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Intracellular accumulation of advanced glycation end products induces apoptosis via endoplasmic reticulum stress in chondrocytes
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/1529-0131(199809)41:9<1632::aid-art14>3.0.co;2-aopenalex-snapshot
Linkage of chondrocyte apoptosis and cartilage degradation in human osteoarthritis
inferredcites →https://doi.org/10.1002/1529-0131(199905)42:5<1003::aid-anr20>3.0.co;2-kopenalex-snapshot
Age-related decrease in proteoglycan synthesis of human articular chondrocytes: The role of nonenzymatic glycation
inferredcites →https://doi.org/10.1002/art.10627openalex-snapshot
Induction of advanced glycation end products and alterations of the tensile properties of articular cartilage
inferredcites →https://doi.org/10.1002/art.20170openalex-snapshot
Accumulation of advanced glycation end products as a molecular mechanism for aging as a risk factor in osteoarthritis
inferredcites →https://doi.org/10.1002/art.21523openalex-snapshot
Activation of receptor for advanced glycation end products in osteoarthritis leads to increased stimulation of chondrocytes and synoviocytes
inferredcites →https://doi.org/10.1002/art.21874openalex-snapshot
Caspase inhibitors reduce severity of cartilage lesions in experimental osteoarthritis
inferredcites →https://doi.org/10.1002/art.27296openalex-snapshot
Akt1 in murine chondrocytes controls cartilage calcification during endochondral ossification under physiologic and pathologic conditions
inferredcites →https://doi.org/10.1006/excr.2001.5224openalex-snapshot
Accumulation of Advanced Glycation End Products Decreases Collagen Turnover by Bovine Chondrocytes
inferredcites →https://doi.org/10.1006/meth.2001.1262openalex-snapshot
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method
inferredcites →https://doi.org/10.1007/s00418-002-0410-6openalex-snapshot
Immunohistochemical demonstration of Nɛ-(carboxymethyl)lysine protein adducts in normal and osteoarthritic cartilage
inferredcites →https://doi.org/10.1007/s00535-009-0193-9openalex-snapshot
The formation of intracellular glyceraldehyde-derived advanced glycation end-products and cytotoxicity
inferredcites →https://doi.org/10.1016/j.acthis.2010.06.007openalex-snapshot
Immunohistochemical distribution of advanced glycation end products (AGEs) in human osteoarthritic cartilage
inferredcites →https://doi.org/10.1016/j.arr.2009.03.001openalex-snapshot
ER and aging—Protein folding and the ER stress response
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ER stress and hormetic regulation of the aging process
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Glycolaldehyde induces growth inhibition and oxidative stress in human breast cancer cells*
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Osteoarthritis development in novel experimental mouse models induced by knee joint instability
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Premature osteoarthritis in the Disproportionate micromelia (Dmm) mouse
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Risk factors for onset of osteoarthritis of the knee in older adults: a systematic review and meta-analysis
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Basic methods in histopathology of joint tissues
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TAGE (toxic AGEs) hypothesis in various chronic diseases
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Glycolaldehyde induces apoptosis in a human breast cancer cell line
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PROTEIN MEASUREMENT WITH THE FOLIN PHENOL REAGENT
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Age-related accumulation of the advanced glycation endproduct pentosidine in human articular cartilage aggrecan: the use of pentosidine levels as a quantitative measure of protein turnover
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Nε-(Carboxymethyl)lysine Protein Adduct Is a Major Immunological Epitope in Proteins Modified with Advanced Glycation End Products of the Maillard Reaction
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S-Nitrosylated protein-disulphide isomerase links protein misfolding to neurodegeneration
inferredcites →https://doi.org/10.1038/ncb0311-184openalex-snapshot
Integrating the mechanisms of apoptosis induced by endoplasmic reticulum stress
inferredcites →https://doi.org/10.1038/sj.embor.7400779openalex-snapshot
Mediators of endoplasmic reticulum stress‐induced apoptosis
inferredcites →https://doi.org/10.1042/0264-6021:3500381openalex-snapshot
Age-related accumulation of Maillard reaction products in human articular cartilage collagen
inferredcites →https://doi.org/10.1073/pnas.191207498openalex-snapshot
Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway
inferredcites →https://doi.org/10.1074/jbc.m006700200openalex-snapshot
Effect of Collagen Turnover on the Accumulation of Advanced Glycation End Products
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Influence of the Hepatic Eukaryotic Initiation Factor 2α (eIF2α) Endoplasmic Reticulum (ER) Stress Response Pathway on Insulin-mediated ER Stress and Hepatic and Peripheral Glucose Metabolism
