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Of Myc and Mnt

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/(sici)1097-0215(19990909)82:6<810::aid-ijc7>3.0.co;2-vopenalex-snapshot

Analysis of the max-binding protein MNT in human medulloblastomas

inferredcites →https://doi.org/10.1002/(sici)1097-4652(200006)183:3<399::aid-jcp13>3.0.co;2-7openalex-snapshot

Analysis of Myc/Max/Mad network members in adipogenesis: Inhibition of the proliferative burst and differentiation by ectopically expressed Mad1

inferredcites →https://doi.org/10.1002/ijc.2910530628openalex-snapshot

Conditional expression and oncogenicity of c‐myc linked to a CD2 gene dominant control region

inferredcites →https://doi.org/10.1002/j.1460-2075.1985.tb03885.xopenalex-snapshot

Metabolism of c‐myc gene products: c‐myc mRNA and protein expression in the cell cycle.

inferredcites →https://doi.org/10.1002/j.1460-2075.1989.tb03435.xopenalex-snapshot

N‐myc transgene promotes B lymphoid proliferation, elicits lymphomas and reveals cross‐regulation with c‐myc.

inferredcites →https://doi.org/10.1002/j.1460-2075.1990.tb07612.xopenalex-snapshot

Negative autoregulation of c‐myc gene expression is inactivated in transformed cells.

inferredcites →https://doi.org/10.1002/j.1460-2075.1991.tb07929.xopenalex-snapshot

The MYC protein activates transcription of the alpha‐prothymosin gene.

inferredcites →https://doi.org/10.1002/j.1460-2075.1994.tb06630.xopenalex-snapshot

c‐Myc‐induced apoptosis in fibroblasts is inhibited by specific cytokines.

inferredcites →https://doi.org/10.1002/j.1460-2075.1995.tb00252.xopenalex-snapshot

Mad3 and Mad4: novel Max‐interacting transcriptional repressors that suppress c‐myc dependent transformation and are expressed during neural and epidermal differentiation.

inferredcites →https://doi.org/10.1002/jcb.10517openalex-snapshot

hMad4, c‐Myc endogenous inhibitor, induces a replicative senescence‐like state when overexpressed in human fibroblasts

inferredcites →https://doi.org/10.1007/978-3-642-60801-8_11openalex-snapshot

Mnt: A Novel Max-interacting Protein and Myc Antagonist

inferredcites →https://doi.org/10.1016/0092-8674(83)90092-2openalex-snapshot

Cell-specific regulation of the c-myc gene by lymphocyte mitogens and platelet-derived growth factor

inferredcites →https://doi.org/10.1016/0092-8674(84)90257-5openalex-snapshot

Spontaneous mammary adenocarcinomas in transgenic mice that carry and express MTV/myc fusion genes

inferredcites →https://doi.org/10.1016/0092-8674(91)90457-aopenalex-snapshot

Association of Myn, the murine homolog of Max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation

inferredcites →https://doi.org/10.1016/0092-8674(92)90123-topenalex-snapshot

Induction of apoptosis in fibroblasts by c-myc protein

inferredcites →https://doi.org/10.1016/0092-8674(93)90661-9openalex-snapshot

Mad: A heterodimeric partner for Max that antagonizes Myc transcriptional activity

inferredcites →https://doi.org/10.1016/0092-8674(93)90662-aopenalex-snapshot

Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sites

inferredcites →https://doi.org/10.1016/0092-8674(95)90355-0openalex-snapshot

Mad-max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3

inferredcites →https://doi.org/10.1016/0092-8674(95)90356-9openalex-snapshot

An amino-terminal domain of Mxi1 mediates anti-myc oncogenic activity and interacts with a homolog of the Yeast Transcriptional Repressor SIN3

inferredcites →https://doi.org/10.1016/s0065-230x(08)60476-5openalex-snapshot

Control of c-myc Regulation in Normal and Neoplastic Cells

inferredcites →https://doi.org/10.1016/s0092-8674(00)80214-7openalex-snapshot

Histone Deacetylase Activity Is Required for Full Transcriptional Repression by mSin3A

