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Cellular Senescence in Aging Primates
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1016/j.devcel.2004.10.019openalex-snapshot
Formation of MacroH2A-Containing Senescence-Associated Heterochromatin Foci and Senescence Driven by ASF1a and HIRA
inferredcites →https://doi.org/10.1016/j.exger.2005.08.005openalex-snapshot
SA β Gal staining: Biomarker or delusion
inferredcites →https://doi.org/10.1016/j.febslet.2004.11.036openalex-snapshot
Telomeres and telomerase: their mechanisms of action and the effects of altering their functions
inferredcites →https://doi.org/10.1016/s0968-0004(02)02110-2openalex-snapshot
Oxidative stress shortens telomeres
inferredcites →https://doi.org/10.1016/s1097-2765(04)00256-4openalex-snapshot
Telomere Shortening Triggers Senescence of Human Cells through a Pathway Involving ATM, p53, and p21CIP1, but Not p16INK4a
inferredcites →https://doi.org/10.1189/jlb.0702361openalex-snapshot
Telomere shortening in leukocyte subpopulations from baboons
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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MYSM1 Suppresses Cellular Senescence and the Aging Process to Prolong Lifespan
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DEL‐1 is an Endogenous Senolytic Protein that Inhibits Senescence‐Associated Bone Loss
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An evolutionary review of human telomere biology: The thrifty telomere hypothesis and notes on potential adaptive paternal effects
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Senescence, brain inflammation, and oligomeric tau drive cognitive decline in Alzheimer's disease: Evidence from clinical and preclinical studies
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Leptin and Neutrophil‐Activating Peptide 2 Promote Mesenchymal Stem Cell Senescence Through Activation of the Phosphatidylinositol 3‐Kinase/Akt Pathway in Patients With Systemic Lupus Erythematosus
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The search for evolutionary developmental origins of aging in zebrafish: A novel intersection of developmental and senescence biology in the zebrafish model system
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Evidence that senescent human prostate epithelial cells enhance tumorigenicity: Cell fusion as a potential mechanism and inhibition by p16INK4a and hTERT
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Aging‐related alterations in the extracellular matrix modulate the microenvironment and influence tumor progression
inferred← citeshttps://doi.org/10.1002/iub.1335openalex-snapshot
Tumour suppressors and cellular senescence
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2015.tb00662.xopenalex-snapshot
Aging Effects on Cardiac Progenitor Cell Physiology
inferred← citeshttps://doi.org/10.1002/j.2040-4603.2023.tb00282.xopenalex-snapshot
Impact of Aging and Cellular Senescence in the Pathophysiology of Preeclampsia
inferred← citeshttps://doi.org/10.1002/jbmr.2892openalex-snapshot
Identification of Senescent Cells in the Bone Microenvironment
inferred← citeshttps://doi.org/10.1002/jbmr.3564openalex-snapshot
The Spectrum of Fundamental Basic Science Discoveries Contributing to Organismal Aging
inferred← citeshttps://doi.org/10.1002/jbmr.3729openalex-snapshot
Independent Roles of Estrogen Deficiency and Cellular Senescence in the Pathogenesis of Osteoporosis: Evidence in Young Adult Mice and Older Humans
inferred← citeshttps://doi.org/10.1002/jcla.24753openalex-snapshot
Does walking during chemotherapy impact p16INK4a levels in women with early breast cancer
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Possibility of mixed progenitor cells in sea star arm regeneration
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Telomerase deficiency in a colonial ascidian after prolonged asexual propagation
inferred← citeshttps://doi.org/10.1002/mc.22428openalex-snapshot
Adiponectin induces CXCL1 secretion from cancer cells and promotes tumor angiogenesis by inducing stromal fibroblast senescence
inferred← citeshttps://doi.org/10.1002/mnfr.202300330openalex-snapshot
Lycopene Improves Bone Quality in SAMP6 Mice by Inhibiting Oxidative Stress, Cellular Senescence, and the SASP
inferred← citeshttps://doi.org/10.1002/oby.21888openalex-snapshot
Immunosenescence Induced by Plasma from Individuals with Obesity Caused Cell Signaling Dysfunction and Inflammation
inferred← citeshttps://doi.org/10.1002/path.2783openalex-snapshot
Regulation of tissue‐ and stimulus‐specific cell fate decisions by p53 in vivo
inferred← citeshttps://doi.org/10.1002/pmic.200800471openalex-snapshot
Proteomic profiling of tumor cells after induction of telomere dysfunction
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Proteomic profiling in distinct cellular compartments of tumor cells reveals p53‐dependent upregulation of S100A6 upon induction of telomere dysfunction
inferred← citeshttps://doi.org/10.1002/ptr.8507openalex-snapshot
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inferred← citeshttps://doi.org/10.1002/smll.202408517openalex-snapshot
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inferred← citeshttps://doi.org/10.1007/978-1-4419-0639-7_13openalex-snapshot
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Chromatin in Senescent Cells: A Conduit for the Anti-Aging Effects of Wnt Signaling?
