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Sodium and Potassium Balance Depends on αENaC Expression in Connecting Tubule

https://doi.org/10.1681/asn.2009101077
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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1002/gene.10041openalex-snapshot
Conditional gene targeting of the Scnn1a (αENaC) gene locus
inferredcites →https://doi.org/10.1007/s00424-009-0656-0openalex-snapshot
Regulated sodium transport in the renal connecting tubule (CNT) via the epithelial sodium channel (ENaC)
inferredcites →https://doi.org/10.1016/0092-8674(94)90250-xopenalex-snapshot
Liddle's syndrome: Heritable human hypertension caused by mutations in the β subunit of the epithelial sodium channel
inferredcites →https://doi.org/10.1016/b978-012088488-9.50035-8openalex-snapshot
Mineralocorticoid Action in the Aldosterone-Sensitive Distal Nephron
inferredcites →https://doi.org/10.1016/s0303-7207(97)00149-4openalex-snapshot
Pseudohypoaldosteronism: mutation found, problem solved?
inferredcites →https://doi.org/10.1038/ng0396-248openalex-snapshot
Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1
inferredcites →https://doi.org/10.1038/ng0396-325openalex-snapshot
Early death due to defective neonatal lung liquid clearance in αENaC-deficient mice
inferredcites →https://doi.org/10.1038/ng0995-76openalex-snapshot
Hypertension caused by a truncated epithelial sodium channel γ subunit: genetic heterogeneity of Liddle syndrome
inferredcites →https://doi.org/10.1073/pnas.90.24.11663openalex-snapshot
Cellular and subcellular immunolocalization of vasopressin-regulated water channel in rat kidney.
inferredcites →https://doi.org/10.1073/pnas.96.4.1727openalex-snapshot
Disruption of the β subunit of the epithelial Na + channel in mice: Hyperkalemia and neonatal death associated with a pseudohypoaldosteronism phenotype
inferredcites →https://doi.org/10.1073/pnas.96.4.1732openalex-snapshot
Salt restriction induces pseudohypoaldosteronism type 1 in mice expressing low levels of the β-subunit of the amiloride-sensitive epithelial sodium channel
inferredcites →https://doi.org/10.1074/jbc.c400080200openalex-snapshot
Epithelial Sodium Channels Are Activated by Furin-dependent Proteolysis
inferredcites →https://doi.org/10.1074/jbc.m708829200openalex-snapshot
Postnatal Requirement of the Epithelial Sodium Channel for Maintenance of Epidermal Barrier Function
inferredcites →https://doi.org/10.1097/01.asn.0000017902.77985.84openalex-snapshot
Blood Pressure, Cardiac, and Renal Responses to Salt and Deoxycorticosterone Acetate in Mice
inferredcites →https://doi.org/10.1152/ajplegacy.1964.206.4.674openalex-snapshot
Micropuncture study of renal potassium excretion in the rat
inferredcites →https://doi.org/10.1152/ajprenal.00257.2005openalex-snapshot
Kidney function in mice lacking aldosterone
inferredcites →https://doi.org/10.1152/ajprenal.00381.2003openalex-snapshot
Na channels in the rat connecting tubule
inferredcites →https://doi.org/10.1152/ajprenal.00383.2005openalex-snapshot
Lithium treatment induces a marked proliferation of primarily principal cells in rat kidney inner medullary collecting duct
inferredcites →https://doi.org/10.1152/ajprenal.00422.2005openalex-snapshot
Regulation of maturation and processing of ENaC subunits in the rat kidney
inferredcites →https://doi.org/10.1152/ajprenal.00454.2003openalex-snapshot
Sodium and potassium handling by the aldosterone-sensitive distal nephron: the pivotal role of the distal and connecting tubule
inferredcites →https://doi.org/10.1152/ajprenal.0085.2001openalex-snapshot
Distribution of transcellular calcium and sodium transport pathways along mouse distal nephron
inferredcites →https://doi.org/10.1152/ajprenal.2000.279.2.f252openalex-snapshot
Differential subcellular localization of ENaC subunits in mouse kidney in response to high- and low-Na diets
inferredcites →https://doi.org/10.1152/ajprenal.2000.279.5.f874openalex-snapshot
Expression of aquaporins in the renal connecting tubule
inferredcites →https://doi.org/10.1152/ajprenal.90495.2008openalex-snapshot
Fluorescence isolation of mouse late distal convoluted tubules and connecting tubules: effects of vasopressin and vitamin D3 on Ca2+ signaling
inferredcites →https://doi.org/10.1172/jci118731openalex-snapshot
Rat renal arcade segment expresses vasopressin-regulated water channel and vasopressin V2 receptor.
