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Neutrophil extracellular traps contribute to immunothrombosis in COVID-19 acute respiratory distress syndrome

https://doi.org/10.1182/blood.2020007008
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1001/jama.2020.8907openalex-snapshot
Postmortem Examination of Patients With COVID-19
inferredcites →https://doi.org/10.1001/jamacardio.2020.0950openalex-snapshot
Association of Cardiac Injury With Mortality in Hospitalized Patients With COVID-19 in Wuhan, China
inferredcites →https://doi.org/10.1002/ajh.25829openalex-snapshot
Hematological findings and complications of COVID ‐19
inferredcites →https://doi.org/10.1016/j.chest.2019.06.012openalex-snapshot
The Emerging Role of Neutrophil Extracellular Traps in Respiratory Disease
inferredcites →https://doi.org/10.1016/j.jacc.2020.04.031openalex-snapshot
COVID-19 and Thrombotic or Thromboembolic Disease: Implications for Prevention, Antithrombotic Therapy, and Follow-Up
inferredcites →https://doi.org/10.1016/j.prrv.2016.03.007openalex-snapshot
Neutrophil Extracellular Traps in Respiratory Disease: guided anti-microbial traps or toxic webs?
inferredcites →https://doi.org/10.1016/j.thromres.2020.04.013openalex-snapshot
Incidence of thrombotic complications in critically ill ICU patients with COVID-19
inferredcites →https://doi.org/10.1016/s0140-6736(20)30566-3openalex-snapshot
Clinical course and risk factors for mortality of adult inpatients with COVID-19 in Wuhan, China: a retrospective cohort study
inferredcites →https://doi.org/10.1016/s0140-6736(20)30628-0openalex-snapshot
COVID-19: consider cytokine storm syndromes and immunosuppression
inferredcites →https://doi.org/10.1016/s1473-3099(20)30120-1openalex-snapshot
An interactive web-based dashboard to track COVID-19 in real time
inferredcites →https://doi.org/10.1038/nri.2017.105openalex-snapshot
Neutrophil extracellular traps in immunity and disease
inferredcites →https://doi.org/10.1038/nri3345openalex-snapshot
Thrombosis as an intravascular effector of innate immunity
inferredcites →https://doi.org/10.1038/s41418-020-0530-3openalex-snapshot
COVID-19 infection: the perspectives on immune responses
inferredcites →https://doi.org/10.1038/s41572-019-0069-0openalex-snapshot
Acute respiratory distress syndrome
inferredcites →https://doi.org/10.1038/s41586-020-2312-yopenalex-snapshot
The pathogenicity of SARS-CoV-2 in hACE2 transgenic mice
inferredcites →https://doi.org/10.1056/nejmc2009787openalex-snapshot
Large-Vessel Stroke as a Presenting Feature of Covid-19 in the Young
inferredcites →https://doi.org/10.1056/nejmoa2002032openalex-snapshot
Clinical Characteristics of Coronavirus Disease 2019 in China
inferredcites →https://doi.org/10.1056/nejmoa2015432openalex-snapshot
Pulmonary Vascular Endothelialitis, Thrombosis, and Angiogenesis in Covid-19
inferredcites →https://doi.org/10.1056/nejmsb2005114openalex-snapshot
Fair Allocation of Scarce Medical Resources in the Time of Covid-19
inferredcites →https://doi.org/10.1073/pnas.1005743107openalex-snapshot
Extracellular DNA traps promote thrombosis
inferredcites →https://doi.org/10.1074/jbc.m112.353557openalex-snapshot
Neutrophils Express Distinct RNA Receptors in a Non-canonical Way
inferredcites →https://doi.org/10.1083/jcb.200606027openalex-snapshot
Novel cell death program leads to neutrophil extracellular traps
inferredcites →https://doi.org/10.1084/jem.186.8.1357openalex-snapshot
Neutrophil Emigration in the Skin, Lungs, and Peritoneum: Different Requirements for CD11/CD18 Revealed by CD18-deficient Mice
inferredcites →https://doi.org/10.1084/jem.20200652openalex-snapshot
Targeting potential drivers of COVID-19: Neutrophil extracellular traps
inferredcites →https://doi.org/10.1097/shk.0b013e31816a6bb1openalex-snapshot
NEUTROPHIL-DERIVED CIRCULATING FREE DNA (cf-DNA/NETs)
inferredcites →https://doi.org/10.1111/j.1365-2141.2009.08052.xopenalex-snapshot
Platelet von Willebrand factor – structure, function and biological importance
inferredcites →https://doi.org/10.1111/jth.14810openalex-snapshot
ISTH interim guidance on recognition and management of coagulopathy in COVID‐19
inferredcites →https://doi.org/10.1111/jth.14842openalex-snapshot
Venous thromboembolism in COVID‐19 patients
inferredcites →https://doi.org/10.1111/resp.12730openalex-snapshot
Neutrophil extracellular traps are associated with inflammation in chronic airway disease
inferredcites →https://doi.org/10.1126/science.1092385openalex-snapshot
Neutrophil Extracellular Traps Kill Bacteria
inferredcites →https://doi.org/10.1126/scitranslmed.aag1711openalex-snapshot
Cancer cells induce metastasis-supporting neutrophil extracellular DNA traps
inferredcites →https://doi.org/10.1160/th17-09-0630openalex-snapshot
The Pathophysiological Role of Neutrophil Extracellular Traps in Inflammatory Diseases
inferredcites →https://doi.org/10.1161/circresaha.119.314581openalex-snapshot
Extracellular DNA NET-Works With Dire Consequences for Health
inferredcites →https://doi.org/10.1172/jci.insight.138999openalex-snapshot
Neutrophil extracellular traps in COVID-19
inferredcites →https://doi.org/10.1172/jci.insight.98178openalex-snapshot
Maladaptive role of neutrophil extracellular traps in pathogen-induced lung injury
inferredcites →https://doi.org/10.1172/jci.insight.99445openalex-snapshot
Neutrophil accumulation and NET release contribute to thrombosis in HIT
inferredcites →https://doi.org/10.1172/jci137244openalex-snapshot
Clinical and immunological features of severe and moderate coronavirus disease 2019
inferredcites →https://doi.org/10.1172/jci61303openalex-snapshot
Platelets induce neutrophil extracellular traps in transfusion-related acute lung injury
inferredcites →https://doi.org/10.1172/jci83873openalex-snapshot
Neonatal NET-inhibitory factor and related peptides inhibit neutrophil extracellular trap formation
inferredcites →https://doi.org/10.1172/jci84538openalex-snapshot
Neutrophil extracellular traps — the dark side of neutrophils
