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Frequent extrachromosomal oncogene amplification drives aggressive tumors

https://doi.org/10.1101/859306
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Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00401-018-1912-1openalex-snapshot
Structure and evolution of double minutes in diagnosis and relapse brain tumors
inferredcites →https://doi.org/10.1016/0092-8674(83)90169-1openalex-snapshot
Transposition and amplification of oncogene-related sequences in human neuroblastomas
inferredcites →https://doi.org/10.1016/0168-9525(91)90103-wopenalex-snapshot
Fluorescence in situ hybridization: applications in cytogenetics and gene mapping
inferredcites →https://doi.org/10.1016/j.ccell.2018.03.007openalex-snapshot
Genomic and Functional Approaches to Understanding Cancer Aneuploidy
inferredcites →https://doi.org/10.1016/j.celrep.2012.12.008openalex-snapshot
Deciphering Signatures of Mutational Processes Operative in Human Cancer
inferredcites →https://doi.org/10.1016/s0021-9258(17)34875-5openalex-snapshot
Selective multiplication of dihydrofolate reductase genes in methotrexate-resistant variants of cultured murine cells.
inferredcites →https://doi.org/10.1038/nature08822openalex-snapshot
The landscape of somatic copy-number alteration across human cancers
inferredcites →https://doi.org/10.1038/nature21356openalex-snapshot
Extrachromosomal oncogene amplification drives tumour evolution and genetic heterogeneity
inferredcites →https://doi.org/10.1038/ng.2760openalex-snapshot
Pan-cancer patterns of somatic copy number alteration
inferredcites →https://doi.org/10.1038/ng.3722openalex-snapshot
Pan-cancer analysis of somatic copy-number alterations implicates IRS4 and IGF2 in enhancer hijacking
inferredcites →https://doi.org/10.1038/nrc2091openalex-snapshot
The impact of translocations and gene fusions on cancer causation
inferredcites →https://doi.org/10.1038/s41467-018-03369-8openalex-snapshot
Circular DNA elements of chromosomal origin are common in healthy human somatic tissue
inferredcites →https://doi.org/10.1038/s41467-018-08200-yopenalex-snapshot
Exploring the landscape of focal amplifications in cancer using AmpliconArchitect
inferredcites →https://doi.org/10.1038/s41568-019-0128-6openalex-snapshot
Extrachromosomal oncogene amplification in tumour pathogenesis and evolution
inferredcites →https://doi.org/10.1038/s41588-018-0105-0openalex-snapshot
Discordant inheritance of chromosomal and extrachromosomal DNA elements contributes to dynamic disease evolution in glioblastoma
inferredcites →https://doi.org/10.1038/s41588-018-0165-1openalex-snapshot
Genome doubling shapes the evolution and prognosis of advanced cancers
inferredcites →https://doi.org/10.1093/nar/gkx1018openalex-snapshot
TumorFusions: an integrative resource for cancer-associated transcript fusions
inferredcites →https://doi.org/10.1101/237313openalex-snapshot
Discovery and characterization of coding and non-coding driver mutations in more than 2,500 whole cancer genomes
inferredcites →https://doi.org/10.1101/gr.221028.117openalex-snapshot
SvABA: genome-wide detection of structural variants and indels by local assembly
inferredcites →https://doi.org/10.1126/science.1213307openalex-snapshot
Extrachromosomal MicroDNAs and Chromosomal Microdeletions in Normal Tissues
inferredcites →https://doi.org/10.1126/science.aaf8399openalex-snapshot
Tumor aneuploidy correlates with markers of immune evasion and with reduced response to immunotherapy
inferredcites →https://doi.org/10.1126/science.aav1898openalex-snapshot
The chromatin accessibility landscape of primary human cancers
inferredcites →https://doi.org/10.1146/annurev-cellbio-092910-154234openalex-snapshot
The Causes and Consequences of Polyploidy in Normal Development and Cancer
inferredcites →https://doi.org/10.1158/1541-7786.mcr-17-0095openalex-snapshot
Normal and Cancerous Tissues Release Extrachromosomal Circular DNA (eccDNA) into the Circulation

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.1016/j.annonc.2020.03.303openalex-snapshot
Extrachromosomal DNA—relieving heredity constraints, accelerating tumour evolution
inferred← citeshttps://doi.org/10.1038/s42003-022-03238-7openalex-snapshot
Rearrangement processes and structural variations show evidence of selection in oesophageal adenocarcinomas
inferred← citeshttps://doi.org/10.1101/2019.12.15.876714openalex-snapshot
Stochastic dynamics of extra-chromosomal DNA
inferred← citeshttps://doi.org/10.1101/2020.10.10.334516openalex-snapshot
Aberrant replication licensing drives Copy Number Gains across species
inferred← citeshttps://doi.org/10.7554/elife.80207openalex-snapshot
Oncogene expression from extrachromosomal DNA is driven by copy number amplification and does not require spatial clustering in glioblastoma stem cells
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