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Bisphenol A Exposure In Utero Disrupts Early Oogenesis in the Mouse

https://doi.org/10.1371/journal.pgen.0030005
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/(sici)1097-0215(19980119)75:2<290::aid-ijc19>3.0.co;2-hopenalex-snapshot
Bisphenol-A induces cellular transformation, aneuploidy and DNA adduct formation in cultured Syrian hamster embryo cells
inferredcites →https://doi.org/10.1002/(sici)1097-0215(20000415)86:2<151::aid-ijc1>3.0.co;2-0openalex-snapshot
Mammalian cell transformation and aneuploidy induced by five bisphenols
inferredcites →https://doi.org/10.1002/1097-0142(197004)25:4<745::aid-cncr2820250402>3.0.co;2-2openalex-snapshot
Adenocarcinoma of the vagina in adolescence.A report of 7 cases including 6 clear-cell carcinomas (so-called mesonephromas)
inferredcites →https://doi.org/10.1002/mrd.1039openalex-snapshot
Estrogen receptor‐ and aromatase‐deficient mice provide insight into the roles of estrogen within the ovary and uterus*
inferredcites →https://doi.org/10.1006/gyno.1999.5471openalex-snapshot
Behavior of Estrogen-Associated Female Genital Tract Cancer and Its Relation to Neoplasia Following Intrauterine Exposure to Diethylstilbestrol (DES)
inferredcites →https://doi.org/10.1007/978-94-011-6621-8_32openalex-snapshot
Adenocarcinoma of the Vagina
inferredcites →https://doi.org/10.1007/bf00328080openalex-snapshot
The effects of ageing on the meiotic chromosomes of male and female mice
inferredcites →https://doi.org/10.1016/0092-8674(95)90318-6openalex-snapshot
Male mice defective in the DNA mismatch repair gene PMS2 exhibit abnormal chromosome synapsis in meiosis
inferredcites →https://doi.org/10.1016/0168-9525(86)90275-1openalex-snapshot
Manipulating the mouse embryo: A laboratory manual
inferredcites →https://doi.org/10.1016/j.cub.2004.11.038openalex-snapshot
Crossover interference
inferredcites →https://doi.org/10.1016/s0165-1218(96)00161-9openalex-snapshot
Interference with microtubules and induction of micronuclei in vitro by various bisphenols
inferredcites →https://doi.org/10.1016/s0960-9822(03)00189-1openalex-snapshot
Bisphenol A Exposure Causes Meiotic Aneuploidy in the Female Mouse
inferredcites →https://doi.org/10.1016/s1097-2765(00)80404-9openalex-snapshot
The Murine SCP3 Gene Is Required for Synaptonemal Complex Assembly, Chromosome Synapsis, and Male Fertility
inferredcites →https://doi.org/10.1023/a:1018445520117openalex-snapshot
SHORT COMMUNICATIONS A drying-down technique for the spreading of mammalian meiocytes from the male and female germline
inferredcites →https://doi.org/10.1038/35066065openalex-snapshot
To err (meiotically) is human: the genesis of human aneuploidy
inferredcites →https://doi.org/10.1038/ncb1135openalex-snapshot
Cohesin SMC1β is required for meiotic chromosome dynamics, sister chromatid cohesion and DNA recombination
inferredcites →https://doi.org/10.1038/ng1672openalex-snapshot
SMC1β-deficient female mice provide evidence that cohesins are a missing link in age-related nondisjunction
inferredcites →https://doi.org/10.1073/pnas.0406212102openalex-snapshot
Distinct DNA-damage-dependent and -independent responses drive the loss of oocytes in recombination-defective mouse mutants
inferredcites →https://doi.org/10.1073/pnas.0500172102openalex-snapshot
Juxtaposition of C(2)M and the transverse filament protein C(3)G within the central region of Drosophila synaptonemal complex
inferredcites →https://doi.org/10.1093/biolreprod/65.4.1215openalex-snapshot
In Utero Exposure to Bisphenol A Alters the Development and Tissue Organization of the Mouse Mammary Gland1
inferredcites →https://doi.org/10.1093/genetics/151.4.1569openalex-snapshot
Distribution of Crossing Over on Mouse Synaptonemal Complexes Using Immunofluorescent Localization of MLH1 Protein
inferredcites →https://doi.org/10.1093/genetics/162.1.297openalex-snapshot
Genetic Control of Mammalian Meiotic Recombination. I. Variation in Exchange Frequencies Among Males From Inbred Mouse Strains