inferredcites →https://doi.org/10.1016/s0092-8674(00)80215-9openalex-snapshot

Histone Deacetylases Associated with the mSin3 Corepressor Mediate Mad Transcriptional Repression

inferredcites →https://doi.org/10.1016/s0092-8674(00)81512-3openalex-snapshot

Drosophila myc Regulates Cellular Growth during Development

inferredcites →https://doi.org/10.1016/s0092-8674(04)00127-8openalex-snapshot

Unbiased Mapping of Transcription Factor Binding Sites along Human Chromosomes 21 and 22 Points to Widespread Regulation of Noncoding RNAs

inferredcites →https://doi.org/10.1016/s0925-4773(01)00566-4openalex-snapshot

A P-insertion screen identifying novel X-linked essential genes in Drosophila

inferredcites →https://doi.org/10.1016/s0960-9822(99)80507-7openalex-snapshot

Control of cell growth by c-Myc in the absence of cell division

inferredcites →https://doi.org/10.1016/s0962-8924(01)02121-3openalex-snapshot

The interplay between Mad and Myc in proliferation and differentiation

inferredcites →https://doi.org/10.1016/s0962-8924(03)00003-5openalex-snapshot

Transcriptional repression by Myc

inferredcites →https://doi.org/10.1016/s1074-7613(01)00088-7openalex-snapshot

Analysis of C-MYC Function in Normal Cells via Conditional Gene-Targeted Mutation

inferredcites →https://doi.org/10.1016/s1097-2765(02)00633-0openalex-snapshot

Negative Regulation of the Mammalian UV Response by Myc through Association with Miz-1

inferredcites →https://doi.org/10.1038/31008openalex-snapshot

Role of Mxi1 in ageing organ systems and the regulation of normal and neoplastic growth

inferredcites →https://doi.org/10.1038/314363a0openalex-snapshot

Levels of c-myc oncogene mRNA are invariant throughout the cell cycle

inferredcites →https://doi.org/10.1038/314366a0openalex-snapshot

c-myc oncogene protein synthesis is independent of the cell cycle in human and avian cells

inferredcites →https://doi.org/10.1038/35070076openalex-snapshot

Repression of p15INK4b expression by Myc through association with Miz-1

inferredcites →https://doi.org/10.1038/35070086openalex-snapshot

TGFβ influences Myc, Miz-1 and Smad to control the CDK inhibitor p15INK4b

inferredcites →https://doi.org/10.1038/359423a0openalex-snapshot

Transcriptional activation by the human c-Myc oncoprotein in yeast requires interaction with Max

inferredcites →https://doi.org/10.1038/359426a0openalex-snapshot

Myc and Max proteins possess distinct transcriptional activities

inferredcites →https://doi.org/10.1038/387049a0openalex-snapshot

Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression

inferredcites →https://doi.org/10.1038/414768aopenalex-snapshot

c-Myc regulates mammalian body size by controlling cell number but not cell size

inferredcites →https://doi.org/10.1038/72761openalex-snapshot

Gene-target recognition among members of the Myc superfamily and implications for oncogenesis

inferredcites →https://doi.org/10.1038/nature01327openalex-snapshot

Direct activation of RNA polymerase III transcription by c-Myc

inferredcites →https://doi.org/10.1038/nature03098openalex-snapshot

Intrinsic tumour suppression

inferredcites →https://doi.org/10.1038/nature03677openalex-snapshot

c-Myc-regulated microRNAs modulate E2F1 expression

inferredcites →https://doi.org/10.1038/nature03845openalex-snapshot

Evasion of the p53 tumour surveillance network by tumour-derived MYC mutants

inferredcites →https://doi.org/10.1038/ncb1223openalex-snapshot

Myc-dependent regulation of ribosomal RNA synthesis during Drosophila development

