On the public citation graph

Exposure-Dependent Control of Malaria-Induced Inflammation in Children

https://doi.org/10.1371/journal.ppat.1004079

0 signed · 254 inferred · view as graph

Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

Is this your work? Sign in with ORCID and put your name on the claims that are yours.

Claim with ORCID

Reference health. See whether every reference in this work resolves to a known work and none has been retracted — and get a dated CiteStamped badge you can embed.

See reference health

What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/cyto.a.21015openalex-snapshot

SPICE: Exploration and analysis of post‐cytometric complex multivariate datasets

inferredcites →https://doi.org/10.1002/eji.1830210523openalex-snapshot

Transient depletion of T cells with high LFA‐1 expression from peripheral circulation during acute Plasmodium falciparum malaria

inferredcites →https://doi.org/10.1002/eji.200839112openalex-snapshot

Homeostatic regulation of T effector to Treg ratios in an area of seasonal malaria transmission

inferredcites →https://doi.org/10.1006/clim.1999.4752openalex-snapshot

CD4+ T Cell Clones Producing both Interferon-γ and Interleukin-10 Predominate in Bronchoalveolar Lavages of Active Pulmonary Tuberculosis Patients

inferredcites →https://doi.org/10.1006/clin.1995.1017openalex-snapshot

Elevated Serum Levels of IL-10 and IFN-γ in Patients with Acute Plasmodium falciparum Malaria

inferredcites →https://doi.org/10.1007/3-540-29967-x_2openalex-snapshot

Early Interactions Between Blood-Stage Plasmodium Parasites and the Immune System

inferredcites →https://doi.org/10.1007/3-540-29967-x_5openalex-snapshot

Glycosylphosphatidylinositols in Malaria Pathogenesis and Immunity: Potential for Therapeutic Inhibition and Vaccination

inferredcites →https://doi.org/10.1007/s00011-007-7104-4openalex-snapshot

Decrease in toll-like receptors 2 and 4 in the spleen of mouse with endotoxic tolerance

inferredcites →https://doi.org/10.1016/0140-6736(90)92827-5openalex-snapshot

TNF concentration in fatal cerebral, non-fatal cerebral, and uncomplicated Plasmodium falciparum malaria

inferredcites →https://doi.org/10.1016/j.cell.2008.01.020openalex-snapshot

Shared Principles in NF-κB Signaling

inferredcites →https://doi.org/10.1016/j.cell.2010.01.022openalex-snapshot

Pattern Recognition Receptors and Inflammation

inferredcites →https://doi.org/10.1016/j.chom.2012.06.006openalex-snapshot

Candida albicans Infection Affords Protection against Reinfection via Functional Reprogramming of Monocytes

inferredcites →https://doi.org/10.1016/j.clim.2008.08.015openalex-snapshot

Gene-specific control of the TLR-induced inflammatory response

inferredcites →https://doi.org/10.1016/j.coi.2012.05.003openalex-snapshot

T cell control of malaria pathogenesis

inferredcites →https://doi.org/10.1016/j.imbio.2008.07.008openalex-snapshot

From expression to signaling: Roles of TREM-1 and TREM-2 in innate immunity and bacterial infection

inferredcites →https://doi.org/10.1016/j.immuni.2005.08.006openalex-snapshot

Upregulation of TGF-β, FOXP3, and CD4+CD25+ Regulatory T Cells Correlates with More Rapid Parasite Growth in Human Malaria Infection

inferredcites →https://doi.org/10.1016/j.immuni.2008.05.012openalex-snapshot

A Modular Analysis Framework for Blood Genomics Studies: Application to Systemic Lupus Erythematosus

inferredcites →https://doi.org/10.1016/j.immuni.2011.05.016openalex-snapshot

Innate Immune Recognition of an AT-Rich Stem-Loop DNA Motif in the Plasmodium falciparum Genome

inferredcites →https://doi.org/10.1016/j.it.2009.07.009openalex-snapshot

Endotoxin tolerance: new mechanisms, molecules and clinical significance

inferredcites →https://doi.org/10.1016/j.it.2009.12.002openalex-snapshot

Regulatory T cells in malaria – friend or foe?

inferredcites →https://doi.org/10.1016/j.pt.2006.06.002openalex-snapshot

Malaria tolerance – for whom the cell tolls?

inferredcites →https://doi.org/10.1016/j.pt.2012.03.006openalex-snapshot

Systems immunology of human malaria

inferredcites →https://doi.org/10.1016/s0035-9203(96)90463-0openalex-snapshot

Malaria: even more chronic in nature than previously thought; evidence for subpatent parasitaemia detectable by the polymerase chain reaction

inferredcites →https://doi.org/10.1016/s0035-9203(99)90329-2openalex-snapshot

11. Premunition in Plasmodium falciparum infection: insights from the epidemiology of multiple infections

inferredcites →https://doi.org/10.1016/s0140-6736(02)08360-5openalex-snapshot

Mechanism of protection induced by vitamin A in falciparum malaria

inferredcites →https://doi.org/10.1016/s0952-7915(02)00368-0openalex-snapshot

Malaria, monocytes, macrophages and myeloid dendritic cells: sticking of infected erythrocytes switches off host cells

inferredcites →https://doi.org/10.1038/6560openalex-snapshot

Immunity to non-cerebral severe malaria is acquired after one or two infections

inferredcites →https://doi.org/10.1038/nature00937openalex-snapshot

Synthetic GPI as a candidate anti-toxic vaccine in a model of malaria

inferredcites →https://doi.org/10.1038/nature05836openalex-snapshot

Gene-specific control of inflammation by TLR-induced chromatin modifications

inferredcites →https://doi.org/10.1038/nature12216openalex-snapshot

Severe malaria is associated with parasite binding to endothelial protein C receptor

inferredcites →https://doi.org/10.1038/ni.f.205openalex-snapshot

Immunity to malaria: more questions than answers

inferredcites →https://doi.org/10.1038/nri1686openalex-snapshot

Immunological processes in malaria pathogenesis

inferredcites →https://doi.org/10.1038/nri1964openalex-snapshot

Tuning inflammation and immunity by chemokine sequestration: decoys and more

inferredcites →https://doi.org/10.1038/nri2097openalex-snapshot

TH1 cells control themselves by producing interleukin-10

inferredcites →https://doi.org/10.1046/j.1365-3024.1997.d01-188.xopenalex-snapshot

