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Increased rate of gluconeogenesis in type II diabetes mellitus. A 13C nuclear magnetic resonance study.
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Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1007/bf00400204openalex-snapshot
On the determination of basal glucose production rate in patients with Type 2 (non-insulin-dependent) diabetes mellitus using primed-continuous 3-3H-glucose infusion
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Fasting hyperglycemia in non-insulin-dependent diabetes mellitus: Contributions of excessive hepatic glucose production and impaired tissue glucose uptake
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Metabolism of [2-14C]acetate and its use in assessing hepatic Krebs cycle activity and gluconeogenesis.
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Quantitation of Muscle Glycogen Synthesis in Normal Subjects and Subjects with Non-Insulin-Dependent Diabetes by13C Nuclear Magnetic Resonance Spectroscopy
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Quantitation of Hepatic Glycogenolysis And Gluconeogenesis in Fasting Humans With 13 C NMR
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Relationships between insulin secretion, insulin action, and fasting plasma glucose concentration in nondiabetic and noninsulin-dependent diabetic subjects.
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Mechanism of increased gluconeogenesis in noninsulin-dependent diabetes mellitus. Role of alterations in systemic, hepatic, and muscle lactate and alanine metabolism.
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Alanine and Gluconeogenesis in Man: Effect of Ethanol
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The Effect of Insulin on the Disposal of Intravenous Glucose: Results from Indirect Calorimetry and Hepatic and Femoral Venous Catheterization
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Increased Rate of Cori Cycle in Obese Subjects With NIDDM and Effect of Weight Reduction
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Predominant Role of Gluconeogenesis in Increased Hepatic Glucose Production in NIDDM
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Liver Glycogen Content in Man in the Postabsorptive State
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Branched-Chain Keto Acid Inhibits Mitochondrial Pyruvate Carrier and Suppresses Gluconeogenesis
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Relationship between gluconeogenesis and phosphoenergetics in rat liver assessed byin vivo13C and31P NMR spectroscopy
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inferred← citeshttps://doi.org/10.1016/b978-0-323-91707-0.00008-5openalex-snapshot
Obesity and insulin resistance: pathophysiology guiding precision therapy
inferred← citeshttps://doi.org/10.1016/b978-0-443-13195-0.00012-0openalex-snapshot
Potent inflammatory biomarkers in insulin resistance
inferred← citeshttps://doi.org/10.1016/b978-0-443-13825-6.00376-9openalex-snapshot
Pathogenesis of type 2 diabetes
inferred← citeshttps://doi.org/10.1016/b978-0-443-32848-0.00023-6openalex-snapshot
Emerging therapeutic approaches in neurobiology of type 2 diabetes
inferred← citeshttps://doi.org/10.1016/b978-012084370-1/50008-3openalex-snapshot
13C Magnetic Resonance Spectroscopy as a Noninvasive Tool for Metabolic Studies on Humans
inferred← citeshttps://doi.org/10.1016/b978-1-4160-2911-3.50032-7openalex-snapshot
10.1016/b978-1-4160-2911-3.50032-7
inferred← citeshttps://doi.org/10.1016/b978-1-4160-5583-9.00041-1openalex-snapshot
Type 2 Diabetes Mellitus
inferred← citeshttps://doi.org/10.1016/b978-1-4377-0324-5.00031-6openalex-snapshot
Type 2 Diabetes Mellitus
inferred← citeshttps://doi.org/10.1016/b978-1-78242-247-1.00006-5openalex-snapshot
Effects of Dietary Lipid Intake on Diabetes
inferred← citeshttps://doi.org/10.1016/j.abb.2015.11.040openalex-snapshot
Keap1/Nrf2 pathway activation leads to a repressed hepatic gluconeogenic and lipogenic program in mice on a high-fat diet