inferredcites →https://doi.org/10.1093/humrep/17.11.2839openalex-snapshot
Determination of bisphenol A concentrations in human biological fluids reveals significant early prenatal exposure
inferredcites →https://doi.org/10.1093/humrep/deh888openalex-snapshot
Exposure to bisphenol A is associated with recurrent miscarriage
inferredcites →https://doi.org/10.1093/molehr/gah179openalex-snapshot
Bisphenol-A induces cell cycle delay and alters centrosome and spindle microtubular organization in oocytes during meiosis
inferredcites →https://doi.org/10.1095/biolreprod.103.025684openalex-snapshot
Estradiol and Its Membrane-Impermeable Conjugate (Estradiol-Bovine Serum Albumin) During In Vitro Maturation of Bovine Oocytes: Effects on Nuclear and Cytoplasmic Maturation, Cytoskeleton, and Embryo Quality
inferredcites →https://doi.org/10.1126/science.1070594openalex-snapshot
Female Germ Cell Aneuploidy and Embryo Death in Mice Lacking the Meiosis-Specific Protein SCP3
inferredcites →https://doi.org/10.1126/science.1071907openalex-snapshot
Sex Matters in Meiosis
inferredcites →https://doi.org/10.1146/annurev.genom.4.070802.110217openalex-snapshot
VARIATION IN HUMAN MEIOTIC RECOMBINATION
inferredcites →https://doi.org/10.1146/annurev.physiol.67.040403.115914openalex-snapshot
LESSONS IN ESTROGEN BIOLOGY FROM KNOCKOUT AND TRANSGENIC ANIMALS
inferredcites →https://doi.org/10.1158/0008-5472.can-06-0516openalex-snapshot
Developmental Exposure to Estradiol and Bisphenol A Increases Susceptibility to Prostate Carcinogenesis and Epigenetically Regulates Phosphodiesterase Type 4 Variant 4
inferredcites →https://doi.org/10.1159/000076809openalex-snapshot
Knockout mice provide novel insights into meiotic chromosome and telomere dynamics
inferredcites →https://doi.org/10.1159/000129914openalex-snapshot
An Air-Drying Method for Chromosome Preparations from Mouse Eggs
inferredcites →https://doi.org/10.1177/074823379801400115openalex-snapshot
A Physiologically Based Approach To the Study of Bisphenol a and Other Estrogenic Chemicals On the Size of Reproductive Organs, Daily Sperm Production, and Behavior
inferredcites →https://doi.org/10.1210/en.2004-0779openalex-snapshot
Developmental Expression of Estrogen Receptor (ER) α and ERβ in the Hamster Ovary: Regulation by Follicle-Stimulating Hormone
inferredcites →https://doi.org/10.1210/en.2005-0565openalex-snapshot
Ontogeny of Rapid Estrogen-Mediated Extracellular Signal-Regulated Kinase Signaling in the Rat Cerebellar Cortex: Potent Nongenomic Agonist and Endocrine Disrupting Activity of the Xenoestrogen Bisphenol A
inferredcites →https://doi.org/10.1210/en.2005-1159openalex-snapshot
Large Effects from Small Exposures. III. Endocrine Mechanisms Mediating Effects of Bisphenol A at Levels of Human Exposure
inferredcites →https://doi.org/10.1210/en.2005-1164openalex-snapshot
Adverse Effects of the Model Environmental Estrogen Diethylstilbestrol Are Transmitted to Subsequent Generations
inferredcites →https://doi.org/10.1210/en.2006-0189openalex-snapshot
Evidence of Altered Brain Sexual Differentiation in Mice Exposed Perinatally to Low, Environmentally Relevant Levels of Bisphenol A
inferredcites →https://doi.org/10.1210/endo.140.1.6438openalex-snapshot
Ontogeny of Estrogen Receptor-β Expression in Rat Testis**This work was supported by grants from the Swedish Medical Research Council (MFR K98–04P-12596–01A) and the Loo och Hans Ostermans Stiftelse (to G.G.J.M.K.) and a grant from the European Union (EU-PL95–1223; to B.v.d.B., P.T.v.d.S., and…
inferredcites →https://doi.org/10.1210/jc.2004-1818openalex-snapshot
Estrogen Receptors and Estrogen-Metabolizing Enzymes in Human Ovaries during Fetal Development
inferredcites →https://doi.org/10.1289/ehp.01109675openalex-snapshot
Perinatal Exposure to Low Doses of Bisphenol A Affects Body Weight, Patterns of Estrous Cyclicity, and Plasma LH Levels
inferredcites →https://doi.org/10.1289/ehp.110-1241091openalex-snapshot
Parent bisphenol A accumulation in the human maternal-fetal-placental unit.
