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Serum Trimethylamine-N-Oxide is Elevated in CKD and Correlates with Coronary Atherosclerosis Burden
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Comparison of Survival Probabilities for Dialysis Patients vs Cadaveric Renal Transplant Recipients
inferredcites →https://doi.org/10.1007/s00240-003-0381-7openalex-snapshot
Bacterial translocation in experimental uremia
inferredcites →https://doi.org/10.1007/s11255-013-0407-1openalex-snapshot
The risk of atherosclerosis in patients with chronic kidney disease
inferredcites →https://doi.org/10.1016/j.jpba.2015.02.040openalex-snapshot
Development and validation of a simple UHPLC–MS/MS method for the simultaneous determination of trimethylamine N-oxide, choline, and betaine in human plasma and urine
inferredcites →https://doi.org/10.1016/s0002-9149(83)80105-2openalex-snapshot
A more meaningful scoring system for determining the severity of coronary heart disease
inferredcites →https://doi.org/10.1016/s0006-2952(98)00218-4openalex-snapshot
Isoform specificity of trimethylamine N-oxygenation by human flavin-containing monooxygenase (FMO) and P450 enzymes
inferredcites →https://doi.org/10.1038/ki.1996.107openalex-snapshot
Enhanced scavenger receptor expression in monocyte-macrophages in dialysis patients
inferredcites →https://doi.org/10.1038/ki.1997.110openalex-snapshot
Uremic serum enhances scavenger receptor expression and activity in the human monocytic cell line U937
inferredcites →https://doi.org/10.1038/ki.2012.345openalex-snapshot
Chronic kidney disease alters intestinal microbial flora
inferredcites →https://doi.org/10.1038/nature09922openalex-snapshot
Gut flora metabolism of phosphatidylcholine promotes cardiovascular disease
inferredcites →https://doi.org/10.1038/nm.3145openalex-snapshot
Intestinal microbiota metabolism of l-carnitine, a nutrient in red meat, promotes atherosclerosis
inferredcites →https://doi.org/10.1053/ajkd.1998.v32.pm9820470openalex-snapshot
Clinical epidemiology of cardiovascular disease in chronic renal disease
inferredcites →https://doi.org/10.1053/j.ajkd.2014.01.416openalex-snapshot
KDOQI US Commentary on the 2012 KDIGO Clinical Practice Guideline for the Evaluation and Management of CKD
inferredcites →https://doi.org/10.1056/nejm199912023412303openalex-snapshot
Comparison of Mortality in All Patients on Dialysis, Patients on Dialysis Awaiting Transplantation, and Recipients of a First Cadaveric Transplant
inferredcites →https://doi.org/10.1056/nejmoa041031openalex-snapshot
Chronic Kidney Disease and the Risks of Death, Cardiovascular Events, and Hospitalization
inferredcites →https://doi.org/10.1056/nejmoa1109400openalex-snapshot
Intestinal Microbial Metabolism of Phosphatidylcholine and Cardiovascular Risk
inferredcites →https://doi.org/10.1080/004982597239949openalex-snapshot
Metabolic disposition of [14C]-trimethylamine N-oxide in rat: variation with dose and route of administration
inferredcites →https://doi.org/10.1093/eurheartj/ehu002openalex-snapshot
Prognostic value of choline and betaine depends on intestinal microbiota-generated metabolite trimethylamine-N-oxide
inferredcites →https://doi.org/10.1093/ndt/12.12.2597openalex-snapshot
Incidence and risk factors of atherosclerotic cardiovascular accidents in predialysis chronic renal failure patients: a prospective study
inferredcites →https://doi.org/10.1093/ndt/12.6.1167openalex-snapshot
Enhanced gene expression of scavenger receptor in peripheral blood monocytes from patients on cuprophane haemodialysis
inferredcites →https://doi.org/10.1093/ndt/gfk056openalex-snapshot
Accumulation of trimethylamine and trimethylamine-N-oxide in end-stage renal disease patients undergoing haemodialysis
inferredcites →https://doi.org/10.1111/j.1523-1755.2005.00551.xopenalex-snapshot
Cardiovascular risk factors in chronic kidney disease
inferredcites →https://doi.org/10.1111/j.1525-1594.2005.29097.xopenalex-snapshot
Expression of Scavenger Receptor CD36 in Chronic Renal Failure Patients
inferredcites →https://doi.org/10.1111/j.1525-1594.2005.29110.xopenalex-snapshot
Aberrant Activation of the TNF‐α System and Production of Fas and Scavenger Receptors on Monocytes in Patients With End‐stage Renal Disease
inferredcites →https://doi.org/10.1136/gut.32.7.754openalex-snapshot
Impaired intestinal barrier function measured by differently sized polyethylene glycols in patients with chronic renal failure.