inferredcites →https://doi.org/10.1182/blood-2003-04-1078openalex-snapshot
Toll-like receptors stimulate human neutrophil function
inferredcites →https://doi.org/10.1182/blood-2008-07-171629openalex-snapshot
Impaired neutrophil extracellular trap (NET) formation: a novel innate immune deficiency of human neonates
inferredcites →https://doi.org/10.1182/blood-2013-07-516484openalex-snapshot
Synchronized integrin engagement and chemokine activation is crucial in neutrophil extracellular trap–mediated sterile inflammation
inferredcites →https://doi.org/10.1182/blood.2020006000openalex-snapshot
COVID-19 and its implications for thrombosis and anticoagulation
inferredcites →https://doi.org/10.1186/s12879-020-05681-5openalex-snapshot
Correlation analysis between disease severity and inflammation-related parameters in patients with COVID-19: a retrospective study
inferredcites →https://doi.org/10.1371/journal.pone.0148142openalex-snapshot
Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis
inferredcites →https://doi.org/10.3324/haematol.2015.140632openalex-snapshot
Histones stimulate von Willebrand factor release in vitro and in vivo
inferredcites →https://doi.org/10.33696/immunology.1.008openalex-snapshot
The Role of NETosis in Systemic Lupus Erythematosus
inferredcites →https://doi.org/10.3389/fimmu.2016.00271openalex-snapshot
Platelets: New Bricks in the Building of Neutrophil Extracellular Traps
inferredcites →https://doi.org/10.3389/fimmu.2016.00311openalex-snapshot
Neutrophil Extracellular Traps in Pulmonary Diseases: Too Much of a Good Thing?
inferredcites →https://doi.org/10.3389/fimmu.2016.00366openalex-snapshot
Neutrophil Extracellular Traps Go Viral
inferredcites →https://doi.org/10.3389/fimmu.2017.00081openalex-snapshot
Neutrophil Extracellular Traps and Its Implications in Inflammation: An Overview
inferredcites →https://doi.org/10.3389/fimmu.2019.02400openalex-snapshot
Platelets and Intravascular Immunity: Guardians of the Vascular Space During Bloodstream Infections and Sepsis
inferredcites →https://doi.org/10.3389/fmed.2018.00121openalex-snapshot
Platelets in Immune Response to Virus and Immunopathology of Viral Infections
inferredcites →https://doi.org/10.3389/fmicb.2018.03174openalex-snapshot
TLR3 Regulated Poly I:C-Induced Neutrophil Extracellular Traps and Acute Lung Injury Partly Through p38 MAP Kinase

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.3646561openalex-extracted
Monocyte-Driven Atypical Cytokine Storm and Aberrant Neutrophil Activation as Key Mediators of COVID19 Disease Severity
inferred← citeshttps://doi.org/10.2139/ssrn.3950749openalex-extracted
Longitudinal Characterization of Circulating Neutrophils Uncovers Distinct Phenotypes Associated with Disease Severity in Hospitalized COVID-19 Patients
inferred← citeshttps://doi.org/10.2139/ssrn.3959670openalex-extracted
Neutrophil Extracellular Traps, Local IL-8 Expression, and Cytotoxic T-Lymphocyte Absence in the Lungs of Fatal COVID-19: A Retrospective Histopathology Cohort Study
inferred← citeshttps://doi.org/10.2139/ssrn.4007576openalex-extracted
Multiplatform Analyses Reveal Distinct Drivers of Systemic Pathogenesis in Adult Versus Pediatric COVID-19
inferred← citeshttps://doi.org/10.2139/ssrn.4052650openalex-extracted
Safety and Efficacy of Mesenchymal Stem Cells in Severe/Critical COVID-19 Patients: A Systematic Review and Meta-Analysis
inferred← citeshttps://doi.org/10.2139/ssrn.4134233openalex-extracted
Prevalence of TTP and its Relationship with Complement Activation in COVID-19 Critically Ill Patients: A Cross-Sectional Study
inferred← citeshttps://doi.org/10.2139/ssrn.4172117openalex-extracted
inferred← citeshttps://doi.org/10.2139/ssrn.4587008openalex-extracted
Association of Vascular Netosis with COVID-19 Severity in Asymptomatic and Symptomatic Patients Infected by Delta and Omicron Variants
inferred← citeshttps://doi.org/10.1001/jama.2022.2361openalex-snapshot
Thromboinflammation and Antithrombotics in COVID-19
inferred← citeshttps://doi.org/10.1001/jama.2022.2910openalex-snapshot
Effect of Antiplatelet Therapy on Survival and Organ Support–Free Days in Critically Ill Patients With COVID-19
inferred← citeshttps://doi.org/10.1002/2211-5463.13779openalex-snapshot
Macrophage‐1 antigen exacerbates histone‐induced acute lung injury and promotes neutrophil extracellular trap formation
inferred← citeshttps://doi.org/10.1002/advs.202300560openalex-snapshot
Exosomes‐transferred LINC00668 Contributes to Thrombosis by Promoting NETs Formation in Inflammatory Bowel Disease
inferred← citeshttps://doi.org/10.1002/advs.202303053openalex-snapshot
Genetically Engineered Cellular Nanovesicle as Targeted DNase I Delivery System for the Clearance of Neutrophil Extracellular Traps in Acute Lung Injury
inferred← citeshttps://doi.org/10.1002/advs.202305554openalex-snapshot
Extracellular Vesicles: The Invisible Heroes and Villains of COVID‐19 Central Neuropathology
inferred← citeshttps://doi.org/10.1002/advs.202504690openalex-snapshot
Engineered Dual‐Function Antibody‐Like Proteins to Combat SARS‐CoV‐2‐Induced Immune Dysregulation and Inflammation
inferred← citeshttps://doi.org/10.1002/advs.202506028openalex-snapshot
Src Reduces Neutrophil Extracellular Traps Generation and Resolves Acute Organ Damage
inferred← citeshttps://doi.org/10.1002/advs.202508695openalex-snapshot
Extracellular Vesicle‐Delivered tRF‐His‐GTG‐1 Reprograms Neutrophil Lipophagy and Triggers Inflammation in COVID‐19
inferred← citeshttps://doi.org/10.1002/ajh.26746openalex-snapshot
Hyper‐inflammation and complement in COVID ‐19
inferred← citeshttps://doi.org/10.1002/art.42094openalex-snapshot
Endothelial Cell–Activating Antibodies in COVID‐19
inferred← citeshttps://doi.org/10.1002/cam4.4753openalex-snapshot
Characterization of coagulopathy and outcomes in cancer patients with severe COVID ‐19 illness: Longitudinal changes in hospitalized cancer patients
inferred← citeshttps://doi.org/10.1002/cti2.1240openalex-snapshot
RGD‐binding integrins and TGF‐β in SARS‐CoV‐2 infections – novel targets to treat COVID‐19 patients?
