On the public citation graph

Antisense Oligonucleotide Induction of Progerin in Human Myogenic Cells

https://doi.org/10.1371/journal.pone.0098306

0 signed · 72 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/1098-1004(200012)16:6<451::aid-humu1>3.0.co;2-9openalex-snapshot

Mutations in the LMNA gene encoding lamin A/C

inferredcites →https://doi.org/10.1002/ana.23982openalex-snapshot

Eteplirsen for the treatment of Duchenne muscular dystrophy

inferredcites →https://doi.org/10.1002/jgm.1288openalex-snapshot

In vivo comparison of 2′‐O‐methyl phosphorothioate and morpholino antisense oligonucleotides for Duchenne muscular dystrophy exon skipping

inferredcites →https://doi.org/10.1002/jgm.295openalex-snapshot

Improved antisense oligonucleotide induced exon skipping in the mdx mouse model of muscular dystrophy

inferredcites →https://doi.org/10.1002/jgm.838openalex-snapshot

Dystrophin expression in the mdx mouse after localised and systemic administration of a morpholino antisense oligonucleotide

inferredcites →https://doi.org/10.1016/j.amjmed.2005.10.049openalex-snapshot

Inflammatory Markers and Loss of Muscle Mass (Sarcopenia) and Strength

inferredcites →https://doi.org/10.1016/j.ceb.2011.11.007openalex-snapshot

The functions of the nuclear envelope in mediating the molecular crosstalk between the nucleus and the cytoplasm

inferredcites →https://doi.org/10.1016/j.febslet.2005.02.047openalex-snapshot

Are splicing mutations the most frequent cause of hereditary disease?

inferredcites →https://doi.org/10.1016/j.freeradbiomed.2006.01.036openalex-snapshot

Absence of CuZn superoxide dismutase leads to elevated oxidative stress and acceleration of age-dependent skeletal muscle atrophy

inferredcites →https://doi.org/10.1016/j.mad.2008.04.003openalex-snapshot

HGPS and related premature aging disorders: From genomic identification to the first therapeutic approaches

inferredcites →https://doi.org/10.1016/j.nmd.2011.06.1063openalex-snapshot

P5.34 Alternative splicing of lamin A leads to age-dependent accumulation of progerin transcript in normal human muscle and sporadic IBM

inferredcites →https://doi.org/10.1016/j.stem.2013.11.006openalex-snapshot

Human iPSC-Based Modeling of Late-Onset Disease via Progerin-Induced Aging

inferredcites →https://doi.org/10.1016/s0072-9752(07)86018-0openalex-snapshot

Muscle diseases and aging

inferredcites →https://doi.org/10.1016/s0140-6736(11)60756-3openalex-snapshot

Exon skipping and dystrophin restoration in patients with Duchenne muscular dystrophy after systemic phosphorodiamidate morpholino oligomer treatment: an open-label, phase 2, dose-escalation study

inferredcites →https://doi.org/10.1016/s0531-5565(01)00220-0openalex-snapshot

The biochemistry of aging muscle

inferredcites →https://doi.org/10.1023/a:1013270512172openalex-snapshot

Mitochondrial alterations, cellular response to oxidative stress and defective degradation of proteins in aging

inferredcites →https://doi.org/10.1038/6799openalex-snapshot

Mutations in the gene encoding lamin A/C cause autosomal dominant Emery-Dreifuss muscular dystrophy

inferredcites →https://doi.org/10.1038/cdd.2010.183openalex-snapshot

Prelamin A-mediated recruitment of SUN1 to the nuclear envelope directs nuclear positioning in human muscle

inferredcites →https://doi.org/10.1038/mtna.2012.40openalex-snapshot

Targeted Exon Skipping to Address “Leaky” Mutations in the Dystrophin Gene

inferredcites →https://doi.org/10.1038/nature01629openalex-snapshot

Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome

inferredcites →https://doi.org/10.1038/ncb1708openalex-snapshot

Lamin A-dependent misregulation of adult stem cells associated with accelerated ageing

inferredcites →https://doi.org/10.1038/nm1204openalex-snapshot

Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome

inferredcites →https://doi.org/10.1038/sj.gt.3302800openalex-snapshot

Antisense oligonucleotide-induced exon skipping restores dystrophin expression in vitro in a canine model of DMD

inferredcites →https://doi.org/10.1038/sj.mt.6300006openalex-snapshot

The Influence of Antisense Oligonucleotide Length on Dystrophin Exon Skipping

inferredcites →https://doi.org/10.1038/sj.mt.6300095openalex-snapshot

Antisense Oligonucleotide-induced Exon Skipping Across the Human Dystrophin Gene Transcript

inferredcites →https://doi.org/10.1073/pnas.0505767102openalex-snapshot

Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes

inferredcites →https://doi.org/10.1073/pnas.0700854104openalex-snapshot

Alterations in mitosis and cell cycle progression caused by a mutant lamin A known to accelerate human aging

inferredcites →https://doi.org/10.1073/pnas.83.17.6450openalex-snapshot

cDNA sequencing of nuclear lamins A and C reveals primary and secondary structural homology to intermediate filament proteins.