inferredcites →https://doi.org/10.1038/ncb1224openalex-snapshot

c-Myc binds to human ribosomal DNA and stimulates transcription of rRNA genes by RNA polymerase I

inferredcites →https://doi.org/10.1038/ncb1225openalex-snapshot

c-Myc associates with ribosomal DNA and activates RNA polymerase I transcription

inferredcites →https://doi.org/10.1038/nrc1393openalex-snapshot

Analysis of genomic targets reveals complex functions of MYC

inferredcites →https://doi.org/10.1038/nrc904openalex-snapshot

c-MYC: more than just a matter of life and death

inferredcites →https://doi.org/10.1038/nrm1551openalex-snapshot

RNA polymerases I and III, growth control and cancer

inferredcites →https://doi.org/10.1038/nrm1703openalex-snapshot

Transcriptional regulation and transformation by Myc proteins

inferredcites →https://doi.org/10.1038/sj.emboj.7600279openalex-snapshot

Myc‐induced proliferation and transformation require Akt‐mediated phosphorylation of FoxO proteins

inferredcites →https://doi.org/10.1038/sj.emboj.7600335openalex-snapshot

MAD1 and c‐MYC regulate UBF and rDNA transcription during granulocyte differentiation

inferredcites →https://doi.org/10.1038/sj.embor.embor821openalex-snapshot

Stimulation of c‐MYC transcriptional activity and acetylation by recruitment of the cofactor CBP

inferredcites →https://doi.org/10.1038/sj.embor.embor861openalex-snapshot

MYC recruits the TIP60 histone acetyltransferase complex to chromatin

inferredcites →https://doi.org/10.1038/sj.onc.1201611openalex-snapshot

Sequential expression of the MAD family of transcriptional repressors during differentiation and development

inferredcites →https://doi.org/10.1038/sj.onc.1203634openalex-snapshot

Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?

inferredcites →https://doi.org/10.1038/sj.onc.1203639openalex-snapshot

A genetic screen to identify genes that rescue the slow growth phenotype of c-myc null fibroblasts

inferredcites →https://doi.org/10.1038/sj.onc.1203880openalex-snapshot

Complementation of Myc-dependent cell proliferation by cDNA expression library screening

inferredcites →https://doi.org/10.1038/sj.onc.1204196openalex-snapshot

Expression of Mad1 in T cells leads to reduced thymic cellularity and impaired mitogen-induced proliferation

inferredcites →https://doi.org/10.1038/sj.onc.1206145openalex-snapshot

Myc represses differentiation-induced p21CIP1 expression via Miz-1-dependent interaction with the p21 core promoter

inferredcites →https://doi.org/10.1038/sj.onc.1207261openalex-snapshot

Myc pathways provoking cell suicide and cancer

inferredcites →https://doi.org/10.1038/sj.onc.1208961openalex-snapshot

Mnt transcriptional repressor is functionally regulated during cell cycle progression

inferredcites →https://doi.org/10.1042/0264-6021:3590361openalex-snapshot

S-phase-specific expression of the Mad3 gene in proliferating and differentiating cells

inferredcites →https://doi.org/10.1073/pnas.050586197openalex-snapshot

Identification of CDK4 as a target of c-MYC

inferredcites →https://doi.org/10.1073/pnas.162369299openalex-snapshot

Myc and Mad bHLHZ domains possess identical DNA-binding specificities but only partially overlapping functions in vivo

inferredcites →https://doi.org/10.1073/pnas.96.23.13180openalex-snapshot

c-Myc enhances protein synthesis and cell size during B lymphocyte development

inferredcites →https://doi.org/10.1074/jbc.273.12.6632openalex-snapshot

Identification and Characterization of Specific DNA-binding Complexes Containing Members of the Myc/Max/Mad Network of Transcriptional Regulators

inferredcites →https://doi.org/10.1074/jbc.274.51.36344openalex-snapshot

Mlx, a Novel Max-like BHLHZip Protein That Interacts with the Max Network of Transcription Factors

inferredcites →https://doi.org/10.1083/jcb.128.6.1197openalex-snapshot

Expression of the mad gene during cell differentiation in vivo and its inhibition of cell growth in vitro.

