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Partial Inactivation of Cardiac 14-3-3 Protein in vivo Elicits Endoplasmic Reticulum Stress (ERS) and Activates ERS-initiated Apoptosis in ERS-induced Mice
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1 of the 36 references listed on this page is in the Retraction Watch database as retracted (export 2026-11-04): 10.1677/joe-07-0230 (notice 2010-10-01).
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1016/j.bbrc.2004.06.023openalex-snapshot
Dominant negative 14-3-3 promotes cardiomyocyte apoptosis in early stage of type I diabetes mellitus through activation of JNK
inferredcites →https://doi.org/10.1016/j.bcp.2008.02.003openalex-snapshot
14-3-3 protein regulates Ask1 signaling and protects against diabetic cardiomyopathy
inferredcites →https://doi.org/10.1016/j.cell.2007.04.027openalex-snapshot
ER Stress Triggers Apoptosis by Activating BH3-Only Protein Bim
inferredcites →https://doi.org/10.1016/j.febslet.2006.02.056openalex-snapshot
Glycogen synthase kinase 3β together with 14‐3‐3 protein regulates diabetic cardiomyopathy: Effect of losartan and tempol
inferredcites →https://doi.org/10.1016/j.febslet.2007.04.040openalex-snapshot
Rapid, transient induction of ER stress in the liver and kidney after acute exposure to heavy metal: Evidence from transgenic sensor mice
inferredcites →https://doi.org/10.1016/j.mehy.2006.01.019openalex-snapshot
The alternative role of 14-3-3 zeta as a sweeper of misfolded proteins in disease conditions
inferredcites →https://doi.org/10.1016/j.tcb.2006.05.006openalex-snapshot
14-3-3 proteins: regulation of endoplasmic reticulum localization and surface expression of membrane proteins
inferredcites →https://doi.org/10.1016/s0021-9258(18)42311-3openalex-snapshot
Thapsigargin inhibits contraction and Ca2+ transient in cardiac cells by specific inhibition of the sarcoplasmic reticulum Ca2+ pump.
inferredcites →https://doi.org/10.1016/s0022-2828(03)00122-6openalex-snapshot
Transcription factor Sp1 regulates SERCA2 gene expression in pressure-overloaded hearts: a study using in vivo direct gene transfer into living myocardium
inferredcites →https://doi.org/10.1016/s0092-8674(00)80405-5openalex-snapshot
Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery
inferredcites →https://doi.org/10.1016/s0092-8674(00)80595-4openalex-snapshot
Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor
inferredcites →https://doi.org/10.1016/s1097-2765(00)80330-5openalex-snapshot
Perk Is Essential for Translational Regulation and Cell Survival during the Unfolded Protein Response
inferredcites →https://doi.org/10.1016/s1097-2765(01)00264-7openalex-snapshot
Diabetes Mellitus and Exocrine Pancreatic Dysfunction in Perk−/− Mice Reveals a Role for Translational Control in Secretory Cell Survival
inferredcites →https://doi.org/10.1023/a:1016175429877openalex-snapshot
Endoplasmic reticulum stress-mediated apoptosis in pancreatic β-cells
inferredcites →https://doi.org/10.1038/nrd2755openalex-snapshot
Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities
inferredcites →https://doi.org/10.1038/sj.embor.7400779openalex-snapshot
Mediators of endoplasmic reticulum stress‐induced apoptosis
inferredcites →https://doi.org/10.1074/jbc.m010677200openalex-snapshot
Activation of Caspase-12, an Endoplastic Reticulum (ER) Resident Caspase, through Tumor Necrosis Factor Receptor-associated Factor 2-dependent Mechanism in Response to the ER Stress
inferredcites →https://doi.org/10.1074/jbc.m401413200openalex-snapshot
Glycogen Synthase Kinase-3β Regulates Growth, Calcium Homeostasis, and Diastolic Function in the Heart
inferredcites →https://doi.org/10.1074/jbc.m807308200openalex-snapshot
Chemical Biology Investigation of Cell Death Pathways Activated by Endoplasmic Reticulum Stress Reveals Cytoprotective Modulators of ASK1
inferredcites →https://doi.org/10.1093/emboj/17.19.5708openalex-snapshot
Cloning of mammalian Ire1 reveals diversity in the ER stress responses
inferredcites →https://doi.org/10.1093/emboj/19.3.349openalex-snapshot
14‐3‐3 proteins block apoptosis and differentially regulate MAPK cascades
inferredcites →https://doi.org/10.1101/gad.992302openalex-snapshot
ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats
inferredcites →https://doi.org/10.1111/j.1471-4159.2008.05447.xopenalex-snapshot
Depletion of 14‐3‐3 zeta elicits endoplasmic reticulum stress and cell death, and increases vulnerability to kainate‐induced injury in mouse hippocampal cultures
inferredcites →https://doi.org/10.1126/science.272.5266.1347openalex-snapshot
Stress-Induced Phosphorylation and Activation of the Transcription Factor CHOP (GADD153) by p38 MAP Kinase
