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Anti‐acne vulgaris effects of chlorogenic acid by anti‐inflammatory activity and lipogenesis inhibition

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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In vitro modeling of unsaturated free fatty acid-mediated tissue impairments seen in acne lesions

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Chlorogenic acid (CGA): A pharmacological review and call for further research

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mTORC2 Signaling Selectively Regulates the Generation and Function of Tissue-Resident Peritoneal Macrophages

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Akt Stimulates Hepatic SREBP1c and Lipogenesis through Parallel mTORC1-Dependent and Independent Pathways

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Propionibacterium acnes and lipopolysaccharide induce the expression of antimicrobial peptides and proinflammatory cytokines/chemokines in human sebocytes

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Acne vulgaris

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Activity-Guided Purification Identifies Lupeol, a Pentacyclic Triterpene, As a Therapeutic Agent Multiple Pathogenic Factors of Acne

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Contribution of GATA6 to homeostasis of the human upper pilosebaceous unit and acne pathogenesis

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Inhibitory effects of superoxide dismutase 3 on Propionibacterium acnes-induced skin inflammation

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IGF-1 Induces SREBP-1 Expression and Lipogenesis in SEB-1 Sebocytes via Activation of the Phosphoinositide 3-Kinase/Akt Pathway

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Chlorogenic acid methyl ester exerts strong anti-inflammatory effects via inhibiting the COX-2/NLRP3/NF-κB pathway

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Gene regulation of mammalian desaturases

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Proinflammatory cytokine production by human keratinocytes stimulated with Propionibacterium acnes and P. acnes GroEL

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FoxO1 Inhibits Sterol Regulatory Element-binding Protein-1c (SREBP-1c) Gene Expression via Transcription Factors Sp1 and SREBP-1c

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Chlorogenic Acid Extends the Lifespan ofCaenorhabditis elegansvia Insulin/IGF-1 Signaling Pathway

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Potential role of F ox O 1 and m TORC 1 in the pathogenesis of W estern diet‐induced acne

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Mechanistic target of rapamycin (mTOR) expression is increased in acne patients' skin

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Loss of Gata6 causes dilation of the hair follicle canal and sebaceous duct

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High bacterial colonization and lipase activity in microcomedones

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What is the pathogenesis of acne?

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Chlorogenic acid: A potent molecule that protects cardiomyocytes from TNF‐α–induced injury via inhibiting NF‐κB and JNK signals

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Acne is an inflammatory disease and alterations of sebum composition initiate acne lesions

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Chlorogenic acid inhibits proliferation and induces apoptosis in A498 human kidney cancer cells via inactivating PI3K/Akt/mTOR signalling pathway

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Induction of proinflammatory cytokines by a soluble factor of Propionibacterium acnes: implications for chronic inflammatory acne

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Review of the Innate Immune Response in Acne vulgaris: Activation of Toll-Like Receptor 2 in Acne Triggers Inflammatory Cytokine Responses

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Thematic review series: Skin Lipids. Sebaceous gland lipids: friend or foe?

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FoxO1 Mediates PTEN Suppression of Androgen Receptor N- and C-Terminal Interactions and Coactivator Recruitment

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Current Concepts of the Pathogenesis of Acne

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Green Tea and Other Tea Polyphenols: Effects on Sebum Production and Acne Vulgaris

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Kaempferia parviflora Extract as a Potential Anti-Acne Agent with Anti-Inflammatory, Sebostatic and Anti-Propionibacterium acnes Activity

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Apigenin reduces the excessive accumulation of lipids induced by palmitic acid via the AMPK signaling pathway in HepG2 cells

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Medicinal Plants for the Treatment of Acne Vulgaris: A Review of Recent Evidences

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