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Deletion of the Mitochondrial Superoxide Dismutase sod-2 Extends Lifespan in Caenorhabditis elegans

https://doi.org/10.1371/journal.pgen.1000361

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Elevated paraquat resistance can be used as a bioassay for longevity in a genetically based long‐lived strain of Drosophila

inferredcites →https://doi.org/10.1006/abbi.1998.1060openalex-snapshot

Characterization of the Antioxidant Status of the Heterozygous Manganese Superoxide Dismutase Knockout Mouse

inferredcites →https://doi.org/10.1006/abbi.1999.1158openalex-snapshot

Mitochondrial Superoxide Decreases Yeast Survival in Stationary Phase

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Superoxide, hydrogen peroxide, and oxygen toxicity in two free-living nematode species

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Permeability of bilayer lipid membranes for superoxide (O2−) radicals

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A methyl viologen-sensitive mutant of the nematode Caenorhabditis elegans

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Metabolism in the Caenorhabditis elegans Mit mutants

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2004.04.021openalex-snapshot

Isolation of long-lived mutants in Caenorhabditis elegans using selection for resistance to juglone

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Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2007.03.034openalex-snapshot

Trends in oxidative aging theories

inferredcites →https://doi.org/10.1016/j.mad.2004.08.012openalex-snapshot

Superoxide dismutase evolution and life span regulation

inferredcites →https://doi.org/10.1016/j.mad.2007.06.005openalex-snapshot

Genetic variation for life span, resistance to paraquat, and spontaneous activity in unselected populations of Drosophila melanogaster: Implications for transgenic rescue of life span

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Reduced mitochondrial SOD displays mortality characteristics reminiscent of natural aging

inferredcites →https://doi.org/10.1016/s1534-5807(01)00071-5openalex-snapshot

Mitochondrial Electron Transport Is a Key Determinant of Life Span in Caenorhabditis elegans

inferredcites →https://doi.org/10.1016/s1567-7249(02)00069-7openalex-snapshot

Complex II inactivation is lethal in the nematode Caenorhabditis elegans

inferredcites →https://doi.org/10.1023/a:1015677828407openalex-snapshot

Longevity genes in the nematode Caenorhabditis elegans also mediate increased resistance to stress and prevent disease

inferredcites →https://doi.org/10.1038/20694openalex-snapshot

Signals from the reproductive system regulate the lifespan of C. elegans

inferredcites →https://doi.org/10.1038/366461a0openalex-snapshot

A C. elegans mutant that lives twice as long as wild type

inferredcites →https://doi.org/10.1038/nature05837openalex-snapshot

PHA-4/Foxa mediates diet-restriction-induced longevity of C. elegans

inferredcites →https://doi.org/10.1038/ng1056openalex-snapshot

A systematic RNAi screen identifies a critical role for mitochondria in C. elegans longevity

inferredcites →https://doi.org/10.1038/ng1295-376openalex-snapshot

Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase

inferredcites →https://doi.org/10.1038/sj.onc.1208207openalex-snapshot

CuZnSOD deficiency leads to persistent and widespread oxidative damage and hepatocarcinogenesis later in life

inferredcites →https://doi.org/10.1042/bj2920605openalex-snapshot

Oxidative stress and ageing in Caenorhabditis elegans

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The kinetic basis of threshold effects observed in mitochondrial diseases: a systemic approach

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RNA interference-mediated silencing of Sod2 in Drosophila leads to early adult-onset mortality and elevated endogenous oxidative stress

inferredcites →https://doi.org/10.1073/pnas.86.8.2761openalex-snapshot

Null mutation of copper/zinc superoxide dismutase in Drosophila confers hypersensitivity to paraquat and reduced longevity.

inferredcites →https://doi.org/10.1073/pnas.90.19.8905openalex-snapshot

Aging and resistance to oxidative damage in Caenorhabditis elegans.