Plasmodium falciparum malaria blood stage parasites preferentially inhibit macrophages with high phagocytic activity

inferredcites →https://doi.org/10.1056/nejm198906153202404openalex-snapshot

Tumor Necrosis Factor and Disease Severity in Children with Falciparum Malaria

inferredcites →https://doi.org/10.1056/nejm199505253322102openalex-snapshot

Indicators of Life-Threatening Malaria in African Children

inferredcites →https://doi.org/10.1073/pnas.0608745104openalex-snapshot

Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9

inferredcites →https://doi.org/10.1073/pnas.1001323107openalex-snapshot

A prospective analysis of the Ab response to Plasmodium falciparum before and after a malaria season by protein microarray

inferredcites →https://doi.org/10.1073/pnas.1120455109openalex-snapshot

Plasmodium falciparum erythrocyte membrane protein 1 domain cassettes 8 and 13 are associated with severe malaria in children

inferredcites →https://doi.org/10.1073/pnas.1120461109openalex-snapshot

A subset of group A-like var genes encodes the malaria parasite ligands for binding to human brain endothelial cells

inferredcites →https://doi.org/10.1073/pnas.1120534109openalex-snapshot

A restricted subset of var genes mediates adherence of Plasmodium falciparum -infected erythrocytes to brain endothelial cells

inferredcites →https://doi.org/10.1073/pnas.1202870109openalex-snapshot

Bacille Calmette-Guérin induces NOD2-dependent nonspecific protection from reinfection via epigenetic reprogramming of monocytes

inferredcites →https://doi.org/10.1074/jbc.m413541200openalex-snapshot

Induction of Proinflammatory Responses in Macrophages by the Glycosylphosphatidylinositols of Plasmodium falciparum

inferredcites →https://doi.org/10.1084/jem.188.1.39openalex-snapshot

Transforming Growth Factor β Production Is Inversely Correlated with Severity of Murine Malaria Infection

inferredcites →https://doi.org/10.1084/jem.20061141openalex-snapshot

Splenic accumulation of IL-10 mRNA in T cells distinct from CD4+CD25+ (Foxp3) regulatory T cells in human visceral leishmaniasis

inferredcites →https://doi.org/10.1084/jem.20061886openalex-snapshot

CD4+CD25−Foxp3− Th1 cells are the source of IL-10–mediated immune suppression in chronic cutaneous leishmaniasis

inferredcites →https://doi.org/10.1084/jem.20062175openalex-snapshot

Conventional T-bet+Foxp3− Th1 cells are the major source of host-protective regulatory IL-10 during intracellular protozoan infection

inferredcites →https://doi.org/10.1086/315016openalex-snapshot

The Prognostic and Pathophysiologic Role of Pro‐ and Antiinflammatory Cytokines in Severe Malaria

inferredcites →https://doi.org/10.1086/339408openalex-snapshot

Absolute Levels and Ratios of Proinflammatory and Anti‐inflammatory Cytokine Production In Vitro Predict Clinical Immunity toPlasmodium falciparumMalaria

inferredcites →https://doi.org/10.1086/592224openalex-snapshot

Sickle Cell Trait Is Associated with a Delayed Onset of Malaria: Implications for Time‐to‐Event Analysis in Clinical Studies of Malaria

inferredcites →https://doi.org/10.1086/599317openalex-snapshot

High Prevalence of AsymptomaticPlasmodium falciparumInfections in a Highland Area of Western Kenya: A Cohort Study

inferredcites →https://doi.org/10.1089/dna.2005.24.410openalex-snapshot

Correlation of Protein and Gene Expression Profiles of Inflammatory Proteins After Endotoxin Challenge in Human Subjects

inferredcites →https://doi.org/10.1093/cid/cit174openalex-snapshot

An Intensive Longitudinal Cohort Study of Malian Children and Adults Reveals No Evidence of Acquired Immunity to Plasmodium falciparum Infection

inferredcites →https://doi.org/10.1097/00003246-200201001-00009openalex-snapshot

Endotoxin tolerance: A review

inferredcites →https://doi.org/10.1111/j.1365-3024.2006.00775.xopenalex-snapshot

Regulating immunity to malaria

inferredcites →https://doi.org/10.1111/j.1365-3024.2006.00808.xopenalex-snapshot

Immune effector mechanisms in malaria

inferredcites →https://doi.org/10.1126/science.1178334openalex-snapshot

Mechanisms Underlying Lineage Commitment and Plasticity of Helper CD4 + T Cells

inferredcites →https://doi.org/10.1128/iai.00689-13openalex-snapshot

Effect of the Leptin Receptor Q223R Polymorphism on the Host Transcriptome following Infection with Entamoeba histolytica

inferredcites →https://doi.org/10.1128/iai.01383-12openalex-snapshot

Effect of the Leptin Receptor Q223R Polymorphism on the Host Transcriptome following Infection with Entamoeba histolytica

inferredcites →https://doi.org/10.1128/iai.67.9.4435-4442.1999openalex-snapshot

A Defect in Interleukin-10 Leads to Enhanced Malarial Disease inPlasmodium chabaudi chabaudiInfection in Mice

inferredcites →https://doi.org/10.1128/iai.72.10.5630-5637.2004openalex-snapshot

Serum Levels of the Proinflammatory Cytokines Interleukin-1 Beta (IL-1β), IL-6, IL-8, IL-10, Tumor Necrosis Factor Alpha, and IL-12(p70) in Malian Children with Severe Plasmodium falciparum Malaria and Matched Uncomplicated Malaria or Healthy Controls

inferredcites →https://doi.org/10.1128/iai.73.7.4190-4197.2005openalex-snapshot

Differential Regulation of β-Chemokines in Children with Plasmodium falciparum Malaria

inferredcites →https://doi.org/10.1136/bmj.2.4994.686openalex-snapshot

Effects of Heavy and Repeated Malarial Infections on Gambian Infants and Children

inferredcites →https://doi.org/10.1146/annurev-immunol-031210-101405openalex-snapshot

