On the public citation graph
Specific Depletion of Ly6Chi Inflammatory Monocytes Prevents Immunopathology in Experimental Cerebral Malaria
0 signed · 118 inferred · view as graph
Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.
Is this your work? Sign in with ORCID and put your name on the
claims that are yours.
Reference health. See whether every reference in this work resolves to a
known work and none has been retracted — and get a dated CiteStamped
badge you can embed.
What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/art.22854openalex-snapshot
Targeting of Gr‐1+,CCR2+ monocytes in collagen‐induced arthritis
inferredcites →https://doi.org/10.1016/0022-1759(94)90012-4openalex-snapshot
Liposome mediated depletion of macrophages: mechanism of action, preparation of liposomes and applications
inferredcites →https://doi.org/10.1016/j.cellimm.2012.07.005openalex-snapshot
In vivo imaging implicates CCR2+ monocytes as regulators of neutrophil recruitment during arthritis
inferredcites →https://doi.org/10.1016/j.chom.2009.10.007openalex-snapshot
Inflammatory Monocytes Facilitate Adaptive CD4 T Cell Responses during Respiratory Fungal Infection
inferredcites →https://doi.org/10.1016/j.coi.2010.04.008openalex-snapshot
Monocytes mediate mucosal immunity to Toxoplasma gondii
inferredcites →https://doi.org/10.1016/j.ijpara.2006.02.016openalex-snapshot
Immunopathogenesis of cerebral malaria
inferredcites →https://doi.org/10.1016/j.immuni.2008.05.019openalex-snapshot
Gr1+ Inflammatory Monocytes Are Required for Mucosal Resistance to the Pathogen Toxoplasma gondii
inferredcites →https://doi.org/10.1016/j.immuni.2012.05.029openalex-snapshot
Inflammatory Monocytes Activate Memory CD8+ T and Innate NK Lymphocytes Independent of Cognate Antigen during Microbial Pathogen Invasion
inferredcites →https://doi.org/10.1016/j.micinf.2011.04.006openalex-snapshot
Induction of pro-inflammatory mediators in Plasmodium berghei infected BALB/c mice breaks blood-brain-barrier and leads to cerebral malaria in an IL-12 dependent manner
inferredcites →https://doi.org/10.1016/j.pt.2012.07.008openalex-snapshot
The diverse roles of monocytes in inflammation caused by protozoan parasitic diseases
inferredcites →https://doi.org/10.1016/j.smim.2006.09.002openalex-snapshot
Adaptations of intestinal macrophages to an antigen-rich environment
inferredcites →https://doi.org/10.1016/s0140-6736(12)60034-8openalex-snapshot
Global malaria mortality between 1980 and 2010: a systematic analysis
inferredcites →https://doi.org/10.1016/s1074-7613(03)00174-2openalex-snapshot
Blood Monocytes Consist of Two Principal Subsets with Distinct Migratory Properties
inferredcites →https://doi.org/10.1038/icb.2008.19openalex-snapshot
Blood monocytes: distinct subsets, how they relate to dendritic cells, and their possible roles in the regulation of T‐cell responses
inferredcites →https://doi.org/10.1038/ni1309openalex-snapshot
Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2
inferredcites →https://doi.org/10.1038/nm914openalex-snapshot
Bone marrow as a priming site for T-cell responses to blood-borne antigen
inferredcites →https://doi.org/10.1046/j.1365-2249.2000.01196.xopenalex-snapshot
Neutrophils play a critical role in the pathogenesis of experimental cerebral malaria
inferredcites →https://doi.org/10.1073/pnas.0806727105openalex-snapshot
Blood-stage Plasmodium infection induces CD8 + T lymphocytes to parasite-expressed antigens, largely regulated by CD8α + dendritic cells
inferredcites →https://doi.org/10.1093/brain/aws058openalex-snapshot
Licensing of myeloid cells promotes central nervous system autoimmunity and is controlled by peroxisome proliferator-activated receptor γ
inferredcites →https://doi.org/10.1093/jmcb/mjt028openalex-snapshot