inferredcites →https://doi.org/10.1126/science.287.5453.664openalex-snapshot
Coupling of Stress in the ER to Activation of JNK Protein Kinases by Transmembrane Protein Kinase IRE1
inferredcites →https://doi.org/10.1128/mcb.21.4.1249-1259.2001openalex-snapshot
Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State
inferredcites →https://doi.org/10.1139/bcb-79-5-569openalex-snapshot
Mechanism of substrate recognition by the chaperonin GroEL
inferredcites →https://doi.org/10.1152/ajpheart.00980.2009openalex-snapshot
The calcineurin-myocyte enhancer factor 2c pathway mediates cardiac hypertrophy induced by endoplasmic reticulum stress in neonatal rat cardiomyocytes
inferredcites →https://doi.org/10.1152/physiolgenomics.00041.2003openalex-snapshot
Mouse cardiac surgery: comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies
inferredcites →https://doi.org/10.1161/01.cir.0000137836.95625.d4openalex-snapshot
Prolonged Endoplasmic Reticulum Stress in Hypertrophic and Failing Heart After Aortic Constriction
inferredcites →https://doi.org/10.1172/jci26373openalex-snapshot
Endoplasmic reticulum stress: cell life and death decisions
inferredcites →https://doi.org/10.1196/annals.1299.032openalex-snapshot
Caspase‐12 and ER‐Stress‐Mediated Apoptosis
inferredcites →https://doi.org/10.1242/jcs.113.21.3697openalex-snapshot
IRE1 and efferent signaling from the endoplasmic reticulum
inferredcites →https://doi.org/10.1248/bpb.28.957openalex-snapshot
Inactivation of 14-3-3 Protein Exacerbates Cardiac Hypertrophy and Fibrosis through Enhanced Expression of Protein Kinase C .BETA.2 in Experimental Diabetes
inferredcites →https://doi.org/10.1677/joe-07-0230openalex-snapshot
Endoplasmic reticulum stress is involved in myocardial apoptosis of streptozocin-induced diabetic rats
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4641193openalex-extracted
Visualizing the Role of ER Stress in Diabetic Cardiomyopathy: A Bibliometric Analysis
inferred← citeshttps://doi.org/10.1002/biof.1280openalex-snapshot
Role of MAPK‐mediated endoplasmic reticulum stress signaling in the heart during aging in senescence‐accelerated prone mice
inferred← citeshttps://doi.org/10.1007/s11064-014-1369-0openalex-snapshot
Endoplasmic Reticulum Stress is Involved in the Neuroprotective Effect of Propofol
inferred← citeshttps://doi.org/10.1016/j.abb.2014.03.008openalex-snapshot
Chaperone-like activity of monomeric human 14-3-3ζ on different protein substrates
inferred← citeshttps://doi.org/10.1016/j.bbapap.2015.09.006openalex-snapshot
Master redox regulator Trx1 upregulates SMYD1 & modulates lysine methylation
inferred← citeshttps://doi.org/10.1016/j.biocel.2017.05.009openalex-snapshot
Role of 14-3-3η protein on cardiac fatty acid metabolism and macrophage polarization after high fat diet induced type 2 diabetes mellitus
inferred← citeshttps://doi.org/10.1016/j.cellsig.2014.12.021openalex-snapshot
Myocardial 14-3-3η protein protects against mitochondria mediated apoptosis
inferred← citeshttps://doi.org/10.1016/j.ijcard.2015.08.142openalex-snapshot
Depletion of cardiac 14-3-3η protein adversely influences pathologic cardiac remodeling during myocardial infarction after coronary artery ligation in mice
inferred← citeshttps://doi.org/10.1038/cddis.2013.114openalex-snapshot
Role of α-crystallin B as a regulatory switch in modulating cardiomyocyte apoptosis by mitochondria or endoplasmic reticulum during cardiac hypertrophy and myocardial infarction
inferred← citeshttps://doi.org/10.1038/s41420-025-02862-6openalex-snapshot
Endoplasmic reticulum stress-mediated programmed cell death in the tumor microenvironment
inferred← citeshttps://doi.org/10.1097/maj.0b013e3182112bafopenalex-snapshot
Endoplasmic Reticulum Stress Caused by Left Ventricular Hypertrophy in Rats: Effects of Telmisartan
inferred← citeshttps://doi.org/10.1152/japplphysiol.01287.2011openalex-snapshot
Heritability of submaximal exercise heart rate response to exercise training is accounted for by nine SNPs
inferred← citeshttps://doi.org/10.1152/physrev.00004.2012openalex-snapshot
Molecular Biology of Atherosclerosis
inferred← citeshttps://doi.org/10.1159/000329970openalex-snapshot
Attenuation of CHOP-mediated Myocardial Apoptosis in Pressure-overloaded Dominant Negative p38α Mitogen-activated Protein Kinase Mice
inferred← citeshttps://doi.org/10.3892/mmr.2017.7841openalex-snapshot
Melatonin ameliorates myocardial apoptosis by suppressing endoplasmic reticulum stress in rats with long‑term diabetic cardiomyopathy
inferred← citeshttps://doi.org/10.4236/jbm.2018.66004openalex-snapshot
Effects of Endoplasmic Reticulum Stress on Pulmonary Hypertension in Rat Induced by Chronic Hypoxia and Hypercapnia
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