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Prognostic value of MET protein overexpression and gene amplification in locoregionally advanced nasopharyngeal carcinoma
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
inferredcites →https://doi.org/10.1002/cncr.28454openalex-snapshot
MET amplification is not rare and predicts unfavorable clinical outcomes in patients with recurrent/metastatic gastric cancer after chemotherapy
inferredcites →https://doi.org/10.1002/path.4268openalex-snapshot
Developing biomarkers to predict benefit from HGF / MET pathway inhibitors
inferredcites →https://doi.org/10.1016/j.ccr.2011.11.005openalex-snapshot
Mosaic Amplification of Multiple Receptor Tyrosine Kinase Genes in Glioblastoma
inferredcites →https://doi.org/10.1016/j.cllc.2012.05.009openalex-snapshot
Treatment Rationale Study Design for the MetLung Trial: A Randomized, Double-Blind Phase III Study of Onartuzumab (MetMAb) in Combination With Erlotinib Versus Erlotinib Alone in Patients Who Have Received Standard Chemotherapy for Stage IIIB or IV Met-Positive Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1016/j.ijrobp.2005.06.037openalex-snapshot
Chemotherapy in locally advanced nasopharyngeal carcinoma: An individual patient data meta-analysis of eight randomized trials and 1753 patients
inferredcites →https://doi.org/10.1016/j.ijrobp.2010.03.024openalex-snapshot
How Does Intensity-Modulated Radiotherapy Versus Conventional Two-Dimensional Radiotherapy Influence the Treatment Results in Nasopharyngeal Carcinoma Patients?
inferredcites →https://doi.org/10.1016/j.semradonc.2012.03.008openalex-snapshot
Current Management of Nasopharyngeal Cancer
inferredcites →https://doi.org/10.1016/s0140-6736(05)66698-6openalex-snapshot
Nasopharyngeal carcinoma
inferredcites →https://doi.org/10.1016/s1097-2765(01)00261-1openalex-snapshot
Coupling Met to Specific Pathways Results in Distinct Developmental Outcomes
inferredcites →https://doi.org/10.1016/s1097-2765(02)00439-2openalex-snapshot
A Mechanism of Cell Survival
inferredcites →https://doi.org/10.1016/s1470-2045(14)70023-3openalex-snapshot
Rilotumumab in combination with epirubicin, cisplatin, and capecitabine as first-line treatment for gastric or oesophagogastric junction adenocarcinoma: an open-label, dose de-escalation phase 1b study and a double-blind, randomised phase 2 study
inferredcites →https://doi.org/10.1016/s1535-6108(02)00017-xopenalex-snapshot
A role for common fragile site induction in amplification of human oncogenes
inferredcites →https://doi.org/10.1038/339155a0openalex-snapshot
Tyrosine kinase receptor indistinguishable from the c-met protein
inferredcites →https://doi.org/10.1038/bjc.2012.237openalex-snapshot
MET in gastric carcinomas: comparison between protein expression and gene copy number and impact on clinical outcome
inferredcites →https://doi.org/10.1038/ncb2257openalex-snapshot
A direct role for Met endocytosis in tumorigenesis
inferredcites →https://doi.org/10.1038/nm.2402openalex-snapshot
Oncogenic PIK3CA-driven mammary tumors frequently recur via PI3K pathway–dependent and PI3K pathway–independent mechanisms
inferredcites →https://doi.org/10.1038/nm.2819openalex-snapshot
Autocrine activation of the MET receptor tyrosine kinase in acute myeloid leukemia
inferredcites →https://doi.org/10.1038/nrc3205openalex-snapshot
Targeting MET in cancer: rationale and progress
inferredcites →https://doi.org/10.1038/nrclinonc.2012.71openalex-snapshot
MET: a promising anticancer therapeutic target
inferredcites →https://doi.org/10.1038/nrd2530openalex-snapshot
Drug development of MET inhibitors: targeting oncogene addiction and expedience
inferredcites →https://doi.org/10.1038/nrm3012openalex-snapshot
MET signalling: principles and functions in development, organ regeneration and cancer
inferredcites →https://doi.org/10.1046/j.1365-2273.2001.00449.xopenalex-snapshot
The aetiology of nasopharyngeal carcinoma
inferredcites →https://doi.org/10.1056/nejmoa0810699openalex-snapshot
Gefitinib or Carboplatin–Paclitaxel in Pulmonary Adenocarcinoma
inferredcites →https://doi.org/10.1073/pnas.0508776103openalex-snapshot