The Inflammasome NLRs in Immunity, Inflammation, and Associated Diseases

inferredcites →https://doi.org/10.1177/003591573803101109openalex-snapshot

Immunity or Tolerance in Malarial Infections

inferredcites →https://doi.org/10.1182/blood.v96.9.3231openalex-snapshot

Nonopsonic monocyte/macrophage phagocytosis of Plasmodium falciparum–parasitized erythrocytes: a role for CD36 in malarial clearance

inferredcites →https://doi.org/10.1186/1741-7007-8-84openalex-snapshot

Assessing the human immune system through blood transcriptomics

inferredcites →https://doi.org/10.1189/jlb.0506312openalex-snapshot

Cellular reprogramming by gram-positive bacterial components: a review

inferredcites →https://doi.org/10.1189/jlb.1111571openalex-snapshot

The emerging role of microRNA in regulation of endotoxin tolerance

inferredcites →https://doi.org/10.1371/journal.ppat.1000364openalex-snapshot

Distinct Roles for FOXP3+ and FOXP3− CD4+ T Cells in Regulating Cellular Immunity to Uncomplicated and Severe Plasmodium falciparum Malaria

inferredcites →https://doi.org/10.1371/journal.ppat.1000402openalex-snapshot

Parasite-Dependent Expansion of TNF Receptor II–Positive Regulatory T Cells with Enhanced Suppressive Activity in Adults with Severe Malaria

inferredcites →https://doi.org/10.1371/journal.ppat.1000559openalex-snapshot

Malarial Hemozoin Activates the NLRP3 Inflammasome through Lyn and Syk Kinases

inferredcites →https://doi.org/10.1371/journal.ppat.1000771openalex-snapshot

Natural Regulatory T Cells in Malaria: Host or Parasite Allies?

inferredcites →https://doi.org/10.1371/journal.ppat.1000912openalex-snapshot

The Plasmodium falciparum-Specific Human Memory B Cell Compartment Expands Gradually with Repeated Malaria Infections

inferredcites →https://doi.org/10.1371/journal.ppat.1003864openalex-snapshot

IFNγ/IL-10 Co-producing Cells Dominate the CD4 Response to Malaria in Highly Exposed Children

inferredcites →https://doi.org/10.3181/00379727-118-29820openalex-snapshot

Malaria Induced Endotoxin Tolerance

inferredcites →https://doi.org/10.4049/jimmunol.0713552openalex-snapshot

Pure Hemozoin Is Inflammatory In Vivo and Activates the NALP3 Inflammasome via Release of Uric Acid

inferredcites →https://doi.org/10.4049/jimmunol.0803350openalex-snapshot

Potent Phagocytic Activity with Impaired Antigen Presentation Identifying Lipopolysaccharide-Tolerant Human Monocytes: Demonstration in Isolated Monocytes from Cystic Fibrosis Patients

inferredcites →https://doi.org/10.4049/jimmunol.1203067openalex-snapshot

Young Lives Lost as B Cells Falter: What We Are Learning About Antibody Responses in Malaria

inferredcites →https://doi.org/10.4049/jimmunol.169.6.2956openalex-snapshot

Innate Immune Response to Malaria: Rapid Induction of IFN-γ from Human NK Cells by Live Plasmodium falciparum -Infected Erythrocytes

inferredcites →https://doi.org/10.4049/jimmunol.177.8.5736openalex-snapshot

Innate Immune Responses to Human Malaria: Heterogeneous Cytokine Responses to Blood-Stage Plasmodium falciparum Correlate with Parasitological and Clinical Outcomes

inferredcites →https://doi.org/10.4049/jimmunol.180.9.5771openalex-snapshot

IL-10: The Master Regulator of Immunity to Infection

inferredcites →https://doi.org/10.4269/tropmed.1999.61-020openalex-snapshot

A Retrospective Examination of Secondary Sporozoite- and Trophozoite-Induced Infections with Plasmodium Falciparum: Development of Parasitologic and Clinical Immunity Following Secondary Infection

inferredcites →https://doi.org/10.4269/tropmed.1999.61-04openalex-snapshot

A Retrospective Examination of Sporozoite- and Trophozoite-Induced Infections with Plasmodium Falciparum: Development of Parasitologic and Clinical Immunity during Primary Infection

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4458921openalex-extracted

inferred← citeshttps://doi.org/10.1002/cti2.1209openalex-snapshot

A population of CD4hiCD38hi T cells correlates with disease severity in patients with acute malaria

inferred← citeshttps://doi.org/10.1002/eji.202350784openalex-snapshot

Disease‐specific plasma protein profiles in patients with fever after traveling to tropical areas

inferred← citeshttps://doi.org/10.1007/978-1-4614-8326-7_131openalex-snapshot

Naturally Acquired Immunity (NAI)

inferred← citeshttps://doi.org/10.1007/978-1-4614-8326-7_132openalex-snapshot

Blood-Stage Immunity to Malaria

inferred← citeshttps://doi.org/10.1007/978-3-319-45210-4_3openalex-snapshot

Adaptive Immunity to Plasmodium Blood Stages

inferred← citeshttps://doi.org/10.1007/s00018-019-03277-0openalex-snapshot

Molecular switches for regulating the differentiation of inflammatory and IL-10-producing anti-inflammatory T-helper cells

inferred← citeshttps://doi.org/10.1007/s00430-018-0547-0openalex-snapshot

Recent advances on T-cell exhaustion in malaria infection

inferred← citeshttps://doi.org/10.1016/b978-0-12-819265-8.00097-8openalex-snapshot

Enhancing Pathogen Resistance: The Gut Microbiota and Malaria

inferred← citeshttps://doi.org/10.1016/j.actatropica.2024.107446openalex-snapshot