inferred← citeshttps://doi.org/10.1002/cti2.1350openalex-snapshot
Endothelial cells are not productively infected by SARS‐CoV‐2
inferred← citeshttps://doi.org/10.1002/cti2.1357openalex-snapshot
Blunted sFasL signalling exacerbates TNF‐driven neutrophil necroptosis in critically ill COVID‐19 patients
inferred← citeshttps://doi.org/10.1002/ctm2.1389openalex-snapshot
METTL3‐mediated N6‐methyladenosine exacerbates ferroptosis via m6A‐IGF2BP2‐dependent mitochondrial metabolic reprogramming in sepsis‐induced acute lung injury
inferred← citeshttps://doi.org/10.1002/ctm2.268openalex-snapshot
Neutrophil extracellular traps and von Willebrand factor are allies that negatively influence COVID‐19 outcomes
inferred← citeshttps://doi.org/10.1002/eji.202048959openalex-snapshot
Complement cascade in severe forms of COVID‐19: Recent advances in therapy
inferred← citeshttps://doi.org/10.1002/eji.202149410openalex-snapshot
Innate immune signaling and immunothrombosis: New insights and therapeutic opportunities
inferred← citeshttps://doi.org/10.1002/eji.202149481openalex-snapshot
Metabolic reprograming shapes neutrophil functions in severe COVID‐19
inferred← citeshttps://doi.org/10.1002/eji.202250010openalex-snapshot
Neutrophil activation and neutrophil extracellular traps (NETs) in COVID‐19 ARDS and immunothrombosis
inferred← citeshttps://doi.org/10.1002/eji.202250011openalex-snapshot
GSDMD‐mediated NETosis promotes the development of acute respiratory distress syndrome
inferred← citeshttps://doi.org/10.1002/iid3.1343openalex-snapshot
Post‐COVID pulmonary injury in K18‐hACE2 mice shows persistent neutrophils and neutrophil extracellular trap formation
inferred← citeshttps://doi.org/10.1002/iid3.500openalex-snapshot
Role of neutrophils in acute viral infection
inferred← citeshttps://doi.org/10.1002/iid3.861openalex-snapshot
SARS‐CoV‐2 induced HDL dysfunction may affect the host's response to and recovery from COVID‐19
inferred← citeshttps://doi.org/10.1002/iid3.949openalex-snapshot
The central role of neutrophil extracellular traps (NETs) and by‐products in COVID‐19 related pulmonary thrombosis
inferred← citeshttps://doi.org/10.1002/jcla.24004openalex-snapshot
Assessment of antiphospholipid antibodies and calprotectin as biomarkers for discriminating mild from severe COVID‐19
inferred← citeshttps://doi.org/10.1002/jev2.12117openalex-snapshot
Extracellular vesicle‐mediated endothelial apoptosis and EV‐associated proteins correlate with COVID‐19 disease severity
inferred← citeshttps://doi.org/10.1002/jlb.1mr1221-345ropenalex-snapshot
Neutrophil subsets and their differential roles in viral respiratory diseases
inferred← citeshttps://doi.org/10.1002/jlb.3covcra0720-359ropenalex-snapshot
Neutrophil calprotectin identifies severe pulmonary disease in COVID-19
inferred← citeshttps://doi.org/10.1002/jlb.3mir0221-100ropenalex-snapshot
Platelet extracellular vesicles in COVID-19: Potential markers and makers
inferred← citeshttps://doi.org/10.1002/jlb.4a0322-164ropenalex-snapshot
Neutrophil functional heterogeneity is a fixed phenotype and is associated with distinct gene expression profiles
inferred← citeshttps://doi.org/10.1002/jlb.4covcra0820-481rrropenalex-snapshot
Neutrophils mediate Th17 promotion in COVID-19 patients
inferred← citeshttps://doi.org/10.1002/jlb.5cova1121-626ropenalex-snapshot
VIP plasma levels associate with survival in severe COVID-19 patients, correlating with protective effects in SARS-CoV-2-infected cells
inferred← citeshttps://doi.org/10.1002/jlb.5covr0621-320rropenalex-snapshot
Scientific premise for the involvement of neutrophil extracellular traps (NETs) in vaccine-induced thrombotic thrombocytopenia (VITT)
inferred← citeshttps://doi.org/10.1002/jmv.28122openalex-snapshot
COVID‐19 immunopathology: From acute diseases to chronic sequelae
inferred← citeshttps://doi.org/10.1002/jmv.28161openalex-snapshot
Single‐cell transcriptome atlas reveals protective characteristics of COVID‐19 mRNA vaccine
inferred← citeshttps://doi.org/10.1002/jmv.28209openalex-snapshot
Persistence of neutrophil extracellular traps and anticardiolipin auto‐antibodies in post‐acute phase COVID‐19 patients
inferred← citeshttps://doi.org/10.1002/jmv.28946openalex-snapshot
New‐onset and relapsed thrombotic microangiopathy post‐COVID‐19 vaccination
inferred← citeshttps://doi.org/10.1002/jmv.29328openalex-snapshot
Nasopharyngeal neutrophilic‐retention signatures could predict disease progression in early SARS‐CoV‐2 infection
inferred← citeshttps://doi.org/10.1002/jmv.29887openalex-snapshot
