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Biological role of miR-204 and miR-211 in melanoma

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Inferred edges come from two machine sources: sweeps of open repository metadata (mostly SSRN and Zenodo), and a bulk OpenAlex citation snapshot. Every row below is labelled with the one it came from. Neither is a complete citation index — absence here is not evidence of absence.

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What this work says

Outgoing claims: the papers this work cites, builds on, or argues with.

inferredcites →https://doi.org/10.1016/j.ccr.2013.02.003openalex-snapshot
Oncogenic BRAF Regulates Oxidative Metabolism via PGC1α and MITF
inferredcites →https://doi.org/10.1038/ncomms14343openalex-snapshot
Microenvironment-derived factors driving metastatic plasticity in melanoma
inferredcites →https://doi.org/10.1111/pcmr.12379openalex-snapshot
miR‐200c/Bmi1 axis and epithelial–mesenchymal transition contribute to acquired resistance to BRAF inhibitor treatment
inferredcites →https://doi.org/10.1158/0008-5472.can-17-1318openalex-snapshot
miR-204-5p and miR-211-5p Contribute to BRAF Inhibitor Resistance in Melanoma
inferredcites →https://doi.org/10.1186/s12943-018-0819-8openalex-snapshot
Loss of miR-204 expression is a key event in melanoma
inferredcites →https://doi.org/10.18632/oncotarget.10669openalex-snapshot
miR-7 reverses the resistance to BRAFi in melanoma by targeting EGFR/IGF-1R/CRAF and inhibiting the MAPK and PI3K/AKT signaling pathways
inferredcites →https://doi.org/10.18632/oncotarget.15915openalex-snapshot
Context-dependent miR-204 and miR-211 affect the biological properties of amelanotic and melanotic melanoma cells

What points at this work

Incoming claims: where this work appears in other papers’ reference lists and arguments.

inferred← citeshttps://doi.org/10.2139/ssrn.4129851openalex-extracted
inferred← citeshttps://doi.org/10.1007/s00210-024-03366-3openalex-snapshot
Non-coding RNAs in BRAF-mutant melanoma: targets, indicators, and therapeutic potential
inferred← citeshttps://doi.org/10.1016/j.jid.2019.06.126openalex-snapshot
MicroRNA Ratios Distinguish Melanomas from Nevi
inferred← citeshttps://doi.org/10.1016/j.pharmthera.2022.108301openalex-snapshot
Targeting the Epigenome in Malignant Melanoma: Facts, Challenges and Therapeutic Promises
inferred← citeshttps://doi.org/10.1016/j.prp.2023.154991openalex-snapshot
MALAT1: A key regulator in lung cancer pathogenesis and therapeutic targeting
inferred← citeshttps://doi.org/10.1038/s41419-019-2090-3openalex-snapshot
Long noncoding RNA LINC00518 acts as a competing endogenous RNA to promote the metastasis of malignant melanoma via miR-204-5p/AP1S2 axis
inferred← citeshttps://doi.org/10.1111/jcmm.14881openalex-snapshot
Long non‐coding RNA F11‐AS1 inhibits HBV‐related hepatocellular carcinoma progression by regulating NR1I3 via binding to microRNA‐211‐5p
inferred← citeshttps://doi.org/10.1155/tbj/2122220openalex-snapshot
Lentinula edodes Cultured Extract Intake at Puberty Mitigates Inflammatory Signals at the Mammary Glands by the Involvement of Epigenetic Mechanisms in BALB/c Mice
inferred← citeshttps://doi.org/10.1186/s12935-022-02738-0openalex-snapshot
Current understanding of epigenetics role in melanoma treatment and resistance
inferred← citeshttps://doi.org/10.1186/s12943-026-02603-yopenalex-snapshot
miRNA-driven cancer cell plasticity, tolerance and therapy resistance: lessons from melanoma
inferred← citeshttps://doi.org/10.1186/s43556-026-00425-4openalex-snapshot
BRAF inhibitor resistance in melanoma: from resistance mechanisms to therapeutic innovations
inferred← citeshttps://doi.org/10.1242/bio.053785openalex-snapshot
Inducible modulation of miR-204 levels in a zebrafish melanoma model
inferred← citeshttps://doi.org/10.14302/issn.2572-3030.jcgb-24-4970openalex-snapshot
Dynamic MicroRNA-Expression In Plasma of Melanoma Patients Correlates With Progression, PD-L1 Status and Overall Survival
inferred← citeshttps://doi.org/10.18632/oncoscience.473openalex-snapshot
Reply to: Biological role of miR-204 and miR-211 in melanoma (published in Oncoscience, Vol 5 (7-8), July 2018)
inferred← citeshttps://doi.org/10.3390/biomedicines12081851openalex-snapshot
Advances in Melanoma: From Genetic Insights to Therapeutic Innovations
inferred← citeshttps://doi.org/10.3390/cancers12113378openalex-snapshot
The Non-Coding Landscape of Cutaneous Malignant Melanoma: A Possible Route to Efficient Targeted Therapy
inferred← citeshttps://doi.org/10.3390/cancers14235858openalex-snapshot
Epigenetic Mechanisms Underlying Melanoma Resistance to Immune and Targeted Therapies
inferred← citeshttps://doi.org/10.3390/cancers15030894openalex-snapshot
A Network of MicroRNAs and mRNAs Involved in Melanosome Maturation and Trafficking Defines the Lower Response of Pigmentable Melanoma Cells to Targeted Therapy
inferred← citeshttps://doi.org/10.3390/ijms21030878openalex-snapshot
The miRNAs Role in Melanoma and in Its Resistance to Therapy
inferred← citeshttps://doi.org/10.3390/ijms242115881openalex-snapshot
Cutaneous Melanoma: A Review of Multifactorial Pathogenesis, Immunohistochemistry, and Emerging Biomarkers for Early Detection and Management
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