inferredcites →https://doi.org/10.1289/ehp.7534openalex-snapshot
Urinary Concentrations of Bisphenol A and 4-Nonylphenol in a Human Reference Population

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Bisphenol A
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Di‐(2‐ethylhexyl) phthalate and bisphenol A exposure impairs mouse primordial follicle assembly in vitro
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Prenatal Exposure to Bisphenol A Disrupts Mouse Fetal Liver Maturation in a Sex-Specific Manner
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Elution of Substances from Dental Composite Materials
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Health Consequences Due to Prenatal Endocrine-Disrupting Chemical Exposure
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Endocrine-Disrupting Chemicals and Female Fertility
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Epigenetics—New Aspects of Chemicals Policy
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The Influence of Environmental Factors on Ovarian Function, Follicular Genesis, and Oocyte Quality
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Pharmacological Regulation of Gametogenesis and Fertilization: To Explore the Effects of Drugs and Environmental Toxins on Gamete Formation and Fertilization in Mammals
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Establishment and depletion of the ovarian reserve: physiology and impact of environmental chemicals
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Bisphenol A suppresses glucocorticoid target gene (ENaCγ) expression via a novel ERβ/NF-κB/GR signalling pathway in lung epithelial cells
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Putative adverse outcome pathways for female reproductive disorders to improve testing and regulation of chemicals
inferred← citeshttps://doi.org/10.1007/s00418-011-0894-zopenalex-snapshot
Bisphenol A exposure modifies methylation of imprinted genes in mouse oocytes via the estrogen receptor signaling pathway
inferred← citeshttps://doi.org/10.1007/s00439-009-0785-8openalex-snapshot
Telomere length is associated with types of chromosome 21 nondisjunction: a new insight into the maternal age effect on Down syndrome birth
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Bisphenol A exposure at an environmentally relevant dose induces meiotic abnormalities in adult male rats
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Measurement in biology is methodized by theory
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Phthalates and bisphenol do not accumulate in human follicular fluid
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Multi-oocyte follicles in the bovine ovary: occurrence, activation, and growth during early in vitro folliculogenesis in a dynamic culture bioreactor
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Structural Analysis of Bisphenol-A and its Methylene, Sulfur, and Oxygen Bridged Bisphenol Analogs
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Disruption of blastomeric F-actin: a potential early biomarker of developmental toxicity in zebrafish
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Fetal exposure to bisphenol A affects the primordial follicle formation by inhibiting the meiotic progression of oocytes
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Effects of diethylhexyl phthalate (DEHP) given neonatally on spermatogenesis of mice
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Polycystic ovary syndrome (PCOS) and endocrine disrupting chemicals (EDCs)
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Ameliorative effect of ginseng extract on phthalate and bisphenol A reprotoxicity during pregnancy in rats
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Implications of environmental toxicants on ovarian follicles: how it can adversely affect the female fertility?
