On the public citation graph
Senolytic treatment targets aberrant p21-expression to restore liver regeneration in adult mice
0 signed · 45 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/hep.26412openalex-snapshot
The Degree of Liver Injury Determines the Role of P21 in Liver Regeneration And Hepatocarcinogenesis in Mice
inferredcites →https://doi.org/10.1002/hep.27094openalex-snapshot
Reversal of Hepatocyte Senescence After Continuous In Vivo Cell Proliferation
inferredcites →https://doi.org/10.1002/hep.510250311openalex-snapshot
Regulation of Cyclin–Dependent Kinase Inhibitor P21Waf1/Cip1/Sdi1 Gene Expression in Hepatic Regeneration
inferredcites →https://doi.org/10.1016/j.cell.2008.06.049openalex-snapshot
Senescence of Activated Stellate Cells Limits Liver Fibrosis
inferredcites →https://doi.org/10.1016/j.cell.2012.12.010openalex-snapshot
Monitoring Tumorigenesis and Senescence In Vivo with a p16INK4a-Luciferase Model
inferredcites →https://doi.org/10.1016/j.cell.2013.10.041openalex-snapshot
Senescence Is a Developmental Mechanism that Contributes to Embryonic Growth and Patterning
inferredcites →https://doi.org/10.1016/j.cell.2017.02.031openalex-snapshot
Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging
inferredcites →https://doi.org/10.1016/j.celrep.2016.12.008openalex-snapshot
Modeling Dynamics and Function of Bone Marrow Cells in Mouse Liver Regeneration
inferredcites →https://doi.org/10.1016/j.celrep.2017.12.092openalex-snapshot
Cellular Senescence Is Induced by the Environmental Neurotoxin Paraquat and Contributes to Neuropathology Linked to Parkinson’s Disease
inferredcites →https://doi.org/10.1016/j.devcel.2014.11.012openalex-snapshot
An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA
inferredcites →https://doi.org/10.1016/j.ydbio.2007.12.025openalex-snapshot
Development and regeneration of the neonatal digit tip in mice
inferredcites →https://doi.org/10.1038/nature10600openalex-snapshot
Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders
inferredcites →https://doi.org/10.1038/nature16932openalex-snapshot
Naturally occurring p16Ink4a-positive cells shorten healthy lifespan
inferredcites →https://doi.org/10.1038/nature22978openalex-snapshot
The extracellular matrix protein agrin promotes heart regeneration in mice
inferredcites →https://doi.org/10.1038/nature23015openalex-snapshot
Cholangiocytes act as facultative liver stem cells during impaired hepatocyte regeneration
inferredcites →https://doi.org/10.1038/ncb3203openalex-snapshot
Hepatic progenitor cells of biliary origin with liver repopulation capacity
inferredcites →https://doi.org/10.1038/ncomms11190openalex-snapshot
Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL
inferredcites →https://doi.org/10.1038/ncomms15691openalex-snapshot
Cellular senescence drives age-dependent hepatic steatosis
inferredcites →https://doi.org/10.1038/nm.4000openalex-snapshot
Cellular senescence in aging and age-related disease: from mechanisms to therapy
inferredcites →https://doi.org/10.1038/nm.4010openalex-snapshot
Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice
inferredcites →https://doi.org/10.1038/nm.4054openalex-snapshot
p16Ink4a-induced senescence of pancreatic beta cells enhances insulin secretion
inferredcites →https://doi.org/10.1038/nprot.2008.80openalex-snapshot
A reproducible and well-tolerated method for 2/3 partial hepatectomy in mice
inferredcites →https://doi.org/10.1038/nrd.2017.116openalex-snapshot
Senescent cells: an emerging target for diseases of ageing
inferredcites →https://doi.org/10.1038/nrm3823openalex-snapshot
Cellular senescence: from physiology to pathology
inferredcites →https://doi.org/10.1038/s41467-018-07825-3openalex-snapshot
Impaired immune surveillance accelerates accumulation of senescent cells and aging
inferredcites →https://doi.org/10.1038/s41586-018-0543-yopenalex-snapshot
Clearance of senescent glial cells prevents tau-dependent pathology and cognitive decline
inferredcites →https://doi.org/10.1038/s41591-018-0092-9openalex-snapshot
Senolytics improve physical function and increase lifespan in old age
inferredcites →https://doi.org/10.1053/j.gastro.2012.08.043openalex-snapshot
Liver Failure After Extended Hepatectomy in Mice Is Mediated by a p21-Dependent Barrier to Liver Regeneration
inferredcites →https://doi.org/10.1073/pnas.122359899openalex-snapshot
Gene expression during the priming phase of liver regeneration after partial hepatectomy in mice
inferredcites →https://doi.org/10.1073/pnas.1818313116openalex-snapshot
Cells exhibiting strong p16 INK4a promoter activation in vivo display features of senescence
inferredcites →https://doi.org/10.1073/pnas.94.4.1441openalex-snapshot
Initiation of liver growth by tumor necrosis factor: Deficient liver regeneration in mice lacking type I tumor necrosis factor receptor
inferredcites →https://doi.org/10.1101/gad.390506openalex-snapshot
c-Jun/AP-1 controls liver regeneration by repressing p53/p21 and p38 MAPK activity
inferredcites →https://doi.org/10.1111/acel.12711openalex-snapshot
Senescence promotes in vivo reprogramming through p16 INK 4a and IL ‐6
inferredcites →https://doi.org/10.1111/acel.12840openalex-snapshot
Tau protein aggregation is associated with cellular senescence in the brain
inferredcites →https://doi.org/10.1111/jgh.12930openalex-snapshot
Dysfunction of liver regeneration in aged liver after partial hepatectomy
inferredcites →https://doi.org/10.1126/science.1200708openalex-snapshot
Transient Regenerative Potential of the Neonatal Mouse Heart
inferredcites →https://doi.org/10.1126/science.aaf4445openalex-snapshot
Tissue damage and senescence provide critical signals for cellular reprogramming in vivo
inferredcites →https://doi.org/10.1126/scitranslmed.aan1230openalex-snapshot
TGFβ inhibition restores a regenerative response in acute liver injury by suppressing paracrine senescence
inferredcites →https://doi.org/10.1172/jci200422475openalex-snapshot
Ink4a/Arf expression is a biomarker of aging
inferredcites →https://doi.org/10.1172/jci22475openalex-snapshot
Ink4a/Arf expression is a biomarker of aging
inferredcites →https://doi.org/10.1387/ijdb.180002jwopenalex-snapshot
An integrative framework for salamander and mouse limb regeneration
inferredcites →https://doi.org/10.15252/embj.201695553openalex-snapshot
p21 maintains senescent cell viability under persistent DNA damage response by restraining JNK and caspase signaling
inferredcites →https://doi.org/10.15252/emmm.201506089openalex-snapshot
Impaired liver regeneration in aged mice can be rescued by silencing Hippo core kinases MST1 and MST2
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.1007/s12015-025-11001-8openalex-snapshot
Liver Regeneration: Polyploidy and Cellular Senescence as Potential Regulators
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.