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Activation of NRF2 ameliorates oxidative stress and cystogenesis in autosomal dominant polycystic kidney disease
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1006/bbrc.1997.6943openalex-snapshot
An Nrf2/Small Maf Heterodimer Mediates the Induction of Phase II Detoxifying Enzyme Genes through Antioxidant Response Elements
inferredcites →https://doi.org/10.1007/128_2012_339openalex-snapshot
Keap1–Nrf2 Signaling: A Target for Cancer Prevention by Sulforaphane
inferredcites →https://doi.org/10.1016/j.bbadis.2017.08.003openalex-snapshot
Genetics and mechanisms of hepatic cystogenesis
inferredcites →https://doi.org/10.1016/j.cell.2013.03.035openalex-snapshot
Master Transcription Factors and Mediator Establish Super-Enhancers at Key Cell Identity Genes
inferredcites →https://doi.org/10.1016/j.cell.2017.02.007openalex-snapshot
A Phase Separation Model for Transcriptional Control
inferredcites →https://doi.org/10.1016/j.cell.2018.10.042openalex-snapshot
Transcription Factors Activate Genes through the Phase-Separation Capacity of Their Activation Domains
inferredcites →https://doi.org/10.1016/j.cellsig.2019.109495openalex-snapshot
Metabolic reprogramming and the role of mitochondria in polycystic kidney disease
inferredcites →https://doi.org/10.1016/j.clinbiochem.2018.04.010openalex-snapshot
Systemic redox biomarkers and their relationship to prognostic risk markers in autosomal dominant polycystic kidney disease and IgA nephropathy
inferredcites →https://doi.org/10.1016/j.ebiom.2016.01.027openalex-snapshot
Fatty Acid Oxidation is Impaired in An Orthologous Mouse Model of Autosomal Dominant Polycystic Kidney Disease
inferredcites →https://doi.org/10.1016/j.freeradbiomed.2019.01.016openalex-snapshot
Redox regulation by NRF2 in aging and disease
inferredcites →https://doi.org/10.1016/j.tips.2017.04.005openalex-snapshot
Targeting Free Radicals in Oxidative Stress-Related Human Diseases
inferredcites →https://doi.org/10.1016/s0140-6736(18)32782-xopenalex-snapshot
Autosomal dominant polycystic kidney disease
inferredcites →https://doi.org/10.1038/ki.2012.439openalex-snapshot
Targeting the transcription factor Nrf2 to ameliorate oxidative stress and inflammation in chronic kidney disease
inferredcites →https://doi.org/10.1038/ki.2014.427openalex-snapshot
Glycogen synthase kinase-3β promotes cyst expansion in polycystic kidney disease
inferredcites →https://doi.org/10.1038/nature11247openalex-snapshot
An integrated encyclopedia of DNA elements in the human genome
inferredcites →https://doi.org/10.1038/nrd4023openalex-snapshot
Pharmacological approaches to restore mitochondrial function
inferredcites →https://doi.org/10.1038/s41573-018-0008-xopenalex-snapshot
Therapeutic targeting of the NRF2 and KEAP1 partnership in chronic diseases
inferredcites →https://doi.org/10.1038/s41581-018-0051-1openalex-snapshot
Metabolism and mitochondria in polycystic kidney disease research and therapy
inferredcites →https://doi.org/10.1038/s41581-019-0183-yopenalex-snapshot
The pathobiology of polycystic kidney disease from a metabolic viewpoint
inferredcites →https://doi.org/10.1038/s41586-018-0174-3openalex-snapshot
Phase-separation mechanism for C-terminal hyperphosphorylation of RNA polymerase II
inferredcites →https://doi.org/10.1038/s41586-019-1464-0openalex-snapshot
Pol II phosphorylation regulates a switch between transcriptional and splicing condensates
inferredcites →https://doi.org/10.1038/s41598-018-20856-6openalex-snapshot
A cleavage product of Polycystin-1 is a mitochondrial matrix protein that affects mitochondria morphology and function when heterologously expressed
inferredcites →https://doi.org/10.1038/s41598-019-45539-8openalex-snapshot
Tolvaptan activates the Nrf2/HO-1 antioxidant pathway through PERK phosphorylation
inferredcites →https://doi.org/10.1039/c3np70065hopenalex-snapshot
Natural product-derived pharmacological modulators of Nrf2/ARE pathway for chronic diseases
inferredcites →https://doi.org/10.1073/pnas.91.21.9926openalex-snapshot
Isolation of NF-E2-related factor 2 (Nrf2), a NF-E2-like basic leucine zipper transcriptional activator that binds to the tandem NF-E2/AP1 repeat of the beta-globin locus control region.