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Implications of endocrine-disrupting chemicals on polycystic ovarian syndrome: A comprehensive review
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Zinc Attenuates Bisphenol A-Induced Reproductive Toxicity in Male Mice by Inhibiting Ferroptosis and Apoptosis Through Improving Zinc Homeostasis
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Dysfunctional Ovarian Stem Cells Due to Neonatal Endocrine Disruption Result in PCOS and Ovarian Insufficiency in Adult Mice
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Endocrine Disruptors Driven Female Reproductive Ailments
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Demystifying Bisphenol A-Induced Alterations in Hypothalamic-Pituitary-Ovarian Functions Leading to Polycystic Ovarian Syndrome
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Bisphenol A (BPA) Exposure In Utero Leads to Immunoregulatory Cytokine Dysregulation in the Mouse Mammary Gland: A Potential Mechanism Programming Breast Cancer Risk
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Effect of Bisphenol A on human health and its degradation by microorganisms: a review
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Genetic Mouse Models for Female Reproductive Toxicology Studies
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Bisphenol A
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Bisphenol A
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Reproductive Toxicity of Environmental Contaminants in the Female
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Environmental Factors and Female Reproduction
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Genetic Mouse Models for Female Reproductive Toxicology Studies
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Environmental Contaminants and Ovarian Toxicity
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Epigenetic Control of Oocyte Development
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Genetic Mouse Models for Female Reproductive Toxicology Studies
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Reproductive Toxicity of Environmental Contaminants in the Female
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Failure to recombine is a common feature of human oogenesis
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Which origin for polycystic ovaries syndrome: Genetic, environmental or both?
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Lack of a heritable reproductive defect in the offspring of male rainbow trout exposed to the environmental estrogen 17α-ethynylestradiol
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Alterations induced by Bisphenol A on cellular organelles and potential relevance on human health
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LncRNA HOTAIR: A master regulator of chromatin dynamics and cancer
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C. elegans as a model organism to study female reproductive health
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Transcriptomic and methylomic analysis reveal the toxicological effect of 2,3,7,8-Tetrachlorodibenzodioxin on human embryonic stem cell
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Parental exposure to bisphenol A affects pharyngeal cartilage development and causes global transcriptomic changes in zebrafish (Danio rerio) offspring
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The impact of bisphenols on reproductive system and on offspring in pigs – A review 2011–2020
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Detrimental effect of Bisphenol S in mouse germ cell cyst breakdown and primordial follicle assembly
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AOP Report: Decreased ALDH1A (RALDH) activity leading to decreased fertility via disrupted meiotic initiation of fetal oogonia
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Oocyte Biology: Do the Wheels Fall Off with Age?
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Replacement Bisphenols Adversely Affect Mouse Gametogenesis with Consequences for Subsequent Generations
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Perinatal exposure to bisphenol A impairs the Mongolian gerbil ovarian follicle dynamics and its extracellular milieu
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Carbon tetrachloride exposure induces ovarian damage through oxidative stress and inflammatory mediated ovarian fibrosis
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Controlled ovarian hyperstimulation (COH) parameters associated with euploidy rates in donor oocytes
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Bisphenol A exposure and risk of thyroid nodules in Chinese women: A case-control study
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Effect of maternal bisphenol exposure on adverse pregnancy and neonatal outcomes: The Japan Environment and Children’s study
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Dibutyl phthalate exposure disrupts the progression of meiotic prophase I by interfering with homologous recombination in fetal mouse oocytes
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Assessing endocrine disruption in freshwater fish species from a “hotspot” for estrogenic activity in sediment
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Involvement of NMDAR/PSD-95/nNOS–NO–cGMP pathway in embryonic exposure to BPA induced learning and memory dysfunction of rats
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Bisphenol A impairs reproductive fitness in zebrafish ovary: Potential involvement of oxidative/nitrosative stress, inflammatory and apoptotic mediators
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In-utero exposure to HT-2 toxin affects meiotic progression and early oogenesis in foetal oocytes by increasing oxidative stress
inferred← citeshttps://doi.org/10.1016/j.envpol.2022.120791openalex-snapshot
Foetal exposure to the bisphenols BADGE and BPAF impairs meiosis through DNA oxidation in mouse ovaries
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Inflammatory cytokines as key players of apoptosis induced by environmental estrogens in the ovary
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A cross-species comparative approach to assessing multi- and transgenerational effects of endocrine disrupting chemicals
inferred← citeshttps://doi.org/10.1016/j.fct.2013.07.007openalex-snapshot
A systematic review of Bisphenol A “low dose” studies in the context of human exposure: A case for establishing standards for reporting “low-dose” effects of chemicals
inferred← citeshttps://doi.org/10.1016/j.fct.2017.12.021openalex-snapshot
Proteomics and phosphoproteomics analysis of liver in male rats exposed to bisphenol A: Mechanism of hepatotoxicity and biomarker discovery
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2007.10.002openalex-snapshot
Proceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: executive summary
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2007.12.028openalex-snapshot
Fetal and early postnatal environmental contaminant exposures and reproductive health effects in the female
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2007.12.043openalex-snapshot
Periconception window: advising the pregnancy-planning couple
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2008.01.060openalex-snapshot
Bisphenol A exposure disrupts egg development in the mouse
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2008.01.065openalex-snapshot
Proceedings of the Summit on Environmental Challenges to Reproductive Health and Fertility: executive summary
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2008.08.067openalex-snapshot
Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2009.07.168openalex-snapshot
Urinary bisphenol a concentrations and ovarian response among women undergoing IVF
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2010.11.008openalex-snapshot
Serum unconjugated bisphenol A concentrations in women may adversely influence oocyte quality during in vitro fertilization
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2013.08.021openalex-snapshot
Chronic exposure to a low concentration of bisphenol A during follicle culture affects the epigenetic status of germinal vesicles and metaphase II oocytes
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2014.01.037openalex-snapshot
Generation of multi-oocyte follicles in the peripubertal rat ovary: link to the invasive capacity of granulosa cells?