inferredcites →https://doi.org/10.1073/pnas.92.14.6264openalex-snapshot

Mice lacking extracellular superoxide dismutase are more sensitive to hyperoxia.

inferredcites →https://doi.org/10.1073/pnas.93.18.9782openalex-snapshot

Neurodegeneration, myocardial injury, and perinatal death in mitochondrial superoxide dismutase-deficient mice.

inferredcites →https://doi.org/10.1073/pnas.95.22.13091openalex-snapshot

The genetics of caloric restriction in Caenorhabditis elegans

inferredcites →https://doi.org/10.1073/pnas.96.3.846openalex-snapshot

Mitochondrial disease in superoxide dismutase 2 mutant mice

inferredcites →https://doi.org/10.1074/jbc.271.21.12275openalex-snapshot

Superoxide Dismutase Activity Is Essential for Stationary Phase Survival in Saccharomyces cerevisiae

inferredcites →https://doi.org/10.1074/jbc.272.45.28652openalex-snapshot

Cloning, Expression, and Characterization of Two Manganese Superoxide Dismutases from Caenorhabditis elegans

inferredcites →https://doi.org/10.1074/jbc.273.43.28510openalex-snapshot

Increased Oxidative Damage Is Correlated to Altered Mitochondrial Function in Heterozygous Manganese Superoxide Dismutase Knockout Mice

inferredcites →https://doi.org/10.1074/jbc.m307700200openalex-snapshot

Compartment-specific Protection of Iron-Sulfur Proteins by Superoxide Dismutase

inferredcites →https://doi.org/10.1074/jbc.m400207200openalex-snapshot

Lack of Peroxisomal Catalase Causes a Progeric Phenotype in Caenorhabditis elegans

inferredcites →https://doi.org/10.1074/jbc.m509142200openalex-snapshot

Activation of CuZn Superoxide Dismutases from Caenorhabditis elegans Does Not Require the Copper Chaperone CCS

inferredcites →https://doi.org/10.1074/jbc.m510764200openalex-snapshot

Phenotypes of Mice Lacking Extracellular Superoxide Dismutase and Copper- and Zinc-containing Superoxide Dismutase

inferredcites →https://doi.org/10.1080/10408440701669959openalex-snapshot

Paraquat Poisonings: Mechanisms of Lung Toxicity, Clinical Features, and Treatment

inferredcites →https://doi.org/10.1093/dnares/3.3.171openalex-snapshot

Cloning, Sequencing and Mapping of a Manganese Superoxide Dismutase Gene of the Nematode Caenorhabditis elegans

inferredcites →https://doi.org/10.1093/dnares/5.1.25openalex-snapshot

A Novel Superoxide Dismutase Gene Encoding Membrane-bound and Extracellular Isoforms by Alternative Splicing in Caenorhabditis elegans

inferredcites →https://doi.org/10.1093/emboj/18.7.1783openalex-snapshot

CLK‐1 controls respiration, behavior and aging in the nematode Caenorhabditis elegans

inferredcites →https://doi.org/10.1093/genetics/139.3.1247openalex-snapshot

Mutations in the clk-1 gene of Caenorhabditis elegans affect developmental and behavioral timing.

inferredcites →https://doi.org/10.1093/genetics/165.4.2295openalex-snapshot

A Sod2 Null Mutation Confers Severely Reduced Adult Life Span in Drosophila

inferredcites →https://doi.org/10.1093/gerona/55.1.b5openalex-snapshot

Ubiquitous overexpression of CuZn superoxide dismutase does not extendlife span in mice

inferredcites →https://doi.org/10.1093/geronj/11.3.298openalex-snapshot

Aging: A Theory Based on Free Radical and Radiation Chemistry

inferredcites →https://doi.org/10.1093/geronj/49.6.b270openalex-snapshot

Thermotolerance of a Long-lived Mutant of Caenorhabditis elegans

inferredcites →https://doi.org/10.1097/00000542-199902000-00031openalex-snapshot

GAS-1

inferredcites →https://doi.org/10.1111/j.1474-9726.2007.00302.xopenalex-snapshot

Functional genomic approach to identify novel genes involved in the regulation of oxidative stress resistance and animal lifespan

inferredcites →https://doi.org/10.1111/j.1532-5415.1972.tb00787.xopenalex-snapshot