inferred← citeshttps://doi.org/10.1007/978-1-4419-0639-7_9openalex-snapshot
Chromosomal Position Effect and Aging
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Cellular Senescence and Tumor Suppression
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Stress-Induced Senescence
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Chromatin Structure in Senescent Cells
inferred← citeshttps://doi.org/10.1007/978-1-4419-1075-2_7openalex-snapshot
A Comparison of Senescence in Mouse and Human Cells
inferred← citeshttps://doi.org/10.1007/978-1-4419-1075-2_9openalex-snapshot
Telomere Dysfunction and Senescence in Stem Cell and Tissues Aging
inferred← citeshttps://doi.org/10.1007/978-1-4419-1770-6_9openalex-snapshot
The Senescence Secretome and Its Impact on Tumor Suppression and Cancer
inferred← citeshttps://doi.org/10.1007/978-1-4419-7002-2_13openalex-snapshot
Post-Translational Modification of Cellular Proteins by Ubiquitin and Ubiquitin-Like Molecules: Role in Cellular Senescence and Aging
inferred← citeshttps://doi.org/10.1007/978-1-4939-0888-2_8openalex-snapshot
The Senescence Arrest Program and the Cell Cycle
inferred← citeshttps://doi.org/10.1007/978-1-4939-6554-0_3openalex-snapshot
Cellular and Molecular Aging
inferred← citeshttps://doi.org/10.1007/978-1-4939-8931-7_12openalex-snapshot
A Novel Quantitative Method for the Detection of Lipofuscin, the Main By-Product of Cellular Senescence, in Fluids
inferred← citeshttps://doi.org/10.1007/978-1-4939-8931-7_17openalex-snapshot
Mouse Models of Accelerated Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-1-60327-507-1_10openalex-snapshot
Life Extension in the Short-Lived Fish Nothobranchius furzeri
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Vascular Aging and Oxidative Stress: Hormesis and Adaptive Cellular Pathways
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_12openalex-snapshot
Detection of Senescence-Associated Heterochromatin Foci (SAHF)
inferred← citeshttps://doi.org/10.1007/978-1-62703-239-1_13openalex-snapshot
Monitoring DNA Damage During Cell Senescence
inferred← citeshttps://doi.org/10.1007/978-1-62703-317-6_7openalex-snapshot
Cardiac Stem Cell Senescence
inferred← citeshttps://doi.org/10.1007/978-1-62703-429-6_8openalex-snapshot
Methods to Study MYC-Regulated Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-1-62703-556-9_4openalex-snapshot
Cell Sorting of Young and Senescent Cells
inferred← citeshttps://doi.org/10.1007/978-3-030-15458-5_3openalex-snapshot
The Molecular Physiology of Ageing: New Targets for Regenerative Medicine
inferred← citeshttps://doi.org/10.1007/978-3-030-22009-9_1032openalex-snapshot
Elastin, Aging-Related Changes in
inferred← citeshttps://doi.org/10.1007/978-3-030-22009-9_790openalex-snapshot
International Society on Aging and Diseases (ISOAD)
inferred← citeshttps://doi.org/10.1007/978-3-030-25890-0_1openalex-snapshot
Muscle and Bone Biology – Similarities and Differences
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_3openalex-snapshot
Interconnection Between Cellular Senescence, Regeneration and Ageing in Salamanders
inferred← citeshttps://doi.org/10.1007/978-3-030-44903-2_4openalex-snapshot
Senolytics Target Senescent Cells and Improve Aging and Age-Related Diseases
inferred← citeshttps://doi.org/10.1007/978-3-030-58532-7_9openalex-snapshot
Lysosomes and Peroxisomes
inferred← citeshttps://doi.org/10.1007/978-3-031-55022-5_4openalex-snapshot
Epigenetic Changes in Aging: The Contribution of SIRT1 to Longevity
inferred← citeshttps://doi.org/10.1007/978-3-031-76469-1_2openalex-snapshot
Biomedical Research in Aging
inferred← citeshttps://doi.org/10.1007/978-3-319-03970-1_7openalex-snapshot
Effects of Aging on the Cellular Function, Healing, and Mechanical Properties of Ligaments
inferred← citeshttps://doi.org/10.1007/978-3-319-06103-0_5openalex-snapshot
The Biology of Aging: Role in Cancer, Metabolic Dysfunction, and Health Disparities
inferred← citeshttps://doi.org/10.1007/978-3-319-16015-3_9openalex-snapshot
The Biology of Immortality
inferred← citeshttps://doi.org/10.1007/978-3-319-26239-0_16openalex-snapshot
Targeting Senescent Cells to Improve Human Health
inferred← citeshttps://doi.org/10.1007/978-3-319-26239-0_4openalex-snapshot
Influence of Donor Age and Species Longevity on Replicative Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-3-319-26239-0_7openalex-snapshot
Telomeres Shortening: A Mere Replicometer?