Amplification of MET may identify a subset of cancers with extreme sensitivity to the selective tyrosine kinase inhibitor PHA-665752
inferredcites →https://doi.org/10.1093/annonc/mdu507openalex-snapshot
A Bayesian network meta-analysis comparing concurrent chemoradiotherapy followed by adjuvant chemotherapy, concurrent chemoradiotherapy alone and radiotherapy alone in patients with locoregionally advanced nasopharyngeal carcinoma
inferredcites →https://doi.org/10.1093/jnci/dji112openalex-snapshot
Epidermal Growth Factor Receptor Gene and Protein and Gefitinib Sensitivity in Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1158/1078-0432.ccr-13-1836openalex-snapshot
Biomarker Analyses from a Placebo-Controlled Phase II Study Evaluating Erlotinib ± Onartuzumab in Advanced Non–Small Cell Lung Cancer: MET Expression Levels Are Predictive of Patient Benefit
inferredcites →https://doi.org/10.1158/1078-0432.ccr-14-0450openalex-snapshot
MET Amplification Status in Therapy-Naïve Adeno- and Squamous Cell Carcinomas of the Lung
inferredcites →https://doi.org/10.1158/1078-0432.ccr-14-1293openalex-snapshot
Analysis of 1,115 Patients Tested for MET Amplification and Therapy Response in the MD Anderson Phase I Clinic
inferredcites →https://doi.org/10.1200/jco.2007.14.8494openalex-snapshot
First-Line Gefitinib in Patients With Advanced Non–Small-Cell Lung Cancer Harboring Somatic EGFR Mutations
inferredcites →https://doi.org/10.1200/jco.2008.19.1635openalex-snapshot
Increased MET Gene Copy Number Negatively Affects Survival of Surgically Resected Non–Small-Cell Lung Cancer Patients
inferredcites →https://doi.org/10.1200/jco.2010.31.4286openalex-snapshot
CCND1 and MET Genomic Amplification During Malignant Transformation of a Giant Cell Tumor of Bone
inferredcites →https://doi.org/10.1200/jco.2011.35.4928openalex-snapshot
MET Amplification Identifies a Small and Aggressive Subgroup of Esophagogastric Adenocarcinoma With Evidence of Responsiveness to Crizotinib
inferredcites →https://doi.org/10.1200/jco.2011.36.7706openalex-snapshot
Genetic Activation of the MET Pathway and Prognosis of Patients With High-Risk, Radically Resected Gastric Cancer
inferredcites →https://doi.org/10.1200/jco.2011.37.7929openalex-snapshot
MET Signaling Pathway: A Rational Target for Cancer Therapy
inferredcites →https://doi.org/10.1200/jco.2011.40.3774openalex-snapshot
Targeting the Hepatocyte Growth Factor–cMET Axis in Cancer Therapy
inferredcites →https://doi.org/10.1200/jco.2012.43.3383openalex-snapshot
Phase II and Biomarker Study of the Dual MET/VEGFR2 Inhibitor Foretinib in Patients With Papillary Renal Cell Carcinoma
inferredcites →https://doi.org/10.1200/jco.2012.43.9422openalex-snapshot
MET As a Possible Target for Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1200/jco.2012.47.4189openalex-snapshot
Randomized Phase II Trial of Onartuzumab in Combination With Erlotinib in Patients With Advanced Non–Small-Cell Lung Cancer
inferredcites →https://doi.org/10.1245/s10434-010-0922-6openalex-snapshot
Altered Expression of E-cadherin by Hepatocyte Growth Factor and Effect on the Prognosis of Nasopharyngeal Carcinoma
inferredcites →https://doi.org/10.1245/s10434-013-3355-1openalex-snapshot
MET Gene Copy Number Gain is an Independent Poor Prognostic Marker in Korean Stage I Lung Adenocarcinomas
inferredcites →https://doi.org/10.1371/journal.pone.0079137openalex-snapshot
c-Met as a Prognostic Marker in Gastric Cancer: A Systematic Review and Meta-Analysis
What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
inferred← citeshttps://doi.org/10.2139/ssrn.4662549openalex-extracted
Design, Synthesis and Pharmacological Evaluation of Novel Thiazole-Based Peptide Like Molecules as C-Met Kinase Inhibitors and Anticancer Agents
inferred← citeshttps://doi.org/10.1002/hed.25655openalex-snapshot
Role of c‐Met expression on prognosis of head and neck cancer: A literature review and meta‐analysis
inferred← citeshttps://doi.org/10.1002/hed.26049openalex-snapshot
Hepatocyte Growth Factor Receptor overexpression predicts reduced survival but its targeting is not effective in unselected HNSCC patients
inferred← citeshttps://doi.org/10.1007/978-3-031-93894-8_6openalex-snapshot