The Biologic Clock: The Mitochondria?

inferredcites →https://doi.org/10.1126/science.1077780openalex-snapshot

Rates of Behavior and Aging Specified by Mitochondrial Function During Development

inferredcites →https://doi.org/10.1126/science.272.5264.1010openalex-snapshot

Determination of Life-Span in Caenorhabditis elegans by Four Clock Genes

inferredcites →https://doi.org/10.1126/science.273.5271.59openalex-snapshot

Oxidative Stress, Caloric Restriction, and Aging

inferredcites →https://doi.org/10.1126/science.275.5302.980openalex-snapshot

Structural and Functional Conservation of the Caenorhabditis elegans Timing Gene clk-1

inferredcites →https://doi.org/10.1146/annurev.bi.64.070195.000525openalex-snapshot

SUPEROXIDE RADICAL AND SUPEROXIDE DISMUTASES

inferredcites →https://doi.org/10.1152/ajpheart.2001.281.3.h1422openalex-snapshot

Knockout mice heterozygous forSod2show alterations in cardiac mitochondrial function and apoptosis

inferredcites →https://doi.org/10.1152/physiolgenomics.00122.2003openalex-snapshot

Life-long reduction in MnSOD activity results in increased DNA damage and higher incidence of cancer but does not accelerate aging

inferredcites →https://doi.org/10.1186/1471-2199-9-9openalex-snapshot

Selection and validation of a set of reliable reference genes for quantitative sod gene expression analysis in C. elegans

inferredcites →https://doi.org/10.1196/annals.1395.055openalex-snapshot

Effects of Deleting Mitochondrial Antioxidant Genes on Life Span

inferredcites →https://doi.org/10.1371/journal.pbio.0050259openalex-snapshot

Relationship Between Mitochondrial Electron Transport Chain Dysfunction, Development, and Life Extension in Caenorhabditis elegans

inferredcites →https://doi.org/10.1534/genetics.107.080788openalex-snapshot

A Measurable Increase in Oxidative Damage Due to Reduction in Superoxide Detoxification Fails to Shorten the Life Span of Long-Lived Mitochondrial Mutants of Caenorhabditis elegans

inferredcites →https://doi.org/10.1534/genetics.167.1.161openalex-snapshot

Caenorhabditis elegans OSR-1 Regulates Behavioral and Physiological Responses to Hyperosmotic Environments

inferredcites →https://doi.org/10.18388/abp.1999_4158openalex-snapshot

Deficiency in superoxide dismutases shortens life span of yeast cells.

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Incoming claims: where this work appears in other papers’ reference lists and arguments.

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inferred← citeshttps://doi.org/10.1016/j.celrep.2014.08.075openalex-snapshot

TOR Signaling Couples Oxygen Sensing to Lifespan in C. elegans

inferred← citeshttps://doi.org/10.1016/j.celrep.2016.07.088openalex-snapshot

FOXO/DAF-16 Activation Slows Down Turnover of the Majority of Proteins in C. elegans

inferred← citeshttps://doi.org/10.1016/j.celrep.2019.04.095openalex-snapshot

Mitohormesis Primes Tumor Invasion and Metastasis

inferred← citeshttps://doi.org/10.1016/j.cmet.2012.02.013openalex-snapshot

Impaired Insulin/IGF1 Signaling Extends Life Span by Promoting Mitochondrial L-Proline Catabolism to Induce a Transient ROS Signal

