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Inclusion bodies enriched for p62 and polyubiquitinated proteins in macrophages protect against atherosclerosis

https://doi.org/10.1126/scisignal.aad5614

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Nrf2 is essential for cholesterol crystal‐induced inflammasome activation and exacerbation of atherosclerosis

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Protein aggregation and aggregate toxicity: new insights into protein folding, misfolding diseases and biological evolution

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Probing the interactions of the solvated electron with DNA by molecular dynamics simulations: bromodeoxyuridine substituted DNA

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The Signaling Adaptor p62 Is an Important NF-κB Mediator in Tumorigenesis

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Homeostatic Levels of p62 Control Cytoplasmic Inclusion Body Formation in Autophagy-Deficient Mice

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Autophagy in the Pathogenesis of Disease

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The Amyloid State of Proteins in Human Diseases

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Prion-like Polymerization Underlies Signal Transduction in Antiviral Immune Defense and Inflammasome Activation

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Nondegradative Role of Atg5-Atg12/ Atg16L1 Autophagy Protein Complex in Antiviral Activity of Interferon Gamma

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“New” hepatic fat activates PPARα to maintain glucose, lipid, and cholesterol homeostasis

inferredcites →https://doi.org/10.1016/j.cmet.2006.01.011openalex-snapshot

Mature-onset obesity and insulin resistance in mice deficient in the signaling adapter p62

inferredcites →https://doi.org/10.1016/j.cmet.2006.10.002openalex-snapshot

ATM-dependent suppression of stress signaling reduces vascular disease in metabolic syndrome

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Autophagy Regulates Cholesterol Efflux from Macrophage Foam Cells via Lysosomal Acid Lipase

inferredcites →https://doi.org/10.1016/j.cmet.2012.01.022openalex-snapshot

Macrophage Autophagy Plays a Protective Role in Advanced Atherosclerosis

inferredcites →https://doi.org/10.1016/j.cmet.2012.02.011openalex-snapshot

Autophagy Links Inflammasomes to Atherosclerotic Progression

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Protein aggregates in Huntington's disease

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Physiological significance of selective degradation of p62 by autophagy

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Disease-associated amyloid and misfolded protein aggregates activate the inflammasome

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Autophagy and innate immunity: Triggering, targeting and tuning

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Self-eating in the plaque: what macrophage autophagy reveals about atherosclerosis

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Effects of heterozygous lipoprotein lipase deficiency on diet-induced atherosclerosis in mice

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Cholesterol crystal formation and growth in model bile solutions.

inferredcites →https://doi.org/10.1016/s0962-8924(00)01852-3openalex-snapshot

Aggresomes, inclusion bodies and protein aggregation

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Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death

inferredcites →https://doi.org/10.1038/nature04724openalex-snapshot

Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

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Autophagy regulates lipid metabolism

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NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

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Autophagy in immunity and inflammation

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The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

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Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction

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Macrophage death and defective inflammation resolution in atherosclerosis

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Macrophages in atherosclerosis: a dynamic balance

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What is the role of protein aggregation in neurodegeneration?

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Ubiquitin signals autophagic degradation of cytosolic proteins and peroxisomes

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The Atypical Protein Kinase C-interacting Protein p62 Is a Scaffold for NF-κB Activation by Nerve Growth Factor

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Caveolin-1 Regulates Transforming Growth Factor (TGF)-β/SMAD Signaling through an Interaction with the TGF-β Type I Receptor

inferredcites →https://doi.org/10.1074/jbc.m105408200openalex-snapshot

Caveolin-1 Null Mice Are Viable but Show Evidence of Hyperproliferative and Vascular Abnormalities

inferredcites →https://doi.org/10.1074/jbc.m111.230508openalex-snapshot

Fatty Acid Synthase Modulates Homeostatic Responses to Myocardial Stress

inferredcites →https://doi.org/10.1074/jbc.m608787200openalex-snapshot

p62 Accumulates and Enhances Aggregate Formation in Model Systems of Familial Amyotrophic Lateral Sclerosis

inferredcites →https://doi.org/10.1083/jcb.200507002openalex-snapshot

p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death

inferredcites →https://doi.org/10.1089/dna.2012.1915openalex-snapshot

Interaction Domains of p62: A Bridge Between p62 and Selective Autophagy

inferredcites →https://doi.org/10.1093/emboj/19.7.1576openalex-snapshot

The atypical PKC‐interacting protein p62 channels NF‐κB activation by the IL‐1–TRAF6 pathway

inferredcites →https://doi.org/10.1093/eurheartj/11.suppl_e.116openalex-snapshot

Macrophages and ceroid in human atherosclerosis

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Autophagy and Neuronal Cell Death in Neurological Disorders