Inflammation‐, immunothrombosis,‐ and autoimmune‐feedback loops may lead to persistent neutrophil self‐stimulation in long COVID
inferred← citeshttps://doi.org/10.1002/jmv.70613openalex-snapshot
Circulating Microclots Are Structurally Associated With Neutrophil Extracellular Traps and Their Amounts Are Elevated in Long COVID Patients
inferred← citeshttps://doi.org/10.1002/jmv.70784openalex-snapshot
Cytokine and Endothelial Activation Patterns Related to Severe and Non‐Severe Respiratory Viral Infections
inferred← citeshttps://doi.org/10.1002/jmv.70932openalex-snapshot
Multiorgan Molecular Landscape of Severe COVID‐19 Revealed by Consensus Gene Signatures and RAB8B Targeting
inferred← citeshttps://doi.org/10.1002/mco2.162openalex-snapshot
Molecular mechanisms and therapeutic target of NETosis in diseases
inferred← citeshttps://doi.org/10.1002/mco2.70063openalex-snapshot
Neutrophil heterogeneity and plasticity: unveiling the multifaceted roles in health and disease
inferred← citeshttps://doi.org/10.1002/mco2.70390openalex-snapshot
Role of Neutrophils in Homeostasis and Diseases
inferred← citeshttps://doi.org/10.1002/mco2.70912openalex-snapshot
Neutrophil Extracellular Traps in Atherosclerosis and Cardiovascular Disease
inferred← citeshttps://doi.org/10.1002/path.5642openalex-snapshot
COVID ‐19: immunopathology, pathophysiological mechanisms, and treatment options
inferred← citeshttps://doi.org/10.1002/pul2.12295openalex-snapshot
Lianhua Qingke ameliorates lipopolysaccharide‐induced lung injury by inhibiting neutrophil extracellular traps formation and pyroptosis
inferred← citeshttps://doi.org/10.1002/rmv.2334openalex-snapshot
Neurological complications associated with Covid‐19; molecular mechanisms and therapeutic approaches
inferred← citeshttps://doi.org/10.1002/rth2.12448openalex-snapshot
Harmonizing hypercoagulable heterogeneity: Baseline VTE risk in COVID‐19
inferred← citeshttps://doi.org/10.1002/rth2.12525openalex-snapshot
Comparison of the coagulopathies associated with COVID‐19 and sepsis
inferred← citeshttps://doi.org/10.1002/rth2.12642openalex-snapshot
The ongoing enigma of SARS‐CoV‐2 and platelet interaction
inferred← citeshttps://doi.org/10.1002/rth2.12709openalex-snapshot
Platelet‐leukocyte interactions in COVID‐19: Contributions to hypercoagulability, inflammation, and disease severity
inferred← citeshttps://doi.org/10.1002/rth2.12808openalex-snapshot
Building a better NET: Neutrophil extracellular trap targeted therapeutics in the treatment of infectious and inflammatory disorders
inferred← citeshttps://doi.org/10.1007/978-1-4939-9544-8_68-1openalex-snapshot
Coronaviruses: Pandemic SARS-CoV-2
inferred← citeshttps://doi.org/10.1007/978-3-030-59261-5_11openalex-snapshot
The Novel Coronavirus and Inflammation
inferred← citeshttps://doi.org/10.1007/978-3-030-70904-4_55-1openalex-snapshot
COVID-19 and Pulmonary Embolism: Incidence and Pathophysiology
inferred← citeshttps://doi.org/10.1007/978-3-030-82938-4_8openalex-snapshot
Endothelial Function and Microcirculation
inferred← citeshttps://doi.org/10.1007/978-3-030-85636-6_14openalex-snapshot
COVID-19 Neurologic Illnesses
inferred← citeshttps://doi.org/10.1007/978-3-030-93433-0_7openalex-snapshot
Nebulized Therapeutics for COVID-19 Pneumonia in Critical Care
inferred← citeshttps://doi.org/10.1007/978-3-031-08045-6_16openalex-snapshot
Association of Alpha 1 Antitrypsin Deficiency with COVID-19 Mortality: Basis for Clinical Trials
inferred← citeshttps://doi.org/10.1007/978-3-031-15478-2_20openalex-snapshot
Antiplatelet Drugs in COVID-19: Mechanism of Action and Effect on Prognosis
inferred← citeshttps://doi.org/10.1007/978-3-031-15478-2_8openalex-snapshot
Acute Vascular Injury in COVID-19
inferred← citeshttps://doi.org/10.1007/978-3-031-21410-3_13openalex-snapshot
An Insight into Platelets at Older Age: Cellular and Clinical Perspectives
inferred← citeshttps://doi.org/10.1007/978-3-031-28012-2_10openalex-snapshot
NMR-Metabolomics in COVID-19 Research
inferred← citeshttps://doi.org/10.1007/978-3-031-59815-9_22openalex-snapshot
COVID-19 Pneumonia and Cytokine Storm Syndrome
inferred← citeshttps://doi.org/10.1007/978-3-031-96352-0_5openalex-snapshot
Platelets in Arterial Thrombosis
inferred← citeshttps://doi.org/10.1007/978-3-031-99609-2_14openalex-snapshot
Microvascular Dysfunction Related to COVID-19 and Systemic Inflammation
inferred← citeshttps://doi.org/10.1007/978-981-19-1008-1_6openalex-snapshot
High Altitude Induced Thrombosis: Challenges and Recent Advancements in Pathogenesis and Management