inferred← citeshttps://doi.org/10.1016/j.cmet.2018.07.011openalex-snapshot

Mitohormesis in Mice via Sustained Basal Activation of Mitochondrial and Antioxidant Signaling

inferred← citeshttps://doi.org/10.1016/j.cub.2014.03.034openalex-snapshot

ROS Function in Redox Signaling and Oxidative Stress

inferred← citeshttps://doi.org/10.1016/j.devcel.2011.09.002openalex-snapshot

A Muscle-Specific p38 MAPK/Mef2/MnSOD Pathway Regulates Stress, Motor Function, and Life Span in Drosophila

inferred← citeshttps://doi.org/10.1016/j.etap.2020.103507openalex-snapshot

Oxidative damage mechanism in Saccharomyces cerevisiae cells exposed to tetrachlorobisphenol A

inferred← citeshttps://doi.org/10.1016/j.etap.2022.103846openalex-snapshot

Copper and Chromium toxicity is mediated by oxidative stress in Caenorhabditis elegans: The use of nanoparticles as an immobilization strategy

inferred← citeshttps://doi.org/10.1016/j.exger.2010.01.002openalex-snapshot

Calorie restriction in the filamentous fungus Podospora anserina

inferred← citeshttps://doi.org/10.1016/j.exger.2010.01.006openalex-snapshot

Increasing mitochondrial superoxide dismutase abundance leads to impairments in protein quality control and ROS scavenging systems and to lifespan shortening

inferred← citeshttps://doi.org/10.1016/j.exger.2010.01.014openalex-snapshot

Effects of sod gene overexpression and deletion mutation on the expression profiles of reporter genes of major detoxification pathways in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2010.02.002openalex-snapshot

Perspectives on the mitochondrial etiology of replicative aging in yeast

inferred← citeshttps://doi.org/10.1016/j.exger.2010.03.014openalex-snapshot

How increased oxidative stress promotes longevity and metabolic health: The concept of mitochondrial hormesis (mitohormesis)

inferred← citeshttps://doi.org/10.1016/j.exger.2013.12.015openalex-snapshot

The interplay between mitochondrial protein and iron homeostasis and its possible role in ageing

inferred← citeshttps://doi.org/10.1016/j.exger.2014.02.002openalex-snapshot

The mitochondrial unfolded protein response and increased longevity: Cause, consequence, or correlation?

inferred← citeshttps://doi.org/10.1016/j.exger.2014.02.015openalex-snapshot

The interplay between mitochondria and autophagy and its role in the aging process

inferred← citeshttps://doi.org/10.1016/j.exger.2014.03.016openalex-snapshot

The paradox of mitochondrial dysfunction and extended longevity

inferred← citeshttps://doi.org/10.1016/j.exger.2014.03.028openalex-snapshot

Effects of the mitochondrial respiratory chain on longevity in C. elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2015.01.042openalex-snapshot

Manganese rescues adverse effects on lifespan and development in Podospora anserina challenged by excess hydrogen peroxide

inferred← citeshttps://doi.org/10.1016/j.exger.2016.04.008openalex-snapshot

Pyrroloquinoline quinone enhances the resistance to oxidative stress and extends lifespan upon DAF-16 and SKN-1 activities in C. elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2017.08.005openalex-snapshot

Mechanisms underlying longevity: A genetic switch model of aging

inferred← citeshttps://doi.org/10.1016/j.exger.2019.02.016openalex-snapshot

Anti-ageing and anti-Parkinsonian effects of natural flavonol, tambulin from Zanthoxyllum aramatum promotes longevity in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.exger.2022.111866openalex-snapshot

Temporal changes in mitochondrial function and reactive oxygen species generation during the development of replicative senescence in human fibroblasts

inferred← citeshttps://doi.org/10.1016/j.febslet.2009.09.051openalex-snapshot

Deceptively simple but simply deceptive – Caenorhabditis elegans lifespan studies: Considerations for aging and antioxidant effects

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2009.12.015openalex-snapshot

Update on the oxidative stress theory of aging: Does oxidative stress play a role in aging or healthy aging?