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Identification in Human Atherosclerotic Lesions of GA-pyridine, a Novel Structure Derived from Glycolaldehyde-modified Proteins
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Hepatocyte inflammation model for cytotoxicity research: fructose or glycolaldehyde as a source of endogenous toxins
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Conventional Antibody against N�-(Carboxymethyl)Lysine (CML) Shows Cross-Reaction to N�-(Carboxyethyl)Lysine (CEL): Immunochemical Quantification of CML with a Specific Antibody
inferredcites →https://doi.org/10.1093/ndt/gfn085openalex-snapshot
Advanced glycation end-products induce cell cycle arrest and hypertrophy in podocytes
inferredcites →https://doi.org/10.1093/rheumatology/kem376openalex-snapshot
Effects of advanced glycation end products on the expression of COX-2, PGE2 and NO in human osteoarthritic chondrocytes
inferredcites →https://doi.org/10.1093/rheumatology/keq380openalex-snapshot
Advanced glycation end products induce the expression of interleukin-6 and interleukin-8 by receptor for advanced glycation end product-mediated activation of mitogen-activated protein kinases and nuclear factor- B in human osteoarthritis chondrocytes
inferredcites →https://doi.org/10.1097/00002281-200309000-00016openalex-snapshot
AGEing and osteoarthritis: a different perspective
inferredcites →https://doi.org/10.1101/gad.12.7.982openalex-snapshot
CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum
inferredcites →https://doi.org/10.1111/j.1365-2613.2010.00758.xopenalex-snapshot
Enhanced apoptotic and reduced protective response in chondrocytes following endoplasmic reticulum stress in osteoarthritic cartilage
inferredcites →https://doi.org/10.1111/j.1756-185x.2011.01618.xopenalex-snapshot
Chondrocyte apoptosis: a cause or consequence of osteoarthritis?
inferredcites →https://doi.org/10.1128/mcb.21.4.1249-1259.2001openalex-snapshot
Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State
inferredcites →https://doi.org/10.1152/physrev.00015.2006openalex-snapshot
Endoplasmic Reticulum Stress Signaling in Disease
inferredcites →https://doi.org/10.1172/jci119176openalex-snapshot
Human neutrophils employ the myeloperoxidase-hydrogen peroxide-chloride system to convert hydroxy-amino acids into glycolaldehyde, 2-hydroxypropanal, and acrolein. A mechanism for the generation of highly reactive alpha-hydroxy and alpha,beta-unsaturated aldehydes by phagocytes at sites of…
inferredcites →https://doi.org/10.1186/ar2807openalex-snapshot
Advanced glycation end products induce cell cycle arrest and proinflammatory changes in osteoarthritic fibroblast-like synovial cells
inferredcites →https://doi.org/10.1371/journal.pone.0000029openalex-snapshot
Critical Role of Methylglyoxal and AGE in Mycobacteria-Induced Macrophage Apoptosis and Activation
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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The link between advanced glycation end products and apoptosis in delayed wound healing
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Glycolaldehyde induces sensory neuron death through activation of the c-Jun N-terminal kinase and p-38 MAP kinase pathways
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Cell death in human articular chondrocyte: a morpho-functional study in micromass model
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Glycation in the cardiomyocyte
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Primary Osteoarthritis Early Joint Degeneration Induced by Endoplasmic Reticulum Stress Is Mitigated by Resveratrol
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A comprehensive review on the advanced glycation end products detection: From conventional to advanced approaches
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SIRT3 alleviates high glucose-induced chondrocyte injury through the promotion of autophagy and suppression of apoptosis in osteoarthritis progression
inferred← citeshttps://doi.org/10.1016/j.jbspin.2023.105642openalex-snapshot
The role of autophagy in mitigating osteoarthritis progression via regulation of chondrocyte apoptosis: A review
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Advanced glycation end products‐induced chondrocyte apoptosis through mitochondrial dysfunction in cultured rabbit chondrocyte
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Chondrocyte Apoptosis in the Pathogenesis of Osteoarthritis
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ER Stress in ERp57 Knockout Knee Joint Chondrocytes Induces Osteoarthritic Cartilage Degradation and Osteophyte Formation
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The Association between Accumulation of Toxic Advanced Glycation End-Products and Cytotoxic Effect in MC3T3-E1 Cells
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Faculty Opinions recommendation of PERK-mediated translational control is required for collagen secretion in chondrocytes.
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Women suffering from systemic lupus erythematosus are characterized by low blood levels of α-dicarbonyl compounds
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