A deficiency in CCR2+ monocytes: the hidden side of Alzheimer's disease
inferredcites →https://doi.org/10.1099/vir.0.021915-0openalex-snapshot
Depletion of Gr-1+, but not Ly6G+, immune cells exacerbates virus replication and disease in an intranasal model of herpes simplex virus type 1 infection
inferredcites →https://doi.org/10.1111/j.1365-3024.2008.01053.xopenalex-snapshot
Control of pathogenic CD8+ T cell migration to the brain by IFN‐γ during experimental cerebral malaria
inferredcites →https://doi.org/10.1126/science.1142883openalex-snapshot
Monitoring of Blood Vessels and Tissues by a Population of Monocytes with Patrolling Behavior
inferredcites →https://doi.org/10.1128/iai.00472-09openalex-snapshot
Inflammatory Monocytes but Not Neutrophils Are Necessary To Control Infection with Toxoplasma gondii in Mice
inferredcites →https://doi.org/10.1128/iai.01349-12openalex-snapshot
Resistance of the Brain to Escherichia coli K1 Infection Depends on MyD88 Signaling and the Contribution of Neutrophils and Monocytes
inferredcites →https://doi.org/10.1128/iai.71.6.3648-3651.2003openalex-snapshot
Chemokine Receptor CCR2 Is Not Essential for the Development of Experimental Cerebral Malaria
inferredcites →https://doi.org/10.1172/jci28549openalex-snapshot
Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques
inferredcites →https://doi.org/10.1182/blood-2009-04-217489openalex-snapshot
Migrating monocytes recruited to the spleen play an important role in control of blood stage malaria
inferredcites →https://doi.org/10.1186/1741-7007-7-87openalex-snapshot
The obesity and inflammatory marker haptoglobin attracts monocytes via interaction with chemokine (C-C motif) receptor 2 (CCR2)
inferredcites →https://doi.org/10.1189/jlb.0407247openalex-snapshot
Use of Ly6G-specific monoclonal antibody to deplete neutrophils in mice
inferredcites →https://doi.org/10.1371/journal.ppat.1003096openalex-snapshot
TNFα-Mediated Liver Destruction by Kupffer Cells and Ly6Chi Monocytes during Entamoeba histolytica Infection
inferredcites →https://doi.org/10.1556/eujmi.2.2012.2.1openalex-snapshot
Monocytes in health and disease — Minireview
inferredcites →https://doi.org/10.1556/eujmi.4.2014.1.6openalex-snapshot
Spinal cord pathology in chronic experimentalToxoplasma gondiiinfection
inferredcites →https://doi.org/10.2307/3274665openalex-snapshot
Parasitemia and Survival in Inbred Strains of Mice Infected with Plasmodium berghei
inferredcites →https://doi.org/10.2307/3274666openalex-snapshot
Some Characteristics of Plasmodium berghei Passed within Inbred Strains of Mice
inferredcites →https://doi.org/10.2353/ajpath.2007.061033openalex-snapshot
A Role for Natural Regulatory T Cells in the Pathogenesis of Experimental Cerebral Malaria
inferredcites →https://doi.org/10.3389/fncel.2012.00051openalex-snapshot
Age-related changes in synaptic markers and monocyte subsets link the cognitive decline of APPSwe/PS1 mice
inferredcites →https://doi.org/10.4049/jimmunol.1200658openalex-snapshot
CD8 T Cell-Initiated Blood–Brain Barrier Disruption Is Independent of Neutrophil Support
inferredcites →https://doi.org/10.4049/jimmunol.1402037openalex-snapshot
Ly6Chigh Monocytes Control Cerebral Toxoplasmosis
inferredcites →https://doi.org/10.4049/jimmunol.157.4.1620openalex-snapshot
Participation of lymphocyte subpopulations in the pathogenesis of experimental murine cerebral malaria
inferredcites →https://doi.org/10.4049/jimmunol.166.7.4697openalex-snapshot
Expression and Characterization of the Chemokine Receptors CCR2 and CCR5 in Mice
inferredcites →https://doi.org/10.4049/jimmunol.169.11.6369openalex-snapshot
On the Pathogenic Role of Brain-Sequestered αβ CD8+ T Cells in Experimental Cerebral Malaria