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2010.03.020openalex-snapshot

Reactive oxygen species: A radical role in development?

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2011.05.010openalex-snapshot

Extending life span by increasing oxidative stress

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2011.07.020openalex-snapshot

Increased life span from overexpression of superoxide dismutase in Caenorhabditis elegans is not caused by decreased oxidative damage

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2013.05.025openalex-snapshot

Repression of the mitochondrial peroxiredoxin antioxidant system does not shorten life span but causes reduced fitness in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2014.03.038openalex-snapshot

Revisiting an age-old question regarding oxidative stress

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2014.10.576openalex-snapshot

Collaboration between mitochondria and the nucleus is key to long life in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2017.04.004openalex-snapshot

Uncoupling of oxidative stress resistance and lifespan in long-lived isp-1 mitochondrial mutants in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2018.06.016openalex-snapshot

Adaptive homeostasis and the free radical theory of ageing

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2021.05.003openalex-snapshot

Senolytics and senomorphics: Natural and synthetic therapeutics in the treatment of aging and chronic diseases

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2022.01.006openalex-snapshot

ER reductive stress caused by Ero1α S-nitrosation accelerates senescence

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2023.09.029openalex-snapshot

The glyoxylate shunt protein ICL-1 protects from mitochondrial superoxide stress through activation of the mitochondrial unfolded protein response

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2024.07.014openalex-snapshot

Superoxide signal orchestrates tetrathiomolybdate-induced longevity via ARGK-1 in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2025.01.028openalex-snapshot

MnSOD non-acetylation mimic knock-in mice exhibit dilated cardiomyopathy

inferred← citeshttps://doi.org/10.1016/j.freeradbiomed.2025.01.032openalex-snapshot

Intestine-specific disruption of mitochondrial superoxide dismutase extends longevity

inferred← citeshttps://doi.org/10.1016/j.gendis.2020.08.004openalex-snapshot

Suppression of a core metabolic enzyme dihydrolipoamide dehydrogenase (dld) protects against amyloid beta toxicity in C. elegans model of Alzheimer's disease

inferred← citeshttps://doi.org/10.1016/j.gene.2012.03.019openalex-snapshot

Living with stress: Regulation of antioxidant defense genes in the subterranean, hypoxia-tolerant mole rat, Spalax

inferred← citeshttps://doi.org/10.1016/j.gene.2016.08.013openalex-snapshot

Comparative RNA-sequencing profiling reveals novel Delta-class glutathione S-transferases relative genes expression patterns in Tribolium castaneum

inferred← citeshttps://doi.org/10.1016/j.ijbiomac.2022.01.071openalex-snapshot

Lentinan extends lifespan and increases oxidative stress resistance through DAF-16 and SKN-1 pathways in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.isci.2022.103874openalex-snapshot

Sanguinarine promotes healthspan and innate immunity through a conserved mechanism of ROS-mediated PMK-1/SKN-1 activation

inferred← citeshttps://doi.org/10.1016/j.jece.2025.116571openalex-snapshot

6-PPD quinone causes alteration in ubiquinone-mediated complex III associated with toxicity on mitochondrial function and longevity in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.jfutfo.2022.12.001openalex-snapshot

Future foods, dietary factors and healthspan

inferred← citeshttps://doi.org/10.1016/j.jgg.2019.03.011openalex-snapshot

Organelle aging: Lessons from model organisms

inferred← citeshttps://doi.org/10.1016/j.jot.2025.03.023openalex-snapshot

The mitochondrial protectant SS31 optimized decellularized Wharton's jelly scaffold improves allogeneic chondrocyte implantation-mediated articular cartilage repair

inferred← citeshttps://doi.org/10.1016/j.lfs.2018.12.029openalex-snapshot

Crosstalk between mitochondrial dysfunction, oxidative stress, and age related neurodegenerative disease: Etiologies and therapeutic strategies

inferred← citeshttps://doi.org/10.1016/j.mad.2009.03.003openalex-snapshot

The effect of different ubiquinones on lifespan in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.mad.2010.01.004openalex-snapshot