Receptor Tyrosine Kinase c-MET: A Biomarker and Therapeutic Target in Cancer
inferred← citeshttps://doi.org/10.1016/b978-0-12-818168-3.00011-5openalex-snapshot
c-Met as a potential therapeutic target in triple negative breast cancer
inferred← citeshttps://doi.org/10.1016/j.bmc.2020.115555openalex-snapshot
Discovery of N-substituted-3-phenyl-1,6-naphthyridinone derivatives bearing quinoline moiety as selective type II c-Met kinase inhibitors against VEGFR-2
inferred← citeshttps://doi.org/10.1016/j.ejmech.2017.08.044openalex-snapshot
Recent advances in the discovery of small molecule c-Met Kinase inhibitors
inferred← citeshttps://doi.org/10.1016/j.lungcan.2020.08.021openalex-snapshot
Characteristics and response to crizotinib in lung cancer patients with MET amplification detected by next-generation sequencing
inferred← citeshttps://doi.org/10.1016/j.molstruc.2024.139074openalex-snapshot
Design, synthesis and pharmacological evaluation of novel thiazole derivatives as c-Met kinase inhibitors and anticancer agents
inferred← citeshttps://doi.org/10.1016/j.tjog.2018.11.020openalex-snapshot
Clinical significance of c-Met and phospho-c-Met (Tyr1234/1235) in ovarian cancer
inferred← citeshttps://doi.org/10.1038/s41388-018-0185-4openalex-snapshot
The multiple paths towards MET receptor addiction in cancer
inferred← citeshttps://doi.org/10.1038/s41388-018-0537-0openalex-snapshot
Overexpression-mediated activation of MET in the Golgi promotes HER3/ERBB3 phosphorylation
inferred← citeshttps://doi.org/10.1038/s41419-022-04686-zopenalex-snapshot
CircTMTC1 contributes to nasopharyngeal carcinoma progression through targeting miR-495-MET-eIF4G1 translational regulation axis
inferred← citeshttps://doi.org/10.1038/s41598-018-28672-8openalex-snapshot
Prognostic role of c-Met in head and neck squamous cell cancer tissues: a meta-analysis
inferred← citeshttps://doi.org/10.1097/mpa.0000000000000604openalex-snapshot
Profiling of cMET and HER Family Receptor Expression in Pancreatic Ductal Adenocarcinomas and Corresponding Lymph Node Metastasis to Assess Relevant Pathways for Targeted Therapies
inferred← citeshttps://doi.org/10.1186/s12885-019-5450-6openalex-snapshot
Increased MET gene copy number negatively affects the survival of esophageal squamous cell carcinoma patients
inferred← citeshttps://doi.org/10.1186/s13000-018-0733-9openalex-snapshot
MET-overexpressing myxofibrosarcoma frequently exhibit polysomy of chromosome 7 but not MET amplification, especially in high-grade cases: clinical and pathological review of 30 myxofibrosarcoma cases
inferred← citeshttps://doi.org/10.1186/s13046-016-0292-4openalex-snapshot
Integrated analysis of microRNA regulatory network in nasopharyngeal carcinoma with deep sequencing
inferred← citeshttps://doi.org/10.1186/s13048-025-01903-zopenalex-snapshot
Mechanistic insights into c-Met rs368750834 mutation and a bifunctional CAR-T strategy for serous ovarian carcinoma
inferred← citeshttps://doi.org/10.18632/oncotarget.21303openalex-snapshot
Clinicopathological impacts of high c-Met expression in head and neck squamous cell carcinoma: a meta-analysis and review
inferred← citeshttps://doi.org/10.18632/oncotarget.23656openalex-snapshot
WITHDRAWN: A meta-analysis of c-Met expression in head and neck cancer
inferred← citeshttps://doi.org/10.3390/cells9020522openalex-snapshot
Detection of MET Alterations Using Cell Free DNA and Circulating Tumor Cells from Cancer Patients
inferred← citeshttps://doi.org/10.70675/689dceb7zaef0z44e6zabc1z752ba1b92b33openalex-snapshot
Implication du récepteur à activité tyrosine kinase (RTK) MET sur la balance survie/apoptose et identification de nouvelles mutations de RTKs dans les cancers colorectaux métastatiques
inferred← citeshttps://doi.org/10.70675/ea435494z4946z4d12za5f5zd338f77b537bopenalex-snapshot
Conséquences de la dérégulation de MET sur le phénotype des cancers bronchiques non à petites cellules EGFR mutés devenus résistant aux inhibiteurs de tyrosine kinase d’EGFR
inferred← citeshttps://doi.org/10.7150/jca.16686openalex-snapshot
Alteration status and prognostic value of MET in head and neck squamous cell carcinoma
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