inferred← citeshttps://doi.org/10.1007/978-3-319-44870-1_71-1openalex-snapshot
Hematopoietic Stem Cell Aging and Malignant Hemopathies
inferred← citeshttps://doi.org/10.1007/978-3-319-54016-0_4openalex-snapshot
Parallels Between Skeletal Muscle and Bone Aging
inferred← citeshttps://doi.org/10.1007/978-3-319-57415-8_71openalex-snapshot
Hematopoietic Stem Cell Aging and Malignant Hemopathies
inferred← citeshttps://doi.org/10.1007/978-3-319-64597-1_142-1openalex-snapshot
Aging and Malignant Hemopathies: A Complex Multistep Process
inferred← citeshttps://doi.org/10.1007/978-3-319-69892-2_1032-1openalex-snapshot
Elastin, Aging-Related Changes in
inferred← citeshttps://doi.org/10.1007/978-3-319-69892-2_790-1openalex-snapshot
International Society on Aging and Diseases (ISOAD)
inferred← citeshttps://doi.org/10.1007/978-3-319-78265-2_2openalex-snapshot
Pearl Chapter: Basis of Photoaging and the Use of Chemical Peelings
inferred← citeshttps://doi.org/10.1007/978-3-319-95387-8_3openalex-snapshot
Biomedical Research in Aging
inferred← citeshttps://doi.org/10.1007/978-3-319-99375-1_142openalex-snapshot
Aging and Malignant Hemopathies: A Complex Multistep Process
inferred← citeshttps://doi.org/10.1007/978-3-540-68976-8_4openalex-snapshot
Bone Marrow-Derived Cells: The Influence of Aging and Cellular Senescence
inferred← citeshttps://doi.org/10.1007/978-3-540-69161-7_10openalex-snapshot
Asymmetric Distribution of DNA Between Daughter Cells with Final Symmetry Breaking During Aging of Human Fibroblasts
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_1openalex-snapshot
Cellular versus Organismal Aging
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_14openalex-snapshot
Werner Syndrome, Telomeres, and Stress Signaling: Implications for Future Therapies?
