On the public citation graph
Synthesis and Propagation of Complement C3 by Microglia/Monocytes in the Aging Retina
0 signed · 124 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1001/archophthalmol.2009.58openalex-snapshot
Forecasting Age-Related Macular Degeneration Through the Year 2050
inferredcites →https://doi.org/10.1002/cne.903630106openalex-snapshot
Development of microglial topography in human retina
inferredcites →https://doi.org/10.1002/emmm.201302692openalex-snapshot
CCR2+ monocytes infiltrate atrophic lesions in age‐related macular disease and mediate photoreceptor degeneration in experimental subretinal inflammation in Cx3cr1 deficient mice
inferredcites →https://doi.org/10.1002/glia.22315openalex-snapshot
Para‐inflammation‐mediated retinal recruitment of bone marrow‐derived myeloid cells following whole‐body irradiation is CCL2 dependent
inferredcites →https://doi.org/10.1007/3-540-26977-0_5openalex-snapshot
Epidemiology of Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1007/978-1-4419-5635-4_1openalex-snapshot
The Case for Complement and Inflammation in AMD: Open Questions
inferredcites →https://doi.org/10.1007/bf01837367openalex-snapshot
Complement deficiency and immune complex disease
inferredcites →https://doi.org/10.1007/bf02150948openalex-snapshot
Senile macular degeneration: The involvement of immunocompetent cells
inferredcites →https://doi.org/10.1007/bf02155808openalex-snapshot
Age-related macular degeneration: ultrastructural studies of the relationship of leucocytes to angiogenesis
inferredcites →https://doi.org/10.1007/s00281-008-0112-9openalex-snapshot
Immunopathological aspects of age-related macular degeneration
inferredcites →https://doi.org/10.1016/0047-6374(90)90042-eopenalex-snapshot
Messenger RNA levels and methlation patterns of GAPDH and β-actin genes in rat liver, spleen and brain in relation to aging
inferredcites →https://doi.org/10.1016/j.exer.2006.11.015openalex-snapshot
Synthesis of complement factor H by retinal pigment epithelial cells is down-regulated by oxidized photoreceptor outer segments
inferredcites →https://doi.org/10.1016/j.imbio.2007.04.001openalex-snapshot
C1q binding and complement activation by prions and amyloids
inferredcites →https://doi.org/10.1016/j.molimm.2004.06.011openalex-snapshot
Complement: a unique innate immune sensor for danger signals
inferredcites →https://doi.org/10.1016/j.molimm.2007.09.008openalex-snapshot
Role of complement and complement regulators in the removal of apoptotic cells
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2012.06.010openalex-snapshot
A2E accumulation influences retinal microglial activation and complement regulation
inferredcites →https://doi.org/10.1016/j.neurobiolaging.2013.03.022openalex-snapshot
Gene expression changes in aging retinal microglia: relationship to microglial support functions and regulation of activation
inferredcites →https://doi.org/10.1016/j.ophtha.2008.09.055openalex-snapshot
Complement Component C3 and Risk of Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1016/j.preteyeres.2009.06.001openalex-snapshot
Para-inflammation in the aging retina
inferredcites →https://doi.org/10.1016/j.preteyeres.2009.11.003openalex-snapshot
The pivotal role of the complement system in aging and age-related macular degeneration: Hypothesis re-visited
inferredcites →https://doi.org/10.1016/j.survophthal.2005.12.001openalex-snapshot
The Role of Inflammation in the Pathogenesis of Age-related Macular Degeneration
inferredcites →https://doi.org/10.1016/s0014-4835(02)00332-9openalex-snapshot