inferredcites →https://doi.org/10.1159/000051199openalex-snapshot
Clinical Subtypes of Alzheimer’s Disease
inferredcites →https://doi.org/10.1159/000186158openalex-snapshot
Increased Intestinal Permeability to Differently Sized Polyethylene Glycols in Uremic Rats: Effects of Low- and High-Protein Diets
inferredcites →https://doi.org/10.1159/000189334openalex-snapshot
Inhibition of the Accumulation of Uremic Toxins in the Blood and Their Precursors in the Feces after Oral Administration of Lebenin®, a Lactic Acid Bacteria Preparation, to Uremic Patients Undergoing Hemodialysis
inferredcites →https://doi.org/10.1159/000353764openalex-snapshot
Chronic Kidney Disease Causes Disruption of Gastric and Small Intestinal Epithelial Tight Junction
inferredcites →https://doi.org/10.1159/000447068openalex-snapshot
Should We STOP Angiotensin Converting Enzyme Inhibitors/Angiotensin Receptor Blockers in Advanced Kidney Disease?
inferredcites →https://doi.org/10.1159/000468530openalex-snapshot
Magnesium Replacement Improves the Metabolic Profile in Obese and Pre-Diabetic Patients with Mild-to-Moderate Chronic Kidney Disease: A 3-Month, Randomised, Double-Blind, Placebo-Controlled Study
inferredcites →https://doi.org/10.1161/01.cir.0000095676.90936.80openalex-snapshot
Kidney Disease as a Risk Factor for Development of Cardiovascular Disease
inferredcites →https://doi.org/10.1161/atvbaha.109.188995openalex-snapshot
Renal Dysfunction Potentiates Foam Cell Formation by Repressing ABCA1
inferredcites →https://doi.org/10.1161/circinterventions.109.876672openalex-snapshot
Diabetes Duration Is Associated With Increased Thin-Cap Fibroatheroma Detected by Intravascular Ultrasound With Virtual Histology
inferredcites →https://doi.org/10.1161/circresaha.116.305360openalex-snapshot
Gut Microbiota-Dependent Trimethylamine N -Oxide (TMAO) Pathway Contributes to Both Development of Renal Insufficiency and Mortality Risk in Chronic Kidney Disease
inferredcites →https://doi.org/10.1681/asn.2009111132openalex-snapshot
Metabolite Profiling Identifies Markers of Uremia
inferredcites →https://doi.org/10.1681/asn.2013080905openalex-snapshot
The Gut Microbiome, Kidney Disease, and Targeted Interventions
inferredcites →https://doi.org/10.2337/dc07-2024openalex-snapshot
Low Adiponectin Levels Are Associated With Atherogenic Dyslipidemia and Lipid-Rich Plaque in Nondiabetic Coronary Arteries
inferredcites →https://doi.org/10.3109/00498258709043962openalex-snapshot
The metabolism of14C-labelled trimethylamine and itsN-oxide in man
inferredcites →https://doi.org/10.7326/0003-4819-150-9-200905050-00006openalex-snapshot
A New Equation to Estimate Glomerular Filtration Rate
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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Trimethylamine-N-Oxide (TMAO) and Predicted Risk of Cardiovascular Events After Partial Nephrectomy
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Association of Trimethylamine N-Oxide and Metabolites With Mortality in Older Adults
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Aldosterone antagonists for people with chronic kidney disease requiring dialysis
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Cardiovascular risk of dietary trimethylamine oxide precursors and the therapeutic potential of resveratrol and its derivatives
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Trimethylamine N‐Oxide Electrochemical Biosensor with a Chimeric Enzyme
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Trimethylamine N‐Oxide Linking Gut Microbiota to Cardiocerebral Disease Is a Novel Biomarker for Stroke Subtypes With Mixed Etiology: A Prospective Cohort Study
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Microbiome‐Derived Metabolites as Molecular Mediators of Gut‐Organ Communication: Mechanisms and Therapeutic Opportunities
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Cardiovascular‐Kidney‐Metabolic Syndrome in People With HIV : An Emerging Frontier for Clinical Pharmacology
inferred← citeshttps://doi.org/10.1002/jcp.30568openalex-snapshot
Trimethylamine N‐oxide exacerbates acetaminophen‐induced liver injury by interfering with macrophage‐mediated liver regeneration
inferred← citeshttps://doi.org/10.1002/mlf2.12079openalex-snapshot
A small, polyphyletic group of Firmicutes synthesizes trimethylamine from l ‐carnitine
inferred← citeshttps://doi.org/10.1002/mnfr.70031openalex-snapshot
Trimethylamine N‐Oxide Plasma Levels Following Red Meat and Cod Fish Intake: A Pilot Crossover Trial in Hemodialysis Patients
inferred← citeshttps://doi.org/10.1002/ptr.7684openalex-snapshot
Perilla frutescens L. alleviates trimethylamine N‐oxide –induced apoptosis in the renal tubule by regulating ASK1‐JNK phosphorylation
inferred← citeshttps://doi.org/10.1007/978-3-030-78177-4_15openalex-snapshot
Role of the Microbiome in Cardiovascular Disease
inferred← citeshttps://doi.org/10.1007/978-3-031-11942-2_4openalex-snapshot
Microbiome Derived Metabolites in CKD and ESRD
inferred← citeshttps://doi.org/10.1007/978-3-031-14952-8_14openalex-snapshot
Role of Renin Angiotensin System in the Pathophysiology of Coronary Heart Disease: Advancements in Diagnosis, Therapy and Preventive Strategies
inferred← citeshttps://doi.org/10.1007/s00204-026-04368-1openalex-snapshot
Trimethylamine N-oxide (TMAO) disrupts endothelial junction integrity through VE-cadherin Tyr658 phosphorylation in vitro
inferred← citeshttps://doi.org/10.1007/s00392-022-01992-6openalex-snapshot
Gut microbiota-dependent metabolite trimethylamine N-oxide (TMAO) and cardiovascular risk in patients with suspected functionally relevant coronary artery disease (fCAD)
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Can diet modulate trimethylamine N-oxide (TMAO) production? What do we know so far?