inferred← citeshttps://doi.org/10.1016/j.amjmed.2006.01.009openalex-snapshot
Etiology of Insulin Resistance
inferred← citeshttps://doi.org/10.1016/j.arcmed.2018.10.005openalex-snapshot
Geniposide and Gentiopicroside Suppress Hepatic Gluconeogenesis via Regulation of AKT-FOXO1 Pathway
inferred← citeshttps://doi.org/10.1016/j.arcmed.2018.11.001openalex-snapshot
Inhibition of SphK2 Stimulated Hepatic Gluconeogenesis Associated with Dephosphorylation and Deacetylation of STAT3
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2023.117237openalex-snapshot
Hepatic fatty acid and glucose handling in metabolic disease: Potential impact on cardiovascular disease risk
inferred← citeshttps://doi.org/10.1016/j.autneu.2013.03.002openalex-snapshot
Autonomic control and bariatric procedures
inferred← citeshttps://doi.org/10.1016/j.bbadis.2008.12.001openalex-snapshot
Plasticity of central autonomic neural circuits in diabetes
inferred← citeshttps://doi.org/10.1016/j.bbadis.2018.09.013openalex-snapshot
Application of Magnetic Resonance Spectroscopy in metabolic research
inferred← citeshttps://doi.org/10.1016/j.bbagrm.2020.194626openalex-snapshot
GCN5 acetyltransferase in cellular energetic and metabolic processes
inferred← citeshttps://doi.org/10.1016/j.bbamcr.2026.120130openalex-snapshot
Activation of phosphoenolpyruvate carboxykinase increases intracellular glucose levels in podocytes under hyperglycemic conditions
inferred← citeshttps://doi.org/10.1016/j.bbapap.2003.11.030openalex-snapshot
Structure/function studies of phosphoryl transfer by phosphoenolpyruvate carboxykinase
inferred← citeshttps://doi.org/10.1016/j.bbrc.2010.06.070openalex-snapshot
S961, an insulin receptor antagonist causes hyperinsulinemia, insulin-resistance and depletion of energy stores in rats
inferred← citeshttps://doi.org/10.1016/j.biocel.2007.03.015openalex-snapshot
How does an enzyme recognize CO2?
inferred← citeshttps://doi.org/10.1016/j.biochi.2012.01.017openalex-snapshot
Insulin-sensitizing properties of adiponectin
inferred← citeshttps://doi.org/10.1016/j.biochi.2016.02.014openalex-snapshot
Valproic acid reduces insulin-resistance, fat deposition and FOXO1-mediated gluconeogenesis in type-2 diabetic rat
inferred← citeshttps://doi.org/10.1016/j.biochi.2017.10.004openalex-snapshot
Glucagon, a key factor in the pathophysiology of type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.biopha.2018.10.077openalex-snapshot
Involvement of AMPK activation in the inhibition of hepatic gluconeogenesis by Ficus carica leaf extract in diabetic mice and HepG2 cells
inferred← citeshttps://doi.org/10.1016/j.biopha.2020.111113openalex-snapshot
Catestatin peptide of chromogranin A as a potential new target for several risk factors management in the course of metabolic syndrome
inferred← citeshttps://doi.org/10.1016/j.bmc.2010.05.041openalex-snapshot
Discovery of potent and orally active tricyclic-based FBPase inhibitors
inferred← citeshttps://doi.org/10.1016/j.bmcl.2003.09.022openalex-snapshot
Novel 3,4-Dihydroquinolin-2(1 H )-one inhibitors of human glycogen phosphorylase a
inferred← citeshttps://doi.org/10.1016/j.bmcl.2004.10.048openalex-snapshot
5-Chloroindoloyl glycine amide inhibitors of glycogen phosphorylase: synthesis, in vitro, in vivo, and X-ray crystallographic characterization
inferred← citeshttps://doi.org/10.1016/j.bmcl.2006.01.015openalex-snapshot
Benzoxazole benzenesulfonamides are novel allosteric inhibitors of fructose-1,6-bisphosphatase with a distinct binding mode
inferred← citeshttps://doi.org/10.1016/j.bmcl.2008.12.085openalex-snapshot
Anthranilimide based glycogen phosphorylase inhibitors for the treatment of type 2 diabetes. Part 3: X-ray crystallographic characterization, core and urea optimization and in vivo efficacy
inferred← citeshttps://doi.org/10.1016/j.bmcl.2009.08.081openalex-snapshot
Synthesis, SAR, and X-ray structure of tricyclic compounds as potent FBPase inhibitors
inferred← citeshttps://doi.org/10.1016/j.bmcl.2009.11.093openalex-snapshot
Sulfonylureido thiazoles as fructose-1,6-bisphosphatase inhibitors for the treatment of Type-2 diabetes