Activated microglia in human retinitis pigmentosa, late-onset retinal degeneration, and age-related macular degeneration
inferredcites →https://doi.org/10.1016/s0039-6257(03)00030-4openalex-snapshot
Age-Related Macular Degeneration: Etiology, Pathogenesis, and Therapeutic Strategies
inferredcites →https://doi.org/10.1016/s0162-3109(00)80302-1openalex-snapshot
Complement components of the innate immune system in health and disease in the CNS
inferredcites →https://doi.org/10.1016/s0162-3109(99)00018-1openalex-snapshot
Systemic lupus erythematosus and complement deficiency: clues to a novel role for the classical complement pathway in the maintenance of immune tolerance
inferredcites →https://doi.org/10.1016/s0531-5565(03)00093-7openalex-snapshot
The aging of the retina
inferredcites →https://doi.org/10.1016/s1350-9462(00)00025-2openalex-snapshot
Immunological and Aetiological Aspects of Macular Degeneration
inferredcites →https://doi.org/10.1017/s0952523805224069openalex-snapshot
Gradients of cone differentiation and FGF expression during development of the foveal depression in macaque retina
inferredcites →https://doi.org/10.1038/eye.1995.192openalex-snapshot
Lipofuscin of the retinal pigment epithelium: A review
inferredcites →https://doi.org/10.1038/nature07201openalex-snapshot
Origin and physiological roles of inflammation
inferredcites →https://doi.org/10.1038/ng2131openalex-snapshot
Variation in complement factor 3 is associated with risk of age-related macular degeneration
inferredcites →https://doi.org/10.1038/nm1709openalex-snapshot
Oxidative damage–induced inflammation initiates age-related macular degeneration
inferredcites →https://doi.org/10.1056/nejmoa072618openalex-snapshot
Complement C3 Variant and the Risk of Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1073/pnas.0501536102openalex-snapshot
A common haplotype in the complement regulatory gene factor H ( HF1/CFH ) predisposes individuals to age-related macular degeneration
inferredcites →https://doi.org/10.1073/pnas.0705079104openalex-snapshot
Complement factor H deficiency in aged mice causes retinal abnormalities and visual dysfunction
inferredcites →https://doi.org/10.1084/jem.192.3.359openalex-snapshot
A Hierarchical Role for Classical Pathway Complement Proteins in the Clearance of Apoptotic Cells in Vivo
inferredcites →https://doi.org/10.1093/hmg/ddn075openalex-snapshot
C3 R102G polymorphism increases risk of age-related macular degeneration
inferredcites →https://doi.org/10.1111/j.1365-2249.2009.03896.xopenalex-snapshot
Complement classical pathway components are all important in clearance of apoptotic and secondary necrotic cells
inferredcites →https://doi.org/10.1111/j.1442-9071.1984.tb01120.xopenalex-snapshot
AN ULTRASTRUCTURAL STUDY OF THE ROLE OF LEUCOCYTES AND FIBROBLASTS IN THE BREAKDOWN OF BRUCH'S MEMBRANE
inferredcites →https://doi.org/10.1126/science.1109557openalex-snapshot
Complement Factor H Polymorphism in Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1126/science.1110189openalex-snapshot
Complement Factor H Polymorphism and Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1126/science.1110359openalex-snapshot
Complement Factor H Variant Increases the Risk of Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1136/bjo.2007.129742openalex-snapshot
Immune cells in the human choroid
inferredcites →https://doi.org/10.1136/bjo.2009.165563openalex-snapshot
Bruch's membrane and choroidal macrophages in early and advanced age-related macular degeneration
inferredcites →https://doi.org/10.1136/bjo.65.1.23openalex-snapshot
Aging and degeneration of the human macula. 1. Outer nuclear layer and photoreceptors.