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Gut microbiota and chronic kidney disease: evidences and mechanisms that mediate a new communication in the gastrointestinal-renal axis
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Navigating the microbial maze: unraveling the connection between gut microbiome and pediatric kidney and urinary tract disease
inferred← citeshttps://doi.org/10.1007/s10157-015-1207-yopenalex-snapshot
Associations among serum trimethylamine-N-oxide (TMAO) levels, kidney function and infarcted coronary artery number in patients undergoing cardiovascular surgery: a cross-sectional study
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Gut microbiota and chronic kidney disease: implications for novel mechanistic insights and therapeutic strategies
inferred← citeshttps://doi.org/10.1007/s11255-019-02349-1openalex-snapshot
Selected cardiovascular risk factors in early stages of chronic kidney disease
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The role of gut-dependent molecule trimethylamine N-oxide as a novel target for the treatment of chronic kidney disease
inferred← citeshttps://doi.org/10.1007/s11255-024-04273-5openalex-snapshot
Is there a correlation between TMAO plasma levels and archaea in the gut of patients undergoing hemodialysis?
inferred← citeshttps://doi.org/10.1007/s11684-021-0857-2openalex-snapshot
FMO3-TMAO axis modulates the clinical outcome in chronic heart-failure patients with reduced ejection fraction: evidence from an Asian population
inferred← citeshttps://doi.org/10.1007/s11739-020-02470-5openalex-snapshot
TMAO as a biomarker of cardiovascular events: a systematic review and meta-analysis
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Gut Microbiota and Atherosclerosis
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Exploring the Microbiome in Heart Failure
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Cerebrovascular Disease, Cardiovascular Disease, and Chronic Kidney Disease: Interplays and Influences
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Targeting the Microbiome in Heart Failure
inferred← citeshttps://doi.org/10.1007/s12035-022-02990-5openalex-snapshot
The Role of a Gut Microbial-Derived Metabolite, Trimethylamine N-Oxide (TMAO), in Neurological Disorders
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Nutritional Assessment and Dietary Interventions in Older Patients with Heart Failure
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Trimethylamine N-Oxide and Risk of Cardiovascular Disease and Mortality
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Advances in Detection of Kidney Transplant Injury
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Drug Metabolism in Chronic Kidney Disease
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Implications of microbiota in the pathogenesis of diabetes mellitus and cardiovascular disease
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The beneficial role of healthy microbiome in metabolic syndrome and cardiovascular health
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Drug metabolism in kidney disease
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New Biomarkers to Evaluate Hyperandrogenemic Women and Hypogonadal Men
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Blood, urine and faecal metabolite profiles in the study of adult renal disease
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Plasma trimethylamine N-oxide (TMAO) levels predict future risk of coronary artery disease in apparently healthy individuals in the EPIC-Norfolk prospective population study
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Choline metabolites and incident cardiovascular disease in a prospective cohort of adults: Coronary Artery Risk Development in Young Adults (CARDIA) Study
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Gut microbiome alterations precede graft rejection in kidney transplantation patients
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Archaea from the gut microbiota of humans: Could be linked to chronic diseases?