An immuno-inflammatory profiling of asymptomatic individuals in a malaria endemic area in Uganda

inferred← citeshttps://doi.org/10.1016/j.ajhg.2025.01.020openalex-snapshot

Single-cell transcriptomics reveals inter-ethnic variation in immune response to Falciparum malaria

inferred← citeshttps://doi.org/10.1016/j.celrep.2016.09.015openalex-snapshot

Type I Interferons Regulate Immune Responses in Humans with Blood-Stage Plasmodium falciparum Infection

inferred← citeshttps://doi.org/10.1016/j.celrep.2020.02.104openalex-snapshot

Acute Plasmodium Infection Promotes Interferon-Gamma-Dependent Resistance to Ebola Virus Infection

inferred← citeshttps://doi.org/10.1016/j.cyto.2023.156262openalex-snapshot

A systematic review and meta-analysis of changes in interleukin-8 levels in malaria infection

inferred← citeshttps://doi.org/10.1016/j.ebiom.2023.104772openalex-snapshot

Malaria-specific Type 1 regulatory T cells are more abundant in first pregnancies and associated with placental malaria

inferred← citeshttps://doi.org/10.1016/j.immuni.2019.08.009openalex-snapshot

A Molecular Signature in Blood Reveals a Role for p53 in Regulating Malaria-Induced Inflammation

inferred← citeshttps://doi.org/10.1016/j.immuni.2025.06.014openalex-snapshot

Systems analysis of clinical malaria reveals proteomic perturbation and innate-adaptive crosstalk linked to disease severity

inferred← citeshttps://doi.org/10.1016/j.isci.2026.115979openalex-snapshot

Mechanistic insights into subclinical Plasmodium infections: Unveiling the silent drivers of malaria transmission

inferred← citeshttps://doi.org/10.1016/j.jep.2025.120326openalex-snapshot

Bitter leaf (Vernonia amygdalina) and siam weed (Chromolaena odorata) aqueous extracts alleviate testicular damage induced by Plasmodium berghei in male mice via modulation of oxidative stress pathways

inferred← citeshttps://doi.org/10.1016/j.mib.2018.09.003openalex-snapshot

Rodent malaria models: insights into human disease and parasite biology

inferred← citeshttps://doi.org/10.1016/j.mib.2026.102752openalex-snapshot

The case for exploring innate memory in Plasmodium infection

inferred← citeshttps://doi.org/10.1016/j.parint.2019.01.003openalex-snapshot

Role of IL-10 in inhibiting protective immune responses against infection with heterologous Plasmodium parasites

inferred← citeshttps://doi.org/10.1016/j.parint.2019.101994openalex-snapshot

Activation and IL-10 production of specific CD4+ T cells are regulated by IL-27 during chronic infection with Plasmodium chabaudi

inferred← citeshttps://doi.org/10.1016/j.pt.2015.11.015openalex-snapshot

Malaria Parasites in the Asymptomatic: Looking for the Hay in the Haystack

inferred← citeshttps://doi.org/10.1016/j.pt.2020.09.017openalex-snapshot

Understanding Host–Pathogen–Vector Interactions with Chronic Asymptomatic Malaria Infections

inferred← citeshttps://doi.org/10.1016/j.pt.2022.01.005openalex-snapshot

The tangled web of autoreactive B cells in malaria immunity and autoimmune disease

inferred← citeshttps://doi.org/10.1016/j.pt.2026.07.016openalex-snapshot

Metabolite-effector immunity against malaria

inferred← citeshttps://doi.org/10.1016/j.sciaf.2020.e00428openalex-snapshot

Cytokine modulation during malaria infections by some medicinal plants

inferred← citeshttps://doi.org/10.1017/s0031182015001092openalex-snapshot

Examining cellular immune responses to inform development of a blood-stage malaria vaccine

inferred← citeshttps://doi.org/10.1017/s0031182015001705openalex-snapshot

Immunological processes underlying the slow acquisition of humoral immunity to malaria

inferred← citeshttps://doi.org/10.1038/s41423-020-0482-zopenalex-snapshot

Variations in killer-cell immunoglobulin-like receptor and human leukocyte antigen genes and immunity to malaria

inferred← citeshttps://doi.org/10.1038/s41467-023-43181-7openalex-snapshot

Single cell transcriptomics shows that malaria promotes unique regulatory responses across multiple immune cell subsets

inferred← citeshttps://doi.org/10.1038/s41467-025-63104-yopenalex-snapshot

Development of clinical immunity to Plasmodium vivax following repeat controlled human malaria infection

inferred← citeshttps://doi.org/10.1038/s41541-017-0035-3openalex-snapshot

Advances in malaria vaccine development: report from the 2017 malaria vaccine symposium

inferred← citeshttps://doi.org/10.1038/s41577-024-01041-5openalex-snapshot

The impact of Plasmodium-driven immunoregulatory networks on immunity to malaria

inferred← citeshttps://doi.org/10.1038/s41598-017-10624-3openalex-snapshot

Vδ2+ T cell response to malaria correlates with protection from infection but is attenuated with repeated exposure

inferred← citeshttps://doi.org/10.1038/s41598-018-22659-1openalex-snapshot

Differential expression pattern of co-inhibitory molecules on CD4+ T cells in uncomplicated versus complicated malaria

inferred← citeshttps://doi.org/10.1038/s41598-019-52388-yopenalex-snapshot

Comparison of whole blood and spleen transcriptional signatures over the course of an experimental malaria infection

inferred← citeshttps://doi.org/10.1038/s41598-024-53519-wopenalex-snapshot

Epigenetic and transcriptional regulation of cytokine production by Plasmodium falciparum-exposed monocytes

inferred← citeshttps://doi.org/10.1038/srep31291openalex-snapshot

Transcriptomic evidence for modulation of host inflammatory responses during febrile Plasmodium falciparum malaria

inferred← citeshttps://doi.org/10.1073/pnas.1708383114openalex-snapshot

Atypical activation of dendritic cells by Plasmodium falciparum

inferred← citeshttps://doi.org/10.1073/pnas.2104166118openalex-snapshot

Complement C1s cleaves PfEMP1 at interdomain conserved sites inhibiting Plasmodium falciparum cytoadherence

inferred← citeshttps://doi.org/10.1080/15384101.2019.1580496openalex-snapshot

NF90 regulation of immune factor expression in response to malaria antigens

inferred← citeshttps://doi.org/10.1080/21505594.2020.1726569openalex-snapshot

Balancing in a black box: Potential immunomodulatory roles for TGF-β signaling during blood-stage malaria

inferred← citeshttps://doi.org/10.1093/cei/uxae015openalex-snapshot

Chronic malaria exposure is associated with inhibitory markers on T cells that correlate with atypical memory and marginal zone-like B cells

inferred← citeshttps://doi.org/10.1093/cid/ciw849openalex-snapshot

Treatment of Chronic Asymptomatic Plasmodium falciparum Infection Does Not Increase the Risk of Clinical Malaria Upon Reinfection