Mitochondrial electron transport chain dysfunction during development does not extend lifespan in Drosophila melanogaster

inferred← citeshttps://doi.org/10.1016/j.mad.2010.09.006openalex-snapshot

The roles of thiol oxidoreductases in yeast replicative aging

inferred← citeshttps://doi.org/10.1016/j.mad.2011.08.006openalex-snapshot

FUdR causes a twofold increase in the lifespan of the mitochondrial mutant gas-1

inferred← citeshttps://doi.org/10.1016/j.mad.2011.11.005openalex-snapshot

Structural features and bioavailability of four flavonoids and their implications for lifespan-extending and antioxidant actions in C. elegans

inferred← citeshttps://doi.org/10.1016/j.mad.2012.03.003openalex-snapshot

Manipulation of in vivo iron levels can alter resistance to oxidative stress without affecting ageing in the nematode C. elegans

inferred← citeshttps://doi.org/10.1016/j.mad.2012.03.008openalex-snapshot

A systems biological analysis links ROS metabolism to mitochondrial protein quality control

inferred← citeshttps://doi.org/10.1016/j.mad.2015.03.008openalex-snapshot

Caloric restriction, resveratrol and melatonin: Role of SIRT1 and implications for aging and related-diseases

inferred← citeshttps://doi.org/10.1016/j.mam.2011.10.004openalex-snapshot

Energy metabolism, proteotoxic stress and age-related dysfunction – Protection by carnosine

inferred← citeshttps://doi.org/10.1016/j.mam.2016.04.005openalex-snapshot

Biology of frailty: Modulation of ageing genes and its importance to prevent age-associated loss of function

inferred← citeshttps://doi.org/10.1016/j.mito.2009.11.003openalex-snapshot

Mitochondrial respiratory chain dysfunction variably increases oxidant stress in Caenorhabditis elegans

inferred← citeshttps://doi.org/10.1016/j.mito.2022.11.005openalex-snapshot

The longest-lived metazoan, Arctica islandica, exhibits high mitochondrial H2O2 removal capacities

inferred← citeshttps://doi.org/10.1016/j.molcel.2010.10.002openalex-snapshot

The Aging Stress Response

inferred← citeshttps://doi.org/10.1016/j.molmed.2010.03.001openalex-snapshot

Genetic background influences mitochondrial function: modeling mitochondrial disease for therapeutic development

inferred← citeshttps://doi.org/10.1016/j.molmed.2016.06.001openalex-snapshot

Autophagy and Immune Senescence

inferred← citeshttps://doi.org/10.1016/j.molmet.2013.02.002openalex-snapshot

Neuronal ROS signaling rather than AMPK/sirtuin-mediated energy sensing links dietary restriction to lifespan extension

inferred← citeshttps://doi.org/10.1016/j.mrrev.2013.09.001openalex-snapshot

Mutations that affect mitochondrial functions and their association with neurodegenerative diseases

inferred← citeshttps://doi.org/10.1016/j.nbd.2016.08.008openalex-snapshot

Oxidative stress is increased in C. elegans models of Huntington’s disease but does not contribute to polyglutamine toxicity phenotypes

inferred← citeshttps://doi.org/10.1016/j.neurobiolaging.2017.08.016openalex-snapshot

