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MicroRNA-709 Mediates Acute Tubular Injury through Effects on Mitochondrial Function

https://doi.org/10.1681/asn.2017040381

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Mitochondrial dysregulation and protection in cisplatin nephrotoxicity

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Augmenter of liver regeneration plays a protective role against hydrogen peroxide-induced oxidative stress in renal proximal tubule cells

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TNF-α regulates miRNA targeting mitochondrial complex-I and induces cell death in dopaminergic cells

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The role of TFAM-associated proteins in mitochondrial RNA metabolism

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miR-483-5p determines mitochondrial fission and cisplatin sensitivity in tongue squamous cell carcinoma by targeting FIS1

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Human RISC Couples MicroRNA Biogenesis and Posttranscriptional Gene Silencing

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miR-709 inhibits 3T3-L1 cell differentiation by targeting GSK3β of Wnt/β-catenin signaling

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miR-709 modulates LPS-induced inflammatory response through targeting GSK-3β

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microRNA regulation of endothelin-1 mRNA in renal collecting duct cells

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Murine microRNAs implicated in liver functions and aging process

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Mitochondrial Function and Disturbances in the Septic Kidney

inferredcites →https://doi.org/10.1016/j.ymgme.2016.07.001openalex-snapshot

Mutations in TFAM, encoding mitochondrial transcription factor A, cause neonatal liver failure associated with mtDNA depletion

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Mitochondria-targeted therapies for acute kidney injury

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Activation of peroxisome proliferator-activated receptor-γ coactivator 1α ameliorates mitochondrial dysfunction and protects podocytes from aldosterone-induced injury

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A microRNA-30e/mitochondrial uncoupling protein 2 axis mediates TGF-β1-induced tubular epithelial cell extracellular matrix production and kidney fibrosis

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Section 3: Prevention and Treatment of AKI

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MicroRNAs act sequentially and asymmetrically to control chemosensory laterality in the nematode

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Many roads to maturity: microRNA biogenesis pathways and their regulation

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Mitochondrial transcription factor A is necessary for mtDNA maintance and embryogenesis in mice

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Acute kidney injury—epidemiology, outcomes and economics

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Gene targets of mouse miR-709: regulation of distinct pools

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The Banff 97 working classification of renal allograft pathology

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Cross-strand binding of TFAM to a single mtDNA molecule forms the mitochondrial nucleoid

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Protein Kinase C-α and ERK1/2 Mediate Mitochondrial Dysfunction, Decreases in Active Na+ Transport, and Cisplatin-induced Apoptosis in Renal Cells

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Predictors of Mortality in Acute Renal Failure in a Developing Country: A Prospective Study

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Therapeutic targeting of the mitochondrial dysfunction in septic acute kidney injury

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Mitochondria as therapeutic targets in acute kidney injury

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RETRACTED: miR‐709 up‐regulated in hepatocellular carcinoma, promotes proliferation and invasion by targeting GPC 5

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Serum miRNA‐122 in acute liver injury induced by kidney injury and sepsis in CD‐1 mouse models

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Banff 07 Classification of Renal Allograft Pathology: Updates and Future Directions

inferredcites →https://doi.org/10.1111/jcmm.12420openalex-snapshot

Mi RNA ‐30a inhibits AEC s‐II apoptosis by blocking mitochondrial fission dependent on Drp‐1

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Architectural Role of Mitochondrial Transcription Factor A in Maintenance of Human Mitochondrial DNA

inferredcites →https://doi.org/10.1136/bmjopen-2014-006497openalex-snapshot

Long-term prognosis after acute kidney injury (AKI): what is the role of baseline kidney function and recovery? A systematic review

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Activation and involvement of p53 in cisplatin-induced nephrotoxicity

inferredcites →https://doi.org/10.1152/ajprenal.00480.2006openalex-snapshot

Aldosterone induces epithelial-mesenchymal transition via ROS of mitochondrial origin

inferredcites →https://doi.org/10.1172/jci111524openalex-snapshot

Oxygen free radicals in ischemic acute renal failure in the rat.

inferredcites →https://doi.org/10.1172/jci58849openalex-snapshot

MicroRNA-30e* promotes human glioma cell invasiveness in an orthotopic xenotransplantation model by disrupting the NF-κB/IκBα negative feedback loop

inferredcites →https://doi.org/10.1172/jci75852openalex-snapshot

Anti–microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathways

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A simple array platform for microRNA analysis and its application in mouse tissues