inferredcites →https://doi.org/10.1080/00313020120034858openalex-snapshot
IMMUNOHISTOCHEMICAL EXPRESSION OF HORMONE RECEPTORS IN INVASIVE BREAST CARCINOMA: CORRELATION OF RESULTS OF H-SCORE WITH PATHOLOGICAL PARAMETERS
inferredcites →https://doi.org/10.1089/ars.2011.4123openalex-snapshot
Redox Regulation of Mitochondrial Function
inferredcites →https://doi.org/10.1089/ars.2016.6804openalex-snapshot
The Kidney: An Organ in the Front Line of Oxidative Stress-Associated Pathologies
inferredcites →https://doi.org/10.1089/ars.2017.7342openalex-snapshot
Transcriptional Regulation by Nrf2
inferredcites →https://doi.org/10.1093/nar/gks409openalex-snapshot
Identification of novel NRF2-regulated genes by ChIP-Seq: influence on retinoid X receptor alpha
inferredcites →https://doi.org/10.1093/nar/gks827openalex-snapshot
Nrf2–MafG heterodimers contribute globally to antioxidant and metabolic networks
inferredcites →https://doi.org/10.1093/ndt/gfz106.fp117openalex-snapshot
FP117SAFETY AND EFFICACY OF BARDOXOLONE METHYL IN PATIENTS WITH RARE CHRONIC KIDNEY DISEASES
inferredcites →https://doi.org/10.1093/toxsci/kfw269openalex-snapshot
Candidate Risk Factors and Mechanisms for Tolvaptan-Induced Liver Injury Are Identified Using a Collaborative Cross Approach
inferredcites →https://doi.org/10.1097/maj.0b013e31821f0552openalex-snapshot
Changes in Urinary Albumin Excretion, Inflammatory and Oxidative Stress Markers in ADPKD Patients with Hypertension
inferredcites →https://doi.org/10.1101/gad.13.1.76openalex-snapshot
Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain
inferredcites →https://doi.org/10.1126/sciadv.aaw3593openalex-snapshot
Activation of P-TEFb by cAMP-PKA signaling in autosomal dominant polycystic kidney disease
inferredcites →https://doi.org/10.1126/science.aaf4382openalex-snapshot
Liquid phase condensation in cell physiology and disease
inferredcites →https://doi.org/10.1126/science.aar3958openalex-snapshot
Coactivator condensation at super-enhancers links phase separation and gene control
inferredcites →https://doi.org/10.1126/science.aar4199openalex-snapshot
Mediator and RNA polymerase II clusters associate in transcription-dependent condensates
inferredcites →https://doi.org/10.1128/mcb.00337-17openalex-snapshot
Mitochondrial Abnormality Facilitates Cyst Formation in Autosomal Dominant Polycystic Kidney Disease
inferredcites →https://doi.org/10.1128/mcb.00785-15openalex-snapshot
The Mediator Subunit MED16 Transduces NRF2-Activating Signals into Antioxidant Gene Expression
inferredcites →https://doi.org/10.1128/mcb.01639-08openalex-snapshot
Acetylation of Nrf2 by p300/CBP Augments Promoter-Specific DNA Binding of Nrf2 during the Antioxidant Response
inferredcites →https://doi.org/10.1128/mcb.15.8.4184openalex-snapshot
Cloning and Characterization of a Novel Erythroid Cell-Derived CNC Family Transcription Factor Heterodimerizing with the Small Maf Family Proteins
inferredcites →https://doi.org/10.1146/annurev-biochem-061516-045037openalex-snapshot
Oxidative Stress
inferredcites →https://doi.org/10.1152/ajprenal.00194.2015openalex-snapshot
Concomitant use of rapamycin and rosiglitazone delays the progression of polycystic kidney disease in Han:SPRD rats: a study of the mechanism of action
inferredcites →https://doi.org/10.1152/ajprenal.00327.2014openalex-snapshot
Endothelial dysfunction and oxidative stress in polycystic kidney disease
inferredcites →https://doi.org/10.1152/physrev.00023.2017openalex-snapshot
The KEAP1-NRF2 System: a Thiol-Based Sensor-Effector Apparatus for Maintaining Redox Homeostasis
inferredcites →https://doi.org/10.1155/2019/2716870openalex-snapshot
Sulforaphane: Its “Coming of Age” as a Clinically Relevant Nutraceutical in the Prevention and Treatment of Chronic Disease
inferredcites →https://doi.org/10.1159/000475890openalex-snapshot
Targeting the KEAP1-NRF2 System to Prevent Kidney Disease Progression
inferredcites →https://doi.org/10.1172/jci72272openalex-snapshot
Genetic mechanisms and signaling pathways in autosomal dominant polycystic kidney disease
inferredcites →https://doi.org/10.14348/molcells.2018.2297openalex-snapshot
Targeting Super-Enhancers for Disease Treatment and Diagnosis.