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2014.03.024openalex-snapshot
Conjugated bisphenol A in maternal serum in relation to miscarriage risk
inferred← citeshttps://doi.org/10.1016/j.fertnstert.2016.07.1114openalex-snapshot
An old culprit but a new story: bisphenol A and “NextGen” bisphenols
inferred← citeshttps://doi.org/10.1016/j.ijheh.2019.09.001openalex-snapshot
The association between maternal occupation and down syndrome: A report from the national Down syndrome project
inferred← citeshttps://doi.org/10.1016/j.isci.2026.115608openalex-snapshot
Prenatal bisphenol A exposure reprograms SF1-lactylation pathways to promote endometriosis susceptibility
inferred← citeshttps://doi.org/10.1016/j.jhazmat.2019.121886openalex-snapshot
Genetic evidence for estrogenicity of bisphenol A in zebrafish gonadal differentiation and its signalling mechanism
inferred← citeshttps://doi.org/10.1016/j.jhazmat.2021.126076openalex-snapshot
Drivers of owning more BPA
inferred← citeshttps://doi.org/10.1016/j.jhazmat.2022.130525openalex-snapshot
Exposure to tris(2,6-dimethylphenyl) phosphate interferes with sexual differentiation via estrogen receptors 2a and 2b in zebrafish
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2011.05.002openalex-snapshot
Bisphenol A: An endocrine disruptor with widespread exposure and multiple effects
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2014.02.002openalex-snapshot
Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2017.01.014openalex-snapshot
Extranuclear-initiated estrogenic actions of endocrine disrupting chemicals: Is there toxicology beyond paracelsus?
inferred← citeshttps://doi.org/10.1016/j.jsbmb.2018.01.004openalex-snapshot
Bisphenol A stimulates adrenal cortical cell proliferation via ERβ-mediated activation of the sonic hedgehog signalling pathway
inferred← citeshttps://doi.org/10.1016/j.mce.2011.12.012openalex-snapshot
Bisphenol-A acts as a potent estrogen via non-classical estrogen triggered pathways
inferred← citeshttps://doi.org/10.1016/j.medre.2017.02.001openalex-snapshot
Efectos del bisfenol A en la reproducción femenina
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2007.08.006openalex-snapshot
Genotoxic effects of environmental estrogen-like compounds in CHO-K1 cells
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2007.10.014openalex-snapshot
Exposure of mouse oocytes to bisphenol A causes meiotic arrest but not aneuploidy
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2013.01.005openalex-snapshot
Melatonin ameliorates bisphenol A-induced DNA damage in the germ cells of adult male rats
inferred← citeshttps://doi.org/10.1016/j.mrgentox.2019.02.004openalex-snapshot
Chemically induced aneuploidy in germ cells. Part II of the report of the 2017 IWGT workgroup on assessing the risk of aneugens for carcinogenesis and hereditary diseases
inferred← citeshttps://doi.org/10.1016/j.neuro.2010.07.008openalex-snapshot
The impact of neonatal bisphenol-A exposure on sexually dimorphic hypothalamic nuclei in the female rat
inferred← citeshttps://doi.org/10.1016/j.rbmo.2010.11.009openalex-snapshot
Oocyte developmental competence and embryo development: impact of lifestyle and environmental risk factors
inferred← citeshttps://doi.org/10.1016/j.rbmo.2014.06.013openalex-snapshot
Bisphenol A, oocyte maturation, implantation, and IVF outcome: review of animal and human data
inferred← citeshttps://doi.org/10.1016/j.reprotox.2007.06.004openalex-snapshot
In vivo effects of bisphenol A in laboratory rodent studies
inferred← citeshttps://doi.org/10.1016/j.reprotox.2007.06.008openalex-snapshot
An evaluation of evidence for the carcinogenic activity of bisphenol A
inferred← citeshttps://doi.org/10.1016/j.reprotox.2007.07.006openalex-snapshot
Long-term adverse effects of neonatal exposure to bisphenol A on the murine female reproductive tract
