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Presynaptic depression maintains stable synaptic strength in developmentally arrested Drosophila larvae
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Outgoing claims: the papers this work cites, builds on, or argues with.
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inferredcites →https://doi.org/10.1371/journal.pone.0005072openalex-snapshot
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inferredcites →https://doi.org/10.1523/jneurosci.19-08-03023.1999openalex-snapshot
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inferredcites →https://doi.org/10.1523/jneurosci.2024-11.2011openalex-snapshot
Stathmin is Required for Stability of the Drosophila Neuromuscular Junction
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inferredcites →https://doi.org/10.1523/jneurosci.3001-04.2004openalex-snapshot
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inferredcites →https://doi.org/10.1523/jneurosci.3068-18.2019openalex-snapshot
Endogenous tagging reveals differential regulation of Ca 2+ channels at single AZs during presynaptic homeostatic potentiation and depression
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Four Different Subunits Are Essential for Expressing the Synaptic Glutamate Receptor at Neuromuscular Junctions ofDrosophila
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Drosophila as an In Vivo Model for Human Neurodegenerative Disease
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Advances in Engineering the Fly Genome with the CRISPR-Cas System
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Extended Synaptotagmin Localizes to Presynaptic ER and Promotes Neurotransmission and Synaptic Growth in Drosophila
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Emerging links between homeostatic synaptic plasticity and neurological disease
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E-I balance and human diseases – from molecules to networking
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Measurement of Lifespan in Drosophila melanogaster
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Homeostatic synaptic depression is achieved through a regulated decrease in presynaptic calcium channel abundance
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Synapse-specific and compartmentalized expression of presynaptic homeostatic potentiation
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Characterization of developmental and molecular factors underlying release heterogeneity at Drosophila synapses
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