inferredcites →https://doi.org/10.1073/pnas.90.18.8673openalex-snapshot

Restoration of correct splicing in thalassemic pre-mRNA by antisense oligonucleotides.

inferredcites →https://doi.org/10.1074/jbc.271.16.9249openalex-snapshot

An Alternative Splicing Product of the Lamin A/C Gene Lacks Exon 10

inferredcites →https://doi.org/10.1083/jcb.200204149openalex-snapshot

Lamin A/C speckles mediate spatial organization of splicing factor compartments and RNA polymerase II transcription

inferredcites →https://doi.org/10.1086/339274openalex-snapshot

Homozygous Defects in LMNA, Encoding Lamin A/C Nuclear-Envelope Proteins, Cause Autosomal Recessive Axonal Neuropathy in Human (Charcot-Marie-Tooth Disorder Type 2) and Mouse

inferredcites →https://doi.org/10.1093/gerona/glr041openalex-snapshot

Neuromuscular Contributions to Age-Related Weakness

inferredcites →https://doi.org/10.1093/hmg/ddh265openalex-snapshot

Lamin A and ZMPSTE24 (FACE-1) defects cause nuclear disorganization and identify restrictive dermopathy as a lethal neonatal laminopathy

inferredcites →https://doi.org/10.1093/hmg/ddi159openalex-snapshot

Loss of ZMPSTE24 (FACE-1) causes autosomal recessive restrictive dermopathy and accumulation of Lamin A precursors

inferredcites →https://doi.org/10.1093/hmg/ddp184openalex-snapshot

Activating the synthesis of progerin, the mutant prelamin A in Hutchinson–Gilford progeria syndrome, with antisense oligonucleotides

inferredcites →https://doi.org/10.1093/hmg/ddr385openalex-snapshot

A conserved splicing mechanism of the LMNA gene controls premature aging

inferredcites →https://doi.org/10.1093/hmg/ddr600openalex-snapshot

A single administration of morpholino antisense oligomer rescues spinal muscular atrophy in mouse

inferredcites →https://doi.org/10.1093/nar/26.1.285openalex-snapshot

The human gene mutation database

inferredcites →https://doi.org/10.1101/gad.1364906openalex-snapshot

Lamin A/C and emerin are critical for skeletal muscle satellite cell differentiation

inferredcites →https://doi.org/10.1101/gad.842600openalex-snapshot

Nuclear organization of DNA replication in primary mammalian cells

inferredcites →https://doi.org/10.1126/science.1090100openalex-snapshot

Genome-Wide Survey of Human Alternative Pre-mRNA Splicing with Exon Junction Microarrays

inferredcites →https://doi.org/10.1126/science.1127168openalex-snapshot

Lamin A-Dependent Nuclear Defects in Human Aging

inferredcites →https://doi.org/10.1146/annurev.biochem.72.121801.161720openalex-snapshot

Mechanisms of Alternative Pre-Messenger RNA Splicing

inferredcites →https://doi.org/10.1161/circulationaha.109.902056openalex-snapshot

Prelamin A Acts to Accelerate Smooth Muscle Cell Senescence and Is a Novel Biomarker of Human Vascular Aging

inferredcites →https://doi.org/10.1172/jci19547openalex-snapshot

Therapeutic potential of antisense oligonucleotides as modulators of alternative splicing

inferredcites →https://doi.org/10.1172/jci200319547openalex-snapshot

Therapeutic potential of antisense oligonucleotides as modulators of alternative splicing

inferredcites →https://doi.org/10.1172/jci66969openalex-snapshot

Reducing TMPRSS6 ameliorates hemochromatosis and β-thalassemia in mice

inferredcites →https://doi.org/10.1186/1471-2121-5-30openalex-snapshot

Cytoskeletal influences on nuclear shape in granulocytic HL-60 cells

inferredcites →https://doi.org/10.1186/gm13openalex-snapshot

The Human Gene Mutation Database: 2008 update

inferredcites →https://doi.org/10.1194/jlr.m400492-jlr200openalex-snapshot

An apolipoprotein B antisense oligonucleotide lowers LDL cholesterol in hyperlipidemic mice without causing hepatic steatosis

inferredcites →https://doi.org/10.1196/annals.1359.001openalex-snapshot

Knock‐down of the Cytoprotective Gene, Clusterin, to Enhance Hormone and Chemosensitivity in Prostate and Other Cancers

inferredcites →https://doi.org/10.1212/01.wnl.0000069460.53438.38openalex-snapshot