inferred← citeshttps://doi.org/10.1016/j.reprotox.2008.01.001openalex-snapshot
No effect of route of exposure (oral; subcutaneous injection) on plasma bisphenol A throughout 24 h after administration in neonatal female mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2008.06.007openalex-snapshot
Impact of acute bisphenol-A exposure upon intrauterine implantation of fertilized ova and urinary levels of progesterone and 17β-estradiol
inferred← citeshttps://doi.org/10.1016/j.reprotox.2008.09.015openalex-snapshot
Perinatal exposure to the xenoestrogen bisphenol-A induces mammary intraductal hyperplasias in adult CD-1 mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2009.03.006openalex-snapshot
Gene expression analysis of human endometrial endothelial cells exposed to Bisphenol A
inferred← citeshttps://doi.org/10.1016/j.reprotox.2009.06.014openalex-snapshot
A CASCADE of effects of bisphenol A
inferred← citeshttps://doi.org/10.1016/j.reprotox.2010.05.012openalex-snapshot
The ReProTect Feasibility Study, a novel comprehensive in vitro approach to detect reproductive toxicants
inferred← citeshttps://doi.org/10.1016/j.reprotox.2010.07.008openalex-snapshot
Neonatal exposure to bisphenol A reduces the pool of primordial follicles in the rat ovary
inferred← citeshttps://doi.org/10.1016/j.reprotox.2010.12.002openalex-snapshot
Effect of perinatal and postnatal bisphenol A exposure to the regulatory circuits at the hypothalamus–pituitary–gonadal axis of CD-1 mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2011.06.118openalex-snapshot
Neonatal exposure to bisphenol A or diethylstilbestrol alters the ovarian follicular dynamics in the lamb
inferred← citeshttps://doi.org/10.1016/j.reprotox.2011.08.010openalex-snapshot
Preimplantation exposure to bisphenol A (BPA) affects embryo transport, preimplantation embryo development, and uterine receptivity in mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2012.02.005openalex-snapshot
Effects of estrogenic compounds on neonatal oocyte development
inferred← citeshttps://doi.org/10.1016/j.reprotox.2012.03.004openalex-snapshot
Transgenerational toxicity of Zearalenone in pigs
inferred← citeshttps://doi.org/10.1016/j.reprotox.2013.09.008openalex-snapshot
The association of bisphenol-A urinary concentrations with antral follicle counts and other measures of ovarian reserve in women undergoing infertility treatments
inferred← citeshttps://doi.org/10.1016/j.reprotox.2014.08.001openalex-snapshot
Use of ovary culture techniques in reproductive toxicology
inferred← citeshttps://doi.org/10.1016/j.reprotox.2014.11.003openalex-snapshot
Urinary bisphenol A and semen quality, the LIFE Study
inferred← citeshttps://doi.org/10.1016/j.reprotox.2015.09.006openalex-snapshot
A review of the carcinogenic potential of bisphenol A
inferred← citeshttps://doi.org/10.1016/j.reprotox.2016.03.045openalex-snapshot
Perinatal exposure to mixtures of endocrine disrupting chemicals reduces female rat follicle reserves and accelerates reproductive aging
inferred← citeshttps://doi.org/10.1016/j.reprotox.2016.11.007openalex-snapshot
Second trimester amniotic fluid bisphenol A concentration is associated with decreased birth weight in term infants
inferred← citeshttps://doi.org/10.1016/j.reprotox.2017.09.013openalex-snapshot
The effects of in utero bisphenol A exposure on ovarian follicle numbers and steroidogenesis in the F1 and F2 generations of mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2018.05.002openalex-snapshot
Perinatal BPA exposure and reproductive axis function in CD-1 mice
inferred← citeshttps://doi.org/10.1016/j.reprotox.2019.08.001openalex-snapshot
BPA interferes with StAR-mediated mitochondrial cholesterol transport to induce germline dysfunctions
inferred← citeshttps://doi.org/10.1016/j.reprotox.2024.108630openalex-snapshot
