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Cerebral Microvascular Accumulation of Tau Oligomers in Alzheimer’s Disease and Related Tauopathies

https://doi.org/10.14336/ad.2017.0112

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Pathological Interface Between Oligomeric Alpha-Synuclein and Tau in Synucleinopathies

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Amyloid-β oligomers as a template for secondary amyloidosis in Alzheimer's disease

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Matrix metalloproteinase 9–mediated intracerebral hemorrhage induced by cerebral amyloid angiopathy

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Impaired cerebral vasoreactivity to CO2 in Alzheimer's disease using BOLD fMRI

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Dendritic spine loss in the hippocampus of young PDAPP and Tg2576 mice and its prevention by the ApoE2 genotype

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Tau Oligomers Impair Artificial Membrane Integrity and Cellular Viability

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Differential activation of mitochondrial apoptotic pathways by vasculotropic amyloid‐β variants in cells composing the cerebral vessel walls

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Identification of oligomers at early stages of tau aggregation in Alzheimer's disease

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APPSW Transgenic Mice Develop Age-related Aβ Deposits and Neuropil Abnormalities, but no Neuronal Loss in CA1

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Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer‐induced AMPA glutamate receptor signaling deficits

inferredcites →https://doi.org/10.1111/j.1365-2990.2005.00663.xopenalex-snapshot

Relationship of neurofibrillary pathology to cerebral amyloid angiopathy in Alzheimer's disease

inferredcites →https://doi.org/10.1111/j.1365-2990.2010.01137.xopenalex-snapshot

Review: Sporadic cerebral amyloid angiopathy

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Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic Mice

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Trimeric Tau Is Toxic to Human Neuronal Cells at Low Nanomolar Concentrations

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Amyloid Oligomers Exacerbate Tau Pathology in a Mouse Model of Tauopathy

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Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice

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inferredcites →https://doi.org/10.1523/jneurosci.3192-13.2014openalex-snapshot

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Formation and Propagation of Tau Oligomeric Seeds

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Unveiling the hidden connection: the blood-brain barrier’s role in epilepsy

inferred← citeshttps://doi.org/10.3389/fnins.2021.731614openalex-snapshot

Retinal Vasculopathy in Alzheimer’s Disease

inferred← citeshttps://doi.org/10.3390/biom13050830openalex-snapshot

Microvascular Contributions to Alzheimer Disease Pathogenesis: Is Alzheimer Disease Primarily an Endotheliopathy?

inferred← citeshttps://doi.org/10.3390/biom14010003openalex-snapshot

Beta-Amyloid Enhances Vessel Formation in Organotypic Brain Slices Connected to Microcontact Prints

inferred← citeshttps://doi.org/10.3390/biomedicines10040742openalex-snapshot

Addressing Blood–Brain Barrier Impairment in Alzheimer’s Disease

inferred← citeshttps://doi.org/10.3390/biomedicines13010029openalex-snapshot

Novel Role of Pin1-Cis P-Tau-ApoE Axis in the Pathogenesis of Preeclampsia and Its Connection with Dementia

inferred← citeshttps://doi.org/10.3390/biomimetics3010004openalex-snapshot

Targeting Early Dementia: Using Lipid Cubic Phase Nanocarriers to Cross the Blood–Brain Barrier

inferred← citeshttps://doi.org/10.3390/biophysica2040039openalex-snapshot

Heterogeneous Tau Oligomers as Molecular Targets for Alzheimer’s Disease and Related Tauopathies

inferred← citeshttps://doi.org/10.3390/cancers14215386openalex-snapshot

Tau Protein as Therapeutic Target for Cancer? Focus on Glioblastoma

inferred← citeshttps://doi.org/10.3390/ijms20246319openalex-snapshot

The Amyloid-Tau-Neuroinflammation Axis in the Context of Cerebral Amyloid Angiopathy

inferred← citeshttps://doi.org/10.3390/ijms22147710openalex-snapshot

Blood–Brain Barrier and Neurodegenerative Diseases—Modeling with iPSC-Derived Brain Cells

inferred← citeshttps://doi.org/10.3390/jcm8050651openalex-snapshot

Vascular Dysfunction in Alzheimer’s Disease: A Prelude to the Pathological Process or a Consequence of It?

inferred← citeshttps://doi.org/10.3390/pharmaceutics12090801openalex-snapshot

CRISPR/Cas9 Delivery Potentials in Alzheimer’s Disease Management: A Mini Review

inferred← citeshttps://doi.org/10.3929/ethz-b-000520246openalex-snapshot

Arterial spin labeling demonstrates preserved regional cerebral blood flow in the P301L mouse model of tauopathy

inferred← citeshttps://doi.org/10.4172/2161-0460.1000443openalex-snapshot

Targeting Prion-like Cis Phosphorylated Tau Pathology in Neurodegenerative Diseases

inferred← citeshttps://doi.org/10.6084/m9.figshare.12825330openalex-snapshot

Additional file 1 of Tau reduction in aged mice does not impact Microangiopathy

inferred← citeshttps://doi.org/10.6084/m9.figshare.13279800openalex-snapshot

Additional file 3 of Retinal capillary degeneration and blood-retinal barrier disruption in murine models of Alzheimer’s disease

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