inferred← citeshttps://doi.org/10.1007/s00005-023-00673-0openalex-snapshot
Immunity in SARS-CoV-2 Infection: Clarity or Mystery? A Broader Perspective in the Third Year of a Worldwide Pandemic
inferred← citeshttps://doi.org/10.1007/s00011-024-01879-4openalex-snapshot
Acute lung injury: a view from the perspective of necroptosis
inferred← citeshttps://doi.org/10.1007/s00011-025-02018-3openalex-snapshot
Unique signatures of airway and systemic immunity in severe COVID-19 patients infected with alpha to Omicron SARS-CoV-2 variants of concern
inferred← citeshttps://doi.org/10.1007/s00011-026-02204-xopenalex-snapshot
Endotheliopathy within the thromboinflammatory network of sepsis-induced coagulopathy
inferred← citeshttps://doi.org/10.1007/s00018-021-03808-8openalex-snapshot
High dimensional profiling identifies specific immune types along the recovery trajectories of critically ill COVID19 patients
inferred← citeshttps://doi.org/10.1007/s00018-022-04126-3openalex-snapshot
Citrullination in the pathology of inflammatory and autoimmune disorders: recent advances and future perspectives
inferred← citeshttps://doi.org/10.1007/s00018-022-04309-yopenalex-snapshot
Role of LL-37 in thrombotic complications in patients with COVID-19
inferred← citeshttps://doi.org/10.1007/s00018-024-05520-9openalex-snapshot
Altered ACE2 and interferon landscape in the COVID-19 microenvironment correlate with the anti-PD-1 response in solid tumors
inferred← citeshttps://doi.org/10.1007/s00059-023-05168-4openalex-snapshot
RAAS inhibition and beyond—cardiovascular medications in patients at risk of or affected by COVID-19
inferred← citeshttps://doi.org/10.1007/s00221-021-06244-zopenalex-snapshot
Neurotropism of SARS-CoV-2 and neurological diseases of the central nervous system in COVID-19 patients
inferred← citeshttps://doi.org/10.1007/s00277-024-05915-5openalex-snapshot
Neutrophil extracellular traps - an a-list-actor in a variety of diseases
inferred← citeshttps://doi.org/10.1007/s00281-022-00928-6openalex-snapshot
COVID-19 Vasculitis and vasculopathy-Distinct immunopathology emerging from the close juxtaposition of Type II Pneumocytes and Pulmonary Endothelial Cells
inferred← citeshttps://doi.org/10.1007/s00292-021-00924-xopenalex-snapshot
COVID-19: Auswirkungen auf das zentrale und periphere Nervensystem
inferred← citeshttps://doi.org/10.1007/s00296-020-04753-8openalex-snapshot
Colchicine as an adjunct to heparin for prophylaxis of venous thromboembolism in patients with COVID-19
inferred← citeshttps://doi.org/10.1007/s00392-024-02464-9openalex-snapshot
NET burden in left atrial blood is associated with biomarkers of thrombosis and cardiac injury in patients with enlarged left atria
inferred← citeshttps://doi.org/10.1007/s00401-024-02725-xopenalex-snapshot
Stage-dependent immunity orchestrates AQP4 antibody-guided NMOSD pathology: a role for netting neutrophils with resident memory T cells in situ
inferred← citeshttps://doi.org/10.1007/s00428-021-03053-1openalex-snapshot
The pulmonary pathology of COVID-19
inferred← citeshttps://doi.org/10.1007/s00428-026-04721-wopenalex-snapshot
Clinicopathological factors potentially relevant to pulmonary intravascular fibrin organisation in patients with COVID-19: an autopsy-based study
inferred← citeshttps://doi.org/10.1007/s10067-020-05529-yopenalex-snapshot
COVID-19, rheumatic diseases and immune dysregulation—a perspective
inferred← citeshttps://doi.org/10.1007/s10142-023-01091-3openalex-snapshot
Using system biology and bioinformatics to identify the influences of COVID-19 co-infection with influenza virus on COPD
inferred← citeshttps://doi.org/10.1007/s10142-025-01603-3openalex-snapshot
Multi-omics integration reveals YWHAE as a key mediator of ferroptosis in ARDS
inferred← citeshttps://doi.org/10.1007/s10555-022-10019-5openalex-snapshot
Of vascular defense, hemostasis, cancer, and platelet biology: an evolutionary perspective
inferred← citeshttps://doi.org/10.1007/s10741-024-10421-xopenalex-snapshot
The role of NETosis in heart failure
inferred← citeshttps://doi.org/10.1007/s10753-024-02102-6openalex-snapshot
PAI-1 Deficiency Promotes NET-mediated Pyroptosis and Ferroptosis during Pseudomonas Aeruginosa-induced Acute Lung Injury by Regulating the PI3K/MAPK/AKT Axis
inferred← citeshttps://doi.org/10.1007/s10753-024-02208-xopenalex-snapshot
Sustained Vascular Inflammatory Effects of SARS-CoV-2 Spike Protein on Human Endothelial Cells
inferred← citeshttps://doi.org/10.1007/s10753-026-02552-0openalex-snapshot
Propofol Inhibits Neutrophil Extracellular Traps Formation and Alleviates Sepsis-Induced Acute Lung Injury
