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Increased Autoreactivity of the Complement-Activating Molecule Mannan-Binding Lectin in a Type 1 Diabetes Model
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/9783527628612.ch1openalex-snapshot
Isolation and Enrichment of Glomeruli Using Sieving Techniques
inferredcites →https://doi.org/10.1002/dmrr.1079openalex-snapshot
Binding of mannose‐binding lectin to fructosamines: a potential link between hyperglycaemia and complement activation in diabetes
inferredcites →https://doi.org/10.1007/bf00953996openalex-snapshot
Lectin binding in the diabetic rat kidney
inferredcites →https://doi.org/10.1007/bf01680452openalex-snapshot
Diabetes mellitus associated changes in glomerular glycocompounds: A fluorescence microscopical study
inferredcites →https://doi.org/10.1007/s00125-004-1594-1openalex-snapshot
Increased levels of mannan-binding lectin in type 1 diabetic patients with incipient and overt nephropathy
inferredcites →https://doi.org/10.1007/s00125-007-0686-0openalex-snapshot
Mannose-binding lectin deficiency attenuates renal changes in a streptozotocin-induced model of type 1 diabetes in mice
inferredcites →https://doi.org/10.1007/s00125-010-1742-8openalex-snapshot
Association between mannose-binding lectin, high-sensitivity C-reactive protein and the progression of diabetic nephropathy in type 1 diabetes
inferredcites →https://doi.org/10.1016/0006-291x(80)90852-9openalex-snapshot
Increased glycosylation of glomerular basement membrane collagen in diabetes
inferredcites →https://doi.org/10.1016/j.molimm.2007.06.005openalex-snapshot
Complement activating soluble pattern recognition molecules with collagen-like regions, mannan-binding lectin, ficolins and associated proteins
inferredcites →https://doi.org/10.1038/ki.2014.387openalex-snapshot
Glycosylation patterns of kidney proteins differ in rat diabetic nephropathy
inferredcites →https://doi.org/10.1038/ni.1923openalex-snapshot
Complement: a key system for immune surveillance and homeostasis
inferredcites →https://doi.org/10.1046/j.1365-3083.2001.00908.xopenalex-snapshot
Characterization and Quantification of Mouse Mannan‐Binding Lectins (MBL‐A and MBL‐C) and Study of Acute Phase Responses
inferredcites →https://doi.org/10.1053/j.ackd.2014.03.009openalex-snapshot
Epidemiology and Risk Factors for Diabetic Kidney Disease
inferredcites →https://doi.org/10.1053/j.ajkd.2015.05.001openalex-snapshot
US Renal Data System 2014 Annual Data Report: Epidemiology of Kidney Disease in the United States
inferredcites →https://doi.org/10.1073/pnas.97.10.5450openalex-snapshot
Molecular basis for a link between complement and the vascular complications of diabetes
inferredcites →https://doi.org/10.1084/jem.139.4.793openalex-snapshot
RENAL TRANSPLANTATION IN DIABETES MELLITUS IN RATS
inferredcites →https://doi.org/10.1084/jem.179.3.985openalex-snapshot
Terminal complement proteins C5b-9 release basic fibroblast growth factor and platelet-derived growth factor from endothelial cells.
inferredcites →https://doi.org/10.1111/j.1440-1797.2007.00796.xopenalex-snapshot
Rodent models of streptozotocin‐induced diabetic nephropathy (Methods in Renal Research)
inferredcites →https://doi.org/10.1155/2012/678381openalex-snapshot
Mannan-Binding Lectin in Diabetic Kidney Disease: The Impact of Mouse Genetics in a Type 1 Diabetes Model
inferredcites →https://doi.org/10.1161/circulationaha.110.981365openalex-snapshot
The Alternative Pathway Is Critical for Pathogenic Complement Activation in Endotoxin- and Diet-Induced Atherosclerosis in Low-Density Lipoprotein Receptor–Deficient Mice
inferredcites →https://doi.org/10.1172/jci116418openalex-snapshot
Terminal complement complex C5b-9 stimulates mitogenesis in 3T3 cells.
inferredcites →https://doi.org/10.1172/jci118221openalex-snapshot
Identification and characterization of a gene regulating enzymatic glycosylation which is induced by diabetes and hyperglycemia specifically in rat cardiac tissue.