inferredcites →https://doi.org/10.1681/asn.2015030231openalex-snapshot
2-Deoxy-d-Glucose Ameliorates PKD Progression
inferredcites →https://doi.org/10.1681/asn.2018060590openalex-snapshot
A Practical Guide for Treatment of Rapidly Progressive ADPKD with Tolvaptan
inferredcites →https://doi.org/10.1681/asn.v134991openalex-snapshot
Oxidant Stress and Reduced Antioxidant Enzyme Protection in Polycystic Kidney Disease
inferredcites →https://doi.org/10.2174/1381612825666190112165206openalex-snapshot
Oxidative Stress and Kidney Function: A Brief Update
inferredcites →https://doi.org/10.2215/cjn.04140510openalex-snapshot
Inflammation, Oxidative Stress, and Insulin Resistance in Polycystic Kidney Disease
inferredcites →https://doi.org/10.2215/cjn.05850518openalex-snapshot
Vascular Dysfunction, Oxidative Stress, and Inflammation in Autosomal Dominant Polycystic Kidney Disease
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3996404openalex-extracted
The C-Terminal Tail of Polycystin-1 Suppresses Cystic Disease in a Mitochondrial Enzyme-Dependent Fashion
inferred← citeshttps://doi.org/10.1002/advs.202104578openalex-snapshot
cAMP‐Induced Nuclear Condensation of CRTC2 Promotes Transcription Elongation and Cystogenesis in Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1002/cbf.4005openalex-snapshot
Icariin alleviates renal inflammation and tubulointerstitial fibrosis via Nrf2‐mediated attenuation of mitochondrial damage
inferred← citeshttps://doi.org/10.1002/exp.20240285openalex-snapshot
EGR1 Nuclear Condensates Promote Renal Cyst Development in Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1002/jcp.30901openalex-snapshot
The role of ferroptosis in the development of acute and chronic kidney diseases
inferred← citeshttps://doi.org/10.1002/jcsm.70166openalex-snapshot
Risk Factors Associated With Sarcopenia in Patients With Chronic Kidney Disease: A Systematic Review and Meta‐Analysis
inferred← citeshttps://doi.org/10.1002/mas.70041openalex-snapshot
Advances in Mass Spectrometry‐Based Proteomics for Kidney Disease Biomarker Discovery and Clinical Translation
inferred← citeshttps://doi.org/10.1002/ptr.8031openalex-snapshot
Taxifolin‐mediated Nrf2 activation ameliorates oxidative stress and apoptosis for the treatment of glucocorticoid‐induced osteonecrosis of the femoral head
inferred← citeshttps://doi.org/10.1002/smll.202404815openalex-snapshot
Natural Polyphenol Delivered Methylprednisolone Achieve Targeted Enrichment for Acute Spinal Cord Injury Therapy
inferred← citeshttps://doi.org/10.1007/s11427-024-2661-3openalex-snapshot
Biomolecular condensates and disease pathogenesis
inferred← citeshttps://doi.org/10.1007/s40265-022-01745-9openalex-snapshot
Drugs in Clinical Development to Treat Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1007/s40620-021-01062-6openalex-snapshot
Therapeutic advances in ADPKD: the future awaits
inferred← citeshttps://doi.org/10.1016/j.ajpath.2024.04.010openalex-snapshot
NAMPT Overexpression Drives Cell Growth in Polycystic Liver Disease through Mitochondrial Metabolism Regulation
inferred← citeshttps://doi.org/10.1016/j.autrev.2024.103576openalex-snapshot
The role of mitochondrial dysfunction in kidney injury and disease
inferred← citeshttps://doi.org/10.1016/j.bbadis.2023.166987openalex-snapshot
Transcriptomic profiling of Polycystic Kidney Disease identifies paracrine factors in the early cyst microenvironment
inferred← citeshttps://doi.org/10.1016/j.bbadis.2024.167652openalex-snapshot
Epigenetics in autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2026.120211openalex-snapshot
Targeted analysis of salicylate binding partners in renal epithelial cells reveals major AMPK dependency
inferred← citeshttps://doi.org/10.1016/j.bbcan.2025.189418openalex-snapshot