inferred← citeshttps://doi.org/10.1007/s10875-021-01061-zopenalex-snapshot
Elevated Expression Levels of Lung Complement Anaphylatoxin, Neutrophil Chemoattractant Chemokine IL-8, and RANTES in MERS-CoV-Infected Patients: Predictive Biomarkers for Disease Severity and Mortality
inferred← citeshttps://doi.org/10.1007/s10875-026-01980-9openalex-snapshot
NET Biomarkers in COVID-19 and Post-COVID Syndrome: a Comprehensive Analysis
inferred← citeshttps://doi.org/10.1007/s11239-020-02324-zopenalex-snapshot
Neutrophil extracellular traps and thrombosis in COVID-19
inferred← citeshttps://doi.org/10.1007/s11239-021-02468-6openalex-snapshot
ICODE: the international COVID-19 thrombosis biomarkers colloquium—novel soluble biomarkers: circulating cell-free nucleic acids and other molecules
inferred← citeshttps://doi.org/10.1007/s11239-021-02583-4openalex-snapshot
Cellular and molecular mechanisms in COVID-19 coagulopathy: role of inflammation and endotheliopathy
inferred← citeshttps://doi.org/10.1007/s11239-022-02667-9openalex-snapshot
Metformin therapy in COVID-19: inhibition of NETosis
inferred← citeshttps://doi.org/10.1007/s11239-024-03028-4openalex-snapshot
The COVID-19 thrombus: distinguishing pathological, mechanistic, and phenotypic features and management
inferred← citeshttps://doi.org/10.1007/s11239-024-03057-zopenalex-snapshot
Impact of antiplatelets, anticoagulants and cyclic nucleotide stimulators on neutrophil extracellular traps (NETs) and inflammatory markers during COVID-19
inferred← citeshttps://doi.org/10.1007/s11302-021-09814-6openalex-snapshot
A novel definition and treatment of hyperinflammation in COVID-19 based on purinergic signalling
inferred← citeshttps://doi.org/10.1007/s11302-024-10062-7openalex-snapshot
The role of the P2X7 receptor in inactivated SARS-CoV-2-induced lung injury
inferred← citeshttps://doi.org/10.1007/s11306-025-02337-wopenalex-snapshot
Proteomic and metabolomic profiling of plasma reveals physiologic changes in patients with convalescent COVID-19
inferred← citeshttps://doi.org/10.1007/s11357-021-00385-3openalex-snapshot
Increased procoagulant platelet levels are predictive of death in COVID-19
inferred← citeshttps://doi.org/10.1007/s11357-022-00724-yopenalex-snapshot
T-cell cellular stress and reticulocyte signatures, but not loss of naïve T lymphocytes, characterize severe COVID-19 in older adults
inferred← citeshttps://doi.org/10.1007/s11427-021-2022-5openalex-snapshot
Treatment of COVID-19 with MSCs: how does it work?
inferred← citeshttps://doi.org/10.1007/s11427-023-2305-0openalex-snapshot
Biomarkers of aging
inferred← citeshttps://doi.org/10.1007/s12026-022-09293-wopenalex-snapshot
A systematic review on neutrophil extracellular traps and its prognostication role in COVID-19 patients
inferred← citeshttps://doi.org/10.1007/s12291-021-00972-3openalex-snapshot
An Integrated Approach of the Potential Underlying Molecular Mechanistic Paradigms of SARS-CoV-2-Mediated Coagulopathy
inferred← citeshttps://doi.org/10.1007/s12291-022-01082-4openalex-snapshot
Circulating Cell-Free DNA Level in Prediction of COVID-19 Severity and Mortality: Correlation of with Haematology and Serum Biochemical Parameters
inferred← citeshttps://doi.org/10.1007/s12325-020-01539-zopenalex-snapshot
Design and Rationale of a Randomized, Double-Blind, Placebo-Controlled, Phase 2/3 Study Evaluating Dociparstat in Acute Lung Injury Associated with Severe COVID-19
inferred← citeshttps://doi.org/10.1007/s12975-022-01039-yopenalex-snapshot
Neutrophil Extracellular Traps (NETs): A New Therapeutic Target for Neuroinflammation and Microthrombosis After Subarachnoid Hemorrhage?
inferred← citeshttps://doi.org/10.1007/s12975-022-01074-9openalex-snapshot
The Role of Neutrophil Extracellular Traps in Early Microthrombosis and Brain Injury After Subarachnoid Hemorrhage in Mice
inferred← citeshttps://doi.org/10.1007/s13365-021-00948-2openalex-snapshot
Potential mechanisms of cerebrovascular diseases in COVID-19 patients
inferred← citeshttps://doi.org/10.1007/s40139-022-00229-5openalex-snapshot
Platelet-Neutrophil Interactions and Thrombo-inflammatory Complications in Type 2 Diabetes Mellitus
inferred← citeshttps://doi.org/10.1007/s43440-022-00394-9openalex-snapshot
The roles of cellular protease interactions in viral infections and programmed cell death: a lesson learned from the SARS-CoV-2 outbreak and COVID-19 pandemic
inferred← citeshttps://doi.org/10.1007/s43440-022-00438-0openalex-snapshot
Platelets in COVID-19 disease: friend, foe, or both?
