
inferredcites →https://doi.org/10.1126/scisignal.2004088openalex-snapshot
Integrative Analysis of Complex Cancer Genomics and Clinical Profiles Using the cBioPortal
inferredcites →https://doi.org/10.1126/scitranslmed.3002394openalex-snapshot
Targeting GLUT1 and the Warburg Effect in Renal Cell Carcinoma by Chemical Synthetic Lethality
inferredcites →https://doi.org/10.1126/scitranslmed.aat5933openalex-snapshot
Sodium-glucose transporter 2 is a diagnostic and therapeutic target for early-stage lung adenocarcinoma
inferredcites →https://doi.org/10.1128/mcb.01621-07openalex-snapshot
Pancreatic Lkb1 Deletion Leads to Acinar Polarity Defects and Cystic Neoplasms
inferredcites →https://doi.org/10.1128/mcb.05124-11openalex-snapshot
Hypoxia Triggers AMPK Activation through Reactive Oxygen Species-Mediated Activation of Calcium Release-Activated Calcium Channels
inferredcites →https://doi.org/10.1152/ajpcell.90654.2007openalex-snapshot
Regulation of H2O2-induced necrosis by PKC and AMP-activated kinase signaling in primary cultured hepatocytes
inferredcites →https://doi.org/10.1152/ajprenal.2001.280.1.f10openalex-snapshot
Renal Na + -glucose cotransporters
inferredcites →https://doi.org/10.1158/0008-5472.can-04-2818openalex-snapshot
Mutations of the Epidermal Growth Factor Receptor Gene in Lung Cancer
inferredcites →https://doi.org/10.1158/0008-5472.can-07-1336openalex-snapshot
Targeting Endoplasmic Reticulum Stress and Akt with OSU-03012 and Gefitinib or Erlotinib to Overcome Resistance to Epidermal Growth Factor Receptor Inhibitors
inferredcites →https://doi.org/10.1158/0008-5472.can-08-4055openalex-snapshot
PTEN Loss Contributes to Erlotinib Resistance in EGFR-Mutant Lung Cancer by Activation of Akt and EGFR
inferredcites →https://doi.org/10.1158/0008-5472.can-14-3304openalex-snapshot
Erlotinib Pretreatment Improves Photodynamic Therapy of Non–Small Cell Lung Carcinoma Xenografts via Multiple Mechanisms
inferredcites →https://doi.org/10.1158/1078-0432.ccr-04-2642openalex-snapshot
Combination Treatment with Erlotinib and Pertuzumab against Human Tumor Xenografts Is Superior to Monotherapy
inferredcites →https://doi.org/10.1158/1078-0432.ccr-05-1981openalex-snapshot
Distinct Epidermal Growth Factor Receptor and KRAS Mutation Patterns in Non–Small Cell Lung Cancer Patients with Different Tobacco Exposure and Clinicopathologic Features
inferredcites →https://doi.org/10.1158/1078-0432.ccr-14-3281openalex-snapshot
Phase II Study of the AKT Inhibitor MK-2206 plus Erlotinib in Patients with Advanced Non–Small Cell Lung Cancer Who Previously Progressed on Erlotinib
inferredcites →https://doi.org/10.1158/1535-7163.mct-11-0497openalex-snapshot
Dual Inhibition of Tumor Energy Pathway by 2-Deoxyglucose and Metformin Is Effective against a Broad Spectrum of Preclinical Cancer Models
inferredcites →https://doi.org/10.1158/1535-7163.mct-12-0131openalex-snapshot
A Small-Molecule Inhibitor of Glucose Transporter 1 Downregulates Glycolysis, Induces Cell-Cycle Arrest, and Inhibits Cancer Cell Growth In Vitro and In Vivo
inferredcites →https://doi.org/10.1158/2159-8290.cd-12-0095openalex-snapshot
The cBio Cancer Genomics Portal: An Open Platform for Exploring Multidimensional Cancer Genomics Data
inferredcites →https://doi.org/10.1158/2159-8290.cd-14-1236openalex-snapshot
Co-occurring Genomic Alterations Define Major Subsets of KRAS -Mutant Lung Adenocarcinoma with Distinct Biology, Immune Profiles, and Therapeutic Vulnerabilities
inferredcites →https://doi.org/10.1158/2159-8290.cd-16-1337openalex-snapshot
Prospective Comprehensive Molecular Characterization of Lung Adenocarcinomas for Efficient Patient Matching to Approved and Emerging Therapies
inferredcites →https://doi.org/10.1161/circulationaha.108.787440openalex-snapshot