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.3299436openalex-extracted
The Liver-Stage Plasmodium Infection is a Critical Checkpoint for Development of Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.2139/ssrn.4826099openalex-extracted
Genome-Wide Analyses Reveal Shared Genetic Architecture and Potential Causal Relationships between Osteoarthritis and Psychiatric Disorders
inferred← citeshttps://doi.org/10.1002/alr.23426openalex-snapshot
Nasal virus infection induces asthma exacerbation through B‐cell‐dependent recruitment of inflammatory monocytes
inferred← citeshttps://doi.org/10.1002/cti2.1273openalex-snapshot
Perivascular macrophages create an intravascular niche for CD8 + T cell localisation prior to the onset of fatal experimental cerebral malaria
inferred← citeshttps://doi.org/10.1002/jlb.1ma0320-731ropenalex-snapshot
Integrative analysis of microRNA and mRNA expression profiles of monocyte-derived dendritic cells differentiation during experimental cerebral malaria
inferred← citeshttps://doi.org/10.1002/jlb.mr0618-233ropenalex-snapshot
The role of myeloid-derived suppressor cells in chronic infectious diseases and the current methodology available for their study
inferred← citeshttps://doi.org/10.1007/978-3-031-15848-3_2openalex-snapshot
Malaria: Cellular Understanding of Disease
inferred← citeshttps://doi.org/10.1007/978-3-319-45210-4_1openalex-snapshot
Innate Immunity to Malaria
inferred← citeshttps://doi.org/10.1007/978-3-319-45210-4_4openalex-snapshot
Pathogenetic Immune Responses in Cerebral Malaria
inferred← citeshttps://doi.org/10.1007/s11033-022-07625-5openalex-snapshot
An update on cerebral malaria for therapeutic intervention
inferred← citeshttps://doi.org/10.1016/b978-0-12-819265-8.00097-8openalex-snapshot
Enhancing Pathogen Resistance: The Gut Microbiota and Malaria
inferred← citeshttps://doi.org/10.1016/j.ajpath.2019.01.011openalex-snapshot
Macrophage-Mediated Phagocytosis and Dissolution of Amyloid-Like Fibrils in Mice, Monitored by Optical Imaging
inferred← citeshttps://doi.org/10.1016/j.bbadis.2015.10.015openalex-snapshot
Myeloid derived suppressor cells in inflammatory conditions of the central nervous system
inferred← citeshttps://doi.org/10.1016/j.bbi.2016.01.002openalex-snapshot
Suppressor of cytokine signaling 2 modulates the immune response profile and development of experimental cerebral malaria
inferred← citeshttps://doi.org/10.1016/j.celrep.2016.04.074openalex-snapshot
Ly6Chi Monocytes Provide a Link between Antibiotic-Induced Changes in Gut Microbiota and Adult Hippocampal Neurogenesis
inferred← citeshttps://doi.org/10.1016/j.ctmp.2026.200272openalex-snapshot
Antimalarial effects of Eugenia ancorifera hydroethanolic leaf extract: Integrating in vitro, in vivo and in silico studies
inferred← citeshttps://doi.org/10.1016/j.cyto.2022.155910openalex-snapshot
IL-6 dependent expansion of inflammatory MDSCs (CD11b+ Gr-1+) promote Th-17 mediated immune response during experimental cerebral malaria
inferred← citeshttps://doi.org/10.1016/j.ibror.2019.12.004openalex-snapshot
Dissociation of neonatal and adult mice brain for simultaneous analysis of microglia, astrocytes and infiltrating lymphocytes by flow cytometry
inferred← citeshttps://doi.org/10.1016/j.imbio.2017.07.001openalex-snapshot
Immunomodulatory effects of myeloid-derived suppressor cells in diseases: Role in cancer and infections
inferred← citeshttps://doi.org/10.1016/j.imlet.2019.06.006openalex-snapshot
Limitations of neutrophil depletion by anti-Ly6G antibodies in two heterogenic immunological models
inferred← citeshttps://doi.org/10.1016/j.mib.2019.08.002openalex-snapshot
Monocyte-derived dendritic cells in malaria
inferred← citeshttps://doi.org/10.1016/j.mucimm.2024.08.002openalex-snapshot
