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Involvement of Voltage-Gated Sodium Channel NaV1.8 in the Regulation of the Release and Synthesis of Substance P in Adult Mouse Dorsal Root Ganglion Neurons

https://doi.org/10.1254/jphs.08163fp
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1007/s00210-007-0211-5openalex-snapshot
The activation of transient receptor potential vanilloid receptor subtype 1 by capsaicin without extracellular Ca2+ is involved in the mechanism of distinct substance P release in cultured rat dorsal root ganglion neurons
inferredcites →https://doi.org/10.1016/0304-3959(88)90209-6openalex-snapshot
A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man
inferredcites →https://doi.org/10.1016/0304-3959(94)90189-9openalex-snapshot
Primary sensory neurons exhibit altered gene expression in a rat model of neuropathic pain
inferredcites →https://doi.org/10.1016/j.neuropharm.2005.01.011openalex-snapshot
Zaltoprofen inhibits bradykinin-induced responses by blocking the activation of second messenger signaling cascades in rat dorsal root ganglion cells
inferredcites →https://doi.org/10.1016/s0006-8993(02)03750-2openalex-snapshot
Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar purkinje cells
inferredcites →https://doi.org/10.1016/s0304-3959(01)00391-8openalex-snapshot
Inhibition of neuropathic pain by decreased expression of the tetrodotoxin-resistant sodium channel, NaV1.8
inferredcites →https://doi.org/10.1016/s0306-4522(01)00342-6openalex-snapshot
Depletion of substance P from rat primary sensory neurons by ATP, an implication of P2X receptor-mediated release of substance P
inferredcites →https://doi.org/10.1038/379257a0openalex-snapshot
A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons
inferredcites →https://doi.org/10.1038/nature05880openalex-snapshot
Sensory neuron sodium channel Nav1.8 is essential for pain at low temperatures
inferredcites →https://doi.org/10.1046/j.1460-9568.1999.00445.xopenalex-snapshot
Substance P release in the dorsal horn assessed by receptor internalization: NMDA receptors counteract a tonic inhibition by GABAB receptors
inferredcites →https://doi.org/10.1073/pnas.0611364104openalex-snapshot
A-803467, a potent and selective Na v 1.8 sodium channel blocker, attenuates neuropathic and inflammatory pain in the rat
inferredcites →https://doi.org/10.1073/pnas.88.24.11344openalex-snapshot
Substance P-mediated slow excitatory postsynaptic potential elicited in dorsal horn neurons in vivo by noxious stimulation.
inferredcites →https://doi.org/10.1091/mbc.6.5.509openalex-snapshot
Delineation of the endocytic pathway of substance P and its seven-transmembrane domain NK1 receptor.
inferredcites →https://doi.org/10.1111/j.1469-7793.1998.771bg.xopenalex-snapshot
Electrophysiological properties of sodium current subtypes in small cells from adult rat dorsal root ganglia
inferredcites →https://doi.org/10.1111/j.1471-4159.2006.03830.xopenalex-snapshot
Substance P release evoked by capsaicin or potassium from rat cultured dorsal root ganglion neurons is conversely modulated with bradykinin
inferredcites →https://doi.org/10.1113/jphysiol.2003.042127openalex-snapshot
The TTX‐Resistant Sodium Channel Nav1.8 (SNS/PN3): Expression and Correlation with Membrane Properties in Rat Nociceptive Primary Afferent Neurons
inferredcites →https://doi.org/10.1152/ajpgi.00154.2004openalex-snapshot
Two TTX-resistant Na+ currents in mouse colonic dorsal root ganglia neurons and their role in colitis-induced hyperexcitability
inferredcites →https://doi.org/10.1152/ajpgi.2001.281.5.g1322openalex-snapshot