inferred← citeshttps://doi.org/10.1007/s44258-023-00009-9openalex-snapshot
Hyperactivity of platelets and increased megakaryopoiesis in COVID-19 patients with acute respiratory distress syndrome
inferred← citeshttps://doi.org/10.1016/b978-0-12-824494-4.00003-5openalex-snapshot
Structure, genomic analysis, and pathogenesis of SARS-CoV-2
inferred← citeshttps://doi.org/10.1016/b978-0-323-82763-8.00258-2openalex-snapshot
Immunomodulating Agents
inferred← citeshttps://doi.org/10.1016/b978-0-323-95374-0.00014-2openalex-snapshot
Understanding the molecular basis of pathogenesis of SARS-CoV-2
inferred← citeshttps://doi.org/10.1016/b978-0-323-96014-4.00174-9openalex-snapshot
COVID-19-Related Coagulopathy
inferred← citeshttps://doi.org/10.1016/b978-0-323-98809-4.00013-9openalex-snapshot
COVID-19
inferred← citeshttps://doi.org/10.1016/b978-0-443-15717-2.00008-1openalex-snapshot
COVID-19-Induced Vascular Coagulopathy
inferred← citeshttps://doi.org/10.1016/b978-0-443-15717-2.00057-3openalex-snapshot
Blood Disorders: Response and Achille Heel in the Framework of SARS-Cov-2- a Short Review
inferred← citeshttps://doi.org/10.1016/b978-0-443-18566-3.00003-7openalex-snapshot
COVID-19 in patients with Systemic Lupus Erythematosus and the antiphospholipid syndrome
inferred← citeshttps://doi.org/10.1016/b978-0-443-19170-1.00002-4openalex-snapshot
Immunothrombotic involvement in COVID-19
inferred← citeshttps://doi.org/10.1016/b978-0-443-30225-1.00013-0openalex-snapshot
Respiratory biomarkers in the pathogenesis of SARS-CoV-2
inferred← citeshttps://doi.org/10.1016/b978-0-7020-7959-7.00028-2openalex-snapshot
COVID-19
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2023.11.003openalex-snapshot
Innate immune responses to SARS-CoV-2
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2025.02.002openalex-snapshot
COVID-19 related complications
inferred← citeshttps://doi.org/10.1016/bs.pmbts.2025.02.003openalex-snapshot
Neutrophils and COVID-19
inferred← citeshttps://doi.org/10.1016/j.abb.2022.109353openalex-snapshot
Structure-biological activity relationships of myeloperoxidase to effect on platelet activation
inferred← citeshttps://doi.org/10.1016/j.ajpath.2020.08.009openalex-snapshot
Immunopathology of Hyperinflammation in COVID-19
inferred← citeshttps://doi.org/10.1016/j.ajpath.2021.09.004openalex-snapshot
Coronavirus Disease 2019 (COVID-19) Coronary Vascular Thrombosis: Correlation with Neutrophil but Not Endothelial Activation.
inferred← citeshttps://doi.org/10.1016/j.ajpath.2023.02.013openalex-snapshot
Severe Acute Respiratory Syndrome Coronavirus 2 Vasculopathy in a Syrian Golden Hamster Model
inferred← citeshttps://doi.org/10.1016/j.amjms.2021.10.012openalex-snapshot
COVID-19 associated coagulopathy: Mechanisms and host-directed treatment
inferred← citeshttps://doi.org/10.1016/j.amjms.2024.01.011openalex-snapshot
Understanding the role of red blood cells in venous thromboembolism: A comprehensive review
inferred← citeshttps://doi.org/10.1016/j.amjsurg.2021.04.004openalex-snapshot
Comparative outcomes between COVID-19 and influenza patients placed on veno-venous extracorporeal membrane oxygenation for severe ARDS
inferred← citeshttps://doi.org/10.1016/j.anai.2020.12.014openalex-snapshot
Monitoring neutrophil-to-lymphocyte ratio in patients with coronavirus disease 2019 receiving tocilizumab
inferred← citeshttps://doi.org/10.1016/j.apsb.2021.10.016openalex-snapshot
Prmt1 upregulated by Hdc deficiency aggravates acute myocardial infarction via NETosis
inferred← citeshttps://doi.org/10.1016/j.atssr.2026.06.032openalex-snapshot
Analysis of Inflammatory Biomarkers in Patients With Chylothorax After Pediatric Cardiac Surgery
inferred← citeshttps://doi.org/10.1016/j.autrev.2022.103206openalex-snapshot
COVID-19 and the antiphospholipid syndrome
inferred← citeshttps://doi.org/10.1016/j.autrev.2026.104164openalex-snapshot
The long shadow of a short life: How neutrophils confer trained immunity
inferred← citeshttps://doi.org/10.1016/j.bbadis.2022.166592openalex-snapshot
Proteomics reveals antiviral host response and NETosis during acute COVID-19 in high-risk patients
inferred← citeshttps://doi.org/10.1016/j.bbapap.2021.140736openalex-snapshot
Immune response pattern across the asymptomatic, symptomatic and convalescent periods of COVID-19
inferred← citeshttps://doi.org/10.1016/j.bbih.2023.100688openalex-snapshot
A potential therapeutic target: The role of neutrophils in the central nervous system
inferred← citeshttps://doi.org/10.1016/j.bbrc.2021.10.068openalex-snapshot
NETs promote pathogenic cardiac fibrosis and participate in ventricular aneurysm formation after ischemia injury through the facilitation of perivascular fibrosis
inferred← citeshttps://doi.org/10.1016/j.bbrc.2023.06.013openalex-snapshot
Precision engineered peptide targeting leukocyte extracellular traps mitigate acute kidney injury in Crush syndrome
inferred← citeshttps://doi.org/10.1016/j.bcmd.2022.102676openalex-snapshot
The significance of surface neutrophilic MPO expression level in NETosis and NETosis-associated coagulopathies in covid-19 infected patients
inferred← citeshttps://doi.org/10.1016/j.bcmd.2023.102782openalex-snapshot
Genetic examination of hematological parameters in SARS-CoV-2 infection and COVID-19
inferred← citeshttps://doi.org/10.1016/j.bcp.2021.114812openalex-snapshot
Pulmonary fibrosis from molecular mechanisms to therapeutic interventions: lessons from post-COVID-19 patients
inferred← citeshttps://doi.org/10.1016/j.bcp.2021.114847openalex-snapshot
The impact of DAMP-mediated inflammation in severe COVID-19 and related disorders
inferred← citeshttps://doi.org/10.1016/j.bcp.2026.118354openalex-snapshot