Rapid identification of reproductive toxicants among environmental chemicals using an in vivo evaluation of gametogenesis in budding yeast Saccharomyces cerevisiae
inferred← citeshttps://doi.org/10.1016/j.scitotenv.2022.153990openalex-snapshot
Effects of Gestational Exposure to Bisphenol a on the Hepatic Transcriptome and Lipidome of Rat Dams: Intergenerational Comparison of Effects in the Offspring
inferred← citeshttps://doi.org/10.1016/j.scitotenv.2022.160431openalex-snapshot
Maternal exposure to 4-vinylcyclohexene diepoxide during pregnancy induces subfertility and birth defects of offspring in mice
inferred← citeshttps://doi.org/10.1016/j.taap.2014.02.009openalex-snapshot
In utero bisphenol A exposure disrupts germ cell nest breakdown and reduces fertility with age in the mouse
inferred← citeshttps://doi.org/10.1016/j.taap.2015.03.003openalex-snapshot
The effects of in utero bisphenol A exposure on reproductive capacity in several generations of mice
inferred← citeshttps://doi.org/10.1016/j.taap.2015.10.010openalex-snapshot
Exposure to bisphenol A in young adult mice does not alter ovulation but does alter the fertilization ability of oocytes
inferred← citeshttps://doi.org/10.1016/j.taap.2018.02.011openalex-snapshot
Bisphenol A alters oocyte maturation by prematurely closing gap junctions in the cumulus cell-oocyte complex
inferred← citeshttps://doi.org/10.1016/j.tig.2007.11.010openalex-snapshot
Human female meiosis: what makes a good egg go bad?
inferred← citeshttps://doi.org/10.1016/j.tiv.2013.09.018openalex-snapshot
Effect of bisphenol A on pluripotency of mouse embryonic stem cells and differentiation capacity in mouse embryoid bodies
inferred← citeshttps://doi.org/10.1016/j.tiv.2014.07.009openalex-snapshot
Exposure to bisphenol A at physiological concentrations observed in Chinese children promotes primordial follicle growth through the PI3K/Akt pathway in an ovarian culture system
inferred← citeshttps://doi.org/10.1016/j.tiv.2019.04.009openalex-snapshot
Bisphenol A disrupts mitotic progression via disturbing spindle attachment to kinetochore and centriole duplication in cancer cell lines
inferred← citeshttps://doi.org/10.1016/j.tox.2014.06.001openalex-snapshot
Involvement of CaM-CaMKII-ERK in bisphenol A-induced Sertoli cell apoptosis
inferred← citeshttps://doi.org/10.1016/j.toxlet.2022.01.014openalex-snapshot
Pubertal exposure to acrylamide disrupts spermatogenesis by interfering with meiotic progression in male mice
inferred← citeshttps://doi.org/10.1016/j.toxlet.2025.04.008openalex-snapshot
Comparative toxicity of eleven bisphenol analogs in the nematode Caenorhabditis elegans
inferred← citeshttps://doi.org/10.1016/j.yexcr.2010.03.024openalex-snapshot
Rapid estrogen signalling in mouse primordial germ cells
inferred← citeshttps://doi.org/10.1016/j.yexcr.2012.02.012openalex-snapshot
Chromosomal, metabolic, environmental, and hormonal origins of aneuploidy in mammalian oocytes
inferred← citeshttps://doi.org/10.1016/j.yfrne.2010.06.003openalex-snapshot
Epigenetic effects of endocrine-disrupting chemicals on female reproduction: An ovarian perspective
inferred← citeshttps://doi.org/10.1016/j.ygcen.2017.11.004openalex-snapshot
Bisphenol A accelerates meiotic progression in embryonic chickens via the estrogen receptor β signaling pathway
inferred← citeshttps://doi.org/10.1016/j.yrtph.2016.01.014openalex-snapshot
The safety of the use of bisphenol A in medical devices
inferred← citeshttps://doi.org/10.1016/s0074-7750(08)00003-7openalex-snapshot
Chapter 3 Trisomy 21
inferred← citeshttps://doi.org/10.1016/s1937-6448(08)00807-1openalex-snapshot
Chapter 7 Oocyte Quality and Maternal Control of Development
inferred← citeshttps://doi.org/10.1017/cbo9781139135030.029openalex-snapshot
Cellular origin of age-related aneuploidy in mammalian oocytes
inferred← citeshttps://doi.org/10.1017/plc.2023.19openalex-snapshot
Microplastics: A threat for developing and repairing organs?