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_4openalex-snapshot
Initiation of Genomic Instability, Cellular Senescence, and Organismal Aging by Dysfunctional Telomeres
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_7openalex-snapshot
Senescence Signatures of Human Hematopoietic Stem Cells
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_8openalex-snapshot
Telomere Shortening Induces Cell Intrinsic Checkpoints and Environmental Alterations Limiting Adult Stem Cell Function
inferred← citeshttps://doi.org/10.1007/978-3-540-73709-4_9openalex-snapshot
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Aging-associated Non-melanoma Skin Cancer: A Role for the Dermis
inferred← citeshttps://doi.org/10.1007/978-3-642-27814-3_58-2openalex-snapshot
Aging-Associated Nonmelanoma Skin Cancer: A Role for the Dermis
inferred← citeshttps://doi.org/10.1007/978-3-642-30018-9_66openalex-snapshot
Role of Oxidative Stress in Vascular Endothelial Cells Through Aging – a Double-Edged Sword
inferred← citeshttps://doi.org/10.1007/978-3-662-47398-6_58openalex-snapshot
Aging-Associated Nonmelanoma Skin Cancer: A Role for the Dermis
inferred← citeshttps://doi.org/10.1007/978-3-662-48250-6_2openalex-snapshot
Der Zellzyklus: Lebenszyklus einer Zelle
inferred← citeshttps://doi.org/10.1007/978-3-662-66576-3_1openalex-snapshot
Neuigkeiten aus der Krebsforschung
inferred← citeshttps://doi.org/10.1007/978-3-7091-1232-8_9openalex-snapshot
Aging of Mammalian Intestinal Stem Cells
inferred← citeshttps://doi.org/10.1007/978-81-322-2032-9_3openalex-snapshot
Oxidative Stress in Metabolic Disorders/Diseases
inferred← citeshttps://doi.org/10.1007/978-90-481-3465-6_5openalex-snapshot
Cell Replication Rates In Vivo and In Vitro and Wound Healing as Affected by Animal Age, Diet, and Species
inferred← citeshttps://doi.org/10.1007/978-94-007-4525-4_16openalex-snapshot
Chromatin Structure and Organization: The Relation with Gene Expression During Development and Disease
inferred← citeshttps://doi.org/10.1007/978-94-007-4525-4_8openalex-snapshot
Small Changes, Big Effects: Chromatin Goes Aging
inferred← citeshttps://doi.org/10.1007/978-94-007-5958-9openalex-snapshot
Tumor Dormancy, Quiescence, and Senescence, Volume 1
inferred← citeshttps://doi.org/10.1007/978-94-007-5958-9_11openalex-snapshot
Stress-Induced Senescence: Molecular Pathways
inferred← citeshttps://doi.org/10.1007/978-94-007-5958-9_19openalex-snapshot
The Two Faces of Senescence-Associated Epigenetic Alterations: Tumor Suppressors and Oncogenic Drivers
inferred← citeshttps://doi.org/10.1007/978-94-007-7726-2_4openalex-snapshot
Regulation of the Novel Senescence Pathway by SKP2 E3 Ligase
inferred← citeshttps://doi.org/10.1007/978-981-13-2835-0_9openalex-snapshot
Telomeres, Telomerase and Ageing
inferred← citeshttps://doi.org/10.1007/978-981-96-1041-9_1openalex-snapshot
Introduction to Cellular Senescence and Aging
inferred← citeshttps://doi.org/10.1007/978-981-99-4436-1_6openalex-snapshot
RPE Senescence and Its Implication in Age-Related Macular Degeneration
inferred← citeshttps://doi.org/10.1007/s00018-007-7123-xopenalex-snapshot
From old organisms to new molecules: integrative biology and therapeutic targets in accelerated human ageing
inferred← citeshttps://doi.org/10.1007/s00018-009-0034-2openalex-snapshot
A comparative analysis of the cell biology of senescence and aging
inferred← citeshttps://doi.org/10.1007/s00018-014-1691-3openalex-snapshot
Physiological and pathological consequences of cellular senescence
inferred← citeshttps://doi.org/10.1007/s00018-018-2879-8openalex-snapshot
Cellular senescence: a view throughout organismal life
inferred← citeshttps://doi.org/10.1007/s00018-021-03798-7openalex-snapshot
Adamantinomatous craniopharyngioma as a model to understand paracrine and senescence-induced tumourigenesis
inferred← citeshttps://doi.org/10.1007/s00105-006-1254-0openalex-snapshot
Ursachen und Folgen zellulärer Seneszenz
inferred← citeshttps://doi.org/10.1007/s00105-023-05201-xopenalex-snapshot
Hautalterung – zelluläre Seneszenz
inferred← citeshttps://doi.org/10.1007/s00109-011-0766-yopenalex-snapshot
Novel ARF/p53-independent senescence pathways in cancer repression
inferred← citeshttps://doi.org/10.1007/s00210-014-1077-yopenalex-snapshot
Inhibition of telomerase activity by NME2: impact on metastasis suppression?