Liquid-liquid phase separation: A dynamic driver of hepatocellular carcinoma
inferred← citeshttps://doi.org/10.1016/j.bcp.2025.117222openalex-snapshot
Targeting TRAF6 inhibits cystogenesis in autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2023.122329openalex-snapshot
Folate conjugated nanomedicines for selective inhibition of mTOR signaling in polycystic kidneys at clinically relevant doses
inferred← citeshttps://doi.org/10.1016/j.biopha.2023.115617openalex-snapshot
CDDO-Me ameliorates podocyte injury through anti-oxidative stress and regulation of actin cytoskeleton in adriamycin nephropathy
inferred← citeshttps://doi.org/10.1016/j.biopha.2024.116802openalex-snapshot
Potential role of molecular hydrogen therapy on oxidative stress and redox signaling in chronic kidney disease
inferred← citeshttps://doi.org/10.1016/j.ekir.2021.04.023openalex-snapshot
Chronic Inflammation in Chronic Kidney Disease Progression: Role of Nrf2
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2021.03.034openalex-snapshot
Regulation of Nrf2 by phosphorylation: Consequences for biological function and therapeutic implications
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2022.06.226openalex-snapshot
Nonalcoholic steatohepatitis and mechanisms by which it is ameliorated by activation of the CNC-bZIP transcription factor Nrf2
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.05.003openalex-snapshot
An antioxidant response element regulates the HIF1α axis in breast cancer cells
inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2025.10.281openalex-snapshot
Arbutin as a therapeutic agent against calcium oxalate-induced crystal deposition and kidney injury
inferred← citeshttps://doi.org/10.1016/j.idairyj.2026.106843openalex-snapshot
Lactiplantibacillus plantarum R6-3 from Sayram Ketteki alleviates oxidative stress in D-galactose-induced aging mice by restoring gut microbiota and revitalizing hepatic Nrf2-antioxidant pathway
inferred← citeshttps://doi.org/10.1016/j.isci.2024.110773openalex-snapshot
β-hydroxybutyrate recapitulates the beneficial effects of ketogenic metabolic therapy in polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.jare.2026.05.006openalex-snapshot
Renal tubular Nfe2l1 protect cisplatin-induced acute kidney injury via suppressing ACSL4-dependent ferroptosis
inferred← citeshttps://doi.org/10.1016/j.jbc.2023.104810openalex-snapshot
A web-based integrative transcriptome analysis, RNAseqChef, uncovers the cell/tissue type-dependent action of sulforaphane
inferred← citeshttps://doi.org/10.1016/j.jbc.2023.105158openalex-snapshot
Cleavage fragments of the C-terminal tail of polycystin-1 are regulated by oxidative stress and induce mitochondrial dysfunction
inferred← citeshttps://doi.org/10.1016/j.jep.2022.115643openalex-snapshot
Jieduquyuziyin prescription promotes the efficacy of prednisone via upregulating Nrf2 in MRL/lpr kidneys
inferred← citeshttps://doi.org/10.1016/j.kint.2022.10.022openalex-snapshot
Transcriptional programming of pathogenic genes in polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.kint.2023.02.010openalex-snapshot
Drug repurposing in autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.lfs.2026.124283openalex-snapshot
Emodin retards renal cyst progression in ADPKD by inhibiting cell proliferation and decreasing oxidative stress
inferred← citeshttps://doi.org/10.1016/j.phanu.2022.100294openalex-snapshot
Adult dominant polycystic kidney disease: A prototypical disease for pharmanutrition interventions
inferred← citeshttps://doi.org/10.1016/j.phrs.2024.107525openalex-snapshot
Antagonising Yin Yang 1 ameliorates the symptoms of lupus nephritis via modulating T lymphocyte signaling