inferredcites →https://doi.org/10.1083/jcb.200411013openalex-snapshot

Mnt–Max to Myc–Max complex switching regulates cell cycle entry

inferredcites →https://doi.org/10.1093/emboj/16.10.2892openalex-snapshot

Rox, a novel bHLHZip protein expressed in quiescent cells that heterodimerizes with Max, binds a non‐canonical E box and acts as a transcriptional repressor

inferredcites →https://doi.org/10.1093/emboj/16.18.5672openalex-snapshot

An alternative pathway for gene regulation by Myc

inferredcites →https://doi.org/10.1093/emboj/17.3.774openalex-snapshot

Targeted disruption of the MYC antagonist MAD1 inhibits cell cycle exit during granulocyte differentiation

inferredcites →https://doi.org/10.1093/emboj/18.19.5321openalex-snapshot

Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27

inferredcites →https://doi.org/10.1093/emboj/18.24.7019openalex-snapshot

Mga, a dual‐specificity transcription factor that interacts with Max and contains a T‐domain DNA‐binding motif

inferredcites →https://doi.org/10.1093/emboj/19.21.5813openalex-snapshot

Induction of cyclin E–cdk2 kinase activity, E2F‐dependent transcription and cell growth by Myc are genetically separable events

inferredcites →https://doi.org/10.1093/emboj/cdf492openalex-snapshot

Modulation of T‐lymphocyte development, growth and cell size by the Myc antagonist and transcriptional repressor Mad1

inferredcites →https://doi.org/10.1093/emboj/cdg442openalex-snapshot

Deletion of Mnt leads to disrupted cell cycle control and tumorigenesis

inferredcites →https://doi.org/10.1093/hmg/ddh116openalex-snapshot

Loss of the Max-interacting protein Mnt in mice results in decreased viability, defective embryonic growth and craniofacial defects: relevance to Miller-Dieker syndrome

inferredcites →https://doi.org/10.1101/gad.1021202openalex-snapshot

N- myc is essential during neurogenesis for the rapid expansion of progenitor cell populations and the inhibition of neuronal differentiation

inferredcites →https://doi.org/10.1101/gad.1066903openalex-snapshot

Genomic binding by the Drosophila Myc, Max, Mad/Mnt transcription factor network

inferredcites →https://doi.org/10.1101/gad.1067003openalex-snapshot

Genomic targets of the human c-Myc protein

inferredcites →https://doi.org/10.1101/gad.11.1.44openalex-snapshot

Mnt, a novel Max-interacting protein is coexpressed with Myc in proliferating cells and mediates repression at Myc binding sites.

inferredcites →https://doi.org/10.1101/gad.14.11.1390openalex-snapshot

N-myc can functionally replace c-myc in murine development, cellular growth, and differentiation

inferredcites →https://doi.org/10.1101/gad.6.1.71openalex-snapshot

Myc and Max associate in vivo.

inferredcites →https://doi.org/10.1101/gad.6.1.81openalex-snapshot

Max: functional domains and interaction with c-Myc.

inferredcites →https://doi.org/10.1101/gad.6.2.166openalex-snapshot

Casein kinase II inhibits the DNA-binding activity of Max homodimers but not Myc/Max heterodimers.

inferredcites →https://doi.org/10.1101/gad.7.11.2110openalex-snapshot

A switch from Myc:Max to Mad:Max heterocomplexes accompanies monocyte/macrophage differentiation.