Resveratrol Prevents the Prohypertrophic Effects of Oxidative Stress on LKB1
inferredcites →https://doi.org/10.1186/1475-4924-2-28openalex-snapshot
Complexes between the LKB1 tumor suppressor, STRADα/β and MO25α/β are upstream kinases in the AMP-activated protein kinase cascade
inferredcites →https://doi.org/10.1186/1741-7007-11-36openalex-snapshot
LKB1 and AMPK and the cancer-metabolism link - ten years after
inferredcites →https://doi.org/10.1200/jco.2005.01.388openalex-snapshot
Predictive and Prognostic Impact of Epidermal Growth Factor Receptor Mutation in Non–Small-Cell Lung Cancer Patients Treated With Gefitinib
inferredcites →https://doi.org/10.1200/jco.2005.09.985openalex-snapshot
Epidermal Growth Factor Receptor Mutations in Non–Small-Cell Lung Cancer: Implications for Treatment and Tumor Biology
inferredcites →https://doi.org/10.1200/jco.2011.29.15_suppl.cra7506openalex-snapshot
Identification of driver mutations in tumor specimens from 1,000 patients with lung adenocarcinoma: The NCI’s Lung Cancer Mutation Consortium (LCMC).
inferredcites →https://doi.org/10.1200/jco.2011.29.18_suppl.cra7506openalex-snapshot
Identification of driver mutations in tumor specimens from 1,000 patients with lung adenocarcinoma: The NCI’s Lung Cancer Mutation Consortium (LCMC).
inferredcites →https://doi.org/10.1200/jco.2017.75.3384openalex-snapshot
Molecular Determinants of Response to Anti–Programmed Cell Death (PD)-1 and Anti–Programmed Death-Ligand 1 (PD-L1) Blockade in Patients With Non–Small-Cell Lung Cancer Profiled With Targeted Next-Generation Sequencing
inferredcites →https://doi.org/10.1242/dmm.004440openalex-snapshot
Lkb1inactivation is sufficient to drive endometrial cancers that are aggressive yet highly responsive to mTOR inhibitor monotherapy
inferredcites →https://doi.org/10.1371/journal.pmed.0020017openalex-snapshot
KRAS Mutations and Primary Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib
inferredcites →https://doi.org/10.1371/journal.pmed.0020073openalex-snapshot
Acquired Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib Is Associated with a Second Mutation in the EGFR Kinase Domain
inferredcites →https://doi.org/10.1371/journal.pone.0005137openalex-snapshot
Somatic LKB1 Mutations Promote Cervical Cancer Progression
inferredcites →https://doi.org/10.1507/endocrj.ej15-0396openalex-snapshot
Effect of dapagliflozin on colon cancer cell [Rapid Communication]
inferredcites →https://doi.org/10.1681/asn.2006101124openalex-snapshot
Reactive Oxygen Species in the Presence of High Glucose Alter Ureteric Bud Morphogenesis
inferredcites →https://doi.org/10.18632/oncotarget.2892openalex-snapshot
Targeting the facilitative glucose transporter GLUT1 inhibits the self-renewal and tumor-initiating capacity of cancer stem cells
inferredcites →https://doi.org/10.2337/db16-0058openalex-snapshot
The Na+/Glucose Cotransporter Inhibitor Canagliflozin Activates AMPK by Inhibiting Mitochondrial Function and Increasing Cellular AMP Levels
inferredcites →https://doi.org/10.2337/diacare.23.10.1563openalex-snapshot
Screening for type 2 diabetes.
inferredcites →https://doi.org/10.7314/apjcp.2015.16.13.5471openalex-snapshot
High Efficiency Apoptosis Induction in Breast Cancer Cell Lines by MLN4924/2DG Co-Treatment
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1007/s11154-021-09675-9openalex-snapshot
Repurposing sodium-glucose co-transporter 2 inhibitors (SGLT2i) for cancer treatment – A Review
inferred← citeshttps://doi.org/10.1016/j.diabres.2025.112925openalex-snapshot
SGLT-2 inhibitors beyond diabetes: a new frontier in cancer treatment
inferred← citeshttps://doi.org/10.3389/fonc.2026.1759702openalex-snapshot
The impact of SGLT2i on the outcome of advanced lung cancer in patients with diabetes
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