inferred← citeshttps://doi.org/10.1016/j.redox.2021.102043openalex-snapshot
Proteasome dysfunction under compromised redox metabolism dictates liver injury in NASH through ASK1/PPARγ binodal complementary modules
inferred← citeshttps://doi.org/10.1016/j.redox.2025.103593openalex-snapshot
Keap1-independent Nrf2 regulation: A novel therapeutic target for treating kidney disease
inferred← citeshttps://doi.org/10.1016/j.slasd.2026.100338openalex-snapshot
From phenotypic screening to target and compound prioritization for autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.1016/j.toxlet.2023.08.002openalex-snapshot
Nrf2 protects against methamphetamine-induced nephrotoxicity by mitigating oxidative stress and autophagy in mice
inferred← citeshttps://doi.org/10.1016/j.xcrm.2026.102720openalex-snapshot
Patient-derived kidney organoids recapitulate ADPKD and facilitate the identification of Rho pathway inhibitors as candidate therapeutics
inferred← citeshttps://doi.org/10.1038/s41421-025-00820-1openalex-snapshot
A phosphorylation switch in the Mediator MED15 controls cellular senescence and cognitive decline
inferred← citeshttps://doi.org/10.1038/s41467-023-37449-1openalex-snapshot
The C-terminal tail of polycystin-1 suppresses cystic disease in a mitochondrial enzyme-dependent fashion
inferred← citeshttps://doi.org/10.1038/s41467-024-45353-5openalex-snapshot
G-quadruplexes promote the motility in MAZ phase-separated condensates to activate CCND1 expression and contribute to hepatocarcinogenesis
inferred← citeshttps://doi.org/10.1038/s41467-025-61887-8openalex-snapshot
Developing serum proteomics based prediction models of disease progression in ADPKD
inferred← citeshttps://doi.org/10.1038/s41573-024-00979-4openalex-snapshot
Targeting ROS in cancer: rationale and strategies
inferred← citeshttps://doi.org/10.1038/s41580-024-00789-xopenalex-snapshot
Transcription regulation by biomolecular condensates
inferred← citeshttps://doi.org/10.1038/s41581-023-00767-0openalex-snapshot
Biomolecular condensates in kidney physiology and disease
inferred← citeshttps://doi.org/10.1038/s41581-026-01131-8openalex-snapshot
Ferroptosis in kidney disease
inferred← citeshttps://doi.org/10.1038/s41598-022-14815-5openalex-snapshot
Renal transcriptome profiles in mice reveal the need for sufficient water intake irrespective of the drinking water type
inferred← citeshttps://doi.org/10.1038/s42003-021-02730-wopenalex-snapshot
Metabolic derangement in polycystic kidney disease mouse models is ameliorated by mitochondrial-targeted antioxidants
inferred← citeshttps://doi.org/10.1038/s44321-024-00167-2openalex-snapshot
Melanin-like nanoparticles slow cyst growth in ADPKD by dual inhibition of oxidative stress and CREB
inferred← citeshttps://doi.org/10.1038/s44321-025-00239-xopenalex-snapshot
Metabolic reprogramming in polycystic kidney disease and other renal ciliopathies
inferred← citeshttps://doi.org/10.1053/j.akdh.2023.01.003openalex-snapshot
Autosomal Dominant Polycystic Kidney Disease Therapies on the Horizon
inferred← citeshttps://doi.org/10.1073/pnas.2317344121openalex-snapshot
A Synthetic Agent Ameliorates Polycystic Kidney Disease by Promoting Apoptosis of Cystic Cells Through Increased Oxidative Stress
inferred← citeshttps://doi.org/10.1080/00498254.2025.2465239openalex-snapshot
Nrf2 signaling pathway studies in Drosophila melanogaster : parallel roles in human health and insect environmental responses
inferred← citeshttps://doi.org/10.1080/0886022x.2023.2205950openalex-snapshot
Association of dietary total antioxidant capacity with all-cause and cardiovascular mortality in patients with chronic kidney disease: based on two retrospective cohort studies of NHANES
inferred← citeshttps://doi.org/10.1080/15548627.2023.2223468openalex-snapshot