Exposure to bacterial PAMPs before RSV infection exacerbates innate inflammation and disease via IL-1α and TNF-α
inferred← citeshttps://doi.org/10.1016/j.pt.2023.07.007openalex-snapshot
Monocytes as primary defenders against Toxoplasma gondii infection
inferred← citeshttps://doi.org/10.1016/j.pt.2023.11.005openalex-snapshot
Unravelling mysteries at the perivascular space: a new rationale for cerebral malaria pathogenesis
inferred← citeshttps://doi.org/10.1016/j.pt.2024.09.006openalex-snapshot
Immunological clues to sex differences in parasitic diseases
inferred← citeshttps://doi.org/10.1016/j.smim.2016.03.014openalex-snapshot
Monocyte-mediated defense against bacteria, fungi, and parasites
inferred← citeshttps://doi.org/10.1021/acsnano.5c02001openalex-snapshot
Transient Macrophage Depletion Circumvents Scavenging and Redirects Biodistribution of mRNA-Lipid Nanoparticles
inferred← citeshttps://doi.org/10.1038/s41467-020-17260-yopenalex-snapshot
Androgens predispose males to monocyte-mediated immunopathology by inducing the expression of leukocyte recruitment factor CXCL1
inferred← citeshttps://doi.org/10.1038/s41467-024-52223-7openalex-snapshot
The spatiotemporal transcriptional profiling of murine brain during cerebral malaria progression and after artemisinin treatment
inferred← citeshttps://doi.org/10.1038/s41598-017-06236-6openalex-snapshot
Inflammatory monocytes regulate Th1 oriented immunity to CpG adjuvanted protein vaccines through production of IL-12
inferred← citeshttps://doi.org/10.1038/s41598-019-42782-xopenalex-snapshot
KCC1 Activation protects Mice from the Development of Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.1038/s41598-019-44181-8openalex-snapshot
Differential Immune Activation in Fetal Macrophage Populations
inferred← citeshttps://doi.org/10.1038/s42003-018-0216-2openalex-snapshot
Experimental severe malaria is resolved by targeting newly-identified monocyte subsets using immune-modifying particles combined with artesunate
inferred← citeshttps://doi.org/10.1039/c9ra08203dopenalex-snapshot
A quantitative ex vivo study of the interactions between reconstituted high-density lipoproteins and human leukocytes
inferred← citeshttps://doi.org/10.1053/j.gastro.2023.01.002openalex-snapshot
F4/80+Ly6Chigh Macrophages Lead to Cell Plasticity and Cancer Initiation in Colitis
inferred← citeshttps://doi.org/10.1074/jbc.m116.770313openalex-snapshot
Small molecule–based inhibition of MEK1/2 proteins dampens inflammatory responses to malaria, reduces parasite load, and mitigates pathogenic outcomes
inferred← citeshttps://doi.org/10.1084/jem.20160880openalex-snapshot
Inflammatory monocytes require type I interferon receptor signaling to activate NK cells via IL-18 during a mucosal viral infection
inferred← citeshttps://doi.org/10.1089/vim.2019.0132openalex-snapshot
The Suppressive Attitude of Inflammatory Monocytes in Antiviral Antibody Responses
inferred← citeshttps://doi.org/10.1093/jleuko/qiaf005openalex-snapshot
Protocols for circulating neutrophil depletion in neonatal C57Bl/6 mice
inferred← citeshttps://doi.org/10.1096/fj.202401237ropenalex-snapshot
Myeloid‐derived suppressor cells inhibit responses of T follicular helper cells during experimental Plasmodium yoelii infection
inferred← citeshttps://doi.org/10.1111/imm.13137openalex-snapshot
Protection of Batf3‐deficient mice from experimental cerebral malaria correlates with impaired cytotoxic T‐cell responses and immune regulation
inferred← citeshttps://doi.org/10.1111/pim.12502openalex-snapshot
Unravelling the roles of innate lymphoid cells in cerebral malaria pathogenesis
inferred← citeshttps://doi.org/10.1111/pim.12912openalex-snapshot
Neutrophils in malaria: The good, the bad or the ugly?