inferredcites →https://doi.org/10.1136/bjo.87.9.1159openalex-snapshot
Differential expression of GFAP in early v late AMD: a quantitative analysis
inferredcites →https://doi.org/10.1159/000019076openalex-snapshot
C1q Knock-Out Mice for the Study of Complement Deficiency in Autoimmune Disease
inferredcites →https://doi.org/10.1167/iovs.03-0038openalex-snapshot
Macrophage Depletion Diminishes Lesion Size and Severity in Experimental Choroidal Neovascularization
inferredcites →https://doi.org/10.1167/iovs.03-0097openalex-snapshot
Macrophage Depletion Inhibits Experimental Choroidal Neovascularization
inferredcites →https://doi.org/10.1167/iovs.08-1738openalex-snapshot
Characteristics of Bone Marrow–Derived Microglia in the Normal and Injured Retina
inferredcites →https://doi.org/10.1167/iovs.08-3231openalex-snapshot
Complement Component 3 ( C3 ) Haplotypes and Risk of Advanced Age-Related Macular Degeneration
inferredcites →https://doi.org/10.1167/iovs.09-3462openalex-snapshot
The Drusenlike Phenotype in Aging Ccl2 -Knockout Mice Is Caused by an Accelerated Accumulation of Swollen Autofluorescent Subretinal Macrophages
inferredcites →https://doi.org/10.1167/iovs.09-4478openalex-snapshot
Age-Related Macular Degeneration: The Molecular Link between Oxidative Damage, Tissue-Specific Inflammation and Outer Retinal Disease
inferredcites →https://doi.org/10.1167/iovs.09-5103openalex-snapshot
Immune Activation in Retinal Aging: A Gene Expression Study
inferredcites →https://doi.org/10.1167/iovs.10-6002openalex-snapshot
Expression of Complement Component 3 (C3) from an Adenovirus Leads to Pathology in the Murine Retina
inferredcites →https://doi.org/10.1167/iovs.10-6010openalex-snapshot
Early Focal Expression of the Chemokine Ccl2 by Müller Cells during Exposure to Damage-Inducing Bright Continuous Light
inferredcites →https://doi.org/10.1167/iovs.10-7119openalex-snapshot
Analysis of Complement Expression in Light-Induced Retinal Degeneration: Synthesis and Deposition of C3 by Microglia/Macrophages Is Associated with Focal Photoreceptor Degeneration
inferredcites →https://doi.org/10.1167/iovs.11-7328openalex-snapshot
Age-Related Macular Degeneration and the Other Double Helix The Cogan Lecture
inferredcites →https://doi.org/10.1167/iovs.11-7815openalex-snapshot
The Effect of Photo-oxidative Stress and Inflammatory Cytokine on Complement Factor H Expression in Retinal Pigment Epithelial Cells
inferredcites →https://doi.org/10.1186/1742-2094-9-137openalex-snapshot
Complement activation in the injured central nervous system: another dual-edged sword?
inferredcites →https://doi.org/10.1186/1742-2094-9-221openalex-snapshot
Small interfering RNA-mediated suppression of Ccl2 in Müller cells attenuates microglial recruitment and photoreceptor death following retinal degeneration
inferredcites →https://doi.org/10.1186/gm315openalex-snapshot
Systems-level analysis of age-related macular degeneration reveals global biomarkers and phenotype-specific functional networks
inferredcites →https://doi.org/10.1371/journal.pone.0002199openalex-snapshot
C2 and CFB Genes in Age-Related Maculopathy and Joint Action with CFH and LOC387715 Genes
inferredcites →https://doi.org/10.3109/02713681003682925openalex-snapshot
Brief Exposure to Damaging Light Causes Focal Recruitment of Macrophages, and Long-Term Destabilization of Photoreceptors in the Albino Rat Retina
inferredcites →https://doi.org/10.4049/jimmunol.1203487openalex-snapshot
Complement in Immune and Inflammatory Disorders: Pathophysiological Mechanisms
inferredcites →https://doi.org/10.4049/jimmunol.158.10.4525openalex-snapshot
C1q binds directly and specifically to surface blebs of apoptotic human keratinocytes: complement deficiency and systemic lupus erythematosus revisited
inferredcites →https://doi.org/10.4049/jimmunol.180.4.2329openalex-snapshot
C1q Binds Phosphatidylserine and Likely Acts as a Multiligand-Bridging Molecule in Apoptotic Cell Recognition
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3811833openalex-extracted
Heterochronic Fecal Microbiota Transfer Reverses Hallmarks of the Aging Murine Gut, Eye and Brain
inferred← citeshttps://doi.org/10.1002/iid3.70203openalex-snapshot
Role of P2X7R in Retinal Diseases: A Review
inferred← citeshttps://doi.org/10.1007/978-3-030-66014-7_5openalex-snapshot
Innate Immunity in Age-Related Macular Degeneration
inferred← citeshttps://doi.org/10.1007/s00018-022-04670-yopenalex-snapshot
Vitreous humor proteome: unraveling the molecular mechanisms underlying proliferative and neovascular vitreoretinal diseases
inferred← citeshttps://doi.org/10.1007/s12035-020-02158-zopenalex-snapshot
Inhibition of microRNA-155 Protects Retinal Function Through Attenuation of Inflammation in Retinal Degeneration
inferred← citeshttps://doi.org/10.1007/s40620-016-0365-7openalex-snapshot
The clinical implications of the unique glomerular complement deposition pattern in transplant glomerulopathy
inferred← citeshttps://doi.org/10.1016/j.bbi.2019.08.004openalex-snapshot
Complement activation sustains neuroinflammation and deteriorates adult neurogenesis and spatial memory impairment in rat hippocampus following sleep deprivation
inferred← citeshttps://doi.org/10.1016/j.ophtha.2019.07.011openalex-snapshot
Complement C3 Inhibitor Pegcetacoplan for Geographic Atrophy Secondary to Age-Related Macular Degeneration
inferred← citeshttps://doi.org/10.1016/j.pneurobio.2018.05.006openalex-snapshot
Retinal microglia – A key player in healthy and diseased retina
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2014.11.004openalex-snapshot
Retinal microglia: Just bystander or target for therapy?