HSPA8 regulates anti-bacterial autophagy through liquid-liquid phase separation
inferred← citeshttps://doi.org/10.1089/ars.2022.0058openalex-snapshot
T Cell Nrf2/Keap1 Gene Editing Using CRISPR/Cas9 and Experimental Kidney Ischemia-Reperfusion Injury
inferred← citeshttps://doi.org/10.1093/advances/nmab131openalex-snapshot
Dietary Interventions in Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1093/gpbjnl/qzaf014openalex-snapshot
PhaSeDis: A Manually Curated Database of Phase Separation–disease Associations and Corresponding Small Molecules
inferred← citeshttps://doi.org/10.1093/nar/gkaf1387openalex-snapshot
Nucleolar MED19 regulates 2′-O-methylation of rRNA in supporting cancer cell growth
inferred← citeshttps://doi.org/10.1096/fj.202101270rropenalex-snapshot
Antioxidant enzyme peroxiredoxin 5 regulates cyst growth and ciliogenesis via modulating Plk1 stability
inferred← citeshttps://doi.org/10.1097/hep.0000000000001711openalex-snapshot
Statins halt polycystic liver disease by reprogramming metabolism and normalizing mitochondrial bioenergetics in cystic cholangiocytes
inferred← citeshttps://doi.org/10.1097/imna-d-24-00026openalex-snapshot
Metabolic–Epigenetic Crosstalk and Traditional Chinese Medicine in Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1101/2021.09.15.460510openalex-snapshot
Metabolic Derangement in Polycystic Kidney Disease Mouse Models Is Ameliorated by Mitochondrial-Targeted Antioxidants
inferred← citeshttps://doi.org/10.1101/2021.12.21.473680openalex-snapshot
The C-terminal tail of polycystin-1 suppresses cystic disease in a mitochondrial enzyme-dependent fashion
inferred← citeshttps://doi.org/10.1101/2025.08.12.669905openalex-snapshot
Mediator subunit MED16 cooperates with UBP1-TFCP2 to control the promoter context-dependent transcriptional activity
inferred← citeshttps://doi.org/10.1111/nep.13957openalex-snapshot
Possible role of the mitochondrial genome in the pathogenesis of autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.1111/ped.70000openalex-snapshot
Inhibition of renal fibrosis via Nrf2 activators for unilateral ureteral obstruction in a rat model
inferred← citeshttps://doi.org/10.1152/ajpcell.00222.2026openalex-snapshot
Modulation of cyst growth in autosomal dominant polycystic kidney disease: mechanistic insights and therapeutic opportunities
inferred← citeshttps://doi.org/10.1152/ajpendo.00067.2026openalex-snapshot
β-Hydroxybutyrate: a renoprotective hormone in polycystic kidney disease
inferred← citeshttps://doi.org/10.1152/ajprenal.00205.2023openalex-snapshot
A combination of β-hydroxybutyrate and citrate ameliorates disease progression in a rat model of polycystic kidney disease
inferred← citeshttps://doi.org/10.1152/ajprenal.00471.2025openalex-snapshot
Disruption of polycystin-1 cleavage impairs mitochondrial bioenergetics and calcium uptake in a substrate-dependent manner
inferred← citeshttps://doi.org/10.1152/physrev.00018.2024openalex-snapshot
Physiologic mechanisms underlying polycystic kidney disease
inferred← citeshttps://doi.org/10.1155/2021/1394486openalex-snapshot
Mitoquinone Protects Podocytes from Angiotensin II‐Induced Mitochondrial Dysfunction and Injury via the Keap1‐Nrf2 Signaling Pathway
inferred← citeshttps://doi.org/10.1158/1541-7786.mcr-22-0935openalex-snapshot
Nrf2 Drives Hepatocellular Carcinoma Progression through Acetyl-CoA–Mediated Metabolic and Epigenetic Regulatory Networks
inferred← citeshttps://doi.org/10.11606/t.5.2023.tde-01122023-173544openalex-snapshot
Efeitos de diferentes perfis de deficiência de Pkd1 sobre o fenótipo vascular em artérias de resistência de camundongos
inferred← citeshttps://doi.org/10.1186/s12882-026-05191-7openalex-snapshot