inferred← citeshttps://doi.org/10.1093/cid/ciy934openalex-snapshot

A Balanced Proinflammatory and Regulatory Cytokine Signature in Young African Children Is Associated With Lower Risk of Clinical Malaria

inferred← citeshttps://doi.org/10.1093/femsre/fuv046openalex-snapshot

The immunological balance between host and parasite in malaria

inferred← citeshttps://doi.org/10.1093/immhor/vlaf070openalex-snapshot

Altered natural killer cell function in children with severe malaria

inferred← citeshttps://doi.org/10.1093/infdis/jiv054openalex-snapshot

Effector Phenotype ofPlasmodium falciparum–Specific CD4+T Cells Is Influenced by Both Age and Transmission Intensity in Naturally Exposed Populations

inferred← citeshttps://doi.org/10.1093/infdis/jiv217openalex-snapshot

The T-Cell Inhibitory Molecule Butyrophilin-Like 2 Is Up-regulated in MildPlasmodium falciparumInfection and Is Protective During Experimental Cerebral Malaria

inferred← citeshttps://doi.org/10.1093/infdis/jiv600openalex-snapshot

Frequent Malaria Drives Progressive Vδ2 T-Cell Loss, Dysfunction, and CD16 Up-regulation During Early Childhood

inferred← citeshttps://doi.org/10.1093/infdis/jiy281openalex-snapshot

Early Changes in CD4+ T-Cell Activation During Blood-Stage Plasmodium falciparum Infection

inferred← citeshttps://doi.org/10.1093/jimmun/vkaf067openalex-snapshot

Regulatory T cell memory: implications for malaria

inferred← citeshttps://doi.org/10.1093/jimmun/vkaf335openalex-snapshot

Different clinical forms of Plasmodium vivax infection result in unique T cell epitope-specific immune responses

inferred← citeshttps://doi.org/10.1093/ofid/ofab302openalex-snapshot

Elevated Levels of the Endothelial Molecules ICAM-1, VEGF-A, and VEGFR2 in Microscopic Asymptomatic Malaria

inferred← citeshttps://doi.org/10.1101/2022.11.16.516822openalex-snapshot

Malaria drives unique regulatory responses across multiple immune cell subsets

inferred← citeshttps://doi.org/10.1101/2023.07.31.551226openalex-snapshot

NK cell-induced damage to P.falciparum -infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG

inferred← citeshttps://doi.org/10.1101/2023.10.06.561264openalex-snapshot

IL-15 complex-induced IL-10 enhances Plasmodium -specific CD4 + Tfh differentiation and antibody production

inferred← citeshttps://doi.org/10.1101/2025.02.04.25321636openalex-snapshot

An experimental model of clinical immunity for human malaria

inferred← citeshttps://doi.org/10.1101/2025.03.26.25324737openalex-snapshot

Adjunctive ruxolitinib attenuates inflammation and enhances antiparasitic immunity in human volunteers experimentally infected with Plasmodium falciparum

inferred← citeshttps://doi.org/10.1101/2025.05.13.653848openalex-snapshot

Natural killer cell function in children with severe malaria differs according to malaria transmission intensity

inferred← citeshttps://doi.org/10.1101/2025.10.20.683440openalex-snapshot

Innate immune responses to Plasmodium falciparum disrupt the blood-brain barrier

inferred← citeshttps://doi.org/10.1111/cei.13653openalex-snapshot

Inflammatory cytokine responses in children with asymptomatic malaria infection living in rural, semi-urban and urban areas in south-eastern Gabon

inferred← citeshttps://doi.org/10.1111/febs.15494openalex-snapshot

γδ T cells in malaria: a double‐edged sword

inferred← citeshttps://doi.org/10.1111/imm.12877openalex-snapshot

Evaluating antidisease immunity to malaria and implications for vaccine design

inferred← citeshttps://doi.org/10.1111/imr.12804openalex-snapshot

The regulation of CD4+ T cells during malaria

inferred← citeshttps://doi.org/10.1111/imr.12814openalex-snapshot

Deciphering host immunity to malaria using systems immunology

inferred← citeshttps://doi.org/10.1111/imr.12817openalex-snapshot

Decoding the complexities of human malaria through systems immunology

inferred← citeshttps://doi.org/10.1111/imr.12835openalex-snapshot

Using two phases of the CD 4 T cell response to blood‐stage murine malaria to understand regulation of systemic immunity and placental pathology in Plasmodium falciparum infection

inferred← citeshttps://doi.org/10.1111/pim.12628openalex-snapshot

Immune effector mechanisms in malaria: An update focusing on human immunity

inferred← citeshttps://doi.org/10.1111/pim.12689openalex-snapshot

Kynurenine elevation correlates with T regulatory cells increase in acute Plasmodium vivax infection: A pilot study

inferred← citeshttps://doi.org/10.1111/pim.12763openalex-snapshot

Host‐pathogen interaction in the tissue environment during Plasmodium blood‐stage infection

inferred← citeshttps://doi.org/10.1126/sciadv.abg8384openalex-snapshot

Shared transcriptional profiles of atypical B cells suggest common drivers of expansion and function in malaria, HIV, and autoimmunity

inferred← citeshttps://doi.org/10.1126/scitranslmed.3009793openalex-snapshot

Loss and dysfunction of Vδ2 + γδ T cells are associated with clinical tolerance to malaria

inferred← citeshttps://doi.org/10.1128/iai.00427-20openalex-snapshot

Nonlethal Plasmodium yoelii Infection Drives Complex Patterns of Th2-Type Host Immunity and Mast Cell-Dependent Bacteremia

inferred← citeshttps://doi.org/10.1128/iai.00482-19openalex-snapshot

Splenic Innate B1 B Cell Plasmablasts Produce Sustained Granulocyte-Macrophage Colony-Stimulating Factor and Interleukin-3 Cytokines during Murine Malaria Infections

inferred← citeshttps://doi.org/10.1128/mbio.02796-25openalex-snapshot

Acute malaria dysregulates specialized lymph node macrophages to suppress vaccine-elicited protection against the Ebola virus

inferred← citeshttps://doi.org/10.1128/mmbr.00071-17openalex-snapshot

Transcriptomic Studies of Malaria: a Paradigm for Investigation of Systemic Host-Pathogen Interactions

inferred← citeshttps://doi.org/10.1128/msystems.00116-20openalex-snapshot

Host and Parasite Transcriptomic Changes upon Successive Plasmodium falciparum Infections in Early Childhood

inferred← citeshttps://doi.org/10.1146/annurev-pathmechdis-012419-032640openalex-snapshot