inferred← citeshttps://doi.org/10.1007/s00335-016-9628-9openalex-snapshot
Old cells, new tricks: chromatin structure in senescence
inferred← citeshttps://doi.org/10.1007/s00418-012-0941-4openalex-snapshot
The peroxisome: an update on mysteries
inferred← citeshttps://doi.org/10.1007/s00418-018-1722-5openalex-snapshot
The peroxisome: an update on mysteries 2.0
inferred← citeshttps://doi.org/10.1007/s00439-023-02565-xopenalex-snapshot
Cellular senescence and neurodegeneration
inferred← citeshttps://doi.org/10.1007/s00441-007-0469-4openalex-snapshot
Telomeres, senescence, and hematopoietic stem cells
inferred← citeshttps://doi.org/10.1007/s10522-008-9207-xopenalex-snapshot
Cell senescence: the future of ageing?
inferred← citeshttps://doi.org/10.1007/s10522-008-9209-8openalex-snapshot
Relationships between cancer and aging: a multilevel approach
inferred← citeshttps://doi.org/10.1007/s10522-009-9216-4openalex-snapshot
Telomere dynamics during replicative senescence are not directly modulated by conditions of oxidative stress in IMR90 fibroblast cells
inferred← citeshttps://doi.org/10.1007/s10522-009-9241-3openalex-snapshot
Cancers and the concept of cell senescence
inferred← citeshttps://doi.org/10.1007/s10522-011-9346-3openalex-snapshot
Cell division and aging of the organism
inferred← citeshttps://doi.org/10.1007/s10522-011-9349-0openalex-snapshot
It’s not all equal: a multiphasic theory of thymic involution
inferred← citeshttps://doi.org/10.1007/s10522-011-9366-zopenalex-snapshot
Cellular aging and senescence characteristics of human T-lymphocytes
inferred← citeshttps://doi.org/10.1007/s10522-012-9407-2openalex-snapshot
p38 MAPK stress signalling in replicative senescence in fibroblasts from progeroid and genomic instability syndromes
inferred← citeshttps://doi.org/10.1007/s10522-013-9473-0openalex-snapshot
Immunosurveillance of senescent cells: the bright side of the senescence program
inferred← citeshttps://doi.org/10.1007/s10522-014-9529-9openalex-snapshot
Senescent cells: SASPected drivers of age-related pathologies
inferred← citeshttps://doi.org/10.1007/s10522-015-9560-5openalex-snapshot
Comparison of senescence-associated miRNAs in primary skin and lung fibroblasts
inferred← citeshttps://doi.org/10.1007/s10522-015-9610-zopenalex-snapshot
Suppression of the senescence-associated secretory phenotype (SASP) in human fibroblasts using small molecule inhibitors of p38 MAP kinase and MK2
inferred← citeshttps://doi.org/10.1007/s10522-020-09874-yopenalex-snapshot
Epigenetic changes during ageing and their underlying mechanisms
inferred← citeshttps://doi.org/10.1007/s10522-020-09893-9openalex-snapshot
Multiparameter flow cytometric detection and quantification of senescent cells in vitro
inferred← citeshttps://doi.org/10.1007/s10522-023-10085-4openalex-snapshot
Telomeres, cellular senescence, and aging: past and future
inferred← citeshttps://doi.org/10.1007/s10555-020-09875-wopenalex-snapshot
Caveolin-1, a master regulator of cellular senescence
inferred← citeshttps://doi.org/10.1007/s10815-015-0483-5openalex-snapshot
Increased DNA damage and repair deficiency in granulosa cells are associated with ovarian aging in rhesus monkey
inferred← citeshttps://doi.org/10.1007/s11010-012-1423-7openalex-snapshot
Hexavalent chromium induces energy metabolism disturbance and p53-dependent cell cycle arrest via reactive oxygen species in L-02 hepatocytes
inferred← citeshttps://doi.org/10.1007/s11010-015-2327-0openalex-snapshot
Calorie restriction increases telomerase activity, enhances autophagy, and improves diastolic dysfunction in diabetic rat hearts
inferred← citeshttps://doi.org/10.1007/s11010-020-03899-0openalex-snapshot
Calorie restriction delays cardiac senescence and improves cardiac function in obese diabetic rats
inferred← citeshttps://doi.org/10.1007/s11224-007-9212-8openalex-snapshot
Symmetry breaking in biological systems. From molecules to tissues
inferred← citeshttps://doi.org/10.1007/s11357-009-9104-5openalex-snapshot
Could Sirt1-mediated epigenetic effects contribute to the longevity response to dietary restriction and be mimicked by other dietary interventions?