A new conceptual framework for PKD1 in ADPKD; integrating DNA structures and inflammation
inferred← citeshttps://doi.org/10.1186/s12882-026-05407-wopenalex-snapshot
Stage-dependent alterations in MaR1 in pediatric chronic kidney disease: a potential candidate biomarker
inferred← citeshttps://doi.org/10.1186/s12995-021-00332-0openalex-snapshot
Genome-wide mRNA profiling identifies the NRF2-regulated lymphocyte oxidative stress status in patients with silicosis
inferred← citeshttps://doi.org/10.1371/journal.pone.0289778openalex-snapshot
In vivo Polycystin-1 interactome using a novel Pkd1 knock-in mouse model
inferred← citeshttps://doi.org/10.14814/phy2.15961openalex-snapshot
NRF2 in kidney physiology and disease
inferred← citeshttps://doi.org/10.1681/asn.0000000592openalex-snapshot
Notch2 Inhibition and Kidney Cyst Growth in Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.1681/asn.2021010101openalex-snapshot
Integrative Cistromic and Transcriptomic Analyses Identify CREB Target Genes in Cystic Renal Epithelial Cells
inferred← citeshttps://doi.org/10.1681/asn.2021111425openalex-snapshot
Nuclear Condensation of CDYL Links Histone Crotonylation and Cystogenesis in Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.21203/rs.3.rs-270262/v1openalex-snapshot
Genome-Wide mRNA Profiling Identifies the NRF2- Regulated Lymphocyte Oxidative Status in Patients with Silicosis
inferred← citeshttps://doi.org/10.2174/0115733998327164240923070313openalex-snapshot
An Association between Bilirubin and Diabetic Retinopathy in Patients with Type 2 Diabetes Mellitus: An Effect Modification by Nrf2 Polymorphisms
inferred← citeshttps://doi.org/10.26599/nr.2025.94907124openalex-snapshot
Facile fabrication of polyphenolic flavonoids nanoparticles via one-pot assembly for treatment of spinal cord injury
inferred← citeshttps://doi.org/10.33393/gcnd.2022.2489openalex-snapshot
Rene policistico autosomico dominante: dalla patogenesi alla terapia
inferred← citeshttps://doi.org/10.3389/fcell.2022.937580openalex-snapshot
Novel method for the genomic analysis of PKD1 mutation in autosomal dominant polycystic kidney disease
inferred← citeshttps://doi.org/10.3389/fendo.2023.1085605openalex-snapshot
Cellular senescence of renal tubular epithelial cells in renal fibrosis
inferred← citeshttps://doi.org/10.3389/fgene.2021.691946openalex-snapshot
Roles of Phase Separation for Cellular Redox Maintenance
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1700004openalex-snapshot
Targeting ferroptosis: novel therapeutic approaches and intervention strategies for kidney diseases
inferred← citeshttps://doi.org/10.3389/fmolb.2022.981963openalex-snapshot
Emerging therapies for autosomal dominant polycystic kidney disease with a focus on cAMP signaling
inferred← citeshttps://doi.org/10.3389/fphys.2020.619740openalex-snapshot
Ophiopogonin A Alleviates Hemorrhagic Shock-Induced Renal Injury via Induction of Nrf2 Expression
inferred← citeshttps://doi.org/10.3390/antiox10121859openalex-snapshot
Bioactive Compounds in Oxidative Stress-Mediated Diseases: Targeting the NRF2/ARE Signaling Pathway and Epigenetic Regulation
inferred← citeshttps://doi.org/10.3390/antiox11010038openalex-snapshot
Obacunone Retards Renal Cyst Development in Autosomal Dominant Polycystic Kidney Disease by Activating NRF2
inferred← citeshttps://doi.org/10.3390/antiox11061112openalex-snapshot
Nrf2 Activation in Chronic Kidney Disease: Promises and Pitfalls
inferred← citeshttps://doi.org/10.3390/antiox12051022openalex-snapshot
Activation of NRF2 Signaling Pathway Delays the Progression of Hyperuricemic Nephropathy by Reducing Oxidative Stress
inferred← citeshttps://doi.org/10.3390/antiox13010070openalex-snapshot
The Multifaceted Roles of NRF2 in Cancer: Friend or Foe?