Improving mitochondrial function significantly reduces metabolic, visual, motor and cognitive decline in aged Drosophila melanogaster

inferred← citeshttps://doi.org/10.1016/j.ntt.2016.10.012openalex-snapshot

The genetics of isoflurane-induced developmental neurotoxicity

inferred← citeshttps://doi.org/10.1016/j.pep.2010.12.008openalex-snapshot

High-level soluble expression of recombinant human manganese superoxide dismutase in Escherichia coli, and its effects on proliferation of the leukemia cell

inferred← citeshttps://doi.org/10.1016/j.pharmthera.2010.10.005openalex-snapshot

At the stem of youth and health

inferred← citeshttps://doi.org/10.1016/j.redox.2016.12.020openalex-snapshot

Caenorhabditis elegans as a model for understanding ROS function in physiology and disease

inferred← citeshttps://doi.org/10.1016/j.redox.2016.12.026openalex-snapshot

The decay of Redox-stress Response Capacity is a substantive characteristic of aging: Revising the redox theory of aging

inferred← citeshttps://doi.org/10.1016/j.redox.2017.08.007openalex-snapshot

Hormesis enables cells to handle accumulating toxic metabolites during increased energy flux

inferred← citeshttps://doi.org/10.1016/j.redox.2022.102335openalex-snapshot

Mitochondrial thioredoxin system is required for enhanced stress resistance and extended longevity in long-lived mitochondrial mutants

inferred← citeshttps://doi.org/10.1016/j.redox.2025.103598openalex-snapshot

Transgenerational inheritance of mitochondrial hormetic oxidative stress mediated by histone H3K4me3 and H3K27me3 modifications

inferred← citeshttps://doi.org/10.1016/j.redox.2026.104319openalex-snapshot

The nuanced role of mitochondrial ROS in modulating aging and aging hallmarks

inferred← citeshttps://doi.org/10.1016/j.redox.2026.104385openalex-snapshot

Elevated mitochondrial superoxide promotes longevity through a mitochondria-to-nucleus kinase signaling pathway

inferred← citeshttps://doi.org/10.1016/j.semcdb.2023.02.010openalex-snapshot

Mitochondrial stress and aging: Lessons from C. elegans

inferred← citeshttps://doi.org/10.1016/j.stem.2013.11.006openalex-snapshot

Human iPSC-Based Modeling of Late-Onset Disease via Progerin-Induced Aging

inferred← citeshttps://doi.org/10.1016/j.tcb.2011.06.008openalex-snapshot

Taking a “good” look at free radicals in the aging process

inferred← citeshttps://doi.org/10.1016/j.tcb.2013.04.007openalex-snapshot

A cytoprotective perspective on longevity regulation

inferred← citeshttps://doi.org/10.1016/j.tem.2019.08.007openalex-snapshot

Hormesis in Health and Chronic Diseases

inferred← citeshttps://doi.org/10.1016/j.tibs.2010.04.002openalex-snapshot

Mitochondrial reactive oxygen species regulate cellular signaling and dictate biological outcomes

inferred← citeshttps://doi.org/10.1016/j.tibs.2024.07.001openalex-snapshot

Cellular oxidants and the proteostasis network: balance between activation and destruction

inferred← citeshttps://doi.org/10.1016/j.tma.2019.10.001openalex-snapshot

Recent studies on anti-aging compounds with Saccharomyces cerevisiae as a model organism

inferred← citeshttps://doi.org/10.1016/s2095-3119(17)61886-0openalex-snapshot

Transcriptome analysis of hsp18.3 functions and regulatory systems using RNA-sequencing in the red flour beetle, Tribolium castaneum

inferred← citeshttps://doi.org/10.1021/acs.est.9b06583openalex-snapshot

Surface-Related Toxicity of Polystyrene Beads to Nematodes and the Role of Food Availability

inferred← citeshttps://doi.org/10.1021/acsomega.9b01367openalex-snapshot

Growth and Virulence of Salmonella Typhimurium Mutants Deficient in Iron Uptake

inferred← citeshttps://doi.org/10.1021/jm2014315openalex-snapshot

Novel 1,2,4-Triazolo[1,5-a]pyridines and Their Fused Ring Systems Attenuate Oxidative Stress and Prolong Lifespan of Caenorhabiditis elegans