inferredcites →https://doi.org/10.1172/jci16956openalex-snapshot
Collecting duct–specific gene inactivation of αENaC in the mouse kidney does not impair sodium and potassium balance
inferredcites →https://doi.org/10.1172/jci3971openalex-snapshot
Role of gammaENaC subunit in lung liquid clearance and electrolyte balance in newborn mice. Insights into perinatal adaptation and pseudohypoaldosteronism.
inferredcites →https://doi.org/10.1681/asn.2006090975openalex-snapshot
Impairment of Sodium Balance in Mice Deficient in Renal Principal Cell Mineralocorticoid Receptor
inferredcites →https://doi.org/10.1681/asn.v134836openalex-snapshot
Human Cortical Distal Nephron

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Comment on Urinary Sodium Level and Its Association with Cardiovascular Risk
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Molecular Physiology of the Medullary Collecting Duct
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Function and Regulation of the Epithelial Na + Channel ENaC
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Dietary potassium and the renal control of salt balance and blood pressure
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Adult nephron-specific MR-deficient mice develop a severe renal PHA-1 phenotype
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Severe hyperkalemia is rescued by low-potassium diet in renal βENaC-deficient mice
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Relative Roles of Principal and Intercalated Cells in the Regulation of Sodium Balance and Blood Pressure
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Pseudohypoaldosteronism type 1
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inferred← citeshttps://doi.org/10.1016/j.coph.2013.11.010openalex-snapshot
Epithelial sodium channel (ENaC) and the control of blood pressure
inferred← citeshttps://doi.org/10.1016/j.coph.2014.12.012openalex-snapshot
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Genetic dissection of sodium and potassium transport along the aldosterone‐sensitive distal nephron: Importance in the control of blood pressure and hypertension
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An angiotensin II type 1 receptor binding molecule has a critical role in hypertension in a chronic kidney disease model
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Aldosterone resistance: Structural and functional considerations and new perspectives
inferred← citeshttps://doi.org/10.1016/j.semnephrol.2013.05.010openalex-snapshot
Epigenetics and the Control of the Collecting Duct Epithelial Sodium Channel
inferred← citeshttps://doi.org/10.1016/j.snb.2019.127376openalex-snapshot
A direct fluorescent signal transducer embedded in a DNA aptamer paves the way for versatile metal-ion detection
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Deletion of the angiotensin II type 1 receptor–associated protein enhances renal sodium reabsorption and exacerbates angiotensin II–mediated hypertension
inferred← citeshttps://doi.org/10.1038/s41598-017-03510-5openalex-snapshot
Aldosterone Modulates the Association between NCC and ENaC
inferred← citeshttps://doi.org/10.1042/bcj20160312openalex-snapshot
The sodium chloride cotransporter (NCC) and epithelial sodium channel (ENaC) associate
inferred← citeshttps://doi.org/10.1042/bsr20130086openalex-snapshot
Physical and functional interaction of Rnf2 with Af9 regulates basal and aldosterone-stimulated transcription of the α-ENaC gene in a renal collecting duct cell line
inferred← citeshttps://doi.org/10.1056/nejmra1404726openalex-snapshot
Molecular Physiology of Water Balance
inferred← citeshttps://doi.org/10.1073/pnas.1221496110openalex-snapshot
Renal intercalated cells are rather energized by a proton than a sodium pump
inferred← citeshttps://doi.org/10.1073/pnas.1406741111openalex-snapshot
The Cap1–claudin-4 regulatory pathway is important for renal chloride reabsorption and blood pressure regulation
inferred← citeshttps://doi.org/10.1073/pnas.1421441112openalex-snapshot
KLHL3 regulates paracellular chloride transport in the kidney by ubiquitination of claudin-8
inferred← citeshttps://doi.org/10.1074/jbc.m112.389148openalex-snapshot
Scaffold Protein Connector Enhancer of Kinase Suppressor of Ras Isoform 3 (CNK3) Coordinates Assembly of a Multiprotein Epithelial Sodium Channel (ENaC)-regulatory Complex
inferred← citeshttps://doi.org/10.1074/jbc.ra119.011789openalex-snapshot
Paraoxonase 3 functions as a chaperone to decrease functional expression of the epithelial sodium channel
inferred← citeshttps://doi.org/10.1093/med/9780199592548.003.0020openalex-snapshot
An overview of tubular function
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Increased SGK1 activity potentiates mineralocorticoid/NaCl-induced hypertension and kidney injury
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Genetic ablation of aquaporin‐2 in the mouse connecting tubules results in defective renal water handling
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International Union of Basic and Clinical Pharmacology. XCI. Structure, Function, and Pharmacology of Acid-Sensing Ion Channels and the Epithelial Na+ Channel
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Renal tubular SGK1 deficiency causes impaired K+ excretion via loss of regulation of NEDD4-2/WNK1 and ENaC
inferred← citeshttps://doi.org/10.1152/ajprenal.00020.2026openalex-snapshot
How does the kidney conserve Na + in a salt-scarce environment?