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Programmed Cell Death in Parkinson's Disease

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Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration

inferredcites →https://doi.org/10.1111/j.1750-3639.2009.00290.xopenalex-snapshot

Inclusion Body Myositis: A Degenerative Muscle Disease Associated with Intra‐Muscle Fiber Multi‐Protein Aggregates, Proteasome Inhibition, Endoplasmic Reticulum Stress and Decreased Lysosomal Degradation

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The C9orf72 GGGGCC Repeat Is Translated into Aggregating Dipeptide-Repeat Proteins in FTLD/ALS

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A Noncanonical Mechanism of Nrf2 Activation by Autophagy Deficiency: Direct Interaction between Keap1 and p62

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Alpha-1-Antitrypsin Deficiency: Importance of Proteasomal and Autophagic Degradative Pathways in Disposal of Liver Disease–Associated Protein Aggregates

inferredcites →https://doi.org/10.1152/physrev.00041.2009openalex-snapshot

Molecular Mechanisms and Potential Therapeutical Targets in Huntington's Disease

inferredcites →https://doi.org/10.1155/jbb/2006/62079openalex-snapshot

Signaling, Polyubiquitination, Trafficking, and Inclusions: Sequestosome 1/p62′s Role in Neurodegenerative Disease

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Autophagy and Viruses: Adversaries or Allies?

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Repertoires of Autophagy in the Pathogenesis of Ocular Diseases

inferredcites →https://doi.org/10.1161/01.atv.7.2.135openalex-snapshot

Decrease in reactive amino groups during oxidation or endothelial cell modification of LDL. Correlation with changes in receptor-mediated catabolism.

inferredcites →https://doi.org/10.1161/atvbaha.110.210906openalex-snapshot

NF-E2–Related Factor 2 Promotes Atherosclerosis by Effects on Plasma Lipoproteins and Cholesterol Transport That Overshadow Antioxidant Protection

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Induction of Lysosomal Biogenesis in Atherosclerotic Macrophages Can Rescue Lipid-Induced Lysosomal Dysfunction and Downstream Sequelae

inferredcites →https://doi.org/10.1161/circresaha.112.265819openalex-snapshot

Mitochondria and Mitophagy

inferredcites →https://doi.org/10.1172/jci64209openalex-snapshot

p62 Links β-adrenergic input to mitochondrial function and thermogenesis

inferredcites →https://doi.org/10.1172/jci9259openalex-snapshot

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

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Disruption of Nrf2, a Key Inducer of Antioxidant Defenses, Attenuates ApoE-Mediated Atherosclerosis in Mice

inferredcites →https://doi.org/10.1371/journal.pone.0011765openalex-snapshot

Cholesterol Crystals Activate the NLRP3 Inflammasome in Human Macrophages: A Novel Link between Cholesterol Metabolism and Inflammation

inferredcites →https://doi.org/10.1371/journal.pone.0052868openalex-snapshot

p62/SQSTM1-Dependent Autophagy of Lewy Body-Like α-Synuclein Inclusions

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Protein Aggregation in the Brain: The Molecular Basis for Alzheimer’s and Parkinson’s Diseases

inferredcites →https://doi.org/10.2217/clp.10.70openalex-snapshot

Lysosomes, cholesterol and atherosclerosis

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Elucidating the crosstalk between endothelial-to-mesenchymal transition (EndoMT) and endothelial autophagy in the pathogenesis of atherosclerosis

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Post-translational regulation of inflammasomes

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Genetic inhibition of CARD9 accelerates the development of atherosclerosis in mice through CD36 dependent-defective autophagy

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High-protein diets increase cardiovascular risk by activating macrophage mTOR to suppress mitophagy

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Protective role of chaperone-mediated autophagy against atherosclerosis

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SNAPIN is critical for lysosomal acidification and autophagosome maturation in macrophages

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N-3 PUFAs induce inflammatory tolerance by formation of KEAP1-containing SQSTM1/p62-bodies and activation of NFE2L2

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The autophagy receptor SQSTM1/p62 mediates anti-inflammatory actions of the selective NR3C1/glucocorticoid receptor modulator compound A (CpdA) in macrophages

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SQSTM1/p62 and PPARGC1A/PGC-1alpha at the interface of autophagy and vascular senescence

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Vitamin D3-VDR-PTPN6 axis mediated autophagy contributes to the inhibition of macrophage foam cell formation