New Insights into Malaria Pathogenesis

inferred← citeshttps://doi.org/10.1172/jci.insight.169362openalex-snapshot

A molecular signature for IL-10–producing Th1 cells in protozoan parasitic diseases

inferred← citeshttps://doi.org/10.1172/jci.insight.183076openalex-snapshot

Phenotype and function of IL-10–producing NK cells in individuals with malaria experience

inferred← citeshttps://doi.org/10.1172/jci153733openalex-snapshot

Human IL-10–producing Th1 cells exhibit a molecular signature distinct from Tr1 cells in malaria

inferred← citeshttps://doi.org/10.1186/1475-2875-13-335openalex-snapshot

Plasmodium falciparum malaria and invasive bacterial co-infection in young African children: the dysfunctional spleen hypothesis

inferred← citeshttps://doi.org/10.1186/s12916-016-0683-6openalex-snapshot

The effect of declining exposure on T cell-mediated immunity to Plasmodium falciparum – an epidemiological “natural experiment”

inferred← citeshttps://doi.org/10.1186/s12916-017-0893-6openalex-snapshot

Resisting and tolerating P. falciparum in pregnancy under different malaria transmission intensities

inferred← citeshttps://doi.org/10.1186/s12916-019-1292-yopenalex-snapshot

Repeated clinical malaria episodes are associated with modification of the immune system in children

inferred← citeshttps://doi.org/10.1186/s12936-015-1036-1openalex-snapshot

Lipid metabolites of the phospholipase A2 pathway and inflammatory cytokines are associated with brain volume in paediatric cerebral malaria

inferred← citeshttps://doi.org/10.1186/s12936-016-1382-7openalex-snapshot

Characterization of blood dendritic and regulatory T cells in asymptomatic adults with sub-microscopic Plasmodium falciparum or Plasmodium vivax infection

inferred← citeshttps://doi.org/10.1186/s12936-017-2148-6openalex-snapshot

Both inflammatory and regulatory cytokine responses to malaria are blunted with increasing age in highly exposed children

inferred← citeshttps://doi.org/10.1186/s12936-018-2410-6openalex-snapshot

Characterization of T cell activation and regulation in children with asymptomatic Plasmodium falciparum infection

inferred← citeshttps://doi.org/10.1186/s12936-019-2702-5openalex-snapshot

Longitudinal analysis of gamma delta T cell subsets during malaria infections in Malian adults

inferred← citeshttps://doi.org/10.1186/s12936-019-3038-xopenalex-snapshot

Changing plasma cytokine, chemokine and growth factor profiles upon differing malaria transmission intensities

inferred← citeshttps://doi.org/10.1186/s12936-020-03533-wopenalex-snapshot

Dendritic cell responses to Plasmodium falciparum in a malaria-endemic setting

inferred← citeshttps://doi.org/10.1186/s12936-021-03849-1openalex-snapshot

Factors influencing phagocytosis of malaria parasites: the story so far

inferred← citeshttps://doi.org/10.1186/s12936-022-04134-5openalex-snapshot

Infection age as a predictor of epidemiological metrics for malaria

inferred← citeshttps://doi.org/10.1186/s12936-022-04199-2openalex-snapshot

Systems biology of malaria explored with nonhuman primates

inferred← citeshttps://doi.org/10.1186/s12936-023-04539-wopenalex-snapshot

The ability of Interleukin–10 to negate haemozoin-related pro-inflammatory effects has the potential to restore impaired macrophage function associated with malaria infection

inferred← citeshttps://doi.org/10.1186/s12936-023-04788-9openalex-snapshot

Plasmodium knowlesi in pig-tailed macaques: a potential new model for malaria vaccine research

inferred← citeshttps://doi.org/10.1186/s12936-026-05996-9openalex-snapshot

Association of interleukin-33 gene polymorphisms with susceptibility to Plasmodium falciparum infection

inferred← citeshttps://doi.org/10.12688/f1000research.74363.1openalex-snapshot

Explaining the unexpected COVID-19 trends and potential impact across Africa.

inferred← citeshttps://doi.org/10.12688/f1000research.74363.2openalex-snapshot

Explaining the unexpected COVID-19 trends and potential impact across Africa.

inferred← citeshttps://doi.org/10.12688/wellcomeopenres.16562.1openalex-snapshot

10-year longitudinal study of malaria in children: Insights into acquisition and maintenance of naturally acquired immunity

inferred← citeshttps://doi.org/10.12688/wellcomeopenres.16562.2openalex-snapshot

10-year longitudinal study of malaria in children: Insights into acquisition and maintenance of naturally acquired immunity

inferred← citeshttps://doi.org/10.12688/wellcomeopenres.16562.3openalex-snapshot

10-year longitudinal study of malaria in children: Insights into acquisition and maintenance of naturally acquired immunity

inferred← citeshttps://doi.org/10.1371/journal.pntd.0003154openalex-snapshot

Co-infection of Long-Term Carriers of Plasmodium falciparum with Schistosoma haematobium Enhances Protection from Febrile Malaria: A Prospective Cohort Study in Mali

inferred← citeshttps://doi.org/10.1371/journal.pntd.0003978openalex-snapshot

Transcription Profiling of Malaria-Naïve and Semi-immune Colombian Volunteers in a Plasmodium vivax Sporozoite Challenge

inferred← citeshttps://doi.org/10.1371/journal.pntd.0004415openalex-snapshot

Combined Immune Therapy for the Treatment of Visceral Leishmaniasis

inferred← citeshttps://doi.org/10.1371/journal.pone.0144654openalex-snapshot

IFN-γ and IL-21 Double Producing T Cells Are Bcl6-Independent and Survive into the Memory Phase in Plasmodium chabaudi Infection

inferred← citeshttps://doi.org/10.1371/journal.pone.0162132openalex-snapshot

A Novel Model of Asymptomatic Plasmodium Parasitemia That Recapitulates Elements of the Human Immune Response to Chronic Infection

