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Galectin-3 induces vascular smooth muscle cells calcification via AMPK/TXNIP pathway

https://doi.org/10.18632/aging.204130

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

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Toxicity of oxidized low density lipoproteins for vascular smooth muscle cells and partial protection by antioxidants

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Thioredoxin interacting protein genetic variation is associated with diabetes and hypertension in the Brazilian general population

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Txnip ablation reduces vascular smooth muscle cell inflammation and ameliorates atherosclerosis in apolipoprotein E knockout mice

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rhEPO inhibited cell apoptosis to alleviate acute kidney injury in sepsis by AMPK/SIRT1 activated autophagy

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AMPK signalling in health and disease

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Ursolic acid exhibits anti-inflammatory effects through blocking TLR4-MyD88 pathway mediated by autophagy

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Diminished AMPK signaling response to fasting in thioredoxin-interacting protein knockout mice

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C-reactive protein induces expression of matrix metalloproteinase-9: A possible link between inflammation and plaque rupture

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Ghrelin improves vascular autophagy in rats with vascular calcification

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TXNIP deficiency mitigates podocyte apoptosis via restraining the activation of mTOR or p38 MAPK signaling in diabetic nephropathy

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HOXA9-induced chemerin signals through CMKLR1/AMPK/TXNIP/NLRP3 pathway to induce pyroptosis of trophoblasts and aggravate preeclampsia

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Thioredoxin interacting protein (TXNIP) rs7212 polymorphism is associated with arterial stiffness in the Brazilian general population

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AMPK regulates NADPH homeostasis to promote tumour cell survival during energy stress

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The structural basis for the negative regulation of thioredoxin by thioredoxin-interacting protein

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Exendin-4 Protects against Hyperglycemia-Induced Cardiomyocyte Pyroptosis via the AMPK-TXNIP Pathway

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NLRP3 Inflammasome and the IL-1 Pathway in Atherosclerosis

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Dietary potassium regulates vascular calcification and arterial stiffness

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Role of ASM/Cer/TXNIP signaling module in the NLRP3 inflammasome activation

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Multiple functions of autophagy in vascular calcification

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Resveratrol Induces Vascular Smooth Muscle Cell Differentiation through Stimulation of SirT1 and AMPK

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Exosomes Derived From Mesenchymal Stromal Cells Pretreated With Advanced Glycation End Product-Bovine Serum Albumin Inhibit Calcification of Vascular Smooth Muscle Cells

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Thioredoxin/Txnip: Redoxisome, as a Redox Switch for the Pathogenesis of Diseases

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Galectin-3-induced oxidized low-density lipoprotein promotes the phenotypic transformation of vascular smooth muscle cells

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Galectin-3 induces the phenotype transformation of human vascular smooth muscle cells via the canonical Wnt signaling

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

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Virgin and Photo-Degraded Microplastics Induce the Activation of Vascular Smooth Muscle Cells

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Role of galectin-3 in vascular calcification

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Excessive accumulation of thioredoxin promotes osteogenic differentiation of mesenchymal stem cells of ligament in ankylosing spondylitis by upregulating autophagy levels

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L-type calcium ion channel-mediated activation of autophagy in vascular smooth muscle cells via thonningianin A (TA) alleviates vascular calcification in type 2 diabetes mellitus

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A novel application perspective of the clinical-used drug verapamil on osteoporosis via targeting Txnip

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Pleiotropic regulatory mechanisms and targeted therapeutic prospects of Galectin-3 in aging-related diseases

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Myocardin related transcription factor and galectin-3 drive lipid accumulation in human blood vessels

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Smooth muscle cell-specific deletion of TXNIP ameliorates medial vascular calcification

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Virgin and photo-degraded microplastics induce the activation of human vascular smooth muscle cells

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TXNIP Suppresses the Osteochondrogenic Switch of Vascular Smooth Muscle Cells in Atherosclerosis

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Understanding Galectin-3’s Role in Diastolic Dysfunction: A Contemporary Perspective

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