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Veno-Occlusive Disease in a Fetus Caused by Pyrrolizidine Alkaloids of Food Origin

https://doi.org/10.1159/000070799
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Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/bf01991912openalex-snapshot
Reversible hepatic veno-occlusive disease in an infant after consumption of pyrrolizidine-containing herbal tea
inferredcites →https://doi.org/10.1016/0002-9343(89)90547-0openalex-snapshot
Comfrey herb tea-induced hepatic veno-occlusive disease
inferredcites →https://doi.org/10.1016/0041-0101(87)90163-2openalex-snapshot
Pulmonary vascular responses induced by the pyrrolizidine alkaloid, monocrotaline, in rats
inferredcites →https://doi.org/10.1016/s0016-5085(85)80027-5openalex-snapshot
Hepatic Venocclusive Disease Associated With the Consumption of Pyrrolizidine Containing Dietary Supplements
inferredcites →https://doi.org/10.1016/s0022-3476(88)80330-5openalex-snapshot
Hepatic veno-occlusive disease in newborn infant of a woman drinking herbal tea
inferredcites →https://doi.org/10.1016/s0027-5107(99)00085-8openalex-snapshot
Cell sensitivity to transplacental mutagenesis by N-ethyl-N-nitrosourea is greatest during early gestation in the Syrian hamster
inferredcites →https://doi.org/10.1016/s0031-9422(01)00192-3openalex-snapshot
Pyrrolizidine alkaloids from Ageratum houstonianum Mill
inferredcites →https://doi.org/10.1016/s0140-6736(56)91692-0openalex-snapshot
VENO-OCCLUSIVE DISEASE OF THE LIVER ESSENTIAL PATHOLOGY
inferredcites →https://doi.org/10.1016/s0140-6736(86)91084-6openalex-snapshot
HELIOTROPIUM LASIOCARPUM FISCH AND MEY IDENTIFIED AS CAUSE OF VENO-OCCLUSIVE DISEASE DUE TO A HERBAL TEA
inferredcites →https://doi.org/10.1016/s0140-6736(89)92154-5openalex-snapshot
COMFREY HERB TEA AND HEPATIC VENO-OCCLUSIVE DISEASE
inferredcites →https://doi.org/10.1016/s0344-0338(99)80108-3openalex-snapshot
Fatal Course of Veno-occlusive Disease of the Liver (Endophlebitis Hepatica obliterans) in a Preterm Infant
inferredcites →https://doi.org/10.1016/s1383-5742(99)00010-1openalex-snapshot
Pyrrolizidine alkaloids in human diet
inferredcites →https://doi.org/10.1017/s1368980000000586openalex-snapshot
The efficacy and safety of comfrey
inferredcites →https://doi.org/10.1021/np50014a001openalex-snapshot
Plant Sources of Hepatotoxic Pyrrolizidine Alkaloids
inferredcites →https://doi.org/10.1055/s-2006-961075openalex-snapshot
Determination of Open Chain Pyrrolizidine Alkaloids by Capillary Gas Chromatography
inferredcites →https://doi.org/10.1055/s-2007-994108openalex-snapshot
Fatal Hepatic Veno-Occlusive Disease in a Newborn Infant
inferredcites →https://doi.org/10.1097/00007691-200006000-00011openalex-snapshot
Clinical and Analytical Aspects of Pyrrolizidine Poisoning Caused by South African Traditional Medicines
inferredcites →https://doi.org/10.1136/bmj.295.6591.183openalex-snapshot
Veno-occlusive disease of the liver secondary to ingestion of comfrey.
inferredcites →https://doi.org/10.2165/00002018-199000051-00020openalex-snapshot
The Harmful Potential of Herbal and Other Plant Products

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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A systematic review and quality assessment of case reports of adverse events for borage (Borago officinalis), coltsfoot (Tussilago farfara) and comfrey (Symphytum officinale)
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Prenatal exposure to integerrimine N‐oxide impaired the maternal care and the physical and behavioral development of offspring rats
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Indirect photodegradation of typical pyrrolizidine alkaloids in water induced by triplet states of dissolved organic matter
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Hepatic sinusoidal obstruction syndrome associated with consumption of Gynura segetum
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Female-specific activation of pregnane X receptor mediates sex difference in fetal hepatotoxicity by prenatal monocrotaline exposure
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Prenatal Exposure to Retrorsine Induces Developmental Toxicity and Hepatotoxicity of Fetal Rats in a Sex-Dependent Manner: The Role of Pregnane X Receptor Activation
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Pyrrolizidinalkaloide und die Seneciose bei Tieren
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Food-Borne Chemical Carcinogens and the Evidence for Human Cancer Risk
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