inferred← citeshttps://doi.org/10.1371/journal.pone.0175864openalex-snapshot

CD4 T-cell expression of IFN-γ and IL-17 in pediatric malarial anemia

inferred← citeshttps://doi.org/10.1371/journal.pone.0242507openalex-snapshot

Red blood cell homeostasis in children and adults with and without asymptomatic malaria infection in Burkina Faso

inferred← citeshttps://doi.org/10.1371/journal.ppat.1004894openalex-snapshot

FCRL5 Delineates Functionally Impaired Memory B Cells Associated with Plasmodium falciparum Exposure

inferred← citeshttps://doi.org/10.1371/journal.ppat.1005041openalex-snapshot

Decline of FoxP3+ Regulatory CD4 T Cells in Peripheral Blood of Children Heavily Exposed to Malaria

inferred← citeshttps://doi.org/10.1371/journal.ppat.1005398openalex-snapshot

Blimp-1-Dependent IL-10 Production by Tr1 Cells Regulates TNF-Mediated Tissue Pathology

inferred← citeshttps://doi.org/10.1371/journal.ppat.1005909openalex-snapshot

Acute Malaria Induces PD1+CTLA4+ Effector T Cells with Cell-Extrinsic Suppressor Function

inferred← citeshttps://doi.org/10.1371/journal.ppat.1005975openalex-snapshot

STING-Licensed Macrophages Prime Type I IFN Production by Plasmacytoid Dendritic Cells in the Bone Marrow during Severe Plasmodium yoelii Malaria

inferred← citeshttps://doi.org/10.1371/journal.ppat.1006046openalex-snapshot

Macrophage Colony Stimulating Factor Derived from CD4+ T Cells Contributes to Control of a Blood-Borne Infection

inferred← citeshttps://doi.org/10.1371/journal.ppat.1009430openalex-snapshot

Plasmodium falciparum malaria drives epigenetic reprogramming of human monocytes toward a regulatory phenotype

inferred← citeshttps://doi.org/10.1371/journal.ppat.1010282openalex-snapshot

Systems analysis of immune responses to attenuated P. falciparum malaria sporozoite vaccination reveals excessive inflammatory signatures correlating with impaired immunity

inferred← citeshttps://doi.org/10.1371/journal.ppat.1011442openalex-snapshot

Symptomatic malaria enhances protection from reinfection with homologous Plasmodium falciparum parasites

inferred← citeshttps://doi.org/10.1371/journal.ppat.1011585openalex-snapshot

NK cell-induced damage to P.falciparum-infected erythrocytes requires ligand-specific recognition and releases parasitophorous vacuoles that are phagocytosed by monocytes in the presence of immune IgG

inferred← citeshttps://doi.org/10.1371/journal.ppat.1014287openalex-snapshot

Hidden hematological, biochemical and immune costs of asymptomatic malaria infections in semi-wild chimpanzees

inferred← citeshttps://doi.org/10.17795/iji-20984openalex-snapshot

Malaria in Children

inferred← citeshttps://doi.org/10.18281/iti.2015.1.3openalex-snapshot

The Role of Regulatory CD4+ CD25+ T Cell Subset in Host Homeostasis during Protozoan Infection: An Overview

inferred← citeshttps://doi.org/10.2217/fmb-2022-0261openalex-snapshot

Systems Biology Of Disease Tolerance to Malaria

inferred← citeshttps://doi.org/10.25913/5eb38f17bade8openalex-snapshot

Functional characterisation of human blood dendritic cell subsets during plasmodium infection

inferred← citeshttps://doi.org/10.30644/rik.v11i2.634openalex-snapshot

Perbandingan ekspresi sitokin proinflamasi interferon-gamma dan antiinflamasi interleukin-10 pada whole blood culture terhadap pajanan mikroorganisme yang distimulasi dengan phytohemagglutinin pada subjek di pemukiman kumuh dan non kumuh

inferred← citeshttps://doi.org/10.32657/10356/72534openalex-snapshot

Control of Plasmodium falciparum infection by human natural killer cells

inferred← citeshttps://doi.org/10.3389/fimmu.2014.00498openalex-snapshot

The Regulation of CD4+ T Cell Responses during Protozoan Infections

inferred← citeshttps://doi.org/10.3389/fimmu.2014.00671openalex-snapshot

CD4 T-Cell Subsets in Malaria: TH1/TH2 Revisited

inferred← citeshttps://doi.org/10.3389/fimmu.2017.00966openalex-snapshot

Chronic Exposure to Malaria Is Associated with Inhibitory and Activation Markers on Atypical Memory B Cells and Marginal Zone-Like B Cells

inferred← citeshttps://doi.org/10.3389/fimmu.2017.01329openalex-snapshot

The Development of Plasmodium falciparum-Specific IL10 CD4 T Cells and Protection from Malaria in Children in an Area of High Malaria Transmission

inferred← citeshttps://doi.org/10.3389/fimmu.2018.02445openalex-snapshot

γδ T Cells in Antimalarial Immunity: New Insights Into Their Diverse Functions in Protection and Tolerance

inferred← citeshttps://doi.org/10.3389/fimmu.2018.02769openalex-snapshot

Novel Strategies for Malaria Vaccine Design

inferred← citeshttps://doi.org/10.3389/fimmu.2019.00229openalex-snapshot

The Role of IL-10 in Malaria: A Double Edged Sword

inferred← citeshttps://doi.org/10.3389/fimmu.2019.00248openalex-snapshot

Infection-Induced Resistance to Experimental Cerebral Malaria Is Dependent Upon Secreted Antibody-Mediated Inhibition of Pathogenic CD8+ T Cell Responses

inferred← citeshttps://doi.org/10.3389/fimmu.2019.01096openalex-snapshot

TH1-Polarized TFH Cells Delay Naturally-Acquired Immunity to Malaria

inferred← citeshttps://doi.org/10.3389/fimmu.2019.02398openalex-snapshot

Understanding P. falciparum Asymptomatic Infections: A Proposition for a Transcriptomic Approach

inferred← citeshttps://doi.org/10.3389/fimmu.2020.575103openalex-snapshot

Evolutionarily Selected Overexpression of the Cytokine BAFF Enhances Mucosal Immune Response Against P. falciparum

inferred← citeshttps://doi.org/10.3389/fimmu.2021.614817openalex-snapshot

Altered Ex-Vivo Cytokine Responses in Children With Asymptomatic Plasmodium falciparum Infection in Burkina Faso: An Additional Argument to Treat Asymptomatic Malaria?