inferredcites →https://doi.org/10.1101/gad.907901openalex-snapshot

Regulation of cyclin D2 gene expression by the Myc/Max/Mad network: Myc-dependent TRRAP recruitment and histone acetylation at the cyclin D2 promoter

inferredcites →https://doi.org/10.1126/science.2006410openalex-snapshot

Max: A Helix-Loop-Helix Zipper Protein That Forms a Sequence-Specific DNA-Binding Complex with Myc

inferredcites →https://doi.org/10.1126/science.274.5292.1523openalex-snapshot

Myc and Max Homologs in Drosophila

inferredcites →https://doi.org/10.1128/mcb.13.1.383openalex-snapshot

Sequence-Specific Transcriptional Activation by Myc and Repression by Max

inferredcites →https://doi.org/10.1128/mcb.13.9.5216openalex-snapshot

Binding of Myc Proteins to Canonical and Noncanonical DNA Sequences

inferredcites →https://doi.org/10.1128/mcb.16.4.1794openalex-snapshot

Expression and Activity of L-Myc in Normal Mouse Development

inferredcites →https://doi.org/10.1128/mcb.20.2.556-562.2000openalex-snapshot

The Essential Cofactor TRRAP Recruits the Histone Acetyltransferase hGCN5 to c-Myc

inferredcites →https://doi.org/10.1128/mcb.21.3.703-712.2001openalex-snapshot

Targeted Deletion of the S-Phase-Specific Myc Antagonist Mad3 Sensitizes Neuronal and Lymphoid Cells to Radiation-Induced Apoptosis

inferredcites →https://doi.org/10.1128/mcb.24.4.1560-1569.2004openalex-snapshot

Mnt Loss Triggers Myc Transcription Targets, Proliferation, Apoptosis, and Transformation

inferredcites →https://doi.org/10.1128/mcb.25.16.7078-7091.2005openalex-snapshot

The Transcriptional Repressor dMnt Is a Regulator of Growth in Drosophila melanogaster

inferredcites →https://doi.org/10.1128/mcb.25.17.7868-7878.2005openalex-snapshot

c-Myc Is Required for the Formation of Intestinal Crypts but Dispensable for Homeostasis of the Adult Intestinal Epithelium

inferredcites →https://doi.org/10.1146/annurev.cellbio.16.1.653openalex-snapshot

The Myc/Max/Mad Network and the Transcriptional Control of Cell Behavior

inferredcites →https://doi.org/10.1242/jcs.02298openalex-snapshot

Endogenous Myc controls mammalian epidermal cell size, hyperproliferation, endoreplication and stem cell amplification

inferredcites →https://doi.org/10.4161/cc.3.9.1145openalex-snapshot

The Life Cycle of C-Myc: From Synthesis to Degradation

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4677331openalex-extracted

Association of Sheep Mcm2 And Mxd3 Gene Polymorphisms with Body Weight and Average Daily Gain In Hu Sheep

inferred← citeshttps://doi.org/10.1002/1873-3468.14097openalex-snapshot

MXD/MIZ1 transcription regulatory complexes activate the expression of MYC‐repressed genes

inferred← citeshttps://doi.org/10.1002/dvdy.22620openalex-snapshot

Conditional deletion of N‐Myc disrupts neurosensory and non‐sensory development of the ear

inferred← citeshttps://doi.org/10.1002/hep.22720openalex-snapshot

Switch from Mnt‐Max to Myc‐Max induces p53 and cyclin D1 expression and apoptosis during cholestasis in mouse and human hepatocytes†

inferred← citeshttps://doi.org/10.1002/iid3.179openalex-snapshot

Alterations of oncogenes expression in NK cells in patients with cancer

inferred← citeshttps://doi.org/10.1002/ijc.22866openalex-snapshot

Camptothecin‐induced apoptosis is enhanced by Myc and involves PKCdelta signaling

inferred← citeshttps://doi.org/10.1002/term.239openalex-snapshot

Growth inhibition of SK-MEL-30 human melanoma cells by antisense c-myc oligonucleotides delivered by poly(N-isopropylacrylamide)/ poly(ethyleneimine) copolymer