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2014.11.005openalex-snapshot
Complement activation and choriocapillaris loss in early AMD: Implications for pathophysiology and therapy
inferred← citeshttps://doi.org/10.1016/j.preteyeres.2016.12.001openalex-snapshot
Pharmacokinetic aspects of retinal drug delivery
inferred← citeshttps://doi.org/10.1016/j.smim.2022.101633openalex-snapshot
Emerging opportunities for C3 inhibition in the eye
inferred← citeshttps://doi.org/10.1038/s41467-025-58708-3openalex-snapshot
Myeloid lineage C3 induces reactive gliosis and neuronal stress during CNS inflammation
inferred← citeshttps://doi.org/10.1038/s41598-018-20733-2openalex-snapshot
Blockade of microglial adenosine A2A receptor impacts inflammatory mechanisms, reduces ARPE-19 cell dysfunction and prevents photoreceptor loss in vitro
inferred← citeshttps://doi.org/10.1038/s41598-020-58849-zopenalex-snapshot
Caspase-1-dependent inflammasomes mediate photoreceptor cell death in photo-oxidative damage-induced retinal degeneration
inferred← citeshttps://doi.org/10.1080/08164622.2023.2279734openalex-snapshot
Understanding how ageing impacts ganglion cell susceptibility to injury in glaucoma
inferred← citeshttps://doi.org/10.1084/jem.20190009openalex-snapshot
C3- and CR3-dependent microglial clearance protects photoreceptors in retinitis pigmentosa
inferred← citeshttps://doi.org/10.1097/icu.0000000000000845openalex-snapshot
Treatment of geographic atrophy: an update on data related to pegcetacoplan
inferred← citeshttps://doi.org/10.1101/413088openalex-snapshot
Cell type-specific complement expression from healthy and diseased retinae
inferred← citeshttps://doi.org/10.1155/2015/673090openalex-snapshot
Contribution of Microglia‐Mediated Neuroinflammation to Retinal Degenerative Diseases
inferred← citeshttps://doi.org/10.1159/000431141openalex-snapshot
Complement Activation and Inhibition in Retinal Diseases
inferred← citeshttps://doi.org/10.1167/iovs.14-15449openalex-snapshot
Identification of miRNAs in a Model of Retinal Degenerations
inferred← citeshttps://doi.org/10.1167/iovs.16-19727openalex-snapshot
Senescence Increases Choroidal Endothelial Stiffness and Susceptibility to Complement Injury: Implications for Choriocapillaris Loss in AMD
inferred← citeshttps://doi.org/10.1167/iovs.17-21672openalex-snapshot
Retinal Macrophages Synthesize C3 and Activate Complement in AMD and in Models of Focal Retinal Degeneration
inferred← citeshttps://doi.org/10.1167/iovs.18-24623openalex-snapshot
MicroRNA-124 Dysregulation Is Associated With Retinal Inflammation and Photoreceptor Death in the Degenerating Retina
inferred← citeshttps://doi.org/10.1167/iovs.18-24627openalex-snapshot
Photoreceptor Survival Is Regulated by GSTO1-1 in the Degenerating Retina
inferred← citeshttps://doi.org/10.1177/15353702211052245openalex-snapshot
Complement factor H deficiency combined with smoking promotes retinal degeneration in a novel mouse model
inferred← citeshttps://doi.org/10.1186/s12974-016-0514-xopenalex-snapshot
The broad-spectrum chemokine inhibitor NR58-3.14.3 modulates macrophage-mediated inflammation in the diseased retina
inferred← citeshttps://doi.org/10.1186/s12974-025-03418-2openalex-snapshot
Moss-derived human complement factor H modulates retinal immune response and attenuates retinal degeneration
inferred← citeshttps://doi.org/10.1186/s13024-017-0175-yopenalex-snapshot
Microglia-derived IL-1β promotes chemokine expression by Müller cells and RPE in focal retinal degeneration