Neutrophil extracellular traps promote glomerular endothelial injury in IgA nephropathy via the cGAS-STING pathway
inferred← citeshttps://doi.org/10.1016/j.beha.2022.101402openalex-snapshot
COVID-19 and antiphospholipid antibodies
inferred← citeshttps://doi.org/10.1016/j.berh.2021.101661openalex-snapshot
The interplay between neutrophils, complement, and microthrombi in COVID-19
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2023.122097openalex-snapshot
Single-Cell Epitope-Transcriptomics Reveal Lung Stromal and Immune Cell Response Kinetics to Nanoparticle delivered RIG-I and TLR4 Agonists
inferred← citeshttps://doi.org/10.1016/j.biomaterials.2025.123183openalex-snapshot
A DNA-based nanorobot for targeting, hitchhiking, and regulating neutrophils to enhance sepsis therapy
inferred← citeshttps://doi.org/10.1016/j.bioorg.2025.109128openalex-snapshot
Discovery of new neutrophil elastase inhibitors through Meloidae genome and transcriptome analyses
inferred← citeshttps://doi.org/10.1016/j.biopha.2023.114530openalex-snapshot
Xuanfei Baidu Decoction regulates NETs formation via CXCL2/CXCR2 signaling pathway that is involved in acute lung injury
inferred← citeshttps://doi.org/10.1016/j.biopha.2025.118100openalex-snapshot
SARS-CoV-2 S1 protein induces IgG-mediated platelet activation and is prevented by 1.8-cineole
inferred← citeshttps://doi.org/10.1016/j.biopha.2026.119180openalex-snapshot
Macrophage extracellular traps in autoimmunity: In vivo definition, pathogenic circuits, and therapeutic targeting
inferred← citeshttps://doi.org/10.1016/j.bj.2020.09.001openalex-snapshot
Understanding the role of neutrophils in acute respiratory distress syndrome
inferred← citeshttps://doi.org/10.1016/j.bvth.2025.100071openalex-snapshot
Elevated tissue factor pathway inhibitor delays thrombin generation in COVID-19 but is not associated with clinical outcomes
inferred← citeshttps://doi.org/10.1016/j.canlet.2025.218103openalex-snapshot
Multitarget natural compounds taming NETosis: A translational strategy for cancer and inflammatory diseases
inferred← citeshttps://doi.org/10.1016/j.cbi.2026.112373openalex-snapshot
Cytochrome P450 1A protects against hyperbaric oxygen-induced lung injury by regulating neutrophil extracellular trap formation
inferred← citeshttps://doi.org/10.1016/j.cca.2025.120180openalex-snapshot
Circulating levels of PADs and citrullinated histone H3 in SARS-CoV-2 infection: Influence of genetic polymorphisms
inferred← citeshttps://doi.org/10.1016/j.cca.2026.121065openalex-snapshot
Both of coagulopathy and neutrophil extracellular trap markers are associated with poor prognosis of dabie bandavirus infection
inferred← citeshttps://doi.org/10.1016/j.ccc.2021.05.004openalex-snapshot
COVID-19–Associated Acute Respiratory Distress Syndrome
inferred← citeshttps://doi.org/10.1016/j.ccc.2022.03.003openalex-snapshot
Role of Acute Thrombosis in Coronavirus Disease 2019
inferred← citeshttps://doi.org/10.1016/j.ccell.2024.01.013openalex-snapshot
Chronic stress increases metastasis via neutrophil-mediated changes to the microenvironment
inferred← citeshttps://doi.org/10.1016/j.cej.2026.182348openalex-snapshot
Precision deployment of cell and cell-derived therapeutics across immune states and lung-injury endotypes in severe pneumonia
inferred← citeshttps://doi.org/10.1016/j.cell.2020.08.001openalex-snapshot
Severe COVID-19 Is Marked by a Dysregulated Myeloid Cell Compartment
inferred← citeshttps://doi.org/10.1016/j.cell.2020.08.028openalex-snapshot
Effects of COVID-19 on the Nervous System
inferred← citeshttps://doi.org/10.1016/j.cell.2021.02.029openalex-snapshot
COVID-19 and the human innate immune system
inferred← citeshttps://doi.org/10.1016/j.cell.2025.11.001openalex-snapshot
The neutrophil collective
inferred← citeshttps://doi.org/10.1016/j.cellimm.2021.104398openalex-snapshot
An outlook on antigen-specific adoptive immunotherapy for viral infections with a focus on COVID-19
inferred← citeshttps://doi.org/10.1016/j.cellimm.2022.104640openalex-snapshot
Neutrophil extracellular traps: Modulation mechanisms by pathogens
inferred← citeshttps://doi.org/10.1016/j.cellsig.2024.111199openalex-snapshot
Identification of S100A8/A9 involved in thromboangiitis obliterans development using tandem mass tags-labeled quantitative proteomics analysis
inferred← citeshttps://doi.org/10.1016/j.cels.2020.10.003openalex-snapshot
Large-Scale Multi-omic Analysis of COVID-19 Severity
inferred← citeshttps://doi.org/10.1016/j.chest.2021.06.016openalex-snapshot
Pulmonary Thrombosis and Thromboembolism in COVID-19
inferred← citeshttps://doi.org/10.1016/j.chest.2022.06.007openalex-snapshot
Neutrophil Extracellular Traps, Local IL-8 Expression, and Cytotoxic T-Lymphocyte Response in the Lungs of Patients With Fatal COVID-19
inferred← citeshttps://doi.org/10.1016/j.chmed.2022.06.014openalex-snapshot
New insights for infection mechanism and potential targets of COVID-19: Three Chinese patent medicines and three Chinese medicine formulas as promising therapeutic approaches
inferred← citeshttps://doi.org/10.1016/j.chom.2024.11.007openalex-snapshot
Persistence of spike protein at the skull-meninges-brain axis may contribute to the neurological sequelae of COVID-19
inferred← citeshttps://doi.org/10.1016/j.clim.2021.108849openalex-snapshot
A hitchhiker's guide through the COVID-19 galaxy
inferred← citeshttps://doi.org/10.1016/j.clim.2023.109687openalex-snapshot
NETosis promotes chronic inflammation and fibrosis in systemic lupus erythematosus and COVID-19
inferred← citeshttps://doi.org/10.1016/j.clim.2023.109836openalex-snapshot
Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production
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