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Cobalt Protoporphyrin Accelerates TFEB Activation and Lysosome Reformation during LPS-Induced Septic Insults in the Rat Heart

inferredcites →https://doi.org/10.1371/journal.pone.0081162openalex-snapshot

Cisplatin Induces a Mitochondrial-ROS Response That Contributes to Cytotoxicity Depending on Mitochondrial Redox Status and Bioenergetic Functions

inferredcites →https://doi.org/10.1371/journal.pone.0119687openalex-snapshot

Overexpression of TFAM or Twinkle Increases mtDNA Copy Number and Facilitates Cardioprotection Associated with Limited Mitochondrial Oxidative Stress

inferredcites →https://doi.org/10.1681/asn.2010040367openalex-snapshot

miR-335 and miR-34a Promote Renal Senescence by Suppressing Mitochondrial Antioxidative Enzymes

inferredcites →https://doi.org/10.1681/asn.2015020177openalex-snapshot

Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI

inferredcites →https://doi.org/10.1681/asn.2015040376openalex-snapshot

Role of Mitochondrial DNA in Septic AKI via Toll-Like Receptor 9

inferredcites →https://doi.org/10.1681/asn.2016070761openalex-snapshot

Mitochondria Protection after Acute Ischemia Prevents Prolonged Upregulation of IL-1β and IL-18 and Arrests CKD

inferredcites →https://doi.org/10.1681/asn.v134858openalex-snapshot

Cisplatin Induces Apoptosis in LLC-PK1 Cells via Activation of Mitochondrial Pathways

inferredcites →https://doi.org/10.3390/ijms17050662openalex-snapshot

Autophagy, Innate Immunity and Tissue Repair in Acute Kidney Injury

inferredcites →https://doi.org/10.4081/jnai.2011.e5openalex-snapshot

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inferredcites →https://doi.org/10.4161/cc.7.23.7186openalex-snapshot

DNA damage-induced upregulation of miR-709 in the germline downregulates BORIS to counteract aberrant DNA hypomethylation

inferredcites →https://doi.org/10.5935/0101-2800.20130052openalex-snapshot

Acute nephrotoxicity of cisplatin: Molecular mechanisms

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Exosomes Highlight Future Directions in the Treatment of Acute Kidney Injury

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Epigenetic Mechanisms Involved in Cisplatin-Induced Nephrotoxicity: An Update

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Parametric MRI Detects Aristolochic Acid Induced Acute Kidney Injury

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Faculty Opinions recommendation of Extracellular vesicle-encapsulated IL-10 as novel nanotherapeutics against ischemic AKI.

inferred← citeshttps://doi.org/10.3892/ijmm.2021.5002openalex-snapshot

Antagonist targeting miR‑106b‑5p attenuates acute renal injury by regulating renal function, apoptosis and autophagy via the upregulation of TCF4

inferred← citeshttps://doi.org/10.3892/ijmm.2022.5087openalex-snapshot

Identification of inflammatory response and alternative splicing in acute kidney injury and experimental verification of the involvement of RNA‑binding protein RBFOX1 in this disease

inferred← citeshttps://doi.org/10.3892/mmr.2019.10383openalex-snapshot

miRNA‑mRNA regulatory network analysis of mesenchymal stem cell treatment in cisplatin‑induced acute kidney injury identifies roles for miR‑210/Serpine1 and miR‑378/Fos in regulating inflammation

inferred← citeshttps://doi.org/10.3892/mmr.2021.12305openalex-snapshot

MicroRNA‑93 inhibits the apoptosis and inflammatory response of tubular epithelial cells via the PTEN/AKT/mTOR pathway in acute kidney injury

inferred← citeshttps://doi.org/10.3892/mmr.2025.13466openalex-snapshot

The key role of miR‑378 in kidney diseases (Review)

inferred← citeshttps://doi.org/10.47749/t/unicamp.2022.1249456openalex-snapshot

MicroRNAs como possíveis biomarcadores de nefrotoxicidade induzida pela cisplatina em pacientes com câncer de cabeça e pescoço

inferred← citeshttps://doi.org/10.6084/m9.figshare.20004701openalex-snapshot

Additional file 1 of Identification of biomarkers, immune infiltration landscape, and treatment targets of ischemia–reperfusion acute kidney injury at an early stage by bioinformatics methods

inferred← citeshttps://doi.org/10.6084/m9.figshare.20004704openalex-snapshot

Additional file 2 of Identification of biomarkers, immune infiltration landscape, and treatment targets of ischemia–reperfusion acute kidney injury at an early stage by bioinformatics methods

inferred← citeshttps://doi.org/10.7150/ijbs.104335openalex-snapshot

TEAD1 Prevents Necroptosis and Inflammation in Cisplatin-Induced Acute Kidney Injury Through Maintaining Mitochondrial Function

inferred← citeshttps://doi.org/10.7150/thno.44051openalex-snapshot

Activation of TFEB-mediated autophagy by trehalose attenuates mitochondrial dysfunction in cisplatin-induced acute kidney injury

inferred← citeshttps://doi.org/10.7150/thno.71064openalex-snapshot

Emerging early diagnostic methods for acute kidney injury

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