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ACE2 imbalance as a key player for the poor outcomes in COVID-19 patients with age-related comorbidities – Role of gut microbiota dysbiosis
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Trimethylamine-N-oxide (TMAO) and the predicted risk of cardiovascular events after partial nephrectomy
inferred← citeshttps://doi.org/10.1016/j.atherosclerosis.2015.10.091openalex-snapshot
Plasma levels of trimethylamine-N-oxide are confounded by impaired kidney function and poor metabolic control
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Metabolic products of the intestinal microbiome and extremes of atherosclerosis
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Diabetic gut microbiota dysbiosis as an inflammaging and immunosenescence condition that fosters progression of retinopathy and nephropathy
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Increased circulating trimethylamine N-oxide contributes to endothelial dysfunction in a rat model of chronic kidney disease
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A novel real-time TMAO detection method based on microbial electrochemical technology
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The impact of gut microbiota on kidney function and pathogenesis
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Trimethylamine-N-oxide (TMAO) promotes balloon injury-induced neointimal hyperplasia via upregulating Beclin1 and impairing autophagic flux
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Renal fibrosis in type 2 cardiorenal syndrome: An update on mechanisms and therapeutic opportunities
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The gut microbiota derived metabolite trimethylamine N-oxide: Its important role in cancer and other diseases
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The mechanisms underlying acute myocardial infarction in chronic kidney disease patients undergoing hemodialysis
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Quantification of trimethylamine-N-oxide (TMAO) and its main related trimethylammonium-containing compounds in human plasma by LC-MS/MS
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A novel dual-function biosensor based on polarity reversal of MFC for bioelectricity generation and detecting trimethylamine-N-oxide
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Regulated Stochasticity in a Bacterial Signaling Network Permits Tolerance to a Rapid Environmental Change
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A Multi-omic Association Study of Trimethylamine N-Oxide
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Metabolomics: Perspectives on potential biomarkers in organ transplantation and immunosuppressant toxicity
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Association of trimethylamine N-oxide and metabolites with kidney function decline in patients with chronic kidney disease
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Meat intake in relation to composition and function of gut microbiota
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Relationship between serum trimethylamine N-oxide and exposure to dioxin-like pollutants
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Trimethylamine-N-oxide disrupts spermatogenesis by inducing mitochondrial oxidative stress injury through Hippo signaling
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Trends in gut-heart axis and heart failure research (1993–2023): A bibliometric and visual analysis
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Intra-hospital variation of gut microbiota product, trimethylamine N-oxide (TMAO), predicts future major adverse cardiovascular events after myocardial infarction
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A review on the beneficial and detrimental roles of gut microbiota-derived trimethylamine-N-oxide (TMAO)
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Loop Diuretics Inhibit Renal Excretion of Trimethylamine N-Oxide
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Long-Term Changes in Gut Microbial Metabolite Trimethylamine N-Oxide and Coronary Heart Disease Risk
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A simple dilute and shoot approach incorporated with pentafluorophenyl (PFP) column based LC-MS/MS assay for the simultaneous determination of trimethylamine N -oxide and trimethylamine in spot urine samples with high throughput
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Simultaneous quantitative analysis of uremic toxins by LC–MS/MS with a reversed-phase/cation-exchange/anion-exchange tri-modal mixed-mode column
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Biosensors for probing trimethylamine and trimethylamine N-oxide: State-of-the-art
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Trimethylamine-N-oxide promotes vascular aging via inhibiting GATA4 degradation in chronic kidney disease rats
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Gut microbial metabolite TMAO portends prognosis in acute ischemic stroke
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The metabolic fate of isotopically labeled trimethylamine-N-oxide (TMAO) in humans
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Proteomic and metabolomic approaches in the search for biomarkers in chronic kidney disease
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An overview of renal metabolomics
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Evaluation of the impact of gut microbiota on uremic solute accumulation by a CE-TOFMS–based metabolomics approach
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The role of trimethylamine N-oxide as a mediator of cardiovascular complications in chronic kidney disease
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Serum metabolites are associated with all-cause mortality in chronic kidney disease
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Interaction between gut microbiome and cardiovascular disease
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METTL14-dependent m6A regulates vascular calcification induced by indoxyl sulfate
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Trimethylamine N-oxide in cardiovascular disease: Pathophysiology and the potential role of statins
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Human risk of diseases associated with red meat intake: Analysis of current theories and proposed role for metabolic incorporation of a non-human sialic acid
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Is increased plasma TMAO a compensatory response to hydrostatic and osmotic stress in cardiovascular diseases?
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Impact of gut microbiota: How it could play roles beyond the digestive system on development of cardiovascular and renal diseases
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Gut microbial metabolites: The bridge connecting diet and atherosclerosis, and next-generation targets for dietary interventions
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Microbiota intestinal en la enfermedad renal crónica
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Gut microbiota in chronic kidney disease
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Trimethylamine N -oxide: A harmful, protective or diagnostic marker in lifestyle diseases?
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Gut microbiota-dependent trimethylamine N-oxide and all-cause mortality: Findings from an updated systematic review and meta-analysis
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Does intestinal dysbiosis contribute to an aberrant inflammatory response to severe acute respiratory syndrome coronavirus 2 in frail patients?
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Using herbal medicine to target the “microbiota-metabolism-immunity” axis as possible therapy for cardiovascular disease
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Pharmacological targeting macrophage phenotype via gut-kidney axis ameliorates renal fibrosis in mice
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Gut microbiome and metabolites, the future direction of diagnosis and treatment of atherosclerosis?