inferred← citeshttps://doi.org/10.3389/fimmu.2022.959697openalex-snapshot

Recent malaria does not substantially impact COVID-19 antibody response or rates of symptomatic illness in communities with high malaria and COVID-19 transmission in Mali, West Africa

inferred← citeshttps://doi.org/10.3389/fimmu.2022.965503openalex-snapshot

A diversified role for γδT cells in vector-borne diseases

inferred← citeshttps://doi.org/10.3390/nu14245303openalex-snapshot

Potential Benefits of Lycopene Consumption: Rationale for Using It as an Adjuvant Treatment for Malaria Patients and in Several Diseases

inferred← citeshttps://doi.org/10.3390/pathogens10101277openalex-snapshot

Immunosuppression in Malaria: Do Plasmodium falciparum Parasites Hijack the Host?

inferred← citeshttps://doi.org/10.3390/vaccines10091525openalex-snapshot

Mouse Models for Unravelling Immunology of Blood Stage Malaria

inferred← citeshttps://doi.org/10.3390/vaccines14050423openalex-snapshot

Cellular Immune Response and T Cell Epitope Mapping of Plasmodium falciparum Chimeric Vaccine Candidate GMZ2.6c and Its Components (MSP-3, GLURP and Pfs48/45) in Individuals Naturally Exposed to Malaria in Brazilian Amazon

inferred← citeshttps://doi.org/10.3978/j.issn.2305-5839.2015.02.22openalex-snapshot

γδ T cells and immunity to human malaria in endemic regions.

inferred← citeshttps://doi.org/10.4049/jimmunol.1403216openalex-snapshot

Early Effector Cells Survive the Contraction Phase in Malaria Infection and Generate Both Central and Effector Memory T Cells

inferred← citeshttps://doi.org/10.4049/jimmunol.1600619openalex-snapshot

The Impact of Established Immunoregulatory Networks on Vaccine Efficacy and the Development of Immunity to Malaria

inferred← citeshttps://doi.org/10.4049/jimmunol.1600968openalex-snapshot

Long-Lived CD4+IFN-γ+ T Cells rather than Short-Lived CD4+IFN-γ+IL-10+ T Cells Initiate Rapid IL-10 Production To Suppress Anamnestic T Cell Responses during Secondary Malaria Infection

inferred← citeshttps://doi.org/10.4049/jimmunol.1701010openalex-snapshot

Cutting Edge: Plasmodium falciparum Induces Trained Innate Immunity

inferred← citeshttps://doi.org/10.4049/jimmunol.1701582openalex-snapshot

Distinct Roles for CD4+ Foxp3+ Regulatory T Cells and IL-10–Mediated Immunoregulatory Mechanisms during Experimental Visceral Leishmaniasis Caused by Leishmania donovani

inferred← citeshttps://doi.org/10.4049/jimmunol.2300525openalex-snapshot

IL-15 Complex–Induced IL-10 Enhances Plasmodium-specific CD4+ T Follicular Helper Differentiation and Antibody Production

inferred← citeshttps://doi.org/10.4314/jasem.v24i5.9openalex-snapshot

Analysis of a Malaria Transmission Model in Children

inferred← citeshttps://doi.org/10.5772/intechopen.1007859openalex-snapshot

Perspective Chapter: The Double Edge Sword – Modulations of Inflammatory Responses – Parasite Survival Strategy or Host Tolerance Mechanisms

inferred← citeshttps://doi.org/10.5772/intechopen.77293openalex-snapshot

Understanding the Importance of Asymptomatic and Low- Density Infections for Malaria Elimination

inferred← citeshttps://doi.org/10.59411/990get50openalex-snapshot

Status in Malaria Vaccine Development: Basic aspects of Vaccine, Mechanism of actions, Vaccine pipelines, Stage oriented immune response ‘Challenges and Opportunities’

inferred← citeshttps://doi.org/10.6084/m9.figshare.22638040openalex-snapshot

Additional file 1 of The ability of Interleukin–10 to negate haemozoin-related pro-inflammatory effects has the potential to restore impaired macrophage function associated with malaria infection

inferred← citeshttps://doi.org/10.6084/m9.figshare.22638043openalex-snapshot

Additional file 2 of The ability of Interleukin–10 to negate haemozoin-related pro-inflammatory effects has the potential to restore impaired macrophage function associated with malaria infection

inferred← citeshttps://doi.org/10.6084/m9.figshare.24804292openalex-snapshot

Additional file 1 of Plasmodium knowlesi in pig-tailed macaques: a potential new model for malaria vaccine research

inferred← citeshttps://doi.org/10.70675/d02ff198zc8f0z48bczb7a8z82613f98bb52openalex-snapshot

Compréhension de la résistance humaine au paludisme : des études génétiques aux approches fonctionnelles

inferred← citeshttps://doi.org/10.7554/elife.107820openalex-snapshot

A natural experiment in Kenya reveals durable immunosuppressive effects of early childhood malaria: a longitudinal cohort study

inferred← citeshttps://doi.org/10.7554/elife.107820.1openalex-snapshot

A natural experiment in Kenya reveals durable immunosuppressive effects of early childhood malaria: a longitudinal cohort study

inferred← citeshttps://doi.org/10.7554/elife.107820.3openalex-snapshot

A natural experiment in Kenya reveals durable immunosuppressive effects of early childhood malaria: a longitudinal cohort study

inferred← citeshttps://doi.org/10.7554/elife.62800openalex-snapshot

Mapping immune variation and var gene switching in naive hosts infected with Plasmodium falciparum

inferred← citeshttps://doi.org/10.7554/elife.63838openalex-snapshot

Inducible mechanisms of disease tolerance provide an alternative strategy of acquired immunity to malaria

inferred← citeshttps://doi.org/10.7759/cureus.6696openalex-snapshot

Malarial Hepatopathy in Children Visiting a Tertiary Healthcare Hospital in Karachi

Editorial review for independent work. ICSAC — the Institute for Complexity Science and Advanced Computing — runs a structured editorial panel for researchers publishing outside journals, and publishes on CiteStamp rails. Submission is free and goes through ORCID: icsacinstitute.org/submit.