inferred← citeshttps://doi.org/10.3390/antiox13121534openalex-snapshot
JP4-039 Mitigates Cisplatin-Induced Acute Kidney Injury by Inhibiting Oxidative Stress and Blocking Apoptosis and Ferroptosis in Mice
inferred← citeshttps://doi.org/10.3390/antiox15040409openalex-snapshot
The AMPK/NRF2/FOXO Axis in CKD—Molecular and Clinical Perspectives
inferred← citeshttps://doi.org/10.3390/antiox15091116openalex-snapshot
Oxidative Stress and Mitochondrial Dysfunction in Chronic Kidney Disease: From Molecular Mechanisms to Biomarkers and Targeted Therapies
inferred← citeshttps://doi.org/10.3390/app14031052openalex-snapshot
Sulforaphane Target Protein Prediction: A Bioinformatics Analysis
inferred← citeshttps://doi.org/10.3390/biomedicines13071596openalex-snapshot
Metabolic Reprogramming in Autosomal Dominant Polycystic Kidney Disease: Role in Cystogenesis and Novel Therapeutic Approaches
inferred← citeshttps://doi.org/10.3390/cancers12113458openalex-snapshot
Role of the KEAP1-NRF2 Axis in Renal Cell Carcinoma
inferred← citeshttps://doi.org/10.3390/cells10092270openalex-snapshot
Glycogen Synthase Kinase-3: A Focal Point for Advancing Pathogenic Inflammation in Depression
inferred← citeshttps://doi.org/10.3390/cells13070610openalex-snapshot
Polycystin-2 Mediated Calcium Signalling in the Dictyostelium Model for Autosomal Dominant Polycystic Kidney Disease
inferred← citeshttps://doi.org/10.3390/genes13081324openalex-snapshot
Nrf2/HO-1 Signaling Stimulation through Acetyl-11-Keto-Beta-Boswellic Acid (AKBA) Provides Neuroprotection in Ethidium Bromide-Induced Experimental Model of Multiple Sclerosis
inferred← citeshttps://doi.org/10.3390/ijms23063317openalex-snapshot
Autosomal Dominant Polycystic Kidney Disease: From Pathophysiology of Cystogenesis to Advances in the Treatment
inferred← citeshttps://doi.org/10.3390/ijms24076053openalex-snapshot
Insights into the Molecular Mechanisms of NRF2 in Kidney Injury and Diseases
inferred← citeshttps://doi.org/10.3390/ijms24097914openalex-snapshot
Proteomics Analysis of R-Ras Deficiency in Oxygen Induced Retinopathy
inferred← citeshttps://doi.org/10.3390/ijms26157471openalex-snapshot
NRF2 Dysregulation and Therapeutic Insights Across Chronic Kidney Diseases
inferred← citeshttps://doi.org/10.3390/jcm12020668openalex-snapshot
Review of the Use of Animal Models of Human Polycystic Kidney Disease for the Evaluation of Experimental Therapeutic Modalities
inferred← citeshttps://doi.org/10.3390/metabo12010058openalex-snapshot
Abnormal Iron and Lipid Metabolism Mediated Ferroptosis in Kidney Diseases and Its Therapeutic Potential
inferred← citeshttps://doi.org/10.7554/elife.108806openalex-snapshot
Mediator subunit MED16 cooperates with UBP1-TFCP2 to control the promoter context-dependent transcriptional activity
inferred← citeshttps://doi.org/10.7554/elife.108806.1openalex-snapshot
Mediator Subunit MED16 Collaborates with UBP1-TFCP2 to Control Transcriptional Activation or Repression via Promoter Positional Specificity
inferred← citeshttps://doi.org/10.7554/elife.108806.2openalex-snapshot
Mediator subunit MED16 cooperates with UBP1-TFCP2 to control the promoter context-dependent transcriptional activity
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