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Shizukaol C alleviates trimethylamine oxide-induced inflammation through activating Keap1-Nrf2-GSTpi pathway in vascular smooth muscle cell
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Trimethylamine N-Oxide increases soluble fms-like tyrosine Kinase-1 in human placenta via NADPH oxidase dependent ROS accumulation
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Gut microbiota dependent trimethylamine N-oxide aggravates angiotensin II–induced hypertension
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Contributory Role of Gut Microbiota and Their Metabolites Toward Cardiovascular Complications in Chronic Kidney Disease
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Gut Microbiota-Kidney Cross-Talk in Acute Kidney Injury
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Gut Microbiota-Targeted Interventions in the Management of Chronic Kidney Disease
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Trimethylamine-N-oxide: the microbial cue in immune-mediated disorders
inferred← citeshttps://doi.org/10.1016/j.thromres.2023.11.003openalex-snapshot
Trimethylamine-N-oxide (TMAO) and basic fibroblast growth factor (bFGF) are possibly involved in corticosteroid resistance in adult patients with immune thrombocytopenia
inferred← citeshttps://doi.org/10.1016/j.trsl.2016.04.007openalex-snapshot
Gut microbiome in chronic kidney disease: challenges and opportunities
inferred← citeshttps://doi.org/10.1016/j.trsl.2016.07.007openalex-snapshot
Microbiome, trimethylamine N-oxide, and cardiometabolic disease
inferred← citeshttps://doi.org/10.1016/j.trsl.2021.10.001openalex-snapshot
Gut microbial metabolite TMAO increases peritoneal inflammation and peritonitis risk in peritoneal dialysis patients
inferred← citeshttps://doi.org/10.1016/j.trsl.2026.04.003openalex-snapshot
Trimethylamine N-oxide exacerbates myocardial ischemia-reperfusion injury by sustaining PERK/eIF2α activation and impairing ferredoxin reductase-dependent mitochondrial function
inferred← citeshttps://doi.org/10.1017/s0954422420000177openalex-snapshot
Trimethylamine N -oxide: heart of the microbiota–CVD nexus?
inferred← citeshttps://doi.org/10.1021/acs.jproteome.1c00851openalex-snapshot
Balancing the Equation: A Natural History of Trimethylamine and Trimethylamine-N-oxide
inferred← citeshttps://doi.org/10.1021/acschemneuro.5c00414openalex-snapshot
Implications of Gut Microbiota-Derived Metabolites in Neurological Disorders
inferred← citeshttps://doi.org/10.1038/nrneph.2015.191openalex-snapshot
The gut microbiome, diet, and links to cardiometabolic and chronic disorders
inferred← citeshttps://doi.org/10.1038/s41419-022-04719-7openalex-snapshot
PRMT5 critically mediates TMAO-induced inflammatory response in vascular smooth muscle cells
inferred← citeshttps://doi.org/10.1038/s41430-022-01104-7openalex-snapshot
Significant correlation between the gut microbiota-derived metabolite trimethylamine-N-oxide and the risk of stroke: evidence based on 23 observational studies
inferred← citeshttps://doi.org/10.1038/s41467-023-39824-4openalex-snapshot
Evidence of a causal and modifiable relationship between kidney function and circulating trimethylamine N-oxide
inferred← citeshttps://doi.org/10.1038/s41569-018-0108-7openalex-snapshot
Dietary metabolism, the gut microbiome, and heart failure
inferred← citeshttps://doi.org/10.1038/s41581-021-00408-4openalex-snapshot
Uraemic solutes as therapeutic targets in CKD-associated cardiovascular disease
inferred← citeshttps://doi.org/10.1038/s41598-017-01387-yopenalex-snapshot
Impaired renal function and dysbiosis of gut microbiota contribute to increased trimethylamine-N-oxide in chronic kidney disease patients
inferred← citeshttps://doi.org/10.1038/s41598-017-13739-9openalex-snapshot
TMAO is Associated with Mortality: Impact of Modestly Impaired Renal Function
inferred← citeshttps://doi.org/10.1038/s41598-019-52032-9openalex-snapshot
Metabolic Activation of Flavin Monooxygenase-mediated Trimethylamine-N-Oxide Formation in Experimental Kidney Disease
inferred← citeshttps://doi.org/10.1038/s41598-019-52082-zopenalex-snapshot
Trimethylamine-N-Oxide (TMAO) Predicts Cardiovascular Mortality in Peripheral Artery Disease
inferred← citeshttps://doi.org/10.1038/s41598-020-76346-1openalex-snapshot
Insights in the regulation of trimetylamine N-oxide production using a comparative biomimetic approach suggest a metabolic switch in hibernating bears
inferred← citeshttps://doi.org/10.1038/s41598-020-80063-0openalex-snapshot