inferred← citeshttps://doi.org/10.1128/mbio.01076-21openalex-snapshot
CCR2 Signaling Promotes Brain Infiltration of Inflammatory Monocytes and Contributes to Neuropathology during Cryptococcal Meningoencephalitis
inferred← citeshttps://doi.org/10.1155/2017/5238402openalex-snapshot
Effects of Acyclovir and IVIG on Behavioral Outcomes after HSV1 CNS Infection
inferred← citeshttps://doi.org/10.1172/jci.insight.89704openalex-snapshot
Acquired resistance to innate immune clearance promotes Klebsiella pneumoniae ST258 pulmonary infection
inferred← citeshttps://doi.org/10.1186/s12936-022-04268-6openalex-snapshot
Increased circulating myeloid-derived suppressor cells in vivax malaria and severe falciparum malaria
inferred← citeshttps://doi.org/10.1186/s12974-025-03531-2openalex-snapshot
TLR9/NF-κB-mediated dendritic cell activation by neutrophil extracellular traps drives pathogenesis in experimental cerebral malaria
inferred← citeshttps://doi.org/10.1186/s40635-024-00707-7openalex-snapshot
Lung immune incompetency after mild peritoneal sepsis and its partial restoration by type 1 interferon: a mouse model study
inferred← citeshttps://doi.org/10.12688/f1000research.12401.1openalex-snapshot
Do we know enough to find an adjunctive therapy for cerebral malaria in African children?
inferred← citeshttps://doi.org/10.1371/journal.ppat.1005210openalex-snapshot
Perivascular Arrest of CD8+ T Cells Is a Signature of Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.1371/journal.ppat.1006022openalex-snapshot
CD8+ T Cells Induce Fatal Brainstem Pathology during Cerebral Malaria via Luminal Antigen-Specific Engagement of Brain Vasculature
inferred← citeshttps://doi.org/10.1371/journal.ppat.1007924openalex-snapshot
Schistosoma mansoni soluble egg antigen (SEA) and recombinant Omega-1 modulate induced CD4+ T-lymphocyte responses and HIV-1 infection in vitro
inferred← citeshttps://doi.org/10.1371/journal.ppat.1008230openalex-snapshot
CXCR4 and MIF are required for neutrophil extracellular trap release triggered by Plasmodium-infected erythrocytes
inferred← citeshttps://doi.org/10.1371/journal.ppat.1009195openalex-snapshot
K18-hACE2 mice develop respiratory disease resembling severe COVID-19
inferred← citeshttps://doi.org/10.18632/oncotarget.14314openalex-snapshot
N-glycosylation converts non-glycoproteins into mannose receptor ligands and reveals antigen-specific T cell responses in vivo
inferred← citeshttps://doi.org/10.31088/cem2024.13.4.29-35openalex-snapshot
Altered proinflammatory cytokine response in colorectal cancer patients: insights into immune dysregulation
inferred← citeshttps://doi.org/10.32657/10356/139653openalex-snapshot
Studies on the immunological mechanisms leading to protection or pathology during malaria infection
inferred← citeshttps://doi.org/10.32657/10356/70327openalex-snapshot
Tissue-Resident CD169+ macrophages as a frontline of host defense in Plasmodium infection
inferred← citeshttps://doi.org/10.3389/fimmu.2019.00830openalex-snapshot
The Ins and Outs of Cerebral Malaria Pathogenesis: Immunopathology, Extracellular Vesicles, Immunometabolism, and Trained Immunity
inferred← citeshttps://doi.org/10.3389/fimmu.2019.02105openalex-snapshot
