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Anti-metastatic potential of somatostatin analog SOM230: Indirect pharmacological targeting of pancreatic cancer-associated fibroblasts
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What this work says
Outgoing claims: the papers this work cites, builds on, or argues with.
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Microenvironmental regulation of cancer development
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Pasireotide (SOM230): Development, mechanism of action and potential applications
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Antitumor effects of somatostatin
inferredcites →https://doi.org/10.1016/j.regpep.2013.10.007openalex-snapshot
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Interleukin-6 in inflammatory and malignant diseases of the pancreas
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Pancreatic cancer genomes reveal aberrations in axon guidance pathway genes
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Pancreatic tumours escape from translational control through 4E-BP1 loss
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Dual roles of hemidesmosomal proteins in the pancreatic epithelium: the phosphoinositide 3-kinase decides
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Somatostatin at nanomolar concentration reduces collagen I and III synthesis by, but not proliferation of activated rat hepatic stellate cells
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Direct binding of p85 to sst2 somatostatin receptor reveals a novel mechanism for inhibiting PI3K pathway
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Loss of Somatostatin Receptor Subtype 2 Promotes Growth of KRAS-Induced Pancreatic Tumors in Mice by Activating PI3K Signaling and Overexpression of CXCL16
inferredcites →https://doi.org/10.1074/jbc.m112.438846openalex-snapshot
Loss of the Transcription Factor GLI1 Identifies a Signaling Network in the Tumor Microenvironment Mediating KRAS Oncogene-induced Transformation
inferredcites →https://doi.org/10.1074/jbc.m114.556563openalex-snapshot
The Transcription Factor GLI1 Modulates the Inflammatory Response during Pancreatic Tissue Remodeling
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StellaTUM: current consensus and discussion on pancreatic stellate cell research
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Cancer-Associated Stromal Fibroblasts Promote Pancreatic Tumor Progression
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What points at this work
Incoming claims: where this work appears in other papers’ reference lists and arguments.
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