Inhibition of microbiota-dependent TMAO production attenuates chronic kidney disease in mice
inferred← citeshttps://doi.org/10.1038/s41598-021-99891-9openalex-snapshot
Ventricular arrhythmias in mouse models of diabetic kidney disease
inferred← citeshttps://doi.org/10.1038/s41598-022-08387-7openalex-snapshot
Blood–brain barrier and gut barrier dysfunction in chronic kidney disease with a focus on circulating biomarkers and tight junction proteins
inferred← citeshttps://doi.org/10.1038/s41598-022-14152-7openalex-snapshot
Utility of SIFT-MS to evaluate volatile organic compounds in nephropathic patients’ breath
inferred← citeshttps://doi.org/10.1038/s42003-024-06718-0openalex-snapshot
Deficiency of flavin-containing monooxygenase 3 protects kidney function after ischemia–reperfusion in mice
inferred← citeshttps://doi.org/10.1038/srep26745openalex-snapshot
Relationship of Serum Trimethylamine N-Oxide (TMAO) Levels with early Atherosclerosis in Humans
inferred← citeshttps://doi.org/10.1038/srep30958openalex-snapshot
Metabolomics Profiling for Obstructive Sleep Apnea and Simple Snorers
inferred← citeshttps://doi.org/10.1039/d0fo01743dopenalex-snapshot
Systematic investigation of the relationships of trimethylamine N -oxide and l -carnitine with obesity in both humans and rodents
inferred← citeshttps://doi.org/10.1042/bsr20190515openalex-snapshot
Serum levels of Trimethylamine-N-oxide in patients with ischemic stroke
inferred← citeshttps://doi.org/10.1042/bsr20221102openalex-snapshot
Two-component carnitine monooxygenase from Escherichia coli: functional characterization, inhibition and mutagenesis of the molecular interface
inferred← citeshttps://doi.org/10.1042/cs20171090openalex-snapshot
Gut microbiota in cardiovascular disease and heart failure
inferred← citeshttps://doi.org/10.1042/cs20171107openalex-snapshot
Altered microbiome in chronic kidney disease: systemic effects of gut-derived uremic toxins
inferred← citeshttps://doi.org/10.1042/cs20190171openalex-snapshot
Quantitative reduction in short-chain fatty acids, especially butyrate, contributes to the progression of chronic kidney disease
inferred← citeshttps://doi.org/10.1053/j.ajkd.2022.02.016openalex-snapshot
Association of Uremic Solutes With Cardiovascular Death in Diabetic Kidney Disease
inferred← citeshttps://doi.org/10.1053/j.jrn.2018.05.007openalex-snapshot
Moderate Renal Impairment and Toxic Metabolites Produced by the Intestinal Microbiome: Dietary Implications
inferred← citeshttps://doi.org/10.1053/j.jrn.2020.04.007openalex-snapshot
Plant-Based Diets, the Gut Microbiota, and Trimethylamine N-Oxide Production in Chronic Kidney Disease: Therapeutic Potential and Methodological Considerations
inferred← citeshttps://doi.org/10.1053/j.jrn.2023.06.011openalex-snapshot
Gut Microbiota and Uremic Retention Solutes in Adults With Moderate CKD: A 6-Day Controlled Feeding Study
inferred← citeshttps://doi.org/10.1074/jbc.r116.765388openalex-snapshot
Targeting of microbe-derived metabolites to improve human health: The next frontier for drug discovery
inferred← citeshttps://doi.org/10.1080/07391102.2022.2053744openalex-snapshot
Urea ameliorates trimethylamine N-oxide-Induced aggregation of intrinsically disordered α-casein protein: the other side of the urea-methylamine counteraction
inferred← citeshttps://doi.org/10.1080/07853890.2023.2215542openalex-snapshot
Gut microbiota-derived trimethylamine N-oxide is associated with the risk of all-cause and cardiovascular mortality in patients with chronic kidney disease: a systematic review and dose-response meta-analysis
inferred← citeshttps://doi.org/10.1080/07853890.2025.2525403openalex-snapshot
Trimethylamine- N -oxide formation, the bacterial taxa involved and intervention strategies to reduce its concentration in the human body
inferred← citeshttps://doi.org/10.1080/0886022x.2020.1781170openalex-snapshot
Trimethylamine- N -oxide is an independent risk factor for hospitalization events in patients receiving maintenance hemodialysis
inferred← citeshttps://doi.org/10.1080/0886022x.2020.1822868openalex-snapshot
Association of trimethylamine N -Oxide with cardiovascular and all-cause mortality in hemodialysis patients
inferred← citeshttps://doi.org/10.1080/0886022x.2021.1890618openalex-snapshot
Trimethylamine N-Oxide and mortality in hemodialysis patients: like a mystery
inferred← citeshttps://doi.org/10.1080/0886022x.2022.2056054openalex-snapshot
Intestinal microbiota dysbiosis in acute kidney injury: novel insights into mechanisms and promising therapeutic strategies
inferred← citeshttps://doi.org/10.1080/0886022x.2022.2131572openalex-snapshot