inferred← citeshttps://doi.org/10.1186/s13024-018-0278-0openalex-snapshot
Subretinal macrophages produce classical complement activator C1q leading to the progression of focal retinal degeneration
inferred← citeshttps://doi.org/10.1186/s40168-022-01243-wopenalex-snapshot
Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.1371/journal.pone.0193740openalex-snapshot
The use of the vaccinia virus complement control protein (VCP) in the rat retina
inferred← citeshttps://doi.org/10.1523/jneurosci.2117-15.2016openalex-snapshot
Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease
inferred← citeshttps://doi.org/10.2174/1570159x15666170720094529openalex-snapshot
Modulation of the Immune System for the Treatment of Glaucoma
inferred← citeshttps://doi.org/10.25911/5d78d614a6941openalex-snapshot
Characterisation of novel therapies to mitigate inflammation in retinal degenerations
inferred← citeshttps://doi.org/10.3389/fimmu.2016.00542openalex-snapshot
Complement Regulator FHR-3 Is Elevated either Locally or Systemically in a Selection of Autoimmune Diseases
inferred← citeshttps://doi.org/10.3389/fimmu.2017.01868openalex-snapshot
Abnormal Complement Activation and Inflammation in the Pathogenesis of Retinopathy of Prematurity
inferred← citeshttps://doi.org/10.3389/fimmu.2020.00154openalex-snapshot
A Systematic Investigation on Complement Pathway Activation in Diabetic Retinopathy
inferred← citeshttps://doi.org/10.3389/fimmu.2025.1671438openalex-snapshot
Unraveling the immune response in optic nerve injury: implications for retinal ganglion cell protection
inferred← citeshttps://doi.org/10.3389/fncel.2021.718087openalex-snapshot
Cytokine and Complement Response in the Glaucomatous βB1-CTGF Mouse Model
inferred← citeshttps://doi.org/10.3389/fphar.2020.591908openalex-snapshot
Control of Complement Activation by the Long Pentraxin PTX3: Implications in Age-Related Macular Degeneration
inferred← citeshttps://doi.org/10.3390/antiox10060848openalex-snapshot
Molecular Mechanisms Related to Oxidative Stress in Retinitis Pigmentosa
inferred← citeshttps://doi.org/10.3390/cells11132091openalex-snapshot
Microglia Phenotypes in Aging and Neurodegenerative Diseases
inferred← citeshttps://doi.org/10.3390/cells12131805openalex-snapshot
Soluble Collectin 11 (CL-11) Acts as an Immunosuppressive Molecule Potentially Used by Stem Cell-Derived Retinal Epithelial Cells to Modulate T Cell Response
inferred← citeshttps://doi.org/10.3390/jcm3041234openalex-snapshot
Age-Related Macular Degeneration: A Disease of Systemic or Local Complement Dysregulation?
inferred← citeshttps://doi.org/10.4252/wjsc.v7.i8.1090openalex-snapshot
Complement activation in the context of stem cells and tissue repair
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695971openalex-snapshot
Additional file 2 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695974openalex-snapshot
Additional file 3 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695977openalex-snapshot
Additional file 4 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695980openalex-snapshot
Additional file 5 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695983openalex-snapshot
Additional file 6 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
inferred← citeshttps://doi.org/10.6084/m9.figshare.19695986openalex-snapshot
Additional file 7 of Fecal microbiota transfer between young and aged mice reverses hallmarks of the aging gut, eye, and brain
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.