inferred← citeshttps://doi.org/10.1021/pr100703aopenalex-snapshot

Metabotyping ofCaenorhabditis elegansand their Culture Media Revealed Unique Metabolic Phenotypes Associated to Amino Acid Deficiency and Insulin-Like Signaling

inferred← citeshttps://doi.org/10.1021/pr9009639openalex-snapshot

Proteomic Analyses of Caenorhabditis elegans Dauer Larvae and Long-Lived daf-2 Mutants Implicates a Shared Detoxification System in Longevity Assurance

inferred← citeshttps://doi.org/10.1038/461486aopenalex-snapshot

A bad boy comes good

inferred← citeshttps://doi.org/10.1038/cdd.2016.123openalex-snapshot

Transient mitochondrial DNA double strand breaks in mice cause accelerated aging phenotypes in a ROS-dependent but p53/p21-independent manner

inferred← citeshttps://doi.org/10.1038/nature13163openalex-snapshot

REST and stress resistance in ageing and Alzheimer’s disease

inferred← citeshttps://doi.org/10.1038/npjamd.2016.10openalex-snapshot

Lifespan-regulating genes in C. elegans

inferred← citeshttps://doi.org/10.1038/nrm2944openalex-snapshot

Progeria syndromes and ageing: what is the connection?

inferred← citeshttps://doi.org/10.1038/s41380-022-01680-xopenalex-snapshot

Targeting the “hallmarks of aging” to slow aging and treat age-related disease: fact or fiction?

inferred← citeshttps://doi.org/10.1038/s41392-024-01839-8openalex-snapshot

Mitochondrial dysfunction: mechanisms and advances in therapy

inferred← citeshttps://doi.org/10.1038/s41418-023-01187-0openalex-snapshot

Identity, structure, and function of the mitochondrial permeability transition pore: controversies, consensus, recent advances, and future directions

inferred← citeshttps://doi.org/10.1038/s41419-020-2458-4openalex-snapshot

SK channel-mediated metabolic escape to glycolysis inhibits ferroptosis and supports stress resistance in C. elegans

inferred← citeshttps://doi.org/10.1038/s41467-021-24634-3openalex-snapshot

Dietary thiols accelerate aging of C. elegans

inferred← citeshttps://doi.org/10.1038/s41598-018-32025-wopenalex-snapshot

Genetic inhibition of an ATP synthase subunit extends lifespan in C. elegans

inferred← citeshttps://doi.org/10.1038/s41598-020-64953-xopenalex-snapshot

Consumption of Oleic Acid During Matriphagy in Free-Living Nematodes Alleviates the Toxic Effects of the Bacterial Metabolite Violacein

inferred← citeshttps://doi.org/10.1038/s41598-021-82466-zopenalex-snapshot

C. elegans electrotaxis behavior is modulated by heat shock response and unfolded protein response signaling pathways

inferred← citeshttps://doi.org/10.1038/s42255-026-01452-9openalex-snapshot

Mitochondrial superoxide regulates nuclear envelope integrity and ageing via redox-mediated lipid metabolism

inferred← citeshttps://doi.org/10.1038/s43587-023-00470-6openalex-snapshot

Tubular lysosome induction couples animal starvation to healthy aging

inferred← citeshttps://doi.org/10.1038/s43587-023-00489-9openalex-snapshot

Combinatorial interventions in aging

inferred← citeshttps://doi.org/10.1039/c0mb00116copenalex-snapshot

Proteomics analysis identifies PARK7 as an important player for renal cell resistance and survival under oxidative stress

inferred← citeshttps://doi.org/10.1042/bst20190054openalex-snapshot

Cross-talk between redox signalling and protein aggregation

inferred← citeshttps://doi.org/10.1042/cs20241851openalex-snapshot

Persistent subclinical renal injury in female rats following renal ischemia-reperfusion injury

showing the first 200 of 490

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