Correlation between serum trimethylamine- N -oxide concentration and protein energy wasting in patients on maintenance hemodialysis
inferred← citeshttps://doi.org/10.1080/0886022x.2023.2165446openalex-snapshot
Serum trimethylamine- N -oxide and protein energy wasting: some factors that should be considered
inferred← citeshttps://doi.org/10.1080/0886022x.2024.2435485openalex-snapshot
Research progress on the association between TMAO and vascular calcification in patients with chronic kidney disease
inferred← citeshttps://doi.org/10.1080/0886022x.2025.2481202openalex-snapshot
The association of plasma TMAO and body composition with the occurrence of PEW in maintenance hemodialysis patients
inferred← citeshttps://doi.org/10.1080/0886022x.2025.2575434openalex-snapshot
Gut microbiota-derived TMAO drives the kidney-bone-vascular axis in chronic kidney disease complications
inferred← citeshttps://doi.org/10.1080/09637486.2016.1256379openalex-snapshot
Dietary, anthropometric, and biochemical factors influencing plasma choline, carnitine, trimethylamine, and trimethylamine-N-oxide concentrations
inferred← citeshttps://doi.org/10.1080/09637486.2017.1318117openalex-snapshot
Ratio of serum levels of AGEs to soluble RAGE is correlated with trimethylamine-N-oxide in non-diabetic subjects
inferred← citeshttps://doi.org/10.1080/10408398.2020.1761771openalex-snapshot
Intestinal microbiota alterations in chronic kidney disease and the influence of dietary components
inferred← citeshttps://doi.org/10.1080/10408398.2020.1770199openalex-snapshot
Novel findings of the association between gut microbiota–derived metabolite trimethylamine N-oxide and inflammation: results from a systematic review and dose-response meta-analysis
inferred← citeshttps://doi.org/10.1080/17425255.2022.2151892openalex-snapshot
Examination of the emerging role of transporters in the assessment of nephrotoxicity
inferred← citeshttps://doi.org/10.1080/17512433.2018.1378095openalex-snapshot
Microbiota-derived uremic retention solutes: perpetrators of altered nonrenal drug clearance in kidney disease
inferred← citeshttps://doi.org/10.1080/19490976.2026.2657048openalex-snapshot
Alteration of gut microbiota contributes to peritoneal fibrosis through increased production of trimethylamine N-oxide
inferred← citeshttps://doi.org/10.1080/19490976.2026.2718621openalex-snapshot
Microbial metabolite–driven mechanisms linking the gut microbiome to atherosclerosis: multi-omic and translational perspectives
inferred← citeshttps://doi.org/10.1080/21691401.2019.1652187openalex-snapshot
Silencing circular ANRIL protects HK-2 cells from lipopolysaccharide-induced inflammatory injury through up-regulating microRNA-9
inferred← citeshttps://doi.org/10.1093/advances/nmab010openalex-snapshot
Gut Microbiota-Derived Trimethylamine N-Oxide and Kidney Function: A Systematic Review and Meta-Analysis
inferred← citeshttps://doi.org/10.1093/advances/nmy068openalex-snapshot
Modulation of the Gut Microbiota by Resistant Starch as a Treatment of Chronic Kidney Diseases: Evidence of Efficacy and Mechanistic Insights
inferred← citeshttps://doi.org/10.1093/ajcn/nqaa015openalex-snapshot
Associations of plasma trimethylamine N-oxide, choline, carnitine, and betaine with inflammatory and cardiometabolic risk biomarkers and the fecal microbiome in the Multiethnic Cohort Adiposity Phenotype Study
inferred← citeshttps://doi.org/10.1093/ajcn/nqaa430openalex-snapshot
Circulating trimethylamine N-oxide in association with diet and cardiometabolic biomarkers: an international pooled analysis
inferred← citeshttps://doi.org/10.1093/ajcn/nqab001openalex-snapshot
Dietary factors, gut microbiota, and serum trimethylamine-N-oxide associated with cardiovascular disease in the Hispanic Community Health Study/Study of Latinos
inferred← citeshttps://doi.org/10.1093/ajcn/nqac074openalex-snapshot
Gut microbiota–derived metabolite trimethylamine-N-oxide and multiple health outcomes: an umbrella review and updated meta-analysis
inferred← citeshttps://doi.org/10.1093/ajcn/nqac278openalex-snapshot
The association between TMAO, CMPF, and clinical outcomes in advanced chronic kidney disease: results from the European QUALity (EQUAL) Study
inferred← citeshttps://doi.org/10.1093/ajcn/nqy116openalex-snapshot
Plasma concentration of trimethylamine-N-oxide and risk of gestational diabetes mellitus
inferred← citeshttps://doi.org/10.1093/cdn/nzaa179openalex-snapshot
Trimethylamine N-oxide (TMAO) Is not Associated with Cardiometabolic Phenotypes and Inflammatory Markers in Children and Adults
inferred← citeshttps://doi.org/10.1093/ckj/sfac113openalex-snapshot