inferred← citeshttps://doi.org/10.1016/j.cca.2017.10.034openalex-snapshot
Circulating asprosin concentrations are increased in type 2 diabetes mellitus and independently associated with fasting glucose and triglyceride
inferred← citeshttps://doi.org/10.1016/j.cell.2013.12.016openalex-snapshot
Microbiota-Generated Metabolites Promote Metabolic Benefits via Gut-Brain Neural Circuits
inferred← citeshttps://doi.org/10.1016/j.cell.2016.02.063openalex-snapshot
Asprosin, a Fasting-Induced Glucogenic Protein Hormone
inferred← citeshttps://doi.org/10.1016/j.cell.2017.03.001openalex-snapshot
Selective Chemical Inhibition of PGC-1α Gluconeogenic Activity Ameliorates Type 2 Diabetes
inferred← citeshttps://doi.org/10.1016/j.cellsig.2021.109959openalex-snapshot
Sphingosine 1-phosphate metabolism and insulin signaling
inferred← citeshttps://doi.org/10.1016/j.celrep.2015.06.041openalex-snapshot
Reduced α-MSH Underlies Hypothalamic ER-Stress-Induced Hepatic Gluconeogenesis
inferred← citeshttps://doi.org/10.1016/j.celrep.2022.111259openalex-snapshot
Hepatocyte Rap1a contributes to obesity- and statin-associated hyperglycemia
inferred← citeshttps://doi.org/10.1016/j.celrep.2023.112641openalex-snapshot
Branched-chain keto acids inhibit mitochondrial pyruvate carrier and suppress gluconeogenesis in hepatocytes
inferred← citeshttps://doi.org/10.1016/j.chembiol.2024.09.001openalex-snapshot
Small molecules targeting selective PCK1 and PGC-1α lysine acetylation cause anti-diabetic action through increased lactate oxidation
inferred← citeshttps://doi.org/10.1016/j.clnu.2004.01.008openalex-snapshot
Glycogenolysis during short-term fasting in malaria and healthy subjects?the potential regulatory role of glycogen content on glycogen breakdown: a hypothesis
inferred← citeshttps://doi.org/10.1016/j.cmet.2009.03.013openalex-snapshot
APPL1 Potentiates Insulin-Mediated Inhibition of Hepatic Glucose Production and Alleviates Diabetes via Akt Activation in Mice
inferred← citeshttps://doi.org/10.1016/j.cmet.2017.04.032openalex-snapshot
Gut-Brain Glucose Signaling in Energy Homeostasis
inferred← citeshttps://doi.org/10.1016/j.cmet.2021.04.006openalex-snapshot
A kinome screen reveals that Nemo-like kinase is a key suppressor of hepatic gluconeogenesis
inferred← citeshttps://doi.org/10.1016/j.cmet.2023.07.003openalex-snapshot
Neddylation of phosphoenolpyruvate carboxykinase 1 controls glucose metabolism
inferred← citeshttps://doi.org/10.1016/j.cmet.2024.09.012openalex-snapshot
Overnutrition causes insulin resistance and metabolic disorder through increased sympathetic nervous system activity
inferred← citeshttps://doi.org/10.1016/j.ddmec.2010.05.002openalex-snapshot
How to modulate FXR activity to treat the Metabolic Syndrome
inferred← citeshttps://doi.org/10.1016/j.ddstr.2007.10.003openalex-snapshot
Fructose-1,6-bisphosphatase as a therapeutic target for type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.diabet.2010.05.001openalex-snapshot
Hypothalamic integration of portal glucose signals and control of food intake and insulin sensitivity
inferred← citeshttps://doi.org/10.1016/j.diabres.2014.04.008openalex-snapshot
Intestinal glucose metabolism revisited
inferred← citeshttps://doi.org/10.1016/j.dsx.2013.06.006openalex-snapshot
Pharmacology of signaling pathways: In type 2 diabetes
inferred← citeshttps://doi.org/10.1016/j.ebiom.2017.08.004openalex-snapshot
Salsalate Activates Skeletal Muscle Thermogenesis and Protects Mice from High-Fat Diet Induced Metabolic Dysfunction
inferred← citeshttps://doi.org/10.1016/j.ecl.2008.09.001openalex-snapshot
Hypothalamic Control of Hepatic Glucose Production and Its Potential Role in Insulin Resistance
inferred← citeshttps://doi.org/10.1016/j.ecoenv.2024.117404openalex-snapshot
Prenatal exposure to PM2.5 disturbs the glucose metabolism of offspring fed with high-fat diet in a gender-dependent manner
inferred← citeshttps://doi.org/10.1016/j.ejmech.2014.11.049openalex-snapshot
Discovery of novel indole derivatives as allosteric inhibitors of fructose-1,6-bisphosphatase
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