Capsaicin vanilloid receptor-1 mediates substance P release in experimental pancreatitis
inferredcites →https://doi.org/10.1152/jn.1999.81.3.1379openalex-snapshot
Primary Afferent Fibers That Contribute to Increased Substance P Receptor Internalization in the Spinal Cord After Injury
inferredcites →https://doi.org/10.1152/jn.2001.86.2.629openalex-snapshot
Contribution of Na v 1.8 Sodium Channels to Action Potential Electrogenesis in DRG Neurons
inferredcites →https://doi.org/10.1186/1744-8069-3-42openalex-snapshot
Activation of the Neurokinin-1 Receptor by Substance P Triggers the Release of Substance P from Cultured Adult Rat Dorsal Root Ganglion Neurons
inferredcites →https://doi.org/10.1254/jphs.fp0061402openalex-snapshot
Prostaglandin E2 Has No Effect on Two Components of Tetrodotoxin-Resistant Na+ Current in Mouse Dorsal Root Ganglion
inferredcites →https://doi.org/10.1254/jphs.fp0071359openalex-snapshot
The Release of Substance P From Cultured Dorsal Root Ganglion Neurons Requires the Non-neuronal Cells Around These Neurons
inferredcites →https://doi.org/10.1523/jneurosci.21-10-03656.2001openalex-snapshot
Differential Contribution of Substance P and Neurokinin A to Spinal Cord Neurokinin-1 Receptor Signaling in the Rat
inferredcites →https://doi.org/10.1523/jneurosci.23-01-00158.2003openalex-snapshot
Redistribution of NaV1.8 in Uninjured Axons Enables Neuropathic Pain
inferredcites →https://doi.org/10.1523/jneurosci.23-32-10338.2003openalex-snapshot
Role of Tetrodotoxin-Resistant Na+Current Slow Inactivation in Adaptation of Action Potential Firing in Small-Diameter Dorsal Root Ganglion Neurons

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.3945933openalex-extracted
Inhibition of Nav1.8 Transport Promotes Axon Regeneration Via Regulating Dynamic Changes in Macrophage Phenotype after Sciatic Nerve Injury
inferred← citeshttps://doi.org/10.1016/j.brainres.2014.07.012openalex-snapshot
Redistribution of voltage-gated sodium channels after nerve decompression contributes to relieve neuropathic pain in chronic constriction injury
inferred← citeshttps://doi.org/10.1016/j.ejphar.2015.11.055openalex-snapshot
Paclitaxel-induced peripheral neuropathy increases substance P release in rat spinal cord
inferred← citeshttps://doi.org/10.1097/aln.0000000000004761openalex-snapshot
Ropivacaine Promotes Axon Regeneration by Regulating Nav1.8-mediated Macrophage Signaling after Sciatic Nerve Injury in Rats
inferred← citeshttps://doi.org/10.1097/fbp.0000000000000422openalex-snapshot
Anti-NGF treatment can reduce chronic neuropathic pain by changing peripheral mediators and brain activity in rats
inferred← citeshttps://doi.org/10.1254/jphs.09033scopenalex-snapshot
Modulation of the Substance P Release From Cultured Rat Primary Afferent Neurons by Zinc Ions
inferred← citeshttps://doi.org/10.1254/jphs.11160fpopenalex-snapshot
Dragon’s Blood Inhibits Chronic Inflammatory and Neuropathic Pain Responses by Blocking the Synthesis and Release of Substance P in Rats
inferred← citeshttps://doi.org/10.1371/journal.pone.0129701openalex-snapshot
Up-Regulation of the Biosynthesis and Release of Substance P through Wnt/β-Catenin Signaling Pathway in Rat Dorsal Root Ganglion Cells
inferred← citeshttps://doi.org/10.33540/424openalex-snapshot
Macrophages and the Art of Controlling Pain in Inflammation and Osteoarthritis
inferred← citeshttps://doi.org/10.3748/wjg.v17.i13.1772openalex-snapshot
β-catenin accumulation in nuclei of hepatocellular carcinoma cells up-regulates glutathione-s-transferase M3 mRNA
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