inferred← citeshttps://doi.org/10.1002/yea.1757openalex-snapshot

Multiple bHLH proteins regulate CIT2 expression in Saccharomyces cerevisiae

inferred← citeshttps://doi.org/10.1007/2789_2008_088openalex-snapshot

The Interplay Between MYC and HIF in the Warburg Effect

inferred← citeshttps://doi.org/10.1007/978-1-60327-227-8_8openalex-snapshot

Transcriptional Networks Regulating Embryonic Stem Cell Fate Decisions

inferred← citeshttps://doi.org/10.1007/978-94-007-6621-1_15openalex-snapshot

Myc in Stem Cell Behaviour: Insights from Drosophila

inferred← citeshttps://doi.org/10.1007/s00109-010-0701-7openalex-snapshot

The role of histone deacetylase 7 (HDAC7) in cancer cell proliferation: regulation on c-Myc

inferred← citeshttps://doi.org/10.1007/s10689-011-9489-zopenalex-snapshot

Mutation and association analyses of the candidate genes ESR1, ESR2, MAX, PCNA, and KAT2A in patients with unexplained MSH2-deficient tumors

inferred← citeshttps://doi.org/10.1007/s11684-018-0650-zopenalex-snapshot

The MYC transcription factor network: balancing metabolism, proliferation and oncogenesis

inferred← citeshttps://doi.org/10.1007/s12015-009-9077-xopenalex-snapshot

The Molecular Mechanism of Induced Pluripotency: A Two-Stage Switch

inferred← citeshttps://doi.org/10.1016/b978-0-12-407173-5.00004-2openalex-snapshot

The Transcription Factor FOXM1 (Forkhead box M1)

inferred← citeshttps://doi.org/10.1016/j.abb.2015.07.005openalex-snapshot

The role of Myc and let-7a in glioblastoma, glucose metabolism and response to therapy

inferred← citeshttps://doi.org/10.1016/j.ajpath.2011.06.008openalex-snapshot

The miR-17-92 MicroRNA Cluster Is Regulated by Multiple Mechanisms in B-Cell Malignancies

inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2013.02.033openalex-snapshot

A repressor protein, Mnt, is a novel negative regulator of vascular smooth muscle cell hypertrophy by angiotensin II and neointimal hyperplasia by arterial injury

inferred← citeshttps://doi.org/10.1016/j.bbagrm.2014.04.004openalex-snapshot

The activities of MYC, MNT and the MAX-interactome in lymphocyte proliferation and oncogenesis

inferred← citeshttps://doi.org/10.1016/j.bbrc.2010.03.030openalex-snapshot

Differentiation-associated miR-22 represses Max expression and inhibits cell cycle progression

inferred← citeshttps://doi.org/10.1016/j.bneo.2025.100149openalex-snapshot

MNT: a new target for AML

inferred← citeshttps://doi.org/10.1016/j.ccell.2014.11.024openalex-snapshot

Deregulated Myc Requires MondoA/Mlx for Metabolic Reprogramming and Tumorigenesis

inferred← citeshttps://doi.org/10.1016/j.celrep.2019.10.022openalex-snapshot

Misregulation of Drosophila Myc Disrupts Circadian Behavior and Metabolism

inferred← citeshttps://doi.org/10.1016/j.dci.2007.04.003openalex-snapshot

Identification and real-time imaging of a myc-expressing neutrophil population involved in inflammation and mycobacterial granuloma formation in zebrafish

inferred← citeshttps://doi.org/10.1016/j.gde.2008.01.013openalex-snapshot

The reprogramming language of pluripotency

inferred← citeshttps://doi.org/10.1016/j.gene.2012.01.095openalex-snapshot

Regulation of the human catalytic subunit of telomerase (hTERT)

inferred← citeshttps://doi.org/10.1016/j.jdermsci.2007.05.005openalex-snapshot

Beyond steric hindrance: The role of adhesion signaling pathways in the pathogenesis of pemphigus

inferred← citeshttps://doi.org/10.1016/j.semcancer.2006.07.009openalex-snapshot

The MAX-interacting transcription factor network

inferred← citeshttps://doi.org/10.1016/j.ydbio.2006.09.047openalex-snapshot

Tbx20 regulation of endocardial cushion cell proliferation and extracellular matrix gene expression