inferredcites →https://doi.org/10.1210/jc.2003-030742openalex-snapshot
Elevated Levels of Mannan-Binding Lectin in Patients with Type 1 Diabetes
inferredcites →https://doi.org/10.2337/db08-1495openalex-snapshot
Elevated MBL Concentrations Are Not an Indication of Association Between the MBL2 Gene and Type 1 Diabetes or Diabetic Nephropathy
inferredcites →https://doi.org/10.2337/db10-1181openalex-snapshot
Transcriptome Analysis of Human Diabetic Kidney Disease
inferredcites →https://doi.org/10.2337/dc15-0851openalex-snapshot
Increased All-Cause Mortality in Patients With Type 1 Diabetes and High-Expression Mannan-Binding Lectin Genotypes: A 12-Year Follow-up Study
inferredcites →https://doi.org/10.2337/diab.23.9.748openalex-snapshot
Pancreatic Islet Transplantation: Effects on the Glomerular Lesions of Experimental Diabetes in the Rat
inferredcites →https://doi.org/10.2337/diab.24.3.280openalex-snapshot
Studies of the Rate of Regression of the Glomerular Lesions in Diabetic Rats Treated with Pancreatic Islet Transplantation
inferredcites →https://doi.org/10.2337/diabetes.53.10.2653openalex-snapshot
Glycation Inactivation of the Complement Regulatory Protein CD59
inferredcites →https://doi.org/10.2337/diabetes.53.6.1570openalex-snapshot
Association Between Mannose-Binding Lectin and Vascular Complications in Type 1 Diabetes
inferredcites →https://doi.org/10.2337/diabetes.54.5.1523openalex-snapshot
Mannose-Binding Lectin as a Predictor of Microalbuminuria in Type 1 Diabetes
inferredcites →https://doi.org/10.4049/jimmunol.166.11.6820openalex-snapshot
Accelerated Nephrotoxic Nephritis Is Exacerbated in C1q-Deficient Mice
inferredcites →https://doi.org/10.4049/jimmunol.173.5.3044openalex-snapshot
Opsonization with C1q and Mannose-Binding Lectin Targets Apoptotic Cells to Dendritic Cells
inferredcites →https://doi.org/10.5897/ajb11.2313openalex-snapshot
Activation of the lectin complement pathway on human renal glomerular endothelial cells triggered by high glucose and mannose-binding lectin
inferredcites →https://doi.org/10.7326/0003-4819-101-4-527openalex-snapshot
Nonenzymatic Glycosylation and the Pathogenesis of Diabetic Complications
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3965383openalex-extracted
Mannose Binding Lectin Activates the Nuclear Factor-κB and Renal Inflammation in the Progression of Diabetic Nephropathy
inferred← citeshttps://doi.org/10.1002/9781119697473.ch46openalex-snapshot
Pathogenesis of Macrovascular Complications in Diabetes
inferred← citeshttps://doi.org/10.1002/dmrr.2895openalex-snapshot
Incident microalbuminuria and complement factor mannan‐binding lectin‐associated protein 19 in people with newly diagnosed type 1 diabetes
inferred← citeshttps://doi.org/10.1007/s11684-017-0569-9openalex-snapshot
Non-genetic mechanisms of diabetic nephropathy
inferred← citeshttps://doi.org/10.1016/bs.ai.2021.09.001openalex-snapshot
Renal diseases and the role of complement: Linking complement to immune effector pathways and therapeutics
inferred← citeshttps://doi.org/10.1016/j.imbio.2025.153113openalex-snapshot
Complement at the crossroads of inflammation and metabolism: implications for diabetes and metabolic functions
inferred← citeshttps://doi.org/10.1016/j.jpha.2023.09.003openalex-snapshot
The role of innate immunity in diabetic nephropathy and their therapeutic consequences
inferred← citeshttps://doi.org/10.1016/j.molimm.2019.07.031openalex-snapshot
Role of complement in diabetes
inferred← citeshttps://doi.org/10.1038/nrneph.2017.31openalex-snapshot
The role of the complement system in diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/s41374-017-0015-2openalex-snapshot
Endothelial heparan sulfate deficiency reduces inflammation and fibrosis in murine diabetic nephropathy
inferred← citeshttps://doi.org/10.1038/s41598-021-88352-yopenalex-snapshot
The pattern-recognition molecule H-ficolin in relation to diabetic kidney disease, mortality, and cardiovascular events in type 1 diabetes
inferred← citeshttps://doi.org/10.1042/cs20160636openalex-snapshot
Diabetic nephropathy – is this an immune disorder?
inferred← citeshttps://doi.org/10.1080/07853890.2025.2561232openalex-snapshot
Progress in the study of the role of C5a-induced tubular cell senescence in the progression of diabetic kidney disease
inferred← citeshttps://doi.org/10.1089/ars.2021.0125openalex-snapshot
The Complement Pathway: New Insights into Immunometabolic Signaling in Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.1096/fj.202101852ropenalex-snapshot
Mannose‐binding lectin activates the nuclear factor‐κB and renal inflammation in the progression of diabetic nephropathy
inferred← citeshttps://doi.org/10.1098/rsif.2025.0138openalex-snapshot
Advantages of using complement components in preventive and therapeutic vaccine strategies for infectious and non-infectious diseases
inferred← citeshttps://doi.org/10.1111/bph.15307openalex-snapshot
Sweet turning bitter: Carbohydrate sensing of complement in host defence and disease
inferred← citeshttps://doi.org/10.1155/2017/6368780openalex-snapshot
Diabetes Is Associated with Increased Autoreactivity of Mannan-Binding Lectin
inferred← citeshttps://doi.org/10.1155/2017/9403754openalex-snapshot
Global Autorecognition and Activation of Complement by Mannan-Binding Lectin in a Mouse Model of Type 1 Diabetes
inferred← citeshttps://doi.org/10.26724/2079-8334-2022-1-79-204-208openalex-snapshot
LECTINS IN THE INVESTIGATION OF RENAL PATHOLOGIES
inferred← citeshttps://doi.org/10.3389/fendo.2019.00459openalex-snapshot
High Expression of Complement Components in the Kidneys of Type 2 Diabetic Rats With Diabetic Nephropathy
inferred← citeshttps://doi.org/10.3389/fimmu.2022.902063openalex-snapshot
The Complement System in Metabolic-Associated Kidney Diseases
inferred← citeshttps://doi.org/10.3389/fmed.2020.599236openalex-snapshot
Complement, a Therapeutic Target in Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.3390/ijerph18052263openalex-snapshot
MALDI-TOF Protein Profiling Reflects Changes in Type 1 Diabetes Patients Depending on the Increased Amount of Adipose Tissue, Poor Control of Diabetes and the Presence of Chronic Complications
inferred← citeshttps://doi.org/10.3390/ijms24108758openalex-snapshot
Therapeutic Potential of Targeting Complement C5a Receptors in Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.3390/ijms25137204openalex-snapshot
Mannan-Binding Lectin Is Associated with Inflammation and Kidney Damage in a Mouse Model of Type 2 Diabetes
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