inferred← citeshttps://doi.org/10.1152/ajprenal.00067.2017openalex-snapshot
Severe acute dehydration in a desert rodent elicits a transcriptional response that effectively prevents kidney injury
inferred← citeshttps://doi.org/10.1152/ajprenal.00093.2015openalex-snapshot
Vasopressin regulation of sodium transport in the distal nephron and collecting duct
inferred← citeshttps://doi.org/10.1152/ajprenal.00177.2013openalex-snapshot
Sp1 trans-activates and is required for maximal aldosterone induction of the αENaC gene in collecting duct cells
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Aldosterone-dependent and -independent regulation of the epithelial sodium channel (ENaC) in mouse distal nephron
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Reducing αENaC expression in the kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading
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Maintaining K+balance on the low-Na+, high-K+diet
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Aldosterone reprograms promoter methylation to regulate αENaC transcription in the collecting cuct
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Increased SGK1 activity potentiates mineralocorticoid/NaCl-induced kidney injury
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Landscape of ENaC regulation in the kidney
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An Af9 cis-element directly targets Dot1a to mediate transcriptional repression of the αENaC gene
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Salt sensitivity of volume and blood pressure in a mouse with globally reduced ENaC γ-subunit expression
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mPGES-1-derived PGE2mediates dehydration natriuresis
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RNA sequencing of kidney distal tubule cells reveals multiple mediators of chronic aldosterone action
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Learning Physiology from Inherited Kidney Disorders
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Independent Regulation of Na+ and K+ Balance by the Kidney
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PPARγ Agonist-Induced Fluid Retention Depends on αENaC Expression in Connecting Tubules
inferred← citeshttps://doi.org/10.1161/hypertensionaha.111.00572openalex-snapshot
Enhanced Angiotensin Receptor-Associated Protein in Renal Tubule Suppresses Angiotensin-Dependent Hypertension
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Impact of Angiotensin Type 1A Receptors in Principal Cells of the Collecting Duct on Blood Pressure and Hypertension
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In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron
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Role of adenylyl cyclase 6 in the development of lithium-induced nephrogenic diabetes insipidus
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The Glucocorticoid-Induced Leucine Zipper (Gilz/Tsc22d3-2) Gene Locus Plays a Crucial Role in Male Fertility
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Epithelial Sodium Channel-Mediated Sodium Transport Is Not Dependent on the Membrane-Bound Serine Protease CAP2/Tmprss4
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AT1 Receptors in the Collecting Duct Directly Modulate the Concentration of Urine
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Aldosterone Regulates MicroRNAs in the Cortical Collecting Duct to Alter Sodium Transport
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Colon-Specific Deletion of Epithelial Sodium Channel Causes Sodium Loss and Aldosterone Resistance
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Severe Salt–Losing Syndrome and Hyperkalemia Induced by Adult Nephron–Specific Knockout of the Epithelial Sodium Channel α-Subunit
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Acetazolamide Attenuates Lithium–Induced Nephrogenic Diabetes Insipidus
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Selective Deletion of the Mechanistic Target of Rapamycin From the Renal Collecting Duct Principal Cell in Mice Down-Regulates the Epithelial Sodium Channel
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Dissecting the Effects of Aldosterone and Hypokalemia on the Epithelial Na+ Channel and the NaCl Cotransporter
inferred← citeshttps://doi.org/10.3390/biomedicines10050981openalex-snapshot
Epithelial Sodium Channel Alpha Subunit (αENaC) Is Associated with Inverse Salt Sensitivity of Blood Pressure
inferred← citeshttps://doi.org/10.3390/biomedicines10092169openalex-snapshot
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Rôle des cellules ß - intercalaires dans le maintien de la balance du sodium et de la pression artérielle
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Potassium dependent rescue of a myopathy with core-like structures in mouse
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