inferred← citeshttps://doi.org/10.1016/j.ydbio.2007.12.026openalex-snapshot

Drosophila growth and development in the absence of dMyc and dMnt

inferred← citeshttps://doi.org/10.1016/j.yexcr.2009.09.009openalex-snapshot

TGF-β enforces senescence in Myc-transformed hematopoietic tumor cells through induction of Mad1 and repression of Myc activity

inferred← citeshttps://doi.org/10.1016/s0065-230x(06)97003-1openalex-snapshot

Mnt Takes Control as Key Regulator of the Myc/Max/Mxd Network

inferred← citeshttps://doi.org/10.1016/s0065-230x(07)99004-1openalex-snapshot

The c‐myc Promoter: Still MysterY and Challenge

inferred← citeshttps://doi.org/10.1021/acs.jpcb.8b07351openalex-snapshot

Occupancies in the DNA-Binding Pathways of Intrinsically Disordered Helix-Loop-Helix Leucine-Zipper Proteins

inferred← citeshttps://doi.org/10.1038/bjc.2011.220openalex-snapshot

Tumour-suppressor microRNAs let-7 and mir-101 target the proto-oncogene MYCN and inhibit cell proliferation in MYCN-amplified neuroblastoma

inferred← citeshttps://doi.org/10.1038/cdd.2017.131openalex-snapshot

Mnt modulates Myc-driven lymphomagenesis

inferred← citeshttps://doi.org/10.1073/pnas.0707418104openalex-snapshot

A Myc–Groucho complex integrates EGF and Notch signaling to regulate neural development

inferred← citeshttps://doi.org/10.1073/pnas.1206406109openalex-snapshot

A critical role for Mnt in Myc-driven T-cell proliferation and oncogenesis

inferred← citeshttps://doi.org/10.1074/jbc.m111.246116openalex-snapshot

The E-box Binding Factors Max/Mnt, MITF, and USF1 Act Coordinately with FoxO to Regulate Expression of Proapoptotic and Cell Cycle Control Genes by Phosphatidylinositol 3-Kinase/Akt/Glycogen Synthase Kinase 3 Signaling

inferred← citeshttps://doi.org/10.1089/cell.2013.0087openalex-snapshot

Sodium Butyrate Efficiently Converts Fully Reprogrammed Induced Pluripotent Stem Cells from Mouse Partially Reprogrammed Cells

inferred← citeshttps://doi.org/10.1091/mbc.e09-06-0456openalex-snapshot

Distinct and Temporal Roles of Nucleosomal Remodeling and Histone Deacetylation in the Repression of the hTERT Gene

inferred← citeshttps://doi.org/10.1093/gbe/evr082openalex-snapshot

Evolution of the Max and Mlx Networks in Animals

inferred← citeshttps://doi.org/10.1101/842799openalex-snapshot

MXD/MIZ1 complexes activate transcription of MYC-repressed genes

inferred← citeshttps://doi.org/10.1101/cshperspect.a014357openalex-snapshot

An Overview of MYC and Its Interactome

inferred← citeshttps://doi.org/10.1101/gad.1712408openalex-snapshot

Myc’s broad reach

inferred← citeshttps://doi.org/10.1128/ec.00144-10openalex-snapshot

Derepression ofINO1Transcription Requires Cooperation between the Ino2p-Ino4p Heterodimer and Cbf1p and Recruitment of the ISW2 Chromatin-Remodeling Complex

inferred← citeshttps://doi.org/10.1128/ec.00383-06openalex-snapshot

Multiple Basic Helix-Loop-Helix Proteins Regulate Expression of the ENO1 Gene of Saccharomyces cerevisiae

inferred← citeshttps://doi.org/10.1128/mcb.00656-06openalex-snapshot

A Novel Role of the Mad Family Member Mad3 in Cerebellar Granule Neuron Precursor Proliferation