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Sex differences in human umbilical vein endothelial cells following ox-LDL injury

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Myeloid deletion and therapeutic activation of AMPK do not alter atherosclerosis in male or female mice

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p62 /SQSTM1 coding plasmid prevents age related macular degeneration in a rat model

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TPM2 as a potential predictive biomarker for atherosclerosis

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Antrodia Camphorata Polysaccharide activates autophagy and regulates NLRP3 degradation to improve liver injury-related inflammatory response

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SIRT1 inhibition promotes atherosclerosis through impaired autophagy

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Luteolin ameliorated CHC-induced autophagy dysfunction in macrophages through the AMPK/mTOR signaling pathway

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Luteolin ameliorated CHC-induced autophagy dysfunction in macrophages through the AMPK/mTOR signaling pathway

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Monocyte-Derived Macrophage Sqstm1/P62 is Essential for Liver Regeneration

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ApoE and ApoE Nascent-Like HDL Particles at Model Cellular Membranes: Effect of Protein Isoform and Membrane Composition

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Trehalose promotes atherosclerosis regression in female mice

inferred← citeshttps://doi.org/10.3389/fimmu.2020.01559openalex-snapshot

PDLIM7 Synergizes With PDLIM2 and p62/Sqstm1 to Inhibit Inflammatory Signaling by Promoting Degradation of the p65 Subunit of NF-κB

inferred← citeshttps://doi.org/10.3389/fimmu.2025.1550226openalex-snapshot

NLRP3 inflammasome in cardiovascular diseases: an update

inferred← citeshttps://doi.org/10.3389/fphar.2018.00438openalex-snapshot

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inferred← citeshttps://doi.org/10.3389/fphys.2020.625974openalex-snapshot

The Heart of the Alzheimer's: A Mindful View of Heart Disease

inferred← citeshttps://doi.org/10.3390/biom13010015openalex-snapshot

Autophagy in Atherosclerotic Plaque Cells: Targeting NLRP3 Inflammasome for Self-Rescue

inferred← citeshttps://doi.org/10.3390/biom15010113openalex-snapshot

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inferred← citeshttps://doi.org/10.3390/biomedicines8060166openalex-snapshot

Mitochondrial Dysfunction and DNA Damage in the Context of Pathogenesis of Atherosclerosis

inferred← citeshttps://doi.org/10.3390/cells10020443openalex-snapshot

Autophagy and Mitophagy as Essential Components of Atherosclerosis

inferred← citeshttps://doi.org/10.3390/cells10030625openalex-snapshot

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inferred← citeshttps://doi.org/10.3390/cells13040353openalex-snapshot

Cellular Senescence, Mitochondrial Dysfunction, and Their Link to Cardiovascular Disease

inferred← citeshttps://doi.org/10.3390/cells7100149openalex-snapshot

Autophagy in Metabolic Age-Related Human Diseases

inferred← citeshttps://doi.org/10.3390/cells9102146openalex-snapshot

Cell Senescence, Multiple Organelle Dysfunction and Atherosclerosis

inferred← citeshttps://doi.org/10.3390/ijms232314741openalex-snapshot

Targeting Mitochondrial Dynamics Proteins for the Development of Therapies for Cardiovascular Diseases

inferred← citeshttps://doi.org/10.3390/ijms26073252openalex-snapshot

Autophagy and Its Association with Macrophages in Clonal Hematopoiesis Leading to Atherosclerosis

inferred← citeshttps://doi.org/10.3390/jcm9030892openalex-snapshot

Mitophagy in Cardiovascular Diseases

inferred← citeshttps://doi.org/10.35537/10915/118961openalex-snapshot

Respuestas celulares a K107del, una mutante natural de apolipoproteína A-I humana con incrementado riesgo aterogénico

inferred← citeshttps://doi.org/10.3892/mmr.2021.12403openalex-snapshot

Salvianolic acid B improves autophagic dysfunction and decreases the apoptosis of cholesterol crystal‑induced macrophages via inhibiting the Akt/mTOR signaling pathway

inferred← citeshttps://doi.org/10.53301/lw/143317openalex-snapshot

Impaired autophagy as an etiological factor of various disease entities

inferred← citeshttps://doi.org/10.53941/ijddp.2024.100021openalex-snapshot

The Role of Autophagy in Heart Disease

inferred← citeshttps://doi.org/10.64898/2026.09.01.748593openalex-snapshot

Impaired Proteostasis is an Early Feature of the Diabetic Heart in Humans and Mice

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