From cardiorenal syndromes to cardionephrology: a reflection by nephrologists on renocardiac syndromes
inferred← citeshttps://doi.org/10.1093/ckj/sfad095openalex-snapshot
The dietary source of trimethylamine N-oxide and clinical outcomes: an unexpected liaison
inferred← citeshttps://doi.org/10.1093/ckj/sfad303openalex-snapshot
Gut-immune axis and cardiovascular risk in chronic kidney disease
inferred← citeshttps://doi.org/10.1093/ckj/sfag112openalex-snapshot
Elevated serum TMAO levels associate with higher risk of arteriovenous vascular access thrombosis in hemodialysis patients
inferred← citeshttps://doi.org/10.1093/ckj/sfy037openalex-snapshot
The metabolomic quest for a biomarker in chronic kidney disease
inferred← citeshttps://doi.org/10.1093/cvr/cvab263openalex-snapshot
Vascular endothelial tissue factor contributes to trimethylamine N-oxide-enhanced arterial thrombosis
inferred← citeshttps://doi.org/10.1093/ehjopen/oeac021openalex-snapshot
High-circulating gut microbiota-dependent metabolite trimethylamine N-oxide is associated with poor prognosis in pulmonary arterial hypertension
inferred← citeshttps://doi.org/10.1093/esj/23969873251366192openalex-snapshot
Trimethylamine N-oxide (TMAO) for risk stratification after acute ischemic stroke: Results from the BIOSIGNAL cohort study
inferred← citeshttps://doi.org/10.1093/eurheartj/ehad089openalex-snapshot
Trimethylamine N-oxide is associated with long-term mortality risk: the multi-ethnic study of atherosclerosis
inferred← citeshttps://doi.org/10.1093/eurheartj/ehw582openalex-snapshot
Gut microbiota-dependent trimethylamine N-oxide in acute coronary syndromes: a prognostic marker for incident cardiovascular events beyond traditional risk factors
inferred← citeshttps://doi.org/10.1093/eurheartj/ehx342openalex-snapshot
Gut microbe-generated metabolite trimethylamine-N-oxide as cardiovascular risk biomarker: a systematic review and dose-response meta-analysis
inferred← citeshttps://doi.org/10.1093/ndt/gfaa335openalex-snapshot
Effect of fructooligosaccharide on endothelial function in CKD patients: a randomized controlled trial
inferred← citeshttps://doi.org/10.1093/ndt/gfae220openalex-snapshot
Dietary protein intake and the tubular handling of indoxyl sulfate
inferred← citeshttps://doi.org/10.1093/ndt/gfaf071openalex-snapshot
Plasma trimethylamine N-oxide concentration and all-cause mortality in kidney transplant recipients
inferred← citeshttps://doi.org/10.1093/ndt/gfw009openalex-snapshot
The influence of renal transplantation on retained microbial–human co-metabolites
inferred← citeshttps://doi.org/10.1093/nutrit/nuaa111openalex-snapshot
Gut microbiota–associated trimethylamineN-oxide and increased cardiometabolic risk in adults: a systematic review and dose-response meta-analysis
inferred← citeshttps://doi.org/10.1093/nutrit/nuad048openalex-snapshot
Blueberry, cranberry, raspberry, and strawberry as modulators of the gut microbiota: target for treatment of gut dysbiosis in chronic kidney disease? From current evidence to future possibilities
inferred← citeshttps://doi.org/10.1097/cm9.0000000000002206openalex-snapshot
Gut microbiota and microbiota-derived metabolites in cardiovascular diseases
inferred← citeshttps://doi.org/10.1097/fjc.0000000000001387openalex-snapshot
Intestinal Flora Metabolite Trimethylamine Oxide Is Inextricably Linked to Coronary Heart Disease
inferred← citeshttps://doi.org/10.1097/md.0000000000037825openalex-snapshot
Gut microbiota metabolites and risk of major adverse cardiovascular events and death: A systematic review and meta-analysis
inferred← citeshttps://doi.org/10.1097/mnh.0000000000000661openalex-snapshot
Microbiome modulation as a novel therapeutic approach in chronic kidney disease
inferred← citeshttps://doi.org/10.1097/mol.0000000000000357openalex-snapshot
Human microbiome as therapeutic intervention target to reduce cardiovascular disease risk
inferred← citeshttps://doi.org/10.1097/qad.0000000000001813openalex-snapshot
The microbial metabolite trimethylamine-N-oxide in association with inflammation and microbial dysregulation in three HIV cohorts at various disease stages
inferred← citeshttps://doi.org/10.1101/2025.10.03.680371openalex-snapshot
Age-related microbiome metabolites alter RNA splicing and chromatin accessibility in the brain
inferred← citeshttps://doi.org/10.1109/jsen.2024.3368409openalex-snapshot
An Off-Chip Capacitance Biosensor Based on Improved Cole–Cole Model for the Detection of Trimethylamine N-Oxide in Early Cardiovascular Disease
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