NFAT1 Regulates Ly6Chi Monocyte Recruitment to the CNS and Plays an Essential Role in Resistance to Toxoplasma gondii Infection
inferred← citeshttps://doi.org/10.3389/fimmu.2021.711876openalex-snapshot
Eosinophils Suppress the Migration of T Cells Into the Brain of Plasmodium berghei-Infected Ifnar1-/- Mice and Protect Them From Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.3389/fimmu.2022.781273openalex-snapshot
Reduced Neutrophil Extracellular Trap Formation During Ischemia Reperfusion Injury in C3 KO Mice: C3 Requirement for NETs Release
inferred← citeshttps://doi.org/10.3389/fimmu.2022.922377openalex-snapshot
Neutrophils in malaria: A double-edged sword role
inferred← citeshttps://doi.org/10.3389/fitd.2021.707100openalex-snapshot
Infection-Derived Monocytic MDSCs Require TGF-β to Suppress Filarial-Specific IFN-γ But Not IL-13 Release by Filarial-Specific CD4+ T Cells In Vitro
inferred← citeshttps://doi.org/10.3390/cells12020269openalex-snapshot
Who Knew? Dopamine Transporter Activity Is Critical in Innate and Adaptive Immune Responses
inferred← citeshttps://doi.org/10.3390/ijms241411332openalex-snapshot
Increased Neutrophil Percentage and Neutrophil–T Cell Ratio Precedes Clinical Onset of Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.3410/f.740541708.793588924openalex-snapshot
Faculty Opinions recommendation of CCR2 Signaling Promotes Brain Infiltration of Inflammatory Monocytes and Contributes to Neuropathology during Cryptococcal Meningoencephalitis.
inferred← citeshttps://doi.org/10.3727/096368916x693482openalex-snapshot
Monocyte-Derived Dendritic Cells Impair Early Graft Function following Allogeneic Islet Transplantation
inferred← citeshttps://doi.org/10.4049/jimmunol.1501562openalex-snapshot
Monocyte- and Neutrophil-Derived CXCL10 Impairs Efficient Control of Blood-Stage Malaria Infection and Promotes Severe Disease
inferred← citeshttps://doi.org/10.4049/jimmunol.1601168openalex-snapshot
MARCH1 E3 Ubiquitin Ligase Dampens the Innate Inflammatory Response by Modulating Monocyte Functions in Mice
inferred← citeshttps://doi.org/10.4049/jimmunol.2300541openalex-snapshot
Plasmodium yoelii Infection Enhances the Expansion of Myeloid-Derived Suppressor Cells via JAK/STAT3 Pathway
inferred← citeshttps://doi.org/10.68327/pexk2543openalex-snapshot
Ubidercarenone Decelerates the Manifestation of Experimental Cerebral Malaria in C57BL/6J Mice by ameliorating the Host Inflammatory Response
inferred← citeshttps://doi.org/10.70675/55902f1cz78ecz4e3czb81fz35dcfd9d2e7aopenalex-snapshot
Etude de l'impact des infections parasitaires et virales sur les maladies inflammatoires médiées par les lymphocytes T
inferred← citeshttps://doi.org/10.7176/jnsr/9-2-05openalex-snapshot
Coenzyme Q10 Protect Mice Against Inflammatory Responses During Experimental Cerebral Malaria
inferred← citeshttps://doi.org/10.7554/elife.77440openalex-snapshot
Immunopathology and Trypanosoma congolense parasite sequestration cause acute cerebral trypanosomiasis
Editorial review for independent work. ICSAC — the Institute for Complexity
Science and Advanced Computing — runs a structured editorial panel for researchers
publishing outside journals, and publishes on CiteStamp rails. Submission is free
and goes through ORCID: icsacinstitute.org/submit.