Single section Western blot

inferredcites →https://doi.org/10.1242/jcs.01642openalex-snapshot

Nesprin-2 is a multi-isomeric protein that binds lamin and emerin at the nuclear envelope and forms a subcellular network in skeletal muscle

inferredcites →https://doi.org/10.1242/jcs.03263openalex-snapshot

DNA damage responses in progeroid syndromes arise from defective maturation of prelamin A

inferredcites →https://doi.org/10.1371/journal.pone.0001269openalex-snapshot

The Mutant Form of Lamin A that Causes Hutchinson-Gilford Progeria Is a Biomarker of Cellular Aging in Human Skin

inferredcites →https://doi.org/10.1371/journal.pone.0003910openalex-snapshot

Premature Aging in Skeletal Muscle Lacking Serum Response Factor

inferredcites →https://doi.org/10.2174/156652305774329249openalex-snapshot

RNA Splicing Manipulation: Strategies to Modify Gene Expression for a Variety of Therapeutic Outcomes

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4057449openalex-extracted

Inhibition of Interferon Alpha by a Peptide-Conjugated Phosphorodiamidate Morpholino Oligomer Increases Influenza Virus Replication in Chicken Embryo Fibroblast (DF-1) Cells; An Alternative Strategy to Increasing Vaccine Production in Cell Culture

inferred← citeshttps://doi.org/10.2139/ssrn.4199811openalex-extracted

Modified Antisense Oligonucleotides Targeting Hutchinson-Gilford Progeria Syndrome Using an Optimized Delivery Strategy

inferred← citeshttps://doi.org/10.1002/humu.22945openalex-snapshot

Enhanced SRSF5 Protein Expression Reinforces Lamin A mRNA Production in HeLa Cells and Fibroblasts of Progeria Patients

inferred← citeshttps://doi.org/10.1007/s12010-021-03514-yopenalex-snapshot

Progeria—a Rare Genetic Condition with Accelerated Ageing Process

inferred← citeshttps://doi.org/10.1016/j.arr.2015.10.002openalex-snapshot

Pluripotent stem cells to model Hutchinson-Gilford progeria syndrome (HGPS): Current trends and future perspectives for drug discovery

inferred← citeshttps://doi.org/10.1016/j.bbrep.2022.101411openalex-snapshot

Nuclear envelope morphology change upon repetitive treatment with modified antisense oligonucleotides targeting Hutchinson-Gilford Progeria Syndrome

inferred← citeshttps://doi.org/10.1038/npjamd.2016.26openalex-snapshot

Metformin decreases progerin expression and alleviates pathological defects of Hutchinson–Gilford progeria syndrome cells

inferred← citeshttps://doi.org/10.1038/s41598-019-49385-6openalex-snapshot

Reduction of integrin alpha 4 activity through splice modulating antisense oligonucleotides

inferred← citeshttps://doi.org/10.1038/s41598-021-94639-xopenalex-snapshot

Induction of cryptic pre-mRNA splice-switching by antisense oligonucleotides

inferred← citeshttps://doi.org/10.1039/9781788015714-00103openalex-snapshot

Splicing Modulation for Therapeutics

inferred← citeshttps://doi.org/10.1042/cs20190066openalex-snapshot

Using nuclear envelope mutations to explore age-related skeletal muscle weakness

inferred← citeshttps://doi.org/10.1099/jmm.0.001807openalex-snapshot

An alternative strategy to increasing influenza virus replication for vaccine production in chicken embryo fibroblast (DF-1) cells by inhibiting interferon alpha and beta using peptide-conjugated phosphorodiamidate morpholino oligomers

inferred← citeshttps://doi.org/10.2147/chc.s59507openalex-snapshot

Emerging perspectives on laminopathies

inferred← citeshttps://doi.org/10.70675/e5ce6d96z7253z4119zb119zc07f3977425dopenalex-snapshot

Role de la PARylation sur l’apparition des troubles cardiovasculaires dans la progeria de Hutchinson-Gilford

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.