inferred← citeshttps://doi.org/10.1007/s11357-009-9108-1openalex-snapshot
Cellular senescence: unravelling complexity
inferred← citeshttps://doi.org/10.1007/s11357-010-9201-5openalex-snapshot
Resveratrol, but not dihydroresveratrol, induces premature senescence in primary human fibroblasts
inferred← citeshttps://doi.org/10.1007/s11357-012-9452-4openalex-snapshot
A role for c-Abl in cell senescence and spontaneous immortalization
inferred← citeshttps://doi.org/10.1007/s11357-015-9764-2openalex-snapshot
Cellular senescence: from growth arrest to immunogenic conversion
inferred← citeshttps://doi.org/10.1007/s11357-015-9789-6openalex-snapshot
Analysis of gene expression during aging of CGNs in culture: implication of SLIT2 and NPY in senescence
inferred← citeshttps://doi.org/10.1007/s11357-015-9810-0openalex-snapshot
Erratum to: Analysis of gene expression during aging of CGNs in culture: implication of SLIT2 and NPY in senescence
inferred← citeshttps://doi.org/10.1007/s11357-018-0030-2openalex-snapshot
Rapamycin increases oxidative metabolism and enhances metabolic flexibility in human cardiac fibroblasts
inferred← citeshttps://doi.org/10.1007/s11357-020-00185-1openalex-snapshot
Heterochronic parabiosis regulates the extent of cellular senescence in multiple tissues
inferred← citeshttps://doi.org/10.1007/s11357-021-00364-8openalex-snapshot
Age-related expression of prominent regulatory elements in mouse brain: catastrophic decline of FOXO3a
inferred← citeshttps://doi.org/10.1007/s11427-019-1629-6openalex-snapshot
Cellular senescence: from anti-cancer weapon to anti-aging target
inferred← citeshttps://doi.org/10.1007/s11914-019-00504-2openalex-snapshot
Targeting Cell Senescence for the Treatment of Age-Related Bone Loss
inferred← citeshttps://doi.org/10.1007/s12035-016-9937-8openalex-snapshot
Anesthetic Isoflurane Induces DNA Damage Through Oxidative Stress and p53 Pathway
inferred← citeshttps://doi.org/10.1007/s12035-025-05504-1openalex-snapshot
Senolytics as Modulators of Critical Signaling Pathways: a Promising Strategy to Combat Brain Aging and Neurodegenerative Disorders
inferred← citeshttps://doi.org/10.1007/s12602-021-09903-3openalex-snapshot
Emerging Interrelationship Between the Gut Microbiome and Cellular Senescence in the Context of Aging and Disease: Perspectives and Therapeutic Opportunities
inferred← citeshttps://doi.org/10.1007/s13577-015-0110-xopenalex-snapshot
Cellular senescence: a hitchhiker’s guide
inferred← citeshttps://doi.org/10.1007/s40610-018-0108-8openalex-snapshot
Cellular Senescence in Intervertebral Disc Aging and Degeneration
inferred← citeshttps://doi.org/10.1007/s40610-020-00129-wopenalex-snapshot
The Pivotal Role of Senescence in Cell Death and Aging: Where Do We Stand?
inferred← citeshttps://doi.org/10.1007/s40610-020-00141-0openalex-snapshot
Senescent Mesenchymal Stem Cells: Disease Mechanism and Treatment Strategy
inferred← citeshttps://doi.org/10.1007/s40843-024-2864-5openalex-snapshot
A precise dual-drug codelivery nanosystem for treating kidney failure by eliminating senescent cells
inferred← citeshttps://doi.org/10.1016/b978-0-12-381332-9.00045-1openalex-snapshot
Cardiac Regeneration and Aging
inferred← citeshttps://doi.org/10.1016/b978-0-12-385493-3.00007-3openalex-snapshot
Fluorescence-Based Detection and Quantification of Features of Cellular Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-394447-4.30066-9openalex-snapshot
Cellular Senescence
inferred← citeshttps://doi.org/10.1016/b978-0-12-397803-5.00017-4openalex-snapshot
Cellular Senescence and Premature Aging in Lung Diseases
inferred← citeshttps://doi.org/10.1016/b978-0-12-405933-7.00002-0openalex-snapshot
Skin Aging and Oxidative Stress
inferred← citeshttps://doi.org/10.1016/b978-0-12-411596-5.00007-1openalex-snapshot
Contributions of Telomere Biology to Human Age-Related Disease
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