inferred← citeshttps://doi.org/10.1155/2018/3286949openalex-snapshot

The Myc/Max/Mxd Network Is a Target of Mutated Flt3 Signaling in Hematopoietic Stem Cells in Flt3-ITD-Induced Myeloproliferative Disease

inferred← citeshttps://doi.org/10.1155/2018/4835491openalex-snapshot

Adult Neural Stem Cells: Basic Research and Production Strategies for Neurorestorative Therapy

inferred← citeshttps://doi.org/10.1158/1541-7786.mcr-12-0244openalex-snapshot

N-myc and Noncoding RNAs in Neuroblastoma

inferred← citeshttps://doi.org/10.1159/000151712openalex-snapshot

Losses of Chromosome Arms 4q, 8p, 13q and Gain of 8q Are Correlated with Increasing Chromosomal Instability in Hepatocellular Carcinoma

inferred← citeshttps://doi.org/10.1177/172460080802300204openalex-snapshot

Expression of Mina53 and its Significance in Gastric Carcinoma

inferred← citeshttps://doi.org/10.1182/blood.2019003014openalex-snapshot

Development and Survival of MYC-driven Lymphomas Requires MYC-Antagonist MNT to Curb MYC-induced Apoptosis

inferred← citeshttps://doi.org/10.1186/1471-2121-15-30openalex-snapshot

MXD3 regulation of DAOY cell proliferation dictated by time course of activation

inferred← citeshttps://doi.org/10.1186/1749-8546-9-18openalex-snapshot

Differential effects of selective frankincense (Ru Xiang) essential oil versus non-selective sandalwood (Tan Xiang) essential oil on cultured bladder cancer cells: a microarray and bioinformatics study

inferred← citeshttps://doi.org/10.1242/dev.02787openalex-snapshot

How is pluripotency determined and maintained?

inferred← citeshttps://doi.org/10.1371/journal.pone.0002722openalex-snapshot

Global Regulation of Nucleotide Biosynthetic Genes by c-Myc

inferred← citeshttps://doi.org/10.1371/journal.pone.0007839openalex-snapshot

Gene Regulation and Epigenetic Remodeling in Murine Embryonic Stem Cells by c-Myc

inferred← citeshttps://doi.org/10.1371/journal.pone.0042021openalex-snapshot

Humanized c-Myc Mouse

inferred← citeshttps://doi.org/10.1371/journal.pone.0042697openalex-snapshot

Hypoxia-Induced Alternative Splicing in Endothelial Cells

inferred← citeshttps://doi.org/10.1387/ijdb.082640egopenalex-snapshot

Mouse induced pluripotent stem cells

inferred← citeshttps://doi.org/10.15252/embj.2021109905openalex-snapshot

Myc suppresses male–male courtship in Drosophila

inferred← citeshttps://doi.org/10.1634/stemcells.2008-0102openalex-snapshot

Polycomb Repressive Complex 2 Is Dispensable for Maintenance of Embryonic Stem Cell Pluripotency

inferred← citeshttps://doi.org/10.21037/tau-20-1187openalex-snapshot

Overexpression of MAX dimerization protein 3 (MXD3) predicts poor prognosis in clear cell renal cell carcinoma

inferred← citeshttps://doi.org/10.2174/138945009788680400openalex-snapshot

Myc - What We have Learned from Flies

inferred← citeshttps://doi.org/10.22028/d291-22420openalex-snapshot

Charakterisierung einer neuen Amplifikationseinheit auf Chromosom 3q25-q26 beim Prostatakarzinom mit Hilfe molekular-zytogenetischer und quantitativer PCR-Techniken

inferred← citeshttps://doi.org/10.33612/diss.134190556openalex-snapshot

The roles of noncoding RNAs in B-cel lymphomas

inferred← citeshttps://doi.org/10.3390/cancers12123546openalex-snapshot

MiR-378a-3p Is Critical for Burkitt Lymphoma Cell Growth

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