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr1openalex-snapshot
Author comment: Microplastics: A threat for developing and repairing organs? — R0/PR1
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr10openalex-snapshot
Decision: Microplastics: A threat for developing and repairing organs? — R1/PR10
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr2openalex-snapshot
Review: Microplastics: A threat for developing and repairing organs? — R0/PR2
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr3openalex-snapshot
Review: Microplastics: A threat for developing and repairing organs? — R0/PR3
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr4openalex-snapshot
Recommendation: Microplastics: A threat for developing and repairing organs? — R0/PR4
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr5openalex-snapshot
Decision: Microplastics: A threat for developing and repairing organs? — R0/PR5
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr6openalex-snapshot
Author comment: Microplastics: A threat for developing and repairing organs? — R1/PR6
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr7openalex-snapshot
Review: Microplastics: A threat for developing and repairing organs? — R1/PR7
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr8openalex-snapshot
Review: Microplastics: A threat for developing and repairing organs? — R1/PR8
inferred← citeshttps://doi.org/10.1017/plc.2023.19.pr9openalex-snapshot
Recommendation: Microplastics: A threat for developing and repairing organs? — R1/PR9
inferred← citeshttps://doi.org/10.1017/s0967199417000363openalex-snapshot
Neo-oogenesis in mammals
inferred← citeshttps://doi.org/10.1017/s2040174421000106openalex-snapshot
Prolonged atrazine exposure beginning in utero and adult uterine morphology in mice
inferred← citeshttps://doi.org/10.1021/acs.chemrestox.6b00106openalex-snapshot
Combination of Metabolomics with Cellular Assays Reveals New Biomarkers and Mechanistic Insights on Xenoestrogenic Exposures in MCF-7 Cells
inferred← citeshttps://doi.org/10.1021/acs.est.2c05212openalex-snapshot
2,4-Dichlorophenol Increases Primordial Germ Cell Numbers via ESR2a-Dependent Pathway in Zebrafish Larvae
inferred← citeshttps://doi.org/10.1021/acs.jproteome.9b00548openalex-snapshot
Bisphenol A Activates an Innate Viral Immune Response Pathway
inferred← citeshttps://doi.org/10.1021/acssuschemeng.8b05432openalex-snapshot
A Vault-Encapsulated Enzyme Approach for Efficient Degradation and Detoxification of Bisphenol A and Its Analogues
inferred← citeshttps://doi.org/10.1021/cb700210uopenalex-snapshot
Bisphenol A Directly Targets Tubulin to Disrupt Spindle Organization in Embryonic and Somatic Cells
inferred← citeshttps://doi.org/10.1021/es402764dopenalex-snapshot
Bisphenol-A (BPA), BPA Glucuronide, and BPA Sulfate in Midgestation Umbilical Cord Serum in a Northern and Central California Population
inferred← citeshttps://doi.org/10.1021/es900235fopenalex-snapshot
Oxidative Removal of Bisphenol A by Manganese Dioxide: Efficacy, Products, and Pathways
inferred← citeshttps://doi.org/10.1038/cddis.2013.203openalex-snapshot
Exposure to bisphenol A disrupts meiotic progression during spermatogenesis in adult rats through estrogen-like activity
inferred← citeshttps://doi.org/10.1038/cddis.2017.350openalex-snapshot
Di (2-ethylhexyl) phthalate exposure impairs meiotic progression and DNA damage repair in fetal mouse oocytes in vitro
inferred← citeshttps://doi.org/10.1038/gim.2013.34openalex-snapshot
The association of low socioeconomic status and the risk of having a child